Treating Diabetes With Enzymes: What We Know Now

[Guest guest]

I have no association with products mentioned here.

 

This article illustrates also how these enzymes positively effect eyes and

also peripheral neuropathy as well veins and blood sugar and diabetes.

blessings

Shan

 

 

Treating Diabetes With Enzymes: What We Know Now

By: Dr. William Wong, ND, PhD.

_http://www.totalityofbeing.com/FramelessPages/Articles/TreatingDiabetes.htm_

(http://www.totalityofbeing.com/FramelessPages/Articles/TreatingDiabetes.htm)

 

Up to a year ago, for anyone asking if systemic enzymes could help lessen

the load of troubles that beset Type 1 diabetic patients, I would have told

them about lowering pancreatic inflammation, and possibly helping with lower

extremity circulatory issues. I would have never suggested that the use of

enzymes could decrease the need for insulin, increase energy or reverse the

seemingly myriad of things diabetics suffer from. Then we started getting

information from Type 1 patients that amazed even me and that have subsequently

sparked new research. Here are two typical case histories.

Case History #1:

A Type 1 diabetic Native American patient from Montana in his mid 40's, very

insulin dependent, with peripheral neuropathy in the lower extremities

(LE's) and presenting paresthesia as well in the upper extremities (UE's)

radiating distally to the hand. Peripheral Vascular Disease (PVD) in the LE's

had

already caused several toes to be amputated.

Patient began taking therapeutic doses of fibrinolytic systemic enzymes

(Vitalzym). Within weeks, circulation was opened in his feet and lower

extremities. Skin there returned to a pink / flesh colour. Remaining toes

now

have full circulation and are no longer candidates for amputation. Lower

extremity and upper extremity pain became paresthesia (tingling and pins and

needles), and as a result is much more bearable.

The patient’s insulin needs were decreased.

Case History #2:

86-year-old male Caucasian from Las Vegas history of Type 1 Diabetes for

over 50 years. One below the knee amputation (left side) already done due to

DVP, the other leg about to be amputated due to general lack of blood flow and

arterial blockage. Poor circulation body wide and a grey / white pallor to

the skin also body wide. Neurological pain was had at both lower extremities.

Urine flow beginning to flag as patients kidneys became laden with scar

tissue (Glomerulosclerosis). Patient was highly insulin dependent. Above

that the patient was functionally blind in one eye from a Lasix procedure that

had generated scar tissue over the retina.

After several weeks of systemic enzyme (Vitalzym) use the patient first

noticed a lessening in his lower extremity neurological pain (neuropathy). His

skin colour in the remaining leg changed to rosy as circulatory pathways were

opening. Outer layer of whitish dead skin shed off leaving what resembled a

“

body wide dandruffâ€, exposing new pink /flesh tone skin beneath. The

existing leg became pink with blood flow, no longer ulcered, no longer had

ischemic

pain and was saved from amputation.

Urine flow increased as fibrin was lysed (eaten away) from the kidneys. If

the urine was allowed to stand in the toilet a layer of tiny bits of fibrin

(component of scar tissue) in what resembled fiberglass floated to the top.

The fibrosis that had blinded one eye was lysed away and the patient now has

better than 20/20 vision in that eye. Most significantly, the patients own

insulin production has returned (thought to be impossible under the auto immune

theory of diabetic pancreatic destruction). He is no longer insulin

dependent. After medical testing the patient is no longer considered diabetic

at

all and is off all medication.

Sound fantastic? It did to me, even as a Naturopath who expects nature to

do fantastic things. Diabetes is one of those diseases you never expect

patients to get better from. Even after several years of working with

systemic

enzymes I had heard of some Type 2 patients improving their energy and

levelling off their sugar highs and lows but I had never expected any form of

improvement in Type 1 patients, the medical literature was very clear. Once

the

immune system destroyed the insulin producing portions of the pancreas, there

was no getting those tissues to function again! That medical “truth†has

turned out to be merely a medical theory.

Lets take a look at the present understanding of the root causes of diabetes

and add our own conjectures based on what we have observed clinically. We

know from the present research work being done that the root cause of

diabetes is inflammation of the pancreas. How and why this inflammation sets

in we

yet do not know. As we also know from the physiology of trauma, inflammation

breeds fibrosis or scar tissue. One follows a chronic course of the other.

 

Fibrosis is also the culprit in the Peripheral Vascular Disease. In this

condition, fibrin plugs form in the micro circulation (tiny blood vessels)

forming blockages to full blood flow. Fibrin also forms the matrix for

arterial

plaque. Inflammation of trauma to the inner lining of an artery (intima),

causes the traumatized or weakened section to shore itself up with scar

tissue. On the spider web of scar tissue fat, calcium and heavy metals accrue

forming what we know as arterial plaque. Once the fibrosis blockages become

extensive enough, the patient presents the signs of PVD, which are cold

extremities, intermittent caludication (pain on walking from lack of oxygen

supply to

the tissues known as ischemia), non healing ulcerations of the skin and

eventual death of tissue creating gangrene leading to amputation.

The high blood sugar levels had during diabetes damages the body’s organs.

One of the first organs to be damaged are the nerves to the legs and then the

arms. Wherever the circulation is poorest the nerve damage follows and

radiating nerve pain is had (neuropathy). The damage begins with, you

guessed it, inflammation and progresses with, you guessed it again, fibrosis.

It

is this inflammation into fibrosis that seems to be a recurring theme in

diabetes.

For a moment lets do some education on orally administered systemic enzymes.

They have a 5 decade history of wide spread medical use in Germany, Central

Europe and Japan with over 150 million patients in Europe alone having

undergone enzyme therapy in the last 4 decades. There are over 200

peer-reviewed studies proving the absorption, therapeutic action and total lack

of

toxicity (no LD-50) of systemic enzymes. Their primary action is

anti-inflammatory, (though not through a COX 1 or Cox 2 action. The enzymes

instead “eatâ€

pro inflammatory cytokines). The enzymes also have a proven lysing action on

all types of fibrosis and scar tissue leaving normal or endogenous tissue

entirely intact and un-bothered. This is due to the body “taggingâ€

excesses of

fibrin as exogenous proteins. (The subject of protein tagging and its

discoverer won the Nobel Prize in biology in the late '90's). Entering the key

words: systemic enzyme, serrapeptase, nattokinase, bromelain, pancreatin,

papain,

trypsin, chymo trypsin into the search engine at Pub Med will bring up some

of the current research on systemic enzymes and their applications. A search

in the “medical fields†section of www.mucos.cz will show abstracts of the

extensive older research done with the first systemic enzyme blends of the

50's and 60's. It has to be said that there is nothing, no drug or substance,

in either the allopathic medical world or in the natural health world that

can remove scar tissue but highly fibrinolytic systemic enzymes.

Current thinking on diabetes is that the body’s immune system attacks the

pancreas creating inflammation. This may be so. Further, the current

thinking is that the inflammation brings about the destruction of the Islets of

Langerhans and its Beta Cells, the places where insulin is made. This may not

be

so. If the studies that are currently being planned and executed further

demonstrate what we are seeing clinically with Type 1 patients on systemic

enzymes, then this point will have to be re-thought. Clinically most of the

Type

1 patients have a significantly lower need for insulin while some no longer

need the insulin at all. This would suggest that the Beta Cells and the

Islets are not destroyed. I conjecture that they are merely clogged by the

fibrosis created by the inflammation. Once the causative inflammation is

reduced and once the fibrinolytic action of the enzymes has eaten away the

fibrosis

and reopened the channels, then what ever production the Islets can make can

actually get into the system.

I believe that the global (body wide) non-toxic, anti-inflammatory effects

of highly fibrinolytic systemic enzymes and the scar tissue eating effects of

the same enzymes are the reasons we are seeing the decrease in pancreatic

inflammation, decrease in diabetic neuropathy, in it's associated Peripheral

Vascular Disease, and the decrease in insulin dependence we are seeing

clinically in Type 1 patients. Let's see if the research further verifies the

observed findings and gives us more insight into the pathways of action.

Resources:

Vitalzym from World Nutrition 1-800-548-2710 or visit their website at:

_www.worldnutrition.info_ (http://www.worldnutrition.info/)

Specialty Enzyme, maker of pharmaceutical grade enzymes and systemic enzyme

blends for industry, pharmaceutical and supplements: 1-909-613-1660 or visit

their website at: _www.specialtyenzymes.com_

(http://www.specialtyenzymes.com/)