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VACCINES AND GENETIC MUTATION

Harold E. Buttram, MD; Susan Kreider, RN; Alan R. Yurko

October 11, 2002

[[Editor's note: This article contains one internet-linked reference that is

currently broken. We are attempting to locate this file. But, due to the

importance of this information, we decided to post the article meantime. Please

forgive any inconvenience.]]

 

Introduction

 

The writers of this article make no claims of being authorities in the

fields of genetics or immunology, but being non-experts may at times carry an

advantage in that, viewing more from a distance, one may sometimes perceive

things that escape those more closely involved in the details and complexities

of

a field or fields. This may be true for the interactions of vaccines with

the human immunology and genetics, about which science truly knows very little.

This article does review the work of three pioneer researchers in this

field, John Martin, PhD, MD, Howard B Urnovitz, PhD, and Dr. MG Montinari, work

which shows fairly convincing evidence that genetic changes are being found in

some patients in whom vaccine reactions appear to be causally involved. There

are no claims that this evidence constitutes proof of genetic change from

vaccines. What we do hope to establish from the work of these researchers is

that it is both possible and plausible that subtle, widespread genetic changes

may be taking place as a result of current childhood vaccine programs,

possibly already affecting large portions of our children.

The burden of proof for vaccine reactions should not rest on parents, as it

does now in our medical-legal system. The burden of proof for the safety of

vaccines; that is, that the vaccines are NOT causing adverse genetic changes,

should rest on the manufacturers, federal and state government health

agencies, and the schools who are now mandating the vaccines. Until this matter

is

settled, does anyone at any level truly have the right to force vaccines in

ever growing numbers on a generation of children?

Basic Immunology for All Ages

 

Although the technical intricacies of the human immune system are extremely

complex, the principles of their operations are the essence of simplicity and

might be compared to the fortifications of a Medieval castle. Using this

analogy, first there might be outlying outposts with sentinels, then a moat,

then the main castle wall, and finally the inner defenses surrounding the

castle

itself, in which reside a royal family. The latter of course represents the

human genetic system, which the human immune system is designed to protect at

all costs.

The sentinels would be represented by a subdivision of lymphocytes (a form

of white blood cell), which are called “memory cells†because of their

having

memory for former exposures to foreign invaders, and which will begin an

explosion of cloning on re-exposure to the same invader. The main castle wall

would be represented by the mucous membranes of the respiratory and

gastrointestinal tract, and the inner defenses by the antibody-producing plasma

cells

(another form of white blood cell) located in the bone marrow.

Immunity

Cellular Immunity

For countless millennia in human evolution, the cellular immunity of the

mucous membranes of the human system have been the primary route of entry of

disease-causing microorganisms into the human body, and therefore through

evolution, the mucous membranes have evolved into the major defense system of

the

body. In health these membranes are coated with an “antiseptic paintâ€

consisting of untold billions and trillions of molecules of secretory

immunoglobulin

A antibodies, whose role is to recognize every single molecule passing into

or through these tracts, sorting out the nutrients in the case of the

intestinal tract, and intercepting all foreign and alien substances, including

incompletely digested foods. It would take several very large computers to

equal

the intelligence of this system when it is working as it should. It is all

that is standing between a very thin (and dumb) gut membrane and a host of

toxic substances which would otherwise pass through the membrane into the blood

stream. In addition to this antiseptic paint, the primary agent of defense of

the mucous membranes against infectious micro-organisms is a cellular

immunity, the primary agents of which are phagocytic (gobbling up) macrophages

and

cytotoxic T lymphocytes.

Humoral Immunity

The inner defenses are represented by plasma cells in the bone marrow with

their antibody production, which normally serve as a secondary defense for the

body, coming into action as an accessory of the mucosal (cellular) immunity,

or as a primary defense when cellular immunity has failed. This immunity is

referred to as humoral immunity. Plasma cells can produce (1)

Macroglobulins, which appear first with an acute infection, being more

primitive, serve

somewhat like a nonspecific natural antibiotic; (2) Immunoglobulin G

antibodies,

which are highly specific for a given foreign invader and appear somewhat

later after onset of an infection after the process of cloning is set in

motion; and (3) IgE antibodies, which are allergy producing.

The Role of Childhood Diseases

There is a school of thought that the so-called minor childhood illnesses of

former times, including measles, mumps, chicken pox, and rubella, which

entered the body through the mucous membranes, served a necessary and positive

purpose in challenging and strengthening the immune system of these membranes.

Vaccines in contrast are injected directly into the body, consequently

bypassing the mucous membranes, leaving the mucosal immunity relatively weak

and

stunted.

In both The New England Journal (1) and the journal Thorax, (2) articles

have appeared stating that a healthy immune system has a “bias†towards the

cellular immune system, whereas people with allergies, asthma, and diseases of

an autoimmune origin have a humoral-dominant system. It has also been shown

that, once one of these subsets become dominant, it is difficult to shift the

system to the other subset. (3)

Genetic Exchanges in the World Around Us

 

Barbara McClintock, the 1983 Nobel laureate “Corn Lady,†was the first to

discover genetic mobility in the so-called jumping genes in the 1930s. For

over 50 years she pursued solitary research with corn, uncovering some of

nature’

s inner most secrets about life.

 

McClintock studied maize, a form of Indian corn, where distribution of red

kernels and yellow kernels is genetically determined. What she perceived was

that some of the genes were moving from one place to another on the cell’s

chromosomes (the floating threads on which genes are lined like beads on a

string). Then she saw patterns in the movements, with sharply differing results

in the colored kernels, and realized that some genes, once moved into

position, switched other genes on or off. It followed that, while most genes

were

workers, others were controllers or managers of genes.

According to an article in World Medicine (September 22, 1971, pp 69-72; New

Medical Journals, Clareville House, Oxendon St., London), scientists at the

University of Geneva have made the startling discovery that biological

substances entering directly into the blood stream may truly become a part of

us

and even a part of our genetic material. The article stated in part:

“When Japanese bacteriologists discovered that bacteria of one species

transferred their own highly specific antibiotic resistance to bacteria of an

entirely different species, they seemed to hit on a unique if not startling

phenomenon. Dr. Maurice Stroun and Dr. Pilippe Anker, with colleagues in the

Department of Plant Physiology at the University of Geneva, have now

accumulated

a wealth of evidence that the transfer of genetic information is not confined

to bacteria but also can occur between bacteria and higher plants and

animals.â€

“The Geneva scientists are convinced that normal animal and plant cells also

shed DNA and that this DNA is also taken up by other cells in the organism.

If they are right, the consequences to virtually every aspect of a cell’s

metabolism would be considerable. The growth and development, diseases, and

even the evolution of an organism would be affected.â€

“Dr. Maurice Stroun and his colleagues did most of their research on plants

but have now turned to animals. In their latest set of experiments they used

the isolated auricles of frogs’ hearts.†(4)

There is no question about the results. They found a high percentage of

RNA-DNA (ribonucleic-deoxyribonucleic) hybridization between bacterial DNA

extracted from bacteria of the same species as that used in the experiment and

titrated RNA extracted from auricles which has been dipped in the bacterial

suspension. (DNA, the characteristic nucleic acid of the nucleus in all cells,

is

the fundamental substance which carries the genetic code within the cells of

the body).

“Since we know that no bacteria got into the frog auricles, we can only

conclude that the bacterial DNA must have been exuded from the bacteria and

absorbed by the animal cells,†says Stroun.

“This transfer phenomenon, or transcession, as Dr. Anker called it, is very

probably a general one, otherwise, he and Dr. Stroun would hardly have

succeeded first go, in getting bacterial RNA synthesized by animal

tissues…â€

“The implications of this work on transcession are enormous, for the Geneva

work suggests that this phenomenon is going on the whole time – even in our

own bodies…Could, for example, the heart damage that can follow after

rheumatic fever and similar bacterial infections be the result of the body’s

immunological system reacting to its own cells producing an alien RNA?â€

Subsequent studies by Anker and Stroun further confirmed observations in the

above report. (5)

Genetic Hybridization

 

As purely genetic material, it would be expected that viruses are more prone

to the process of jumping genes than other microorganisms. The following

publication tends to support this hypothesis: In a study of 24 passages of a

nuclear polyhedrosis virus through cell cultures there were both insertions and

deletions in the virus, appearing to suggest that the virus both donated

genetic material to and received genetic material from the cells in which it

was

cultured, therefore also suggesting the possibility of similar viral

exchanges in the human system (our interpretation). (6)

As another possible complication of viral infections (presumably also viral

vaccines) similarities have been found between certain viral proteins and

proteins related to myelin sheaths of the brain and nervous system.(7) As a

result of this protein mimicry between viral proteins and homologous areas of

the

nervous system, immunological cross reactions may take place resulting in

post-infectious or post-vaccinal encephalitis, myelitis, or neuritis. These

viruses include measles, Epstein-Barr, influenza A and B, and others that cause

upper respiratory infections.

Following this line of thought one step further, in an article entitled, “

Vaccination and autoimmunity-‘vaccinosis’: a dangerous liaison?,†the

authors

pointed to the potential problem of “molecular mimicry†in vaccines, in

which a structural similarity existing between some viral antigen and a

self-anti

gen could, by bringing about a slight modification of the antigenic

character of tissues, cause it to appear foreign to the immune system and thus

a fair

target for antibody production†(and autoimmunity) (8)

Endogenous and Exogenous Assaults on the Human Immune System – Stripping

Away the Outer Defenses

 

Returning to the analogy of the Medieval castle with its series of immune

defenses protecting our genetics, in health the human body can stand a great

deal of abuse, toxic or otherwise, but when these outer defenses are stripped

away leaving our genetics relatively unprotected, it is in this type of

scenario where, theoretically, genetic damage could take place. Situations

leading

to this genetic vulnerability could include one or more of the following:

• At a conference a number of years ago, Dr. H.H. Fudenberg, world-renowned

immunologist with hundreds of publications to his credit, made the following

comments: “One vaccine decreases cell-mediated immunity by 50%, two vaccines

by 70%…all triple vaccines (MMR, DTaP) markedly impair cell-mediated

immunity, which predisposes to recurrent viral infections, especially otitis

media,

as well as yeast and fungi infections.â€

• Severe and/or prolonged stress raises both endogenous adrenalin and serum

cortisol levels. It has long been known that cortisone medications tend to

depress the immune system. Endogenous elevations of cortisone can do the same.

• Toxic chemicals, as in the Persian Gulf War Syndrome (9) or toxic

industrial waste sites, which have been associated with increases in

chromosomal

congenital anomalies in residents living near these sites. (10)

• Nutritional deficiencies, especially deficiencies in folic acid, which

performs a critical function in making and repairing chromosomes. As reviewed

in

a monograph on folic acid by Sidney M Baker, M.D., precancerous chromosomal

damage has been found in cell cultures when the culture medium contains low

levels of folic acid. Smokers with low blood levels of folic acid have more

pre-cancerous chromosomal changes than smokers or nonsmokers with normal folic

acid levels. (11)

• As reviewed in standard pediatric textbooks, newborn babies and infants,

having little immunity of their own, are largely dependent on antibodies

received from their mother for about 6 months following birth, as indicated by

their small lymph nodes, few plasma cells in their bone marrow, and very low

rates of immunoglobulin synthesis. Normally about 6 years are required before

various immune parameters are well established. At least theoretically,

because of the immaturity of the immune system in infancy and early childhood,

the

child’s genetics during these early ages would be more vulnerable to injury.

** Please see _Addendum_

(http://us.mg2.mail./dc/launch?.rand=bem6fmtmn1ied#addendum)

• Although final proof is as yet lacking, there is much indirect evidence

that vaccines may be skewing the human immune system away from cellular immune

system, which is normally dominant in health, towards the weaker humoral

system, which is associated with allergies and autoimmunity as well as

increased

vulnerability to viral and fungal infections. This conclusion can hardly be

escaped because most if not all childhood vaccines in current use are

injected directly into the body and are directed at stimulating antibody

production

in the bone marrow. Bypassing the mucous membranes of the body as they do,

the cellular immune system remains weak and relatively stunted due to lack of

stimulation. As previously noted, once the humoral system attains dominance,

as demonstrated in the following study, this dominance tends to be

self-perpetuating.

Each of the two systems has identifying markers called cytokines (peptides

which act as messengers), and this is how they are identified. A study by

Sudhir Gupta of 20 autistic children, a condition thought by growing numbers of

parents and physicians to be largely vaccine-related, showed consistent

elevations of humoral cytokines and lowering of cellular cytokines.(12)

Consequently, if vaccines are skewing infants’ immune systems by inducing a

humoral-dominant system at a highly vulnerable time of life, they could be

creating

double-jeopardy from the standpoint of genetic mutations.

Stealth Viruses and the Work of John Martin, MD, PhD

 

A stealth virus is one that can establish a persistent infection in people

over a period of years, while at the same time escaping detection by the human

immune system because of its genetic fragmentation and polyglot mixture of

genetic elements. The story begins years ago when Dr. Martin was serving as

director of the viral oncology branch within the U.S. Food and Drug

Administration when he found foreign DNA in the oral polio vaccine being

manufactured

at the time. He later learned that a Simian (monkey) cytomegalic virus (CMV)

had been found in all of the eleven African green monkeys imported for

production of the polio vaccine.(13)

After leaving the FDA, Dr. Martin took a position as professor of pathology

with the University of Southern California. There he tested blood samples

from patients with chronic fatigue syndrome, autism, and other nervous system

disorders. This work led to his discovery of unique cell-destroying viruses

that were not recognized by the immune system. Termed “stealth viruses,â€

some

of which he thought had clearly originated from the simian cytomegalic virus,

these viruses were missing specific genes which, if expressed, would induce

immune responses from the host. (14-18)

By way of explanation, the stealth virus, which, according to the work of

Dr. Martin had its origins from a CMV contaminant of the oral polio vaccine,

had become extremely fragile and unstable, possibly as a result of numerous

serial passages through a variety of hosts in the commercial development of the

vaccine. Being more unstable, it would theoretically be more prone to

exchange nuclear material with its various hosts, in the end becoming somewhat

like

a genetic Rubik's cube with a polyglot of nuclear material. This polyglot

mixture remains unidentifiable to the immune system of the infected human host.

Martin has reported on finding the stealth virus of Simian-CMV-origin in

chronic fatigue (15) and in an autistic child.(18) The findings of chromosomal

changes by Urnovitz in studies of veterans suffering from Persian Gulf War

Syndrome (20) reported the findings of “many enteroviral-similar segmentsâ€

in

the abnormal chromosomes. It was also pointed out by Urnovitz that virtually

all of the Gulf War veterans received the oral polio vaccine, the implication

being that the polio vaccine with its CMV contaminant could have been a

source of the enteroviral segments. (Polio is an enteric virus).

Considering the possible consequences of these early findings of Dr. Martin,

one wonders if there are plans for further investigation of these disturbing

findings, or must this be left to future generations?

The Work of Howard B. Urnovitz and The Chronic Illness Foundation

 

Dr. Urnovitz and his colleagues have been studying the implications of

vaccines in cancer, Persian Gulf War Syndrome, multiple sclerosis, and AIDS.

Urnovitz, who holds doctorates in Immunology and Microbiology from the

University

of Michigan where he studied vaccines, has become one of the most vocal

proponents for scientists to become aware of vaccine-associated genetic

mutations.(19) His work in this area has supported the concepts that:

I Our bodies have a “genetic memory†of foreign substances it encounters,

including vaccines.

II There is a limit on how much foreign material our bodies can handle

before genetic damage occurs and/or progresses into a chronic illness.

III Each person has their own unique genetic blueprint which responds to

foreign substances differently.

 

Although Urnovitz did not elaborate further on the subject of “genetic

memory,†his reference to it can be interpreted as an inference that the

genetic

blueprints we inherit from our parents are influenced and potentially changed

in adaptation to environmental exposures during our lifetimes.

Perhaps Urnovitz and colleagues are best known for the work they have

published on the Gulf War Syndrome (GWS), where they found evidence of genetic

alterations in Chromosome 22q11.2, a known genetic “hot spot†for

mutations,

which appear to have a role in the pathogenesis of GWS.(20) Even more striking

is that when they sequenced their findings, many enteroviral-similar segments

were found suggesting that this may have played a role in causing the changes

in 22q11.2. As previously stated, most Gulf War veterans received the oral

poliovirus vaccine, an enterovirus, presumably along with its Simian CMV

contaminant.

Also, in the introductory paragraph to the report, the authors included a

list of chemicals to which the veterans had been exposed in the Gulf War,

including low-level chemical warfare agents; investigational drugs (including

pyridostigmine bromide), organophosphate, carbamate, and other pesticides and

insect repellants; and toxic combustion products from oil well fires and diesel

exhaust products. Although not specifically stated, the inclusion of this

list clearly implies the authors’ opinion that toxic chemical exposures may

also have played a causal role in the Gulf War Syndrome and its accompanying

genetic changes.

To expand on this further, some of the genetic sequences were found to come

from other, unidentified non-human sources. This raises the question of

whether or not there was a connection between the work of Urnovitz and John

Martin, (14-18) with genetic residues from the oral polio vaccines, the oral

polio

virus in turn having been cultured in monkey kidney tissues, and thus

contributing to non-human segments described in the Urnovitz report.

The work of Urnovitz (9, 20-22) places a serious light on the implications

of vaccines in bringing about genetic alterations. Our parents provide our

genetic blueprints at birth, but this raw genetic material now appears to be

malleable to environmental influences, including toxic chemicals and vaccines.

Based on the foregoing information it is both possible and plausible that

genetic translocations are taking place as a result of vaccines. Surely this is

a credible cause for concern.

Immunogenetics

 

The genetics of our immune system are not well understood by scientists.

However, there are many studies which pose serious implications. As one

example, MG Montinari and colleagues investigated the relationship between

post-vaccine central nervous system (CNS) diseases and human leukocyte antigens

(HLA),

which essentially strips the body’s brain and nerve tissues of their outer

myelin coating.(23)

By way of explanation, the HLA system is one which aids an individual’s

immune system to differentiate that which is “self†from that which is “

non-self.†Although the mechanisms are complex, it is a system which, during

embryonic life, learns to recognize healthy or normal cells of the body as

“selfâ€

so that these cells will remain unmolested by the search and destroy

mechanisms of the immune system, leaving the latter free to protect the body

from

foreign invaders.

Of special concern is that the HLA system also carries an increased

proneness to polymorphism (mutation), the mutations in turn possibly resulting

in an

impairment of self-recognition. This process may be the fundamental cause, or

one of the primary causes of underlying autoimmune disorders in which the

immune system attacks the cells within the body. The HLA system plays an

integral part of this process.(24) When the alleles of the HLA system are

mutated,

as sometimes seen in viral infections, viral vaccines, or environmental

illness from toxic chemicals, the body’s immunogenetic memory is altered. The

presentation of an antigen to the immune system is important, and interference

with this presentation may cause the body to mistake normal tissue, such as

brain and nerve myelin, and thus attack its own tissues (autoimmunity).

Montinari found that certain alleles of HLA (A3 & DR7) were more frequent in

patients with post vaccine-induced illness. This indicates an immunogenetic

basis for such illnesses. What caused much concern was that Montinari

implicated vaccine preservatives such as thimerosal as causing genetic

mutations by

modifying the amino acids in presenting antigen proteins, (25-29) which may

be responsible for confusing the body into autoimmune reactions.

Further Concerns of Vaccine-Induced Genetic Mutation

 

Many of us are aware of The Human Genome Project which is an attempt to map

out the entire chromosomal locations of human genes. It is important to note

that a technique for the mapping of genes actually fuses human and rat cells

in tissue cultures. These cells, called human-rodent somatic cells, actually

have both rat and human chromosomes combined. This hybridization of human and

non-human cells is done by placing both in a tissue culture and put through

repeated cell-cycle passage, where human chromosomes are “lost.†This

allows

for scientists to mark certain protein-expressing genes to individual human

chromosomes. (30)

Knowing that such hybridization occurs in laboratory processes and can be

repeated, one must wonder if vaccines, which are contaminated and made with

various human, animal, and non-human cells/DNA, can have the same effect in the

human body.

Returning now to the subject of genetic contamination, and to the work of

Anker and Stroun as well as to the human-rat cell fusion, we know that many

vaccines use “immortal cell lines†which are actually cancerous types of

cells

with no limit on how many times they can divide. The most commonly known type

of tissue used is of the human diploid variety extracted from aborted fetal

tissue. It is possible that these cells could actually hybridize with our

own. In fact, it is likely in light of what we know about human-rodent somatic

cells.

As well, there is concern that these cell lines are easily contaminated with

pathogens and spread cancer (mutation-promoting) material to humans. (31-34)

Certain vaccines called “recombinant,†“sub-unit,†and “naked DNAâ€

use

methods of genetic engineering in their production. These techniques pose major

concerns because of the unknown interaction of the vaccine and human

proteins/DNA. The FDA actually acknowledges this concern where mutations take

place

through the activation of oncogenes or inactivation of tumor suppressor

genes allowing cancers to thrive. Moreover they concede that free nucleic acids

are easily taken up and integrated into a cell’s genome, thus potentially

resulting in genetic mutations. (35,36) A detailed and technical report which

details the many cancerous and genetic consequences of vaccine contamination

notes that each vaccine dose is allowed 100,000,000 alllowable pieces of DNA,

not including the DNA in the viral and viral-contaminated portions. We believe

that any allowable piece of DNA is a risk.

Summation

 

In a Letter-to-the-Editor of Science Magazine, October, 1967, Joshua

Lederberg, Department of Genetics, Stanford University School of Medicine,

warned

about live-virus vaccines:

“In point of fact we (are practicing) biological engineering on a rather

large scale by use of live viruses in mass immunization campaigns. . . . Crude

virus preparations, such as some in common use at the present time, are also

vulnerable to frightful mishaps of contamination and misidentification.†(38)

In a larger sense, the question about possible effects of vaccines in

causing adverse genetic changes might be considered as the “black hole†of

scientific knowledge. Even if it is taking place, do we have the technology to

identify it, and if not, do we have the time to await the slow processes of

science to prove such a relationship? Studies from Africa, England, Sweden, and

New

Zealand have consistently shown a greater incidence of allergic problems

such as asthma and eczema, along with increasing patterns of sickness, among

fully vaccinated children as compared to those with limited or no vaccines.

(39-42) It seems inconceivable to us that health could be one thing and

genetics

another, or that these patterns of deteriorating health would not be

accompanied by corresponding genetic changes.

In our view there is one fundamental issue with which we are confronted, and

that is for parents to gain the right of free choice to accept or reject

vaccines for their children based on informed consent. Wherever one looks in

the

natural world one finds systems of checks and balances. It is the

fundamental system on which the US Constitution was framed and intended to

function.

The same principle should apply with childhood vaccines. Only by this can

things be set right.

References

 

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2. Holt PG, Sly PD, Allergic respiratory disease: strategic targets for

primary prevention during childhood, Thorax, 1997; 52:1-4.

 

3. Immunobiology; The Immune System in Health and Disease, Charles Janeway,

Paul Travers, Mark Walport. Donald Capra, Fourth Edition, Garland Publishing,

New York, 1999:394-395.

 

4. Stroun M, Anker P, Transcription of spontaneously released bacterial

deoxyribonucleic acid in frog auricles, J Bacteriology, April, 1973; Vol 114:

114-120.

 

5. Anker P, Stroun M, Bacterial ribonucleic acid in the frog brain after a

bacterial peritoneal infection, Science, Nov. 10, 1972; 178:621-623.

 

6. Kumar S, Miller LK, Effects of serial passage of Autographa Californica

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7. Jahnke U, Fischer EH, Alvord EC, Sequence homology between certain viral

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8. Shoenfeld Y, Aron-Maor A, Vaccination and autoimmunity-‘vaccinosis’: a

dangerous liaison?, J Autoimmun, Feb 2000, 14(1):1-10.

 

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Gulf War Veterans Have Segments Homologous to Chromosome 22q11.2, Clinical and

Laboratory Diagnostic Immunology, May, 1999; 6(3):330-335.

 

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11. Folic Acid, the Vital Nutrient that Fights Birth Defects, Cancer, and

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12. Gupta S et al, Th1 and Th2-like cytokines in CD4+ and CD8+ T cells in

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13. This story is related in the book, Emerging Viruses, AIDS and Ebola, by

Leonard G Horowitz, D.M.D., M.A., M.P.H., Tetrahedron, Inc., Rockport, MA,

1997, pp 488-493.

 

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human tissue, (Presentation July 1, 1996 sponsored by the National Institute of

Allergy and Infectious Disease (NIAID), held in the Solar Building,

Rockville, MD).

 

18. Martin WJ, Stealth virus isolated from an autistic child, (letter to the

editor),

J Autism Developmental Disorders, 1995; 25(2):258.

 

19. Written testimony of Dr. Howard B Urnovitz, August 3rd, 1999, at the

Committee on Government Reform and Oversight

 

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Persian Gulf War veterans have segments homologous to chromosome 22q11.2, Clin

Diagn Lab Immunol, May, 1999; 6(3):330-335.

 

21. Urnovitz HB et al, Increased sensitivity of HIV-2 antibody detection,

Natural Med, 1997; 3:1258

 

22. Urnovitz HB, Murphy WH, Human endogenous retroviruses: nature,

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1996;

9:72-99.

 

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Lilostampa Bari, 1999.

 

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26. Shrana I et al, Mitosis and numerical chromosome aberration analyses in

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1996; 12.-9.-20 Riken, Isukuba, Japan, _http://www.

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ADDENDUM

by Edda West, Founder & President

Vaccine Risk Awareness Network _(VRAN)_ (http://www.vran.org/)

The human infant's vulnerability to infection hinges on whether or not

he/she is breastfed. Breastfeeding adds the most essential and powerful immune

dimension, which protects the newborn and young baby from a myriad of

infectious disease. Many years ago, medical researcher Dr. Alan Cunnigham, MD

found

that breastfed babies have more than a 12-fold reduced risk of infectious

diseases, particularly the big killers, which are respiratory and

gastrointestinal

diseases.(1)

 

New Zealand researcher, Hilary Butler, has written extensively about the

newborn immune system being skewed towards TH2, a necessity so that the

mother's body does not reject the fetus. After birth, however, the baby's

immune

system starts to shift to cell mediated, TH1. THE MAJOR factor enabling this

transition is breastfeeding, which provides the baby with a highly complex

protective immunological ecology.(2)

 

Therefore, although the infant is born with an immature and vulnerable

immune system, breastfeeding provides the crucial immunological bridge insuring

that the neonate has the following: a plentiful supply of cell-mediating

secretory IgA; tremendous amounts of macrophages, which engulf and destroy

pathogens; and a highly complex enzyme system that provides appropriate

nutrients

and functions in multiple immune capacities. An example is lactoferrin, the

remarkable iron-binding protein that insures iron remains unavailable to

bacteria, hence minimizing risk of infection.(3)

 

Fundamentally, human milk insures continuing oral passive immunity as it

lays down essential gut protection that prevents enteroviruses from taking

hold.

It also maintains gut impermeability so that antigens don't seep through,

triggering allergic mechanisms. Breastmilk is the most vital element by which

immune strength and integrity is built. It sets the immune foundation for

life!

 

While doctors pay lip service to the advantages of breastfeeding, little is

done to teach parents the profound and far reaching immune benefits of it. If

any mother understands her body's capacity to provide her baby with the

means to build a strong and disease resistant immune foundation, one could

venture to predict that fear of this or that disease would disappear, along

with

the idea of the necessity of vaccinations.

 

 

Addendum References

1. Dr. Alan Cunningham, Comparison of hospital admissions of breastfed &

bottlefed babies at The Mary Imogene Bassett Hospital, Cooperstown, N.Y. Letter

to U.S. Department of Agriculture, 1980.

2. Hilary Butler, Position Paper on the Role of Vaccines in SIDS. February,

2000.

3. Derrick B. Jelliffe, MD, Human Milk in the Modern World. 1979, Oxford

Medical Publications, Oxford University Press, Oxford, U.K.T

 

 

(Note: This is an excellent article I sent out a couple of years ago but

wanted to send again as there are so many new rs. When I tried to

access it on the original Alan Yurko site, I could not. Luckily I saved the

original copy....Ed)

 

 

 

 

 

 

 

 

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