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Viruses

_http://freespace.virgin.net/ahcare.qua/literature/science/viruses.html_

(http://freespace.virgin.net/ahcare.qua/literature/science/viruses.html)

By Patrick Quanten MD

 

Let's start with a medically well-known fact: viruses aren't themselves

alive. They are smaller and simpler than bacteria and by themselves they are

inert and harmless. So, the immediate question then has to be: How can you

*catch* a virus if it isn't a living thing?

 

The answer is: You can't.

 

Experimenters have incubated viruses for the common cold, placed them

directly on the mucous lining of the nose, and found that their subjects came

down

with colds only 12% of the time. These odds could not be increased by

exposing the subjects to cold drafts, putting their feet in ice water to give

them

chills, or anything else that was purely physical.

 

Swine flu (viral infection) arose as a normal, non-lethal flu in the spring

of 1918, but somehow, over the following months, it mutated into something

more severe. In an attempt to devise a vaccine, medical authorities conducted

experiments on volunteers at a military prison on Deer Island in Boston

Harbour. The prisoners were promised pardon if they survived a battery of

tests.

These tests were rigorous to say the least. First, the subjects were injected

with infected lung tissue taken from the dead and then sprayed in the eyes,

nose and mouth with infectious aerosols. If they still failed to succumb, they

had their throats swabbed with discharges taken from the sick and dying. If

all else failed, they were required to sit open-mouthed while a gravely ill

victim was sat up slightly and made to cough into their faces. The doctors chose

sixty-two of the volunteers for the tests. None contracted the flu, not one.

The only person who did grow ill was the ward doctor, who swiftly died.

 

One of the mysteries of viral epidemics is how it can erupt suddenly all

over, in places separated by oceans, mountain ranges and other earthly

impediments. Although a virus is not alive in itself, it also loses its

potential of

hijacking the genetic material of a living host cell within a few hours of

being outside the host body. The commonly heard answer that it travels in

" carriers " (people who have no symptoms but carry and distribute the virus)

cannot

be proven and after decades of using it as " the " explanation remains nothing

more than a shaky and desperate theory. It is made even more unlikely in the

light of the fact that you cannot catch a viral infection, as proven above,

so even if it did travel that way, how would it " jump " from the carrier to the

victim? Furthermore, how does a virus manage to lie low for several months,

in the case of HIV or variant CJD we are to believe it can be up to 20 years,

before erupting so explosively at more or less the same time all over? What's

the trigger and why instantaneously in all those different places?

 

Some of these viral epidemics have been known to be more devastating to

people in their prime rather than infants and the elderly, who are more likely

to

have a more vulnerable immune system. Strange, to say the least.

 

From time to time certain strains of virus return. A disagreeable Russian

virus known as H1N1 caused severe outbreaks over wide areas in 1933, then again

in the 1950s and again in the 1970s. Where it went in the meantime each time

is uncertain. Could it have survived, lying " dormant " , in humans or animals

for all that time? This raises the same old two questions: Why did it not

cause any symptoms wherever it was hiding? and If it was hiding somewhere, how

did it spread so quickly when it did, as you can't catch it - not from a

human, not from an animal?

 

What do we know about Viruses?

 

We have already mentioned that they are very small, and they weren't

detected until 1943 with the invention of the electron microscope. Many,

including

HIV, have ten or fewer genes, whereas the simplest bacteria require several

thousand. To create a living thing you need properly organised DNA of a

substantial quantity, which the virus hasn't got.

 

We define **a living organism** as something that performs three tasks in

succession: taking in stuff (eating, breathing), metabolising stuff (digesting,

absorbing), and excreting waste. A fourth necessary task is reproduction. A

virus doesn't do any of these. No virus does. Within the viral capsule there

are no other structures that are required for a metabolic process. There is

no activity at all inside the viral capsule.

 

Not only doesn*t it look structurally as if its alive, it also isn*t alive

in physiological terms.

 

So what is it then? As we all know, viruses can have devastating effects on

the health of plants, animals - great and small, including bacteria - and

humans. How does it produce these effects, if it is not alive, can*t be caught

and doesn*t reproduce?

 

Known scientific facts about viruses and the way they function are obtained

from chemical analysis and looking at still pictures from electron

microscopes. The story is pieced together, not actually observed! This means

that what

you are told happens, is actually a theory at best, and a fantasy story at

worst. What has actually, in simple terms, been discovered?

 

-- Viruses contain either RNA or DNA, a small amount and mostly one or the

other, but there are exceptions. Bits of genetic material of whatever kind,

really; but only bits.

 

-- Viruses are marked species and organ specific, and on the whole, viruses

infecting plants, insects, rickettsiae, bacteria and other animals are

distinct from their human counterparts, but this is now thought not to be

entirely

the case. They are specific, but then again they are not.

 

-- Viruses may be naked with the genome only protected by a protein capsid,

or they may have a lipid envelop surrounding the capsid. Bits of genetic

material in a thin simple bag, and sometimes put in a fatty bubble.

 

-- Viruses are seen to be *encapsulated* by the body cells that have

specific receptors for the virus. Once inside the cell, it seems that the virus

capsule is removed and the exposed bit of DNA or RNA is *read* and the host

cell

seems to duplicate it. These bits of genetic materials are encapsulated once

again, and with the host cell bursting with complete viruses it will explode

and the viruses are spilled into the cellular surroundings. So, we see a lot

of genetic bits within the cell; these bits are then encapsulated and

eventually the cell burst open to release the now bagged up genetic material

into

the extra-cellular environment.

 

-- Viruses in the intercellular environment are engulfed by cells from the

immune system (macrophages and lymphocytes), which collect them and destroy

them. These bags that contain bits of genetic material are collected into cells

from the immune system.

 

-- Viruses are very difficult to demonstrate (they are extremely small) and

the diagnosis of viral infection is mostly made on clinical symptoms alone

and the assumption that it fits into a known disease pattern for which there is

no causative factor known. Virtually every time a diagnosis of viral

infection is pronounced no proof is offered for this diagnosis.

 

-- Materials for virus isolation must be obtained as early as possible

during illness. It is at the very early stages of the illness that the highest

titres are found and the most likely it is one can produce a positive test

result. There are more viruses present right at the beginning of the illness

than

at any other stage of the disease process. If the viruses were multiplying

you would expect the number to rise as the disease developed.

 

-- Identification of viruses is done in laboratories by measuring the level

of antibodies against specific viruses, not by measuring or demonstrating the

virus itself. Measuring a higher protection level is diagnosed as the

illness itself!

 

Summarising this scientific knowledge, we can say that viral infections are

not diagnosed by finding the specific virus, but by guessing a virus is the

cause of the symptoms. In practical terms, this happens when the doctor

doesn't really know what the cause is.

 

As regards the story of the viral infection is concerned, we now know that

as soon as the symptoms start the number of viruses will very quickly be

dramatically reduced. There is no evidence of rapid number proliferation once

the

disease manifests itself.

 

Before we move on to explain the real virus story, it is worthwhile to

remind ourselves of what we now know:

 

1. A virus is not alive.

 

2. You cannot catch a virus.

 

3. A virus disintegrates very quickly outside the host.

 

4. A virus consists of small bits of genetic material, variable from virus

to virus, surrounded by a thin coating, either protein (water-soluble) or fat.

 

5. Viral materials are seen in large numbers inside the host cell.

 

6. A full host cell breaks open and the viruses are spilled into the

environment.

 

7. In the environment the viruses are bagged up by the cells of the immune

system (See **The Inflammation Process**

_http://freespace.virgin.net/ahcare.qua/literature/medical/inflammationprocess.h\

tml_

(http://freespace.virgin.net/ahcare.qua/literature/medical/inflammationprocess.h\

tml) ).

 

The Virus Story

 

If viruses are not living things they cannot multiplicate and they don't

need a specific environment to *survive*. They cannot appear from nowhere and

they can*t spread and infect other cells.

 

When a cell becomes diseased and the function of the cell begins to falter,

it may start to come apart at the seams. Bits of its essential structure, the

DNA and RNA, may become detached as it is falling apart. The cell will try

and clean up these bits by preparing them for the rubbish bin. The small

pieces of genetic material, which are now floating around in the intracellular

fluid, will be isolated by means of encapsulating them. As the cellular

disintegration continues more and more of these bits are seen inside the cell

and

more and more small *bags* of useless genetic material will appear. Once the

cell is totally dysfunctional and filled with rubbish the cellular wall itself

bursts and the contents will be spilled into the cellular environment. Here,

the cleanup continues by packaging these small bags up even further into what

has been called the lymphocytes and macrophages of the immune system. These

large vesicles now drift away into the lymphatic fluid and the blood stream,

from where they will be filtered out at appropriate draining stations, like the

spleen and the lymph nodes. This process continues until the whole lot has

been cleared.

 

This explains why the numbers of *viruses* is the highest at the very

beginning of the disease and continues to decline steadily throughout the

disease

process, even without treatment. This also accounts for the thousand and

thousand of different *viruses* that have been identified and for the

*mutation*

of viruses. Viral behaviour is essentially totally unpredictable because the

cells and the way they disintegrate is never the same, not because this is an

animal that changes its behaviour so quickly and intelligently that nothing

can keep up with it. It also does away with the idea that the *virus* can lay

dormant for an indefinite period of time and become activated without any

triggers or reasons having been identified.

 

How do we then explain **viral epidemics**? Why is it then that we get a

cold the day after someone in the office starts to cough and sneeze a lot?

 

The medical profession knows that viruses have incubation periods. These are

said to vary from virus to virus from a few days to several years. A cold

virus has an average incubation period of about a week. Now, first of all, you

can't catch a virus; and secondly, if you could catch the cold virus, it

would take a week before it had established itself within your body and starts

to

show symptoms. Consequently, your cold cannot have been caused by the other

person's cold in the office the day before!

 

What is seen and has been named *a virus* starts after the cellular

structure begins to disintegrate. Why does a cell start to fall apart? Because

it is

diseased. The disease is already there, long before any viral particles show

up in any pictures. So, then we have to ask the question why the cell has

become diseased? The answer to this lies in the build-up of toxic material

within the cellular structure. As the cell gets loaded up with inappropriate

material it will eventually be unable to cope and it will start to fall to

pieces.

It is exactly those pieces that are photographed by the electron microscope

and have been named *viruses*.

 

The influences that can lead to an increased pressure on the system are many

and are varied. They range from the weather, to living and working

environment, to life style and diet, to the balance of activity and rest, to

mental

balance, stress and worries. Because a lot of these influences, such as working

conditions and the weather, are general circumstances which affect all of

us, it is very likely that a great number of us, in the same environment, will

fall ill at or around the same time, succumbing to the environmental

influences. Add to this that people who are working in the same environment are

very

likely to have similar life styles and another factor has been identified

explaining why similar disease pattern occur within certain groups of people at

certain times. On top of that, we now know that worry reduces our immunity

capacity and increases the likelihood of illness. The belief that, if one

person close to you has a cold you are going to get it, increases the

likelihood

of this actually happening dramatically, as you become more vulnerable through

the immune reducing effect of the worry itself.

 

Epidemics occur because people in similar circumstances, living environments

and conditions, have similar imbalances within their systems, leading

directly to similar disease patterns. This causes fear and apprehension all

around

them, making others more vulnerable to start showing a breakdown of health

themselves. The disease is spreading. More accurately, the fear of the disease

is spreading first, resulting in a lowered resistance, which allows each

individual*s imbalances to show up through the inability to cope with the

problems the system has already been faced with for a long time. More and more

people are becoming ill and showing signs of the fact that their bodies have

been

under extreme pressure for quite a while to maintain health. The showing of

an illness is the end result of a long process, even an *acute* illness, of a

slow deterioration of the system*s normal functioning. Disease is a process,

not a state of being.

 

It is time to learn the facts of life.

 

It is time to do away with ignorance and the resulting fear.

 

It is time to focus on individual health and the factors that influence it.

 

Viruses are dead, but diseases are very much alive. Let's concentrate on the

living, not the dead, if we want to be healthy.

 

(http://www.papercut.biz/emailStripper.htm)

 

 

 

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