Viruses by Patrick Quanten, MD.

February 27, 2005

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Sun, 27 Feb 2005 16:09:09 -0000

Viruses

By Patrick Quanten, MD.

Let's start with a medically well-known fact: viruses aren't

themselves alive. They are smaller and simpler than bacteria and by

themselves they are inert and harmless. So, the immediate question

then has to be: How can you " catch " a virus if it isn't a living thing?

The answer is: You can't.

Experimenters have incubated viruses for the common cold, placed them

directly on the mucous lining of the nose, and found that their

subjects came down with colds only 12% of the time. These odds could

not be increased by exposing the subjects to cold drafts, putting

their feet in ice water to give them chills, or anything else that was

purely physical.

Swine flu (viral infection) arose as a normal, non-lethal flu in the

spring of 1918, but somehow, over the following months, it mutated

into something more severe. In an attempt to devise a vaccine, medical

authorities conducted experiments on volunteers at a military prison

on Deer Island in Boston Harbour. The prisoners were promised pardon

if they survived a battery of tests. These tests were rigorous to say

the least. First, the subjects were injected with infected lung tissue

taken from the dead and then sprayed in the eyes, nose and mouth with

infectious aerosols. If they still failed to succumb, they had their

throats swabbed with discharges taken from the sick and dying. If all

else failed, they were required to sit open-mouthed while a gravely

ill victim was sat up slightly and made to cough into their faces. The

doctors chose sixty-two of the volunteers for the tests. None

contracted the flu, not one. The only person who did grow ill was the

ward doctor, who swiftly died.

One of the mysteries of viral epidemics is how it can erupt suddenly

all over, in places separated by oceans, mountain ranges and other

earthly impediments. Although a virus is not alive in itself, it also

loses its potential of hijacking the genetic material of a living host

cell within a few hours of being outside the host body. The commonly

heard answer that it travels in " carriers " (people who have no

symptoms but carry and distribute the virus) cannot be proven and

after decades of using it as " the " explanation remains nothing more

than a shaky and desperate theory. It is made even more unlikely in

the light of the fact that you cannot catch a viral infection, as

proven above, so even if it did travel that way, how would it " jump "

from the carrier to the victim? Furthermore, how does a virus manage

to lie low for several months, in the case of HIV or variant CJD we

are to believe it can be up to 20 years, before erupting so

explosively at more or less the same time all over? What's the trigger

and why instantaneously in all those different places?

Some of these viral epidemics have been known to be more devastating

to people in their prime rather than infants and the elderly, who are

more likely to have a more vulnerable immune system. Strange, to say

the least.

From time to time certain strains of virus return. A disagreeable

Russian virus known as H1N1 caused severe outbreaks over wide areas in

1933, then again in the 1950s and again in the 1970s. Where it went in

the meantime each time is uncertain. Could it have survived, lying

" dormant " , in humans or animals for all that time? This raises the

same old two questions: Why did it not cause any symptoms wherever it

was hiding? and If it was hiding somewhere, how did it spread so

quickly when it did, as you can't catch it - not from a human, not

from an animal?

What do we know about Viruses?

We have already mentioned that they are very small, and they weren't

detected until 1943 with the invention of the electron microscope.

Many, including HIV, have ten or fewer genes, whereas the simplest

bacteria require several thousand. To create a living thing you need

properly organised DNA of a substantial quantity, which the virus

hasn't got.

We define " a living organism " as something that performs three tasks

in succession: taking in stuff (eating, breathing), metabolising stuff

(digesting, absorbing), and excreting waste. A fourth necessary task

is reproduction. A virus doesn't do any of these. No virus does.

Within the viral capsule there are no other structures that are

required for a metabolic process. There is no activity at all inside

the viral capsule.

Not only doesn't it look structurally as if its alive, it also isn't

alive in physiological terms.

So what is it then? As we all know, viruses can have devastating

effects on the health of plants, animals - great and small, including

bacteria - and humans. How does it produce these effects, if it is not

alive, can't be caught and doesn't reproduce?

Known scientific facts about viruses and the way they function are

obtained from chemical analysis and looking at still pictures from

electron microscopes. The story is pieced together, not actually

observed! This means that what you are told happens, is actually a

theory at best, and a fantasy story at worst. What has actually, in

simple terms, been discovered?

* Viruses contain either RNA or DNA, a small amount and mostly one

or the other, but there are exceptions. Bits of genetic material of

whatever kind, really; but only bits.

* Viruses are marked species and organ specific, and on the whole,

viruses infecting plants, insects, rickettsiae, bacteria and other

animals are distinct from their human counterparts, but this is now

thought not to be entirely the case. They are specific, but then again

they are not.

* Viruses may be naked with the genome only protected by a protein

capsid, or they may have a lipid envelop surrounding the capsid. Bits

of genetic material in a thin simple bag, and sometimes put in a fatty

bubble.

* Viruses are seen to be " encapsulated " by the body cells that

have specific receptors for the virus. Once inside the cell, it seems

that the virus capsule is removed and the exposed bit of DNA or RNA is

" read " and the host cell seems to duplicate it. These bits of genetic

materials are encapsulated once again, and with the host cell bursting

with complete viruses it will explode and the viruses are spilled into

the cellular surroundings. So, we see a lot of genetic bits within the

cell; these bits are then encapsulated and eventually the cell burst

open to release the now bagged up genetic material into the cellular

environment.

* Viruses in the intercellular environment are engulfed by cells

from the immune system (macrophages and lymphocytes), which collect

them and destroy them. These bags that contain bits of genetic

material are collected into cells from the immune system.

* Viruses are very difficult to demonstrate (they are extremely

small) and the diagnosis of viral infection is mostly made on clinical

symptoms alone and the assumption that it fits into a known disease

pattern for which there is no causative factor known. Virtually every

time a diagnosis of viral infection is pronounced no proof is offered

for this diagnosis.

* Materials for virus isolation must be obtained as early as

possible during illness. It is at the very early stages of the illness

that the highest titres are found and the most likely it is one can

produce a positive test result. There are more viruses present right

at the beginning of the illness than at any other stage of the disease

process. If the viruses were multiplying you would expect the number

to rise as the disease developed.

* Identification of viruses is done in laboratories by measuring

the level of antibodies against specific viruses, not by measuring or

demonstrating the virus itself. Measuring a higher protection level is

diagnosed as the illness itself!

Summarising this scientific knowledge, we can say that viral

infections are not diagnosed by finding the specific virus, but by

guessing a virus is the cause of the symptoms. In practical terms,

this happens when the doctor doesn't really know what the cause is.

As regards the story of the viral infection is concerned, we now know

that as soon as the symptoms start the number of viruses will very

quickly be dramatically reduced. There is no evidence of rapid number

proliferation once the disease manifests itself.

Before we move on to explain the real virus story, it is worthwhile to

remind ourselves of what we now know:

1. A virus is not alive.

2. You cannot catch a virus.

3. A virus disintegrates very quickly outside the host.

4. A virus consists of small bits of genetic material, variable

from virus to virus, surrounded by a thin coating, either protein

(water-soluble) or fat.

5. Viral materials are seen in large numbers inside the host cell.

6. A full host cell breaks open and the viruses are spilled into

the environment.

7. In the environment the viruses are bagged up by the cells of the

immune system (See " The Inflammation Process " , available on

www.activehealthcare.co.uk).

The Virus Story

If viruses are not living things they cannot multiplicate and they

don't need a specific environment to " survive " . They cannot appear

from nowhere and they can't spread and infect other cells.

When a cell becomes diseased and the function of the cell begins to

falter, it may start to come apart at the seams. Bits of its essential

structure, the DNA and RNA, may become detached as it is falling

apart. The cell will try and clean up these bits by preparing them for

the rubbish bin. The small pieces of genetic material, which are now

floating around in the intracellular fluid, will be isolated by means

of encapsulating them. As the cellular disintegration continues more

and more of these bits are seen inside the cell and more and more

small " bags " of useless genetic material will appear. Once the cell is

totally dysfunctional and filled with rubbish the cellular wall itself

bursts and the contents will be spilled into the cellular environment.

Here, the cleanup continues by packaging these small bags up even

further into what has been called the lymphocytes and macrophages of

the immune system. These large vesicles now drift away into the

lymphatic fluid and the blood stream, from where they will be filtered

out at appropriate draining stations, like the spleen and the lymph

nodes. This process continues until the whole lot has been cleared.

This explains why the numbers of " viruses " is the highest at the very

beginning of the disease and continues to decline steadily throughout

the disease process, even without treatment. This also accounts for

the thousand and thousand of different " viruses " that have been

identified and for the " mutation " of viruses. Viral behaviour is

essentially totally unpredictable because the cells and the way they

disintegrate is never the same, not because this is an animal that

changes its behaviour so quickly and intelligently that nothing can

keep up with it. It also does away with the idea that the " virus " can

lay dormant for an indefinite period of time and become activated

without any triggers or reasons having been identified.

How do we then explain " viral epidemics " ? Why is it then that we get a

cold the day after someone in the office starts to cough and sneeze a lot?

The medical profession knows that viruses have incubation periods.

These are said to vary from virus to virus from a few days to several

years. A cold virus has an average incubation period of about a week.

Now, first of all, you can't catch a virus; and secondly, if you could

catch the cold virus, it would take a week before it had established

itself within your body and starts to show symptoms. Consequently,

your cold cannot have been caused by the other person's cold in the

office the day before!

What is seen and has been named " a virus " starts after the cellular

structure begins to disintegrate. Why does a cell start to fall apart?

Because it is diseased. The disease is already there, long before any

viral particles show up in any pictures. So, then we have to ask the

question why the cell has become diseased? The answer to this lies in

the build-up of toxic material within the cellular structure. As the

cell gets loaded up with inappropriate material it will eventually be

unable to cope and it will start to fall to pieces. It is exactly

those pieces that are photographed by the electron microscope and have

been named " viruses " .

The influences that can lead to an increased pressure on the system

are many and are varied. They range from the weather, to living and

working environment, to life style and diet, to the balance of

activity and rest, to mental balance, stress and worries. Because a

lot of these influences, such as working conditions and the weather,

are general circumstances which affect all of us, it is very likely

that a great number of us, in the same environment, will fall ill at

or around the same time, succumbing to the environmental influences.

Add to this that people who are working in the same environment are

very likely to have similar life styles and another factor has been

identified explaining why similar disease pattern occur within certain

groups of people at certain times. On top of that, we now know that

worry reduces our immunity capacity and increases the likelihood of

illness. The belief that, if one person close to you has a cold you

are going to get it, increases the likelihood of this actually

happening dramatically, as you become more vulnerable through the

immune reducing effect of the worry itself.

Epidemics occur because people in similar circumstances, living

environments and conditions, have similar imbalances within their

systems, leading directly to similar disease patterns. This causes

fear and apprehension all around them, making others more vulnerable

to start showing a breakdown of health themselves. The disease is

spreading. More accurately, the fear of the disease is spreading

first, resulting in a lowered resistance, which allows each

individual's imbalances to show up through the inability to cope with

the problems the system has already been faced with for a long time.

More and more people are becoming ill and showing signs of the fact

that their bodies have been under extreme pressure for quite a while

to maintain health. The showing of an illness is the end result of a

long process, even an " acute " illness, of a slow deterioration of the

system's normal functioning. Disease is a process, not a state of being.

It is time to learn the facts of life.

It is time to do away with ignorance and the resulting fear.

It is time to focus on individual health and the factors that

influence it.

Viruses are dead, but diseases are very much alive. Let's concentrate

on the living, not the dead, if we want to be healthy.

November 2004

ahcare.qua

www.activehealthcare.co.uk

for knowledge and well-being - in truth and in health

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March 1, 2005

Dear Dr. Patrick Quanten, MD.;

There are a number of statements in this Post that are true.And what makes this

post difficult for me, is there are a number of statements that are not true or

correct. In a number of errored statements, they are much more than just

partially incorrect. I am writing this letter to you, to ask if I may provide

some critique commentary throughout the post for your consideration and review.

e.g. Viruses contain additional proteins, enzymes required for translation and

replication.

e.g. Some viruses such as the Herpes Family containe huge genomes.

e.g. Many virus a very infectious, especially those that mutate and are

air-borne such as Rhino and Influenza Viruse families.

e.g. Immunity may be acquired by limited exposure to some non-potent viral

mutants. Such as with milk-maids.

e,g, Some virus require more than one strain to become potent and infectious

such as Hepatitus B,C,D etc. and also such as Epstein-Barr and Cytomegliovirus

concommitent activation.

e.g. Many virus, more than just the Herpes virus have a long dormant stage and

are capable of long term latency. Primary initial infection may have occured

months prior to symptomatic pathologies.

e.g. It is true, as you say, that Medical Doctors do NOT have a diagnostic tool

to actually detect a virus or know what virus. This is scary. IgM and IgG

antibody titers are only circumstantial evidence and some antibody levels may be

present to a past infection or temporary systemic hightened reactivation during

onset of any new infection.

e.g. You can most definately catch a virus. I have performed research that

unequivicably prove this with Pappiloma, moluscum contagiousum, HSV1, Rhino and

several others in non-human hosts such as rats, pigs, mice, chicken, etc..

This post, with respect to the subject material is very useful. if you are

interested in considering a few corrections please do not hesitate to

professionally reply.

Greg Keyock

xigent

Molecular Virologist

califpacific <califpacific wrote: