AUTISM ADD ADHD - 1. BRAIN-GUT Parasites

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1.Brain-Gut connection - Parasites.

 

There are some interesting parallels between autism and tetanus.

Individuals with tetanus have extreme sensory sensitivity and may need

to be placed in dimly lighted rooms.

 

Loud noises had to be avoided. In addition, patients with this disorder

might have difficulty chewing and swallowing; lockjaw is the other name

for tetanus.

 

Children with autism are reported to frequently suffer from

constipation, diarrhea and upset stomachs. Gibson's team found high

levels of CLOSTRIDIUM bacteria in feces of children with autism.

" Some clostridium bacteria produce neurotoxins, which may affect the

brain at early stages of development, "

 

Clostridium tetani overgrowth of the intestinal tract has been

demonstrated in rats

 

FREE EBOOK Dr Shawn

 

Biological Treatments for Autism and PDD

 

http://gplstoreonline.com/bookstore/index.php?cPath=28

<http://gplstoreonline.com/bookstore/index.php?cPath=28>

 

You'll find it here:

 

http://gplstoreonline.com/bookstore/AutismandPDD.pdf

<http://gplstoreonline.com/bookstore/AutismandPDD.pdf>

 

the possibility of a subacute, chronic tetanus infection of the

intestinal tract as the underlying cause for symptoms of autism observed

in some individuals. A significant percentage of individuals with autism

have a history of extensive antibiotic use. Oral antibiotics

significantly disrupt protective intestinal microbiota, creating a

favorable environment for colonization by opportunistic pathogens.

Clostridium tetani is an ubiquitous anaerobic bacillus that produces a

potent neurotoxin. Intestinal colonization by C. tetani, and subsequent

neurotoxin release, have been demonstrated in laboratory animals which

were fed vegetative cells. The vagus nerve is capable of transporting

tetanus neurotoxin (TeNT) and provides a route of ascent from the

intestinal tract to the CNS. This route bypasses TeNT's normal

preferential binding sites in the spinal cord, and therefore the

symptoms of a typical tetanus infection are not evident. Once in the

brain, TeNT disrupts the release of neurotransmitters by the proteolytic

cleavage of synaptobrevin, a synaptic vesicle membrane protein. This

inhibition of neurotransmitter

release would explain a wide variety of behavioral deficits apparent in

autism. Lab animals injected in the brain with TeNT have exhibited many

of these behaviors. Some children with autism have also shown a

significant reduction in stereotyped behaviors when treated with

antimicrobials effective against intestinal clostridia. When viewed as

sequelae to a subacute, chronic tetanus infection, many of the puzzling

abnormalities of autism have a logical basis. A review of atypical

tetanus cases, and strategies to test the validity of this paper's

hypothesis, are included.

 

Clostridium tetani that causes tetanus,

 

Clostridium botulinum that causes the food poisoning botulism,

Clostridium perfringens

 

Clostridium difficile that cause diarrhea

 

Clostridium perfringens, Clostridium novyi, Clostridium bifermentans,

Clostridium histolyticum, Clostridium septicum, and Clostridium fallax

may all cause gangrene

 

This organism is usually treated with either metronidazole (Flagyl) or

vancomycin followed by a replenishment of the intestine with

Lactobacillus acidophilus.

 

http://www.greatplainslaboratory.com/book/bk4sect1.html

<http://www.greatplainslaboratory.com/book/bk4sect1.html>

 

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