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>>>

Hi all,

 

I have a patient with a main complain of metallic taste in her mouth,

she is not taking any medications and her liver\gb labs result are ok.

I was trying to find any comment about metallic taste in various tcm

books but couldn't find a thing.

Is there any correlation between this taste and any tcm organ?

 

Thank You,

Dan<<<

 

Dan,

As I understand it, a metallic taste simply refers to the metal element--lung and/or large intestine. Also, from my experience with Western holistic doctors testing for mineral deficiencies (vacuities?), various mineral deficiencies can result in metallic and other tastes, as well as sugar cravings.

Joseph Garner

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> Dan,

> As I understand it, a metallic taste simply refers to the metal

element--lung

> and/or large intestine.

 

Do you have a reference on this?

 

-

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Hi All,

 

Joseph Garner wrote:

> Dan, As I understand it, a metallic taste simply refers to the

> metal element--lung and/or large intestine. Also, from my

> experience with Western holistic doctors testing for mineral

> deficiencies (vacuities?), various mineral deficiencies can result

> in metallic and other tastes, as well as sugar cravings.

 

A friend of mine for >35 years was diagnosed recently as having

haemochromatosis. His case is complicated by a history of

multiple angina pectoris attacks and 4 minor heart attacks that

required hospitalisation. He has angiographically confirmed severe

blockage of two major coronary arteries but his specialists have

told him that they cannot do bypass surgery because his vessels

are too fragile. He has been on conventional medication for

myocardial ischaemia/angina for nearly 2 years now.

 

As regards a metal influence (Metal Revolting on Fire?), he was an

unbelievablly heavy smoker. Before he quit (on medical advice

when his heart problem was diagnosed) he smoked between 60

and 120 cigarettes daily. As one might expect, he has had

throat/bronchial and LU (Metal) irritation for years. My friend is a

typical Steel (Metal) Man - he was physically very tough, with great

stamina before the heart attacks, but rigid and almost inflexible in

his opinions.

 

A pathologically high iron concentration in liver and blood is a

feature of haemochromatosis [a genetically-transmitted disorder,

as evidenced by its confirmation in two of his brothers also]. To

reduce the Fe load from his body, about 7 weeks ago he began a

programme to have 1 unit of blood removed per week.

 

Within 2 weeks of the start of the bleeding programme, he noticed

the following:

(a) strong metallic taste in the mouth;

(b) bilateral tenderness/pain on palpation near LU01 and BL13;

© strange sensations, sometimes painful, in the fingers,

especially the thumb and index finger (LU-LI fingers).

 

Two weeks ago, his blood Fe (or ferritin) levels went offscale and

the weekly phlebotomy was discontinued for the moment. Whether

or not haemochromatosis was a causal factor in his heart problem

has not been confirmed, but that seems possible.

 

Another nice anecdote for TCM?

 

Arch Mal Coeur Vaiss 1978 Apr;71(4):371-9 [The cardiomyopathy

of idiopathic hemochromatosis] [Article in French] Mattheyses M,

Hespel JP, Brissot P, Daubert JC, Hita de Nercy Y, Lancien G, Le

Treut A, Pony JC, Simon M, Ferrand B, Gouffault J, Bourel M. A

retrospective study of the case histories of 216 patients with

idiopathic haemochromatosis has highlighted the frequency of

cardiac involvement in this condition (53%). Two forms can be

distinguished: a latent one (65%), in which the changes are

predominantly electrocardiographic, and a clinical form (35%) with

the features of congestive cardiomyopathy, notable for the rapidity

of onset after right heart failure, the degree of cardiomegaly, the

constant finding of abnormalities of ventricular repolarisation, the

relative frequency of latent disorders of supra-His atrio-ventricular

conduction, and the finding of elongation of the isovolumic

contraction time on the phonomechanocardiogram. A

haemodynamic profile is the same as for non-obstructive hypotonic

cardiomyopathies, and is usually associated with a slow rise in left

ventricular pressure. The cardiomyopathy, which is the most

frequent cause of death, determines the prognosis in this condition.

It may be found in association with diabetes and gonad failure. The

finding of cardiomyopathy indicates basic treatment by veresection,

which may be the only means of establishing a favourable

outcome. PMID: 96755 [PubMed - indexed for MEDLINE]

 

Drugs 1991 Jun;41(6):875-82 Current concepts in rational therapy

for haemochromatosis. Crawford DH, Halliday JW. Liver Unit,

University of Queensland, Royal Brisbane Hospital, Australia.

Genetic haemochromatosis is characterised by an inappropriately

high rate of iron absorption by the small intestine. The disease is

transmitted as an autosomal recessive condition. The gene

frequency in the Caucasian population is approximately 1 in 20 and

the disease frequency is 1 in 400. Excessive iron deposition

occurs in the liver, pancreas, heart, pituitary and joints and hepatic

iron concentrations above approximately 400 mumol/g dry weight

are always associated with fibrosis and usually with cirrhosis and

progressive liver failure. Accurate diagnosis depends upon the

demonstration of elevated hepatic iron stores. An hepatic iron index

[hepatic iron concentration (in mumol/g dry weight) divided by

patient age] of greater than 2.0 distinguishes homozygous subjects

from the other conditions in which slight increases in hepatic iron

concentration may occur, e.g. in a subject heterozygous for

haemochromatosis or alcoholic liver disease. If cirrhosis is present,

patients are at a high risk of developing hepatocellular carcinoma.

Therefore, they should undergo regular abdominal ultrasound and

alpha-fetoprotein estimation. In the absence of cirrhosis,

phlebotomy restores life expectancy to normal. Venesection

should be continued until all excess iron stores are removed as

judged by failure of a rise in haemoglobin concentration on

cessation of phlebotomy. Screening of first degree relatives should

commence from a young age (e.g. 10 years). If serum ferritin or

transferrin saturation are abnormal, liver biopsy should be

undertaken. HLA typing of the family allows for the identification of

those siblings who are most likely to develop the disease.

Secondary iron overload is often multifactorial in origin. Iron

chelation therapy with subcutaneous deferoxamine

(desferrioxamine) should only commence after careful

consideration of the potential benefits in each individual patient.

Publication Types: Review Review, Tutorial PMID: 1715264

[PubMed - indexed for MEDLINE]

 

Hosp Med 1999 Dec;60(12):884-90 Haemochromatosis: a time for

guidelines? Hall CJ, Critchley AT, Norfolk DR. Harrogate District

Hospital, North Yorks. Hereditary haemochromatosis is an

autosomal recessive metabolic abnormality which causes

excessive absorption of dietary iron. Iron accumulation leads to

potentially fatal damage to organs such as the heart, liver and

pancreas. Normal life expectancy can be restored simply by

therapeutic venesection. Discovery of the gene, HFE, has rekindled

interest in pathogenesis, management and screening strategies.

Publication Types: Review Review, Tutorial PMID: 10707173

[PubMed - indexed for MEDLINE]

 

Lakartidningen 1998 Aug 5;95(32-33):3430-5 [Defective iron

metabolism in genetic hemochromatosis. The mechanisms remain

unknown in spite of genetic advances] [Article in Swedish] Stal P,

Hagen K, Hultcrantz R. Gastroenterologiskt Centrum, Huddinge

Sjukhus. Genetic haemochromatosis (GH) is one of the most

common hereditary diseases, with a prevalence of 1-5/1000 in the

Western world. In 90 per cent of cases a mutation is found in an

MHC-class-like gene designated HFE, involving a substitution at

position 282 of the HFE protein and resulting in defective binding of

beta(2)-microglobulin. Animals with beta(2)-microglobulin deficiency

develop iron overload, indicating this protein to be involved in the

regulation of iron metabolism. Hepatic iron overload results in

increased production of oxygen free radicals and peroxidation of

membrane lipids, thus causing damage to lysosomes,

mitochondria and the endoplasmic reticulum. These cellular events

may progress to cell death, fibrogenesis, and the development of

liver cirrhosis which is associated with a 200-fold increase in risk of

hepatocellular carcinoma. In addition to the risk of diabetes,

arthralgia, cardiac arrhythmia, pituitary insufficiency and

hypogonadism, iron excess is also associated with aggravation of

the cytotoxic effects exerted on hepatocytes by other agents such

as alcohol or hepatotrophic viruses. The treatment of iron overload

in GH consists of weekly venesection until the serum ferritin level is

normalized, followed by maintenance therapy. Survival rates are

normal if the disease is detected and treated before complications

have developed. Publication Types: Review Review, Tutorial PMID:

9725962 [PubMed - indexed for MEDLINE]

 

Z Orthop Ihre Grenzgeb 1987 Mar-Apr;125(2):206-8 [Arthropathy in

idiopathic hemochromatosis] [Article in German] Zogg T, Fuchs W,

Kissling R. Idiopathic hemochromatosis is normally associated

with hepatic cirrhosis, myocardial disease and diabetes mellitus. A

characteristic arthropathy occurs in approximately 40% of patients

with hemochromatosis. The onset may precede other detectable

clinical manifestations of the disease. In these cases a early

diagnosis and treatment may improve the prognosis. A review of

the recent literature is presented. A typical hemochromatosis

arthropathy is described in one patient. The significance of a

frequent associated chondrocalcinosis is discussed. PMID:

3617892 [PubMed - indexed for MEDLINE]

 

Best regards,

 

 

WORK : Teagasc Staff Development Unit, Sandymount Ave., Dublin 4, Ireland

WWW :

Email: <

Tel : 353-; [in the Republic: 0]

 

HOME : 1 Esker Lawns, Lucan, Dublin, Ireland

WWW : http://homepage.eircom.net/~progers/searchap.htm

Email: <

Tel : 353-; [in the Republic: 0]

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> Dan,

> As I understand it, a metallic taste simply refers to the metal

element--lung

> and/or large intestine.

 

Do you have a reference on this?

 

-<<<

 

Jason-

No, I do not. I seem to recall having heard it in class. My Chinese colleague Qun Li says that it could be a liver detoxification problem. She has no particular reference for this, either.

Joseph

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I had a patient who's main complaint was that everything they ate tasted

bitter. His lips had a burning sensation and salty food made his tongue

burn terribly. This occured during his chemo therapy for lung cancer

(he had smoked heavily for years). It made sense to me that there had

to be Heat in the Lu, and any materials I could find (can't remember the

reference) all indicated Heat of various organs were to blame for

abnormal tastes in the mouth. I tried clearing heat points and tried

herbs to clear heat from the Lungs, but I was only able to get his hair

to grow back & turn black, and to make his leg spasms cease. His

oncologist told him that it sometimes happens during chemotherapy and

that sometimes the taste would return to normal and sometimes not. I

don't know what part of the chemotherapy would be to blame though,

perhaps that would have given me a better direction to try.

 

Geoff

 

> __________

>

> Message: 16

> Tue, 13 May 2003 14:50:11 EDT

> acugrpaz

> Re: Metallic Taste

>

> > Dan,

> > As I understand it, a metallic taste simply refers to the metal

> element--lung

> > and/or large intestine.

>

> Do you have a reference on this?

>

> -<<<

>

> Jason-

> No, I do not. I seem to recall having heard it in class. My

> Chinese colleague

> Qun Li says that it could be a liver detoxification problem.

> She has no

> particular reference for this, either.

> Joseph

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Just yesterday one of my teachers was discussing what it means when

people have these different tastes in their mouth. She recently

attended a Jeffrey Yuen conference and this is what she relayed about

his take on the matter:

 

Taste is due to Spleen and if someone had a metallic taste in their

mouth that would be the LU expressing itself through the SP. Bitter

taste would be HT expressing itself through SP.

 

She continued to explain that LU dealt with sound, so if someone was

had a weeping sound to their voice that would be LU expressing itself

through LU. An angry shouting voice would be LV expressing itself

through LU.

 

So, SP=taste, LU=sound, K=discharge, HT=complexion, LV=I forget what

LV was, Vision perhaps?

 

Dave

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"Simon" at <s.becker wrote in response to "Dave" at <seacell :

-------

>>>Taste is due to Spleen and if someone had a metallic taste in their

mouth that would be the LU expressing itself through the SP. Bitter

taste would be HT expressing itself through SP.>>>>>

 

The question here is: how does this influence your treatment. It sounds

nice: the lung expressing itself through the spleen. What actually does this

mean in terms of treatment principles or disease mechanism?

Simon Becker

-------

 

Dave and Simon:

 

I also attended the Jeffrey Yuen workshop last weekend here in L.A. In reference to this point, he was discussing five elements according to the Nan Jing, and difficulty 49 in particular.

 

The idea is that each of the five zang is susceptible to damage by a particular type of xie/evil, and has a characteristic way that it expresses its pathology: heart=odor, spleen=taste, lung=sound/voice, kidney=discharge/fluid, liver=color/complexion. (Note: this varies slightly from other statements of correspondences, such as in some parts of the Nei Jing) Further, transmission of the pathology of a zang to a second zang will characteristically express a quality corresponding to the second affected zang.

 

Therefore, spleen xie/evil affecting the lung would express a taste (spleen expression) of pungent/acrid/metallic (lung quality of taste). Bob Flaws' "The Classic of Difficulties: A Translation of the Nan Jing" is one reference for this. As a general mechanism, there is an implication that other pathology of a zang, aside from that caused by the specific xie/evil mentioned in the Nan Jing, can transmit to another zang and express similarly.

 

If you to a Nan Jing-based five elements approach, then you would use this information diagnostically as support for the hypothesis that there is spleen pathology which is entwined with the lung. However, you would still need to consider the other diagnostic information and determine (1) if the spleen and lung are the primary areas of pathology, consistent with tongue, pulse, s/s, etc. (in particular, you would expect the metal pulse position to be relaxed/slippery (earth quality), indicating earth invading metal), (2) where excess and deficiency lie within the spleen and lung, (3) which type of perverse/pathological five element relationship holds (disease mechanism), and (4) which five element treatment protocol to use to improve the situation (treatment principle).

 

Also, as a reference for a previous question as to the source of LU/LI correspondence to metal and metallic taste etc., the Nei Jing seems to be often-quoted. Maoshing Ni's "The Yellow Emperor's Classic of Medicine" is one reference for this, for example, chapters 2 and 4.

 

Hope this helps.

 

John Barber

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  • 2 years later...
Guest guest

In a variety of situations, patients have volunteered having a

metalic taste. Unusual? Its not one of the big 5 that we learned?

Dr.Holmes?

thanx

jch

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Heat in the gallbladder

 

--- cameronhollister <cameronhollister

wrote:

 

> In a variety of situations, patients have

> volunteered having a

> metalic taste. Unusual? Its not one of the big 5

> that we learned?

> Dr.Holmes?

> thanx

> jch

>

>

>

>

>

>

 

 

 

 

 

 

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if illness is recent, acute, and non-serious, metallic taste denotes a

LU illness

if illness is belated, chronic, and serious, metallic taste denotes a HT

illness

 

best

holmes

1.888.TCM.CEUS 826.2387

 

 

 

cameronhollister wrote:

 

> In a variety of situations, patients have volunteered having a

> metalic taste. Unusual? Its not one of the big 5 that we learned?

> Dr.Holmes?

> thanx

> jch

>

>

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Guest guest

more on metallic taste:

 

one:

if taste is transient the condition involves LU

treatment - settle LU

 

two:

if taste is pervasive over days the condition involves HT

work only with HT

 

care!!

if one tones LU this will weaken LV and have HT run rampant and cause

irreparable harm

 

three:

if taste is pervasive over months and has tended to change to sour, HT

has embroiled LU which has

shifted oppression to LV - a serious stage of things

 

here also, work only with HT, taste will settle

 

four:

if taste is pervasive over days and has tended to saltish, nothing

serious is happening, Son K is embroiled

work only with LU

 

best

holmes

1.888.TCM.CEUS 826.2387

 

 

 

holmes wrote:

 

> if illness is recent, acute, and non-serious, metallic taste denotes a

> LU illness

> if illness is belated, chronic, and serious, metallic taste denotes a HT

> illness

>

> best

> holmes

> 1.888.TCM.CEUS 826.2387

>

>

>

> cameronhollister wrote:

>

> > In a variety of situations, patients have volunteered having a

> > metalic taste. Unusual? Its not one of the big 5 that we learned?

> > Dr.Holmes?

> > thanx

> > jch

> >

> >

>

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