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http://www.pyxis.com.au/soma/documents/Articles/Grave%20new%20world%201.doc

 

 

GRAVE NEW WORLD : GENETICALLY MODIFIED FOOD – GRAVE CAUSE FOR MANKIND.

Dr C M Reading, B.Sc.,Dip.Ag.Sci.,M.B.,B.S.,F.R.A.N.Z.C.P.,A.C.N.E.M.

and J.M. Sulima

 

I am very concerned about the safety of genetically modified (GM) foods and

strongly believe far more research is required before humans should regard

them as safe, either for themselves or for future generations. This

research should be completed before we are FORCED to live on GM foods, there

being no longer any choice.

 

GM foods have already been marketed for public consumption without proper

research or discussion into their long term effects.

 

The food we eat is broken down into ‘food fractions’ which are absorbed and

used by the body. GM foods may possibly contain toxic fractions which could

have a serious effect not only on the immediate health of some individuals

but also on the health of future generations.

It is entirely possible that these toxic fractions could affect us in many

ways, for example :-

· Ova and sperm may be affected;

· Chromosomes, or telomeres, could be altered. Telomeres are on the

ends of chromosomes and are full-length at birth, shortening throughout

life;

· Some GM foods could act as carcinogens and exacerbate or cause

cancer;

· The may act as mutagens or teratogens, causing mutations or

congenital abnormalities;

· They could cause autoimmune diseases such as collagen/connective

tissue disorder like Systemic Lupus Erythematosus (SLE), Rheumatoid

Arthritis/Scleroderms, or Arthritis, etc. ;

· Neurotoxins could cause or flare-up Multiple Sclerosis (MS) or

Motor Neurone Disease (ALS) later in life;

 

Is it possible that they could act like prions, causing illnesses such as

Mad Cow Disease (BSE/CJD) to appear years later ?

The frightening prospect is not that there will be an immediate epidemic or

illnesses but that we may have caused an irreversible reversal of human life

in, say, 50 years’ time, when teenagers start to die of coronary artery

disease, diabetes becomes rampant, and old age arrives before the age of 50.

Blighted ova or sperm, damage done in utero, can possibly cause innumerable

life-threatening conditions.

 

We already see the effects of autoimmune diseases, many of which should be

renamed ‘toxic lectinopathy’ (or poisonous allergies / sensitivities /

reactions to food fractions and chemicals). These are not always recognized

as such, even today. If, or when, we have damaged ourselves with a totally

genetically modified food chain, there will be no time for recognition or

reversal. It will already be too late. The damage will have been done.

 

Our greatest asset – our children – will be born with short telomeres and

short futures. We may be introducing a threat with GM crops and foods

greater than plagues, wars, volcanoes, earthquakes, global warming, ozone

depletion or a stray asteroid.

 

Remember what happened to Pottenger’s Cats ? Three generations of eating

junk and they went ‘down the tube’.

(Originally published SOMA NEWSLETTER October 2000, Vol.22, No.2. Reprint

by request.)

SOMA HEALTH ASSOCIATION OF AUSTRALIA LIMITED

 

UNDER-INVESTIGATION OF SYMPTOMS IN CHRONIC FATIGUE SYNDROME (CFS) AND

MULTIPLE CHEMICAL SENSITIVITY SYNDROME (MCSS).

Statement by Dr C M Reading: B.Sc.,

Dip.Ag.Sci.,M.B.,B.S.,F.R.A.N.Z.C.P.,A.C.N.E.M.

 

Since 1980 I have treated over two thousand patients for CHRONIC FATIGUE

SYNDROME and well over a hundred for MULTIPLE CHEMICAL SENSITIVITY. All

these patients, when thoroughly investigated, showed food and chemical

sensitivities / intolerances on the CYTOTOXIC TEST, especially to cow’s

milk, gluten-containing grains, legumes and beans, all of which can cause

severe malabsorption state for vitamins, minerals, amino acids, etc.

These patients tend to have severe nutrient deficiencies due to

malabsorption, and are usually low in Vits. B1, B2, B6, C, A, Folic Acid

and, less often, B 12. They often have anaemia due to the above

deficiencies but may have sideropaenia – low iron without anaemia. Many

have low serum zinc, and hair analysis shows low calcium, magnesium,

manganese, molybdenum, iron, chromium, zinc, selenium and cobalt, raised

copper and aluminum, and unsatisfactory lead, mercury and nickel levels.

(The Cytotoxic Test is accepted by the LANCET, Letter, January 24, 1987.)

 

The glare/photophobia so often observed in CFS patients indicates low iron,

low Retinol A and low Zinc – hence white dots in nails often, despite a

normal haemoglobin/film, and thus not anaemia. Many patients also

have raised IgE and a host of inhalant allergies to pollens (grass, weeds,

trees – pollinosis), moulds, mites, etc., as well as food allergies, and are

sensitive to fumes, chemicals, perfumes, petrol, etc.

 

Most CFS patients have missed coeliac disease (nine out of ten in a row,

diagnosed as CFS by a leading Sydney hospital – POW) and missed because

most psychiatrists do not routinely have measurements done of Endomysial

IgA, Gluten IgA and IgG or alpha-gliadin IgA and IgG, reticulin antibodies

and IgM to see if these are raised, nor do they look for low C3, C4 and

raised immune complexes as seen in coeliac disease, IgM can be raised-to-low

also in coeliac disease.

 

An extremely high percentage of CFS/MCSS patients are MISSED COELIACS and

about 5% are MISSED SLE –which is not usually even considered.

All CFS patients should be tested for ANF and, if positive, then do dsDNA;

and, if not raised (diagnosing SLE), then ENA screen, C3, C4 complements,

immune complexes, anti-lymphocyte antibodies, immunoglobulins (IgA, IgM,

IgG) and if any of the above are abnormal (despite a negative ENA screen)

then a skin biopsy on unexposed skin with immunofluorescent technique to

confirm/diagnose SLE.

 

Many patients with CFS show white dots in their nails which is associated

with low B6, zinc and pyroluria with kryptopyrroles in the urine, and if

these levels are high there is high risk for acute intermittent poryphyria,

especially if reacting to drugs , chemicals, fumes, perfumes and

chlorpyrifos/pesticides and herbicides which can cause a flare-up of

porphyria, as also can barbiturates, sulphnamides, neuroleptics, etc.

 

Most CFS/MCSS patients have severe autoimmune disease (as seen with SLE,

coeliac disease, and show gastritis, thyroidiotis, cholangitis, vasculitis,

autoimmune neuritis,etc.) and need a gluten-free diet or a diet design to

reverse SLE. These patients, in my experience, have severe

cow’s milk alpha-casein, +/- alpha-lactalbumin, +/- beta-lactalbumin,

sensitivity/intolerance when antibodies to these peptides of cow’s milk are

measured - as well over a thousand patients since 1980.

(cont.)

 

Under-investigation of symptoms in CFS and MCSS: (cont.)

 

When intolerant/hypersensitive to cow’s milk albumin-globulin, then these

cross-react with egg and beef albumin and globulin, and also need to be

avoided. In addition, because of the suppressed/compromised immune system

and often low cortisol DHEA, MCSS patients (often being coeliacs) are at

risk for opportunist pathogenes such as candida albicans, mycoplasma,

rickettsia, helicobacter pylori, chlamydia pneumoniae and campylobacter

jejuni aggravating/complicating their treatment. These pathogens need to

be identified and killed off.

 

High levels of aluminium, copper and , less often, lead and mercury are seen

in CFS/MCSS patients and show especially on hair analysis. They also have

low serum cortisol +/- low serum DHEAS, i.e. adrenal exhaustion, and many

are hypothyroid. Most have a marked tendency to hypoglycaemia

and need to eat protein two-hourly in order not to become hypyglocaemic.

Severe amino acid deficiencies are common in my experience (also refer

Newcastle University research) and may have low Vit B3 in addition to

nutrient deficiencies mentioned above. High levels of

pesticides/herbicides are common in CFS/MCSS patients (work of Dr Mark

Donohoe and Tim Roberts et al of Newcastle University). They also have

abnormal metabolites in the urine (and Kryptopyrolles / pyroluria ).

 

SPECIFIC INSTANCES OF UNDER-INVESTIGATION:

 

(a) I have seen moribund patients with severe pellagra who were

called ‘hysterical’ and not allowed to have Vit. B3. (Pellagra is a Vit.

B3 deficiency). One patient at the Cummins Unit of Royal North Shore

Hospital crawled out of hospital and caught a taxi to Manly Hospital where

she made a rapid recovery with B3 treatment, etc.

 

(b) Another with severe pallor/weakness at Camperdown Children’s

Hospital with severe CFS picture was called hysterical until I showed the

treating psychiatrist she had antibodies to alpha-casein, alpha-lactalbumin,

proving she had severe cow’s milk sensitivity/intolerance. The ice-cream

and milk products they were giving her out of kindness were making her

seriously ill and moribund. Off all diary products she rapidly recovered.

 

© Another patient with tardive dyskinesia due to Serenace, and

wrongly diagnosed as schizophrenic instead of toxic

encephalopathy/chloropyrifos poisoning, etc., did well for many months on

extra nutrients and food allergy-free diet, etc., and no neuroleptics at

all, or minimal Melleril, until she came into contact with more spraying

with pesticides/herbicides. She was hospitalized and given high amounts of

neuroleptics, making her very ill. I saw her regularly while she was on

high dosage IMI neuroleptic weekly for her Acute Brain Syndrome which had

severe side-effects, agitation, depression. This was to be her treatment

indefinitely, all her complaints about side-effects being ignored. Each day

off the neuroleptics she rapidly improved. Fortunately, she again did well

on extra nutrients, special diet and minimal Melleril. The evidence

supporting increased investigation into causes of symptoms in CFS and MCSS

is overwhelming.

JMS

 

(Originally published SOMA NEWSLETTER, October 1999, Vol.21, No.2.

Reprint by request.)

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

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