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" Peter Myers "

 

October 30, 2005

 

 

(1) aluminum and alzheimer's

(2) Aluminum fluoride and alzheimer's

(3) aluminum and fluoride

(4) Fluoride Combines with Aluminium in Drinking Water

 

 

(1) aluminum and alzheimer's

 

Sun, 30 Oct 2005 00:40:25 -0700 Phil Eversoul <Philev

>

 

http://www.ecotalk.org/Alzheimer'sAluminumPhosphateIndustry.htm<http://www.ecota\

lk.org/Alzheimer%27sAluminumPhosphateIndustry.htm>

 

Lynn Investigates ...

 

Alzheimer's in America: The Aluminum - Phosphate Fertilizer Connection

 

by Lynn Landes 8/19/02

 

Americans are losing their minds to Alzheimer's disease. It's an epidemic.

And

it's not typical of what's going on in the rest of the world.

 

The World Health Organization (WHO) estimates that there are 18 million

people

with Alzheimer's. Over 4 1/2 million Americans have the disease. We account

for

25% of all Alzheimer's cases, even though we represent only 4.6% of the

world's

population. Europe is experiencing half our rate of disease. For Americans

over

85 years of age, 50% are thought to have Alzheimer's.

 

The question is, " Why? "

 

Alzheimer's was first discovered in 1906. It is not a part of normal aging,

says

the National Institutes of Health (NIH). The NIH contends that the cause of

Alzheimer's is " not known. " They say, " Prior theories regarding the

accumulation

of aluminum, lead, mercury, and other substances in the brain have been

disproved. "

 

Don't believe that. Federal agencies have a talent for not finding

environmental

causes for many diseases. They live by the motto, " Do not seek and thou

shall

not find. " Genetic triggers and lifestyle choices get the research dollars

for

pretty obvious reasons - their findings don't hurt polluters' profits.

 

The world's scientists and government researchers have not taken aluminum

off

the scientific table as a causal factor in Alzheimer's. Research scientists

with

the International Aluminum Network report, " Aluminum has been implicated ...

as

a potential factor or cofactor in the Alzheimer's syndrome, as well as in

the

etiopathogenesis of other neurodegenerative diseases, Parkinsonism,

Amyotrophic

Lateral Sclerosis and other diseases. " That's a mouthful, but you get the

picture.

 

Initially, it was thought that aluminum might be the sole cause of

Alzheimer's.

Persons with Alzheimer's have been found to experience increased absorption

of

aluminum in the brain, as well as exhibit densities of senile plaques and

neurofibrillary tangles. However, there are reports that suggest plaques and

tangles do not always signify Alzheimer's, and vice versa.

 

Further clouding the issue are patients on kidney dialysis machines. They

are

unable to excrete aluminum, plus they may also be treated with medicines

that

include aluminum. However, reports say that dialysis patients don't develop

Alzheimer's, although they can develop dialysis dementia if the equipment

doesn't filter out aluminum. And therein lies a clue.

 

The process of kidney dialysis requires very purified, non-fluoridated

water.

What does this mean? Perhaps fluoride is aluminum's partner-in-crime.

 

In 1998 Julie Varner and two colleagues published research on the effects of

aluminum-fluoride and sodium-fluoride on the nervous system of rats. They

concluded, " Chronic administration of aluminum-fluoride and sodium-fluoride

in

the drinking water of rats resulted in distinct morphological alterations of

the

brain, including the effects on neurons and cerebrovasculature. " In layman's

terms, it looked like fluoride and aluminum could cause Alzheimer's.

 

That was not a definitive study, but they may have been onto something.

Aluminum

is in our drinking water, foods, and many consumer products. Adding fluoride

to

drinking water in the U.S. started in the 1950's. America's drinking water

is

now over 60% fluoridated. Fluoride appears in many processed foods and

beverages

made with fluoridated water. Keep in mind, Europe has half our rate of

Alzheimer's. They don't fluoridate their water supplies, but they do use

fluoride supplements and dental products. Is there a connection?

 

There are other intriguing issues. Why do people with thyroid disease have

an

increased risk for Alzheimer's? In the U.S., thyroid disease has reached

even

greater epidemic levels than Alzheimer's, with as many as 20 million

American

victims. Besides problems with iodine intake, a common cause of thyroid

disease

is radiation.

 

There are also striking similarities between Alzheimer's,

Creutzfeldt-Jacob-Disease (CJD), and mad cow disease. Mad cow has been

linked to

livestock feed and fertilizer.

 

So, what do radiation, livestock feed, fluoride, and fertilizer have in

common

which may have led to the emergence of the Alzheimer's epidemic? The

phosphate

fertilizer industry.

 

" Fertilizer use was not a common practice in the United States until after

1870,

when phosphate and lime were applied to crops like cotton and tobacco. By

the

end of World War II, an era of intensive agriculture began., " says Cargill

Fertilizer. " Of the phosphate produced in Florida, about 95% is used in

agriculture (90% goes into fertilizer and 5% into livestock feed

supplements). "

The remaining 5% is used in a variety of foods and beverages, plus personal

care, consumer and industrial products.

 

George Glasser writes in the Earth Island Journal, " Radium wastes from

filtration systems at phosphate fertilizer facilities are among the most

radioactive types of naturally occurring radioactive material

wastes...Uranium

and all of its decay-rate products are found in phosphate rock,

fluorosilicic

acid (fluoride) and phosphate fertilizer. "

 

The Florida Institute of Phosphate Research says, " Removal of uranium as a

product is no longer profitable and all of the extraction facilities have

been

dismantled. The uranium that remains in the phosphoric acid and fertilizer

products is at a low enough level that it is safe for use. " That's not

reassuring. Chronic exposure to low levels of contamination can be as

dangerous,

or more so, than chronic high levels of exposure or acute occurrences.

 

Of particular interest is calcium silicate, another byproduct of the

phosphate

fertilizer industry. One of its uses is as an anti-caking agent in iodized

table

salt. Is calcium silicate also radioactive? Would that have a significant

impact

on the thyroid? Given the relationship between Alzheimer's and thyroid

disease,

Alzheimer's may be destined to increase exponentially.

 

The phosphate fertilizer industry seems to be the common thread in

Alzheimer's -

and maybe also in thyroid and mad cow type diseases. Aluminum by itself may

not

cause Alzheimer's, but in combination with the radioactive products of the

phosphate fertilizer industry, it could be wreaking havoc on our health.

 

Whatever the cause, we deserve real answers to the Alzheimer's epidemic, not

the

red herrings of research on genetics and lifestyle. The number of American

victims is totally out of proportion to the incidence of Alzheimer's

worldwide.

Something truly has gone terribly wrong.

 

Links:

 

http://www.bio.unipd.it/~zatta/alumin.htm<http://www.bio.unipd.it/%7Ezatta/alumi\

n.htm>

http://www.fluoride-journal.com/98-31-2/31291-95.htm

http://www.earthisland.org/eijournal/fluoride/fluoride_phosphates.html

 

Lynn Landes is the publisher of EcoTalk.org and a news reporter for DUTV in

Philadelphia, PA. Formerly Lynn was a radio show host for WDVR in New Jersey

and

a regular commentator for a BBC radio program. She can be reached at (215)

629-3553 / lynnlandes.

 

 

 

 

(2) Aluminum fluoride and alzheimer's

 

Sun, 30 Oct 2005 00:47:59 -0700 Phil Eversoul <Philev

>

 

Number Five

 

http://members.tripod.com/~safewater/brainre.html<http://members.tripod.com/%7Es\

afewater/brainre.html>

 

NEWS RELEASE April 16, 1998 Contact: Jeff Green

 

Citizens For Safe Drinking Water 3243 Madrid Street San Diego, CA 92110

 

Email: jgreen (800) 728-3833 Email: davidkennedy-dds

 

Research Links Low Levels of Fluoride and Aluminum to Alzheimer's and Kidney

Damage

 

In a study just published in the peer-reviewed journal Brain Research the

presence of low levels of fluoride in the drinking water of test animals,

equal

to the amount of elemental fluorine found in fluoridated water, caused

damage to

the tissue of the brain that the authors identified as similar to the

pathological changes found in humans with Alzheimer's and other forms of

dementia.

 

While the purpose of this study was to assess the factors that enhance or

inhibit the bioavailability of aluminum and its effects on the nervous

system,

the study looked at the effects of aluminum-fluoride and sodium-fluoride

separately.

 

The authors report, " Histological evidence of glomerular distortion and

other

signs of kidney disorder were found in animals in both the aluminum-fluoride

and

sodium fluoride groups... "

 

" While the small amount of aluminum-fluoride in the drinking water of rats

required for neurotoxic effects is surprising, perhaps even more surprising

are

the neurological results of the sodium-fluoride at the dose given in the

present

study (2.1 ppm). {the amount used to achieve 1 ppm of elemental fluorine

used in

fluoridation}.

 

" Fluoride has diverse actions on a variety of cellular and physiological

functions, including the inhibition of a variety of enzymes, a corrosive

action

in acid mediums, hypocalcemia, hyperkalemia, and possibly cerebral

impairment. "

 

The authors summarize, " Chronic administration of aluminum-fluoride and

sodium-fluoride in the drinking water of rats resulted in distinct

morphological

alterations of the brain, including the effects on neurons and

cerebrovasculature. "

 

While there are numerous studies linking fluoride to increased risk of hip

fracture, cancer, genetic damage, bone pathology, and dental fluorosis, as

identified in July 1997 by the union which consists of all of the scientists

and

other professionals at the Environmental Protection Agency, Washington, D.C

..,

this study adds further definition to a series of recent studies that have

illuminated fluoride's adverse neurological impact and have anticipated the

results from this research that focuses on the hippocampus region of the

brain,

and interaction with other neurotoxins.

 

A previous study by Mullenix, et al. in Neurotoxicology and Teratology,

1995,

documents abnormal behavioral responses by animals exposed to fluoride at

various stages of gestation, which resulted in the exposed animals

exhibiting

either permanent hyperactivity if exposed prenatally, or what layman refer

to as

" the rat version of couch potato " if exposed after birth.

 

In " Psychopharmacology of Fluoride: A Review " , 1994, the author A. Spittle

concludes, " There would appear to be some evidence that chronic exposure to

fluoride may be associated with cerebral impairment affecting particularly

the

concentration and memory of some individuals. These symptoms are reminiscent

of

those seen in the chronic fatigue syndrome. "

 

In the " Effect of Fluoride on the Physiology of the Pineal Gland " , 1994, the

author, J. A. Luke suggests that fluoride also effects the gland in the

brain

that produces melatonin, which has been established as critical to those

people

suffering from sleep disorders.

 

The follow-up question should be obvious: In light of this scientific

evidence,

is it in the best interest of our nation to continue a public policy - a

public

policy that has already been rejected by 98% of Europe - that forces each

man,

woman, and child to ingest a known cumulative neurotoxin, which is added to

our

water supply with no control over total intake from all sources, or

variances in

susceptibility?

 

The 1996 Safe Drinking Water Act requires that each chemical that appears in

our

drinking water be re-assessed with a new criteria for assuring the safety of

drinking water for the most susceptible segments of our population. The

coming

days will reveal whether the agencies that have been established to protect

our

health will act.

 

30-30-30

 

References

 

Chronic administration of aluminum-fluoride or sodium-fluoride to rats in

the

drinking water: alterations in neuronal and cerebrovascular integrity, Julie

A.

Verner, Karl F. Jensen, William Horvath, Robert L. Isaacson, Brain Research,

vol. 784, pp. 284-298, 1998.

 

Neurotoxicity of Sodium Fluoride in Rats, Mullenix et al., Neurotoxicology

and

Teratology, Vol. 17, no. 2, pp. 169-177, 1995

 

Effect of Fluoride on the Physiology of the Pineal Gland, J. A Luke, Caries

Research, Vol. 28, p204, 1994.

 

Psychopharmacology of Fluoride: A Review, A. Spittle, International Clinical

Psychopharmacology, Vol. 9, 1994.

 

Websites

 

http://www.sonic.net/~kryptox/fluoride.htm<http://www.sonic.net/%7Ekryptox/fluor\

ide.htm>

http://www.cadvision.com/fluoride/index.htm

 

 

 

 

(3) aluminum and fluoride

 

Sun, 30 Oct 2005 00:38:16 -0700 Phil Eversoul <Philev

>

 

http://home.att.net/~gtigerclaw/dead_rats.html<http://home.att.net/%7Egtigerclaw\

/dead_rats.html>

 

Fluoride Study Produces Too Many

 

USEPA MINIMIZES 80% DEATH RATE

 

IN EXPERIMENTAL LAB RATS.

 

George Glasser

 

" In l999, EPA convened a group of experts to carefully consider the results

of

the Varner et al. (1998) study, " USEPA spokesman, Charles Fox wrote in a

September 5, 2000 letter to US Congressman Ken Calvert, Chairman, House

Subcommittee on Energy and the Environment. Fox continued, " As a result of

that

conference, EPA has requested that the National Toxicology Program consider

the

possibility of conducting additional studies of the neurotoxicity of

aluminum

that include verification of the results observed in the Varner et al.

Study. "

 

Fox carefully avoided mentioning the fact that the reviewed study he cited

was

replicated in two previous studies by Dr. Julie Varner. All three studies

found

that aluminum-fluoride interactions are associated with brain and kidney

damage

in laboratory animals. Aside from brain and kidney damage, there was an 80%

mortality rate in the animals fed doses of sodium fluoride and aluminum

similar

to those found in artificially fluoridated drinking water.

 

Alum (aluminum sulfate) is most frequently used by water companies to

improve

the appearance of drinking water, to make it clear. For many years, aluminum

has

been known to be neurotoxic to humans and animals.

 

The original Varner, et al, study published in Neuroprotective Agents, 1997,

was

designed to determine whether aluminum and fluoride (aluminum fluoride) in

drinking water play a role in age-related neurological damage similar to

Alzheimer's disease. It was the first scientific study to deal with

fluoride/aluminum interaction.

 

The researchers considered that fluoride and aluminum could combine in the

stomach and be more readily transported to the brain. The combination, they

believed, could enhance neurological damage and cause conditions such as

presenile dementia or Alzheimer's like dementia (ALD). During the first

experiment, the researchers had noted and were perplexed by the alarmingly

high

death rate in the group of animals receiving aluminum and fluoride in their

drinking water (80% of the animals in the low-dose group died before

completion

of the experiment).

 

They also noted that the amounts of aluminum and fluoride fed to the animals

was

about the same as the amounts people are exposed to in artificially

fluoridated

public water supplies. The reasons for the high number of animal deaths is

still

unexplained as was the fact that the greatest number of mental impairments

appeared in the low-dose group of animals.

 

It was also observed that the animals who drank the aluminum/fluoride-laced

water developed sparse hair and abnormal, copper-colored underlying skin

which

is related to premature aging. Researchers said that most often this

condition

is the result of several diseases including chronic kidney failure. Further

autopsy results showed serious kidney abnormalities in animals that drank

water

containing both sodium fluoride and aluminum fluoride.

 

The Varner team said that, " Striking parallels were seen between

aluminum-induced alterations " in cerebral blood vessels that are associated

with

Alzheimer's disease and other forms of presenile dementia. They concluded

that

the alterations of the blood vessels may be a primary event triggering

neuro-degenerative diseases.

 

Astounded, the researchers went on to say: " Not only did the rats in the

lowest

dose groups die more often during the experiment, they looked poorly well

before

their deaths. Even the rats in the lowest dose group that managed to survive

the

45 weeks looked to be in poor health. "

 

Subsequently, the researchers reported that the results of the " THIRD "

animal

study confirmed the findings of the previous studies. This red flag alarmed

the

USEPA panel of experts because the same physical and neurological damage can

also be occurring in humans in areas where both aluminum sulfate and

fluorides

are added to the public drinking water.

 

Based on the conclusions drawn from the third Varner study, the USEPA

experts

requested further research be commissioned by the National Toxicology

Program.

In spite of the disturbing Varner team revelations it will take two to three

years for the National Toxicology to review the request.

 

Almost 60% of the United States is fluoridated, and the odds of an American

developing some form of dementia by the age of 65 is estimated at 1 in every

10

people, and at the age of 85, the odds are 3 in every 10 people.

 

In the United Kingdom, which is 10% fluoridated, 7% of the population over

65

years will develop some form of dementia.

 

Recent USEPA concerns over arsenic, a Group 1 (a) carcinogen, caused the

government-contracted water treatment chemical certification laboratory,

National Sanitation Foundation International, to say that there will be

" increased product failures " due to high arsenic levels in fluorosilicic

acid.

USEPA has asked the US Government to dramatically reduce arsenic levels in

drinking water from 50 parts per billion to 5 parts per billion. The EPA is

keen

to change the regulations because arsenic in known to cause a wide range of

cancers in humans.

 

More recently, the primary component of fluorosilicic acid and sodium

fluorosilicate - silicon - has also been discovered in the brain plaque of

Alzheimer's and Alzheimer's-like dementia (ALD) victims. Silica has also

been

found in the brain tissue of people with primary brain tumors, which is

considered a terminal condition. Although aware of these new developments,

no

responsible government agency including the USEPA will request that any

research

be done with the actual toxic waste " product " used to fluoridate public

drinking

water. ==

 

 

 

 

(4) Fluoride Combines with Aluminium in Drinking Water

 

NEW STUDY SHOWS GRAVE IMPLICATIONS FROM INTERACTION OF ALUMINUM AND LOW DOSE

FLUORIDE

 

http://www.actionpa.org/fluoride/aluminum.html

 

Promoters of fluoridation can no longer get away with the " unequivocal

statement " that fluoride is a " free ion " in water " , OR that " it completely

dissociates and doesn't react with other minerals in drinking water. "

 

Following the Varner, et al aluminium fluoride studies in which 80% of the

experimental rats died before the end of the experiment the United States

Environmental Protection Agency was sufficiently alarmed to push the

National

Toxicology Program (NTP) to do further research.

 

Varner and associates appear to have found TOXIC SYNERGISTIC ACTION between

FLUORIDE and ALUMINIUM in drinking water. This has now been made a part of

PUBLIC RECORD in the US FEDERAL REGISTER as of December 4, 2000.

 

The National Institute of Environmental Health Sciences (NIEHS) concurs with

the

EPA and has formally called for NTP to commission studies.

 

WHAT DOES THIS MEAN?

 

For the first time, synergistic action is officially acknowledged, along

with

the fact that FLUORIDE in the water COMBINES WITH OTHER MINERALS.

 

The National Institute of Environmental Health Sciences has commissioned a

Review of Toxicological Literature on Aluminium Compounds.

 

[Federal Register: December 4, 2000 (Volume 65, Number 233)] [Notices] [Page

75727-75730] From the Federal Register Online via GPO Access

[wais.access.gpo.gov <http://wais.access.gpo.gov/>] [DOCID:fr04de00-106] ...

==

 

http://www.fluoridealert.org/ifin-28.htm

 

Aluminum, Fluoride, and Hormones

 

International Fluoride Information Network

 

December 15, 1999

 

IFIN Bulletin #28: Aluminum, Fluoride, and Hormones

 

by Paul Connett, PhD

 

These days there is a great deal of concern about industrial chemicals, such

as

pesticides, additives to plastics and incinerator emissions, which disrupt

the

activities of hormones. Hormones are chemical messengers which finely tune

and

regulate the body's chemistry. They are produced in specialiazed glands (for

example, adrenaline is produced in the adrenal glands) in the body and are

then

circulated in the bloodstream until they find their target tissues. Hormones

can

be divided into two large classes: ones which are fat soluble and others

which

are water soluble. The fat soluble ones ( e.g. estrogen and testerone) are

able

to cross the cell membranes (which are made of fat) without difficulty.

Inside

the cells they find a protein ( a receptor) to which they bind like a key

fitting into a lock. This combination then attaches to the DNA, resulting in

the

switching on of genes and thus the production of new proteins in the cell.

The

water soluble hormones ( e.g. insulin, adrenaline, glucagon etc) as well as

some

neurotransmitters) are not able to enter the cell. Thus they have to deliver

their message from the outside. They combine with receptors on the outside

of

the cell. This combination triggers of a change in the G-proteins - proteins

which stretch out across the membrane-, this change in turn leads to the

activation or release of a " secondary messenger " (usually cyclic AMP or

Ca2+),

inside the cell, which then goes on to make changes inside the cell -usually

the

activation of enzymes involved in the breakdown of sugars and fats to

produce

more energy and growth. Thus the G-proteins are seen as an " On-OFF switch "

between the delivery of the hormonal message at the outside of the cell and

the

ensuing activity inside the cell. When the hormone ( or neurotransmitter)

arrives the G-proteins are switched ON and when the message has been

delivered

the G-proteins are switched off again.

 

The actual mechanism is actually fairly simple. Sitting in a crevice of the

G-protin is a molecule called GDP (Guanosine diphosphate)- this consists of

one

of the bases in nucleic acids (guanine)- attached to a sugar (ribose)- and

then

to two phosphate groups. The G-protein is switched on when a third phosphate

group attaches to the end of the other two and forms GTP. This attachment

occurs

when the hormone attaches to the receptor.

 

Now this is where the devastating news about fluoride comes in. In the

presence

of a little aluminum, fluoride forms an ion called AF4-: that is an aluminum

ion

surrounded by four fluoride ions. To the G-protein this AlF4- looks just

like a

phosphate ion. Thus when there is fluoride and aluminum around, it is this

species that can combine with the GDP in the G-protein, AND without any

involvement of the hormone it can deliver the message (ie activate the

cellular

change) that the hormone would have delivered. Should this happen it would

cause

serious problems for the tissue involved. It would be getting the wrong

message

at the wrong time. We describe chemicals that disrupt hormonal signals as

" endocrine disruptors " .

 

Could fluoride and aluminum at the levels to which we are exposed to them

cause

pathological problems. This issue was reviewed by two Czech scientists in

the

following article sent to me by Andreas Schuld of Parents of Fluoride

Poisoned

Children. Even if you don't underestand all the science involved, please

keep

hold of this article and share it with any doctor, dentist or scientist you

know. The information here is potentially the silver bullet that could get

the

fluoride out of our water, out of our tooth paste and out of industry.

Please

read on.

 

Paul Connett.

 

Charles University, Faculty of Sciences, Department of Physiology and

Developmental Physiology, Prague Department of Toxicology, Purkyn� Military

Medical Academy, Hradec Kr�lov�, Czech Republic

 

Running title: Pharmacological implications of aluminofluoride complexes

 

Corresponding author: Anna Struneck� Department of Physiology and

Developmental

Physiology, Faculty of Sciences, Charles University, Vini�n� 7, 128 00,

Prague

2, Czech Republic Telephone: (42) - 02/21953239 Fax no.: (42) - 02/299713

E-mail: astrun

 

Abstract

 

Aluminofluoride complexes have been widely used in laboratory investigations

for

stimulation of various guanine nucleotide binding proteins. These

fluorometallic

complexes cannot be obtained through any catalogue or drug store. They are

formed in water solutions containing fluoride and traces of aluminium in the

form of the soluble ionic complexes, Aluminofluoride complexes have been

recognised to act as phosphate analogues. Reflecting many studies which

utilise

aluminofluoride complexes in laboratory investigations, the effects of these

fluorometallic complexes on various cells and tissues as observed, can be

reviewed. With the appearance of acid rain and the use of aluminium in

industry,

there has been a dramatic increase in the amount of uncomplexed aluminium in

ecosystems. In view of the ubiquity of phosphate in cell metabolism,

aluminofluoride complexes represent a strong potential danger for living

organisms including humans. The possibility of pathophysiological

consequences

of their long-term action is not fully recognised at this point. ...

 

Supported by Grant Agency of Charles University, Prague (Grant

No.113/1998/BBio/P?F).

 

�Anna Struneck� & Jir� Patocka ...

 

Fluoride Action Network | 802-859-3363 | info ==

 

August 26, 1998

 

Many older papers claiming that the concentration of fluoride in public

water

systems is so small that we can be sure it does no harm have been published

in

scientific journals. New research, reported at the twenty-second conference

of

the International Association for Fluoride Research on August 25, 1998, has

uncovered a fatal flaw in the research design of experiments with laboratory

rats. It was the custom for laboratory experiments about fluoride toxicity

to be

done using sodium fluoride in distilled water for the rats' drinking water.

It

was presumed that if sodium fluoride at very low levels in distilled water

did

not harm the rats then fluoride in tap water must be safe.

 

Now, scientists have found that there is a toxic chemical reaction with

aluminum

and fluoride even at very low levels just like they put in the water. K.

Jensen

and coworkers found that when fluoride and aluminum combine to make AlF3 at

very

low concentrations in water, aluminum gets into the brain and kidney more

easily. The accumulation of aluminum in the brain results in damage to the

neurons thus resulting in an Alzheimer's-like condition with memory loss.

Alum

is commonly used for processing water for municipal water systems. This

leaves

small amounts of aluminum in the water which combine with fluoride.

 

ISFR Conference: Dr. Mullenix Presents More Evidence that Fluoride Damages

the

Brain

 

August 25, 1998

 

Presentations of scientific research at the twenty-second conference of the

International Society for Fluoride Research (ISFR) began today. The

highlight of

the day was Dr. Mullenix's report that her laboratory studies that fluorine

damages the brain. The toxic effect of fluorine in the brain is relevant to

childhood leukemia. Leukemia is a cancer of the bone marrow which causes

excess

production of white blood cells The treatment for leukemia causes the cancer

cells to go to the brain. Drugs must be used to kill the cancer cells in the

brain if the treatment is to be successful. Prednisoline and dexamethasone

have

been used for this purpose. The molecular structures of these two drugs is

almost the same. Dexamethasone has one fluorine atom in each molecule and

prednisolone does not. Dexamethasone has become the drug of choice because

it is

more powerful. However, children who were treated with dexamethasone and who

already had mediocre IQ lost ten more IQ points after treatment with

dexamethasone. These children also grew more slowly, had narrower skull

shapes

and their teeth stopped developing.

 

In her latest laboratory studies, Dr. Mullenix found that dexamethasone

impaired

the behavior of rats in a way that is equivalent to hyperactivity in humans.

Dr.

Mullenix pioneered a computerized technology for these studies. Video

cameras

record the activity of rats at specified intervals. The activities of the

rats

are classified and then the sequence of activities can be compared. The rats

on

the dexamethasone had " dispersed " sequences. This means that their behavior

sequences did not follow the usual patterns of healthy rats. The disrupted

patterns of activity are reminiscent of hyperactivity in children.

 

Dr. Mullenix is in the Pscyhiatry Department at Children's Hospital in

Boston,

Massachusetts.

 

Another presentation at the ISFR conference showed photographs of

microscopic

brain damage of rats which drank water with sodium fluoride (NaF). Dr.

Chubek

and co-workers found that the rats on the fluoridated water for 21 days had

the

highest concentrations of NaF and had brain cells that were smaller and

mishapen. The myelin, a substance which surrounds certain axons and nerve

fibers, was swollen:

 

" A neuropathological study and computerized morphometric analyses revealed

revealed a marked shrinkage of cerebellar granular and Purkinje cells,

perivascular myelin swelling, and astroglia reaction, especially in the

white

matter of brains in the NaF-treated animals. Neuronal and myelin changes

appeared to be more pronounced ... " ...

 

Part of this information came from the Seattle Times, June 23, 1998, p. A1.

 

Notes:

 

1.Robert Lenzner, " A monster beverage event, " Forbes, October 20, 1997, p.

64.

2.Time, June 10, 1996, p. 70. 3.BA <http://3.ba/> Auslander, " Toronto Tap

Water: Perception of

its Quality and Use of Alternatives, " Canadian Journal of Public Health,

March-April, 1993, pp. 99-102. 4.S Van Winkle, et al., " Water and Formula

Fluoride Concentrations: Significance for Infants Fed Formula, " Pediatric

Dentistry, July-August, 1995, pp. 305-310. 5.AM <http://5.am/> Weissman,

" Bottled Water Use in

an Immigrant Community: A Public Health Issue? " American Journal of Public

Health, August, 1997, pp. 1397-1380. 6.JT Chan, et al., " Fluoride Content of

Bottled Waters: Implications for Dietary Supplementation, " Texas Dental

Journal,

April, 1990, pp. 17-21. 7.S Van Winkle, op. cit. 8.GM

<http://8.gm/>Whitford, GM, " Intake and

Metabolism of Fluoride, " Advances in Dental Research, June, 1994, pp. 5-14.

 

--

Peter Myers, 381 Goodwood Rd, Childers 4660, Australia ph +61 7 41262296

http://users.cyberone.com.au/myers Mirror: http://mailstar.net/index.html I

use a Mac & the old Mac OS; I send text from a word-processor, without

attachments. If mail does not arrive, email, write or ring me. To

,

reply with " " in the subject line; allow 1 day.

 

 

 

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