What no one is telling you about statins that you need to know!

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What no

one is telling you about statins that you need to know!

 

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Posted by Dr. Garry Gordon

 

 

Finally we have a comprehensive published paper about the negative side effects of statins.

I do not have any patients on statins.

I’ve recently shared with you all important research indicating why lead alone can raise cholesterol. This

suggests that getting lead as low as possible is providing

benefits similar to what statins are prescribed to do – without the adverse

side-effects. So of course I will not go a day without oral EDTA, and we can

all gain from the $10 million Lester Morrison spent proving that

mucopolysaccarides also decrease excessive blood clotting. I strongly suggest

you learn more about Essential Daily Defense as found in all packets of Beyond

Chelation and Beyond Vitamins.

You may be very interested to learn that taking EDD, along with Boluoke and

Endokinase, has brought INR values to 2 for a patient with a metal heart valve.

This person submitted angiograms showing PERFECTLY CLEAN CORONARY arteries

after 8 years of taking Beyond Chelation Improved. He had started BC-I after a heart attack where they had put in a

stent. Eventually this stent did close which caused a second heart attack. The angiogram revealed

the only blockage was from the stent, while the rest of his vessels are all

perfect clean!

Thankfully he is alive and well. He is adding Boluoke and/or Endokinase to

his prevention program, and has declared that he will not go a day without

BC-I. Statins are not the answer to achieving your maximum intended USEFUL LIFE

SPAN; BC-I is!!!

Garry F. Gordon MD,DO,MD(H)

President, Gordon Research Institute

www.gordonresearch.com

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Public release date: 26-Jan-2009

Contact: Debra Kain

ddkain

619-543-6163

University of California - San Diego

First comprehensive paper on statins’ adverse effects released

A paper co-authored by Beatrice Golomb, MD, PhD, associate professor of

medicine at the University of California, San Diego School of Medicine and

director of UC San Diego’s Statin Study group cites nearly 900 studies on the

adverse effects of HMG-CoA reductase inhibitors (statins), a class of drugs

widely used to treat high cholesterol.

The result is a review paper, currently published in the on-line edition of

American Journal of Cardiovascular Drugs, that provides the most complete

picture to date of reported side effects of statins,

showing the state of evidence for each. The paper also helps explain why some

people have a higher risk than others for such adverse effects.

“Muscle problems are the best known of statin drugs’ adverse side effects,†said

Golomb. “But cognitive problems and peripheral neuropathy, or pain or numbness

in the extremities like fingers and toes, are also widely reported.†A spectrum

of other problems, ranging from blood glucose elevations to tendon problems,

can also occur as side effects from statins.

The paper cites clear evidence that higher statin doses or more powerful statins

those with a stronger ability to lower cholesterol as well as certain genetic

conditions, are linked to greater risk of developing side effects.

“Physician awareness of such side effects is reportedly low,†Golomb said.

“Being vigilant for adverse effects in their patients is necessary in order for

doctors to provide informed treatment decisions and improved patient care.â€

The paper also summarizes powerful evidence that statin-induced injury to the function of

the body’s energy-producing cells, called mitochondria, underlies many of the

adverse effects that occur to patients taking statin drugs.

Mitochondria produce most of the oxygen free radicals in the body, harmful

compounds that “antioxidants†seek to protect against. When mitochondrial

function is impaired, the body produces less energy and more “free radicalsâ€

are produced. Coenzyme Q10 (â€Q10″) is a compound central to the process of

making energy within mitochondria and quenching free radicals. However, statins

lower Q10 levels because they work by blocking the pathway involved in

cholesterol production the same pathway by which Q10 is produced. Statins also

reduce the blood cholesterol that transports Q10 and other fat-soluble

antioxidants.

“The loss of Q10 leads to loss of cell energy and increased free radicals

which, in turn, can further damage mitochondrial DNA,†said Golomb, who

explained that loss of Q10 may lead to a greater likelihood of symptoms

arising from statins in patients with existing mitochondrial damage since these

people especially rely on ample Q10 to help bypass this damage. Because statins

may cause more mitochondrial problems over time and as these energy powerhouses

tend to weaken with age new adverse effects can also develop the longer a

patient takes statin drugs.

“The risk of adverse effects goes up as age goes up, and this helps explain

why,†said Golomb. “This also helps explain why statins’ benefits have not been

found to exceed their risks in those over 70 or 75 years old, even those with heart disease.†High blood pressure

and diabetes are linked to higher rates of mitochondrial problems, so these

conditions are also clearly linked to a higher risk of statin complications, according to Golomb

and co-author Marcella A. Evans, of UC San Diego and UC Irvine Schools of

Medicine.

The connection between statins’ antioxidant properties and mitochondrial

risk helps explain a complicated finding that statins can protect against the

very same problems, in some people, to which they may predispose others

problems such as muscle and kidney function or heart arrhythmia.

This paper was funded in part by a Robert Wood Johnson Generalist Physician

Faculty Scholar award to Dr Golomb.

University of California San Diego

LaJolla

CA 92093

United States

 

 

 

 

 

 

 

 

 

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