Type 2 Diabetes Linked to Diet Soda

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Type 2 Diabetes Linked to Diet Soda

http://www.naturalnews.com/025644.html

A new study published in January 16, 2009 edition of Diabetes Care indicates

that daily consumption of diet soda plays a key role in the development of

metabolic syndrome and Type 2 Diabetes. Metabolic syndrome is a cluster of

findings known to increase the risk of diabetes and cardiovascular disease and

is also known as Syndrome X. Metabolic syndrome includes insulin resistance or

actual Type 2 diabetes, low HDL, elevated triglycerides and central obesity

(high waist circumference).

 

Two previous studies have shown a positive correlation between diet soda and

metabolic syndromeand Type 2 diabetes. This study, the Multi-Ethnic Study of

Atherosclerosis (MESA), was designed to test the association between diet

sodaand the overall risk of metabolic syndrome, the various components of

metabolic syndrome and Type 2 diabetes. The study conclusions support the

findings of the previous two studies. Another study published in the Annals of

Epidemiology in Sept, 2006, found that adults with diabeteswho drank one or more

diet sodasper day had hemaglobin A1C levels 0.7 percent higher than those who

drank none. HemaglobinA1C is a measurement that evaluates blood glucosecontrol

over a period of approximately 3 months. That difference is what would expect to

be found when one group started a new, effective diabetic medicationand another

group left untreated.

 

Those who consumed diet soda at least daily had a 36% greater risk for metabolic

syndrome and a huge 67% greater risk for the development of Type 2 diabetes.

Drinking diet soda daily increased the risk of Type 2 diabetes regardless of

initial obesityand changes during the study. When metabolic syndrome was looked

at by individual components, only high fastingblood sugar (>100mg/dL) and high

waist circumference (men: 102 cm/39.78inches; women: 88 cm/34.3inches) were

significantly correlated with daily consumption of diet soda.

 

Baseline questionnaires regarding diet soda consumptionwere collected between

2000 and 2002 followed by three follow up examinations: 2002-2003, 2004-2005 and

2005-2007. These exams were to specifically identify the presence of Type 2

diabetes and the various components of metabolic syndrome. For this study, Type

2 diabetes was defined as a fasting blood glucose>126mg/dL, self reported

diagnosis or use of glucose lowering medication.

 

After all data was compiled, adjustments were made for demographics, lifestyle

and dietary differences. Hazard ratios were then calculated for Type 2 diabetes,

metabolic syndrome, and the components of metabolic syndrome. The participants

who consumed diet soda were then compared to those participants who did not

drink diet soda.

 

Limitations of the study included: observational design which precluded findings

of causality, the possibility of unaccounted for differences in diet and

lifestyle, difficulties accurately estimating intake of diet sodas and different

artificial sweeteners.

 

The study authors conclude that: " These results corroborate findings from the

ARIC [Atherosclerosis Risk in Communities] and Framingham studies and show

stronger adverse associations exist between diet soda and Type 2 diabetes. Diet

soda consumption, either independently or in conjunction with other dietary and

lifestyle behaviors, may lead to weight gain, impaired glucose control, and

eventual diabetes. " While this data cannot establish causality, daily

consumption of diet soda was associated with significantly increased risk of

certain components of metabolic syndrome and Type 2 diabetes.

 

This study was done by:

Division of Epidemiology, University of Texas Health Sciences Center, Houston,

TX, the

Division of Epidemiology and Community Health, University of Minnesota,

Minneapolis, MN, Center for Human Nutrition, Department of International Health,

Division of Cardiology, Johns Hopkins University, Baltimore, MD; the

Department of Nutrition, University of Oslo, Oslo, Norway

 

http://care.diabetesjournals.org/

http://cme.medscape.com:80/viewarti...

http://www.annalsofepidemiology.org...)00387-X/abstract