high bilirubin, normal SGOT

[Guest guest]

Namaste Marilette

I have a friend who is suufering from jaundice since

Sept 2004,the bilrubin reading was 3.5 which had

increased to 27 by October '04 and the

normal range is less than 1.He was taking allopathic

medication and under going treatment at CMC Vellour

and the reading went down to 3 by end

of Nov'04.

The Bilrubin reading has always been high but the

SGPT reading has always been normal which should be

below 32 and his reading is now around

15.

 

For the last one year his bilrubin reading is

varying between 3 and 5 and has never come down to the

normal level.Since his SGPT reading has

always been normal his liver functioning is not

affected at all and currently has no symptoms of

jaundice but his bilrubin reading is still

high and would like to know what could be the affects

in the long run due to high bilrubin and also would

like to have a protocol for his healing

to help him get rid of this problem.

Thanks and best wishes

Rajesh.Chablani

 

=====================================================

 

 

Dear Rajesh,

 

Namaste.

 

Thank you for your email.

 

Medical Background:

 

Jaundice is indicated by a yellow tone in the skin and

eyes, and alone it is not a health concern. Jaundice

may occur if bilirubin levels increase, which can

occur with high levels of red blood cell destruction.

Bilirubin is the final product of hemoglobin

degradation, and is typically removed from the

bloodstream by the liver. Therefore, jaundice can also

be a sign of a poorly functioning liver, which may

also be evidenced by an enlarged liver. Increased

bilirubin also leads to increased chance for

gallstones in children with sickle cell disease.

 

According to DAVID E. JOHNSTON, M.D., University of

New Mexico School of Medicine in Albuquerque, New

Mexico, there are special considerations in

interpreting liver function tests.

 

A number of pitfalls can be encountered in the

interpretation of common blood liver function tests.

These tests can be normal in patients with chronic

hepatitis or cirrhosis. The normal range for

aminotransferase levels is slightly higher in males,

nonwhites and obese persons. Severe alcoholic

hepatitis is sometimes confused with cholecystitis or

cholangitis. Conversely, patients who present soon

after passing common bile duct stones can be

misdiagnosed with acute hepatitis because

aminotransferase levels often rise immediately, but

alkaline phosphatase and gamma-glutamyltransferase

levels do not become elevated for several days.

Asymptomatic patients with isolated, mild elevation of

either the unconjugated bilirubin or the

gamma-glutamyltransferase value usually do not have

liver disease and generally do not require extensive

evaluation. Overall hepatic function can be assessed

by applying the values for albumin, bilirubin and

prothrombin time in the modified Child-Turcotte

grading system.

 

The commonly used liver function tests (LFTs)

primarily assess liver injury rather than hepatic

function. Indeed, these blood tests may reflect

problems arising outside the liver, such as hemolysis

(elevated bilirubin level) or bone disease (elevated

alkaline phosphatase [AP] level).

 

Abnormal LFTs often, but not always, indicate that

something is wrong with the liver, and they can

provide clues to the nature of the problem. However,

normal LFTs do not always mean that the liver is

normal. Patients with cirrhosis and bleeding

esophageal varices can have normal LFTs. Of the

routine LFTs, only serum albumin, bilirubin and

prothrombin time (PT) provide useful information on

how well the liver is functioning.

 

The general subject of LFTs1,2 and the differential

diagnosis of abnormal LFTs in asymptomatic patients3-5

have been well reviewed. This article discusses some

common pitfalls in the interpretation of LFTs.

 

Helpful Hints for Interpreting Liver Function Tests

 

- Mildly elevated ALT level

(less than 1.5 times normal):ALT value could be normal

for gender,ethnicity or body mass index.

Consider muscle injury or myopathy.

 

- Alcoholic hepatitis: Laboratory values can appear

cholestatic, and symptoms can mimic cholecystitis.

Minimal elevations of AST and ALT often occur.

 

- AST level greater than 500 U per L: The AST

elevation is unlikely to result from alcohol intake

alone. In a heavy drinker, consider acetaminophen

toxicity.

 

- Common bile duct stone: Condition can simulate

acute hepatitis.

AST and ALT become elevated immediately, but elevation

of AP and GGT is delayed.

 

- Isolated elevation of GGT level: This situation may

be induced by alcohol and aromatic medications,

usually with no actual liver disease.

 

- Isolated elevation of AP level (asymptomatic patient

with normal GGT level): Consider bone growth or

injury, or primary biliary cirrhosis.

AP level rises in late pregnancy.

 

- Isolated elevation of unconjugated bilirubin level:

Consider Gilbert syndrome or hemolysis.

 

- Low albumin level: Low albumin is most often caused

by acute or chronic inflammation, urinary loss, severe

malnutrition or liver disease; it is sometimes caused

by gastrointestinal loss (e.g., colitis or some

uncommon small bowel disease).

 

Normal values are lower in pregnancy.

 

- Blood ammonia level: Blood ammonia values are not

necessarily elevated in patients with hepatic

encephalopathy. Determination of blood ammonia levels

is most useful in patients with altered mental status

of new onset or unknown origin.

 

ALT=alanine aminotransferase;

AST=aspartate aminotransferase;

AP=alkaline phosphatase;

GGT=gamma-glutamyltransferase.

 

 

Markers of Hepatocellular Injury

 

The most commonly used markers of hepatocyte injury

are aspartate aminotransferase (AST, formerly serum

glutamic-oxaloacetic transaminase [sGOT]) and alanine

aminotransferase (ALT, formerly serum

glutamate-pyruvate transaminase [sGPT]). While ALT is

cytosolic, AST has both cytosolic and mitochondrial

forms.

 

Hepatocyte necrosis in acute hepatitis, toxic injury

or ischemic injury results in the leakage of enzymes

into the circulation. However, in chronic liver

diseases such as hepatitis C and cirrhosis, the serum

ALT level correlates only moderately well with liver

inflammation. In hepatitis C, liver cell death occurs

by apoptosis (programmed cell death) as well as by

necrosis. Hepatocytes dying by apoptosis presumably

synthesize less AST and ALT as they wither away. This

probably explains why at least one third of patients

infected with hepatitis C virus have persistently

normal serum ALT levels despite the presence of

inflammation on liver biopsy.6,7 Patients with

cirrhosis often have normal or only slightly elevated

serum AST and ALT levels. Thus, AST and ALT lack some

sensitivity in detecting chronic liver injury. Of

course, AST and ALT levels tend to be higher in

cirrhotic patients with continuing inflammation or

necrosis than in those without continuing liver

injury.

 

Source - American Academy of Family Physicians

 

Pranic Healing:

 

1. Invoke and scan before, during andafter treatment.

 

2. Generla sweeping several times.

 

3. Localized thorough sweeping on the front and back

solar plexus chakra with LWG.

 

4. Localized thorough sweeping on the front, side and

back of the liver alterately with LWG and LWO ( or

very LWO).

 

5. Energize the solar plexus chkara with LWG, LWB

then ordinary LWV. Visualize the energy going into

the liver.

 

Repeat steps 2 to 5 three times per day for the first

few days or until until the bilirubin level is

normalized and stabilized.

 

6. Localized thorough sweeping on the front and back

heart chakra. Energize the thymus through the back

heart chakra with LWg then with ordinary LWV.

 

7. Localized thorouh sweeping on the lungs. Energize

the lungs directly through the back of the lungs with

LWG then with LWO. Point your fingers away form the

patient's head when energizing with O.

 

8. Localized thorough sweeping on the front and back

spleen chakra. Energize the spleen chakra with LWG

then ordinary LWV. This has to be done with caution.

 

 

9. Localized thorough cleansing on the navel chakra.

Energize the navel with W.

 

10. Localized thorough sweeping on the basic chakra.

 

 

11. Localzied thorough sweeping on the arms and legs.

Energize th ehand and sole minor chakras with

ordinary LWV. Do not repeat this step more than once

per day.

 

12. Localized thorough sweeping on the crown chakra,

forehead chakra, ajna chakra, throat chakra, and back

head minor chakra. Energize them with LWG then with

more of ordinary LWV.

 

13. Stabilize and release projected energy.

 

14. Repeat 3 or more time per week until the the

patient is completely healed.

 

15. Once the patient's condition is stabilized and

completely healed per medical tests, continue the

entire treatment as follows:

 

15.1 Invoke and scan before during and after

treatment.

 

15.2. Localized thorough cleansing on the front and

back solar plexus chakra, the front, sides and back of

the liver using LWG then ordinary LWV.

 

15.3. Energize the liver with LWB for localizing

effect.

 

15.4. Energize the liver with LWG, LWO then LWR.

 

15.6. Localized thorough sweeping on the front and

back sola rplexus chakra and energize it with LWG then

with more of oridnary LWV.

 

15.7. Localized thorough sweeping on the navel and

the basci chakras. Energize them with LWR.

 

15.8. Stabilize and release projected pranic energy.

 

15.9. Reeat treatment 3 times per wek for as long as

necessry. Treatment may take 6 months to a year or

more.

 

The rate of healing depends upon the degree of liver

damage, the physical and psychological conditions of

th epatient, the karmic factor and the healer's degree

of proficiency in healing.

 

Source - Advanced Pranic Healing by Master Choa Kok

Sui.

 

Love,

 

Marilette

 

 

 

 

 

 

 

 

 

 

 

 

1. Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer.

 

2. Pranic Healers who are are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments. ~ Master Choa

Kok Sui

 

Miracles do not happen in contradiction to nature, but only to that which is

known to us in nature. ~ St. Augustine

 

Reference material for Pranic Healing protocols are the following books

written by Master Choa Kok Sui:

Science and Art of Modern Pranic Healing, Advanced Pranic Healing, Pranic

Psychotherapy, Pranic Crystal Healing.

 

Ask or read the up to date Pranic Healing protocols by joining the group through

http://health./

 

MCKS Pranic Healing gateway website: http://www.pranichealing.org.