Acupuncture in chronic prostatitis & AP mechanisma (placebo V sham V verum)

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Hi All,

 

See these:

 

Zhang J, Liu CD, Ding Y, Tang QB. [Clinical observation on

therapeutic effect of electroacupuncture on chronic prostatitis and

detection of urethral sphincter EMG] [Article in Chinese] Zhongguo

Zhen Jiu. 2010 Jan;30(1):13-7. Department of Urinary Surgery, The

First Affiliated Hospital of Chongqing Medical University, Chongqing

400016, China. OBJECTIVE: To observe the clinical therapeutic effect

of electroacupuncture for chronic prostatitis and investigate its

mechanism. METHODS: 48 cases were randomly divided into an

electroacupuncture group and a western medicine group, 24 cases in

each group. The EAP group was treated by EAP at CV04-Guanyuan, CV03-

Zhongji, BL32-Ciliao and BL35-Huiyang as main acupoints. The western

medicine group was treated by oral administration of Sparfloxacin

tablets and Prostat tablets. The scores of NIH-CPSI, changes in

uroflow rate-urethral sphincter EMG and their therapeutic effects

were observed. RESULTS: The total effective rate was 87.5 % in the

electroacupuncture group which, was better than 62. 5% in the western

medicine group (P(<0. 05). There were significant differences in the

scores of NIH-CPSI and Q(max), Q(ave), TL value before and after

treatment in the electroacupuncture group (all P<0.05), and with a

significant difference in the scores of NIH-CPSI and Q(max). Q(ave),

TL value after treatment between the two groups (all P<0.05).

CONCLUSION: EAP based on syndrome differentiation has better

therapeutic effect on chronic prostatitis than that of routine

clinical medicine. PMID: 20353107 [PubMed - in process]

 

Enck P, Klosterhalfen S, Zipfel S. Acupuncture, psyche and the

placebo response. Auton Neurosci. 2010 Mar 30. [Epub ahead of print]

University Hospital Tübingen, Dept. of Psychosomatic Medicine,

Tübingen, Germany. With growing use of AP treatment in various

clinical conditions, the question has been posed whether the reported

effects reflect specific mechanisms of AP or whether they represent

placebo responses, as they often are similar in effect size and

resemble similarities to placebo analgesia and its mechanisms. We

reviewed the available literature for different placebos (sham

procedures) used to control the AP effects, for moderators and

potential biases in respective clinical trials, and for central and

peripheral mechanisms involved that would allow differentiation of

placebo effects from AP and sham AP effects. While the evidence is

still limited, it seems that biological differences exist between a

placebo response, e.g. in placebo analgesia, and analgesic response

during AP that does not occur with sham AP. It seems advisable that

clinical trials should include potential biomarkers of AP, e.g.

measures of the autonomic nervous system function to verify that AP

and sham AP are different despite similar clinical effects. Copyright

© 2010 Elsevier B.V. All rights reserved. PMID: 20359961 [PubMed - as

supplied by publisher]

 

Best regards,