Guest guest Posted July 30, 2008 Report Share Posted July 30, 2008 Poster's Comment: I thought this was interesting. Below I copied a few paragraphs from this article. Lyme disease: A Look Beyond Antibiotics _http://www.neuraltherapy.com/LymeALookBeyond6.pdf_ (http://www.neuraltherapy.com/LymeALookBeyond6.pdf) extract........................... Making the diagnosis It appears that many patients with MS, ALS, Parkinson’s disease, autism, joint arthritis, chronic fatigue, sarcoidosis and even cancer are infected with Borrelia burgdorferi. But is the infection causing the illness or is it an opportunistic infection simply occurring in people weakened by other illnesses. My experience is based on: a) using direct microscopic proof of the presence of Borrelia burgdoferi (Bb) and other spirochetes (4, 5) b) the information many affected clients have brought to me c) my own clinical training and experience (30 years in Medical practice, 15 years Bb cognizant) d) ART testing (autonomic response testing), which is the most advanced and scientifically validated method of muscle testing (6) e) regular lab parameters affected by Lyme: • Abnormal lipid profile (moderate cholesterol elevation with significant LDL elevation) • insulin resistance • borderline low wbc, normal SED rate and CRP • normal thyroid hormone tests but positive Barnes test and excellent response to giving T3 • type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low cortisol and DHEA) • low testosterone and DHEA • decreased urine concentration (low specific gravity) • complex changes in cytokines, interferones, NK cells, white blood cell indicators, etc. Bb tends to infect the B-lymphocytes and other components of the immune system which are responsible for creating the antibodies, which are then measured by an ELISA test or Western Blot test. Since antibody production is greatly compromised in infected individuals, it makes no sense to use these tests as the gold standard or benchmark for the presence of Bb (7). We also are aware that in endemic areas in the US up to 22% of stinging flies and mosquitoes (2, 8, 9, and 10) are carriers of Bb and co-infections. In South East Germany and Eastern Europe 12 % of mosquitoes have been shown to be infected. Also many spiders, flees, lice and other stinging insects carry spirochetes and co-infections. Making the history of a tick bite a condition for a physician to be willing to even consider the possibility of a Bb infection seems cynical and cruel. To use conventional diagnostic tests such as the Western Blot, one has to think in paradoxes: the patient has to be treated with an effective treatment modality first before the patient recovers enough to produce the antibodies, which then are looked for in the test. A positive Western Blot proves that the treatment given worked to some degree. A negative Western Blot does not and cannot prove the absence of the infection. Having taken another route altogether, we have recognized that today many if not most Americans are carriers of the infection. Most infected people are symptomatic, but the severity and type of the symptoms varies greatly. The microbes often invade tissues that had been injured: your chronic neck pain or sciatica really may be a Bb infection. The same may be true for your chronic TMJ problem, your adrenal fatigue, your thyroid dysfunction, your GERD and many other seemingly unrelated symptoms. Many Bb symptoms are mistaken for problems of natural or premature aging. In most places the diagnosis of an active Bb infection is made only if the symptoms are severe, persistent, obvious, and many non-specific and fruitless avenues of treatment have been exhausted. Acute new “typical†cases of Bb infection are rare in my practice. Symptoms tend to get stranger and more obscure every year. Frequently, if the patient is fortunate enough to Quote Link to comment Share on other sites More sharing options...
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