GENETICALLY MODIFIED FOOD - GRAVE CAUSE FOR MANKIND.

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GENETICALLY MODIFIED FOOD - GRAVE CAUSE FOR MANKIND.

Oct 02, 2004 12:21 PDT

 

SOMA HEALTH ASSOCIATION OF AUSTRALIA LIMITED

GRAVE NEW WORLD :

GENETICALLY MODIFIED FOOD - GRAVE CAUSE FOR MANKIND.

 

http://www.laleva.cc/food/gmo_soma.html

 

I am very concerned about the safety of genetically modified (GM)

foods and strongly believe far more research is required before

humans should regard them as safe, either for themselves or for

future generations.

 

This research should be completed before we are FORCED to live on GM

foods, there being no longer any choice.

 

GM foods have already been marketed for public consumption without

proper research or discussion into their long term effects.

 

The food we eat is broken down into Ôfood fractionsÕ which are

absorbed and used by the body. GM foods may possibly contain toxic

fractions which could have a serious effect not only on the

immediate health of some individuals but also on the health of

future generations.

 

It is entirely possible that these toxic fractions could affect us

in many ways, for example:

 

Ova and sperm may be affected;

 

Chromosomes, or telomeres, could be altered. Telomeres are on the

ends of chromosomes and are full-length at birth, shortening

throughout life;

 

 

Some GM foods could act as carcinogens and exacerbate or cause

cancer;

They may act as mutagens or teratogens, causing mutations or

congenital abnormalities;

 

They could cause autoimmune diseases such as collagen/connective

tissue disorder like Systemic Lupus Erythematosus (SLE), Rheumatoid

Arthritis/Scleroderms, or Arthritis, etc. ;

 

Neurotoxins could cause or flare-up Multiple Sclerosis (MS) or Motor

Neurone Disease (ALS) later in life;

 

Is it possible that they could act like prions, causing illnesses

such as Mad Cow Disease (BSE/CJD) to appear years later ?

 

The frightening prospect is not that there will be an immediate

epidemic or illnesses but that we may have caused an irreversible

reversal of human life in, say, 50 yearsÕ time, when teenagers start

to die of coronary artery disease, diabetes becomes rampant, and old

age arrives before the age of 50.

 

Blighted ova or sperm, damage done in utero, can

possibly cause innumerable life-threatening conditions.

 

We already see the effects of autoimmune diseases, many of which

should be renamed " toxic lectinopathy " (or poisonous allergies /

sensitivities/ reactions to food fractions and chemicals).

 

These are not always recognized as such, even today. If, or when,

we have damaged ourselves with a totally genetically modified food

chain, there will be no time for recognition or reversal. It will

already be too late.

 

The damage will have been done.

 

Our greatest asset < our children < will be born with short

telomeres and short futures. We may be introducing a threat with GM

crops and foods greater than plagues, wars, volcanoes, earthquakes,

global warming, ozone depletion or a stray asteroid.

 

Remember what happened to Pottenger's Cats?

 

Three generations of eating

junk and they went " down the tube " .

 

(Originally published SOMA NEWSLETTER October 2000, Vol.22, No.2.

Reprint by request.)

 

 

 

SOMA HEALTH ASSOCIATION OF AUSTRALIA LIMITED

UNDER-INVESTIGATION OF SYMPTOMS IN CHRONIC FATIGUE SYNDROME (CFS)

AND MULTIPLE CHEMICAL SENSITIVITY SYNDROME (MCSS).

 

 

Since 1980 I have treated over two thousand patients for CHRONIC

FATIGUE SYNDROME and well over a hundred for MULTIPLE CHEMICAL

SENSITIVITY. All these patients, when thoroughly investigated,

showed food and chemical

sensitivities / intolerances on the CYTOTOXIC TEST, especially to

cow's milk which can cause a severe malabsorption state for

vitamins, minerals, amino acids,

etc.

These patients tend to have severe nutrient deficiencies due to

malabsorption, and are usually low in Vits. B1, B2, B6, C, A, Folic

Acid

and, less often, B 12.

 

They often have anaemia due to the above

deficiencies but may have sideropaenia < low iron without anaemia.

 

Many have low serum zinc, and hair analysis shows low calcium,

magnesium, manganese, molybdenum, iron, chromium, zinc, selenium and

cobalt, raised copper and aluminum, and unsatisfactory lead, mercury

and nickel levels.

 

(The Cytotoxic Test is accepted by the LANCET, Letter, January 24,

1987.)

 

The glare/photophobia so often observed in CFS patients indicates

low iron, low Retinol A and low Zinc < hence white dots in nails

often, despite a normal haemoglobin/film, and thus not anaemia.

 

 

Many patients also have raised IgE and a host of inhalant allergies

to pollens (grass, weeds, trees < pollinosis), moulds, mites, etc.,

as well as food allergies, and are sensitive to fumes, chemicals,

perfumes, petrol, etc.

 

Most CFS patients have missed coeliac disease (nine out of ten in a

row, diagnosed as CFS by a leading Sydney hospital < POW) and missed

because most psychiatrists do not routinely have measurements done

of Endomysial IgA, Gluten IgA and IgG or alpha-gliadin IgA and IgG,

reticulin antibodies and IgM to see if these are raised, nor do they

look for low

C3, C4 and raised immune complexes as seen in coeliac disease, IgM

can be raised-to-low also in coeliac disease.

 

An extremely high percentage of CFS/MCSS patients are MISSED

COELIACS

and about 5% are MISSED SLE <which is not usually even considered.

All CFS patients should be tested for ANF and, if positive, then do

dsDNA; and, if not raised (diagnosing SLE), then ENA screen, C3, C4

complements, immune complexes, anti-lymphocyte antibodies,

immunoglobulins (IgA, IgM, IgG) and if any of the above are abnormal

(despite a negative ENA screen) then a skin biopsy on unexposed skin

with immunofluorescent technique to confirm/diagnose SLE.

 

Many patients with CFS show white dots in their nails which is

associated with low B6, zinc and pyroluria with kryptopyrroles in

the urine, and if these levels are high there is high risk for acute

intermittent poryphyria, especially if reacting to drugs ,

chemicals,

fumes, perfumes and chlorpyrifos/pesticides and herbicides which can

cause a flare-up of porphyria, as also can barbiturates,

sulphnamides, neuroleptics, etc.

 

Most CFS/MCSS patients have severe autoimmune disease (as seen with

SLE,

coeliac disease, and show gastritis, thyroidiotis, cholangitis,

vasculitis, autoimmune neuritis,etc.) and need a

diet design to reverse SLE.

 

These patients, in my experience, have

severe cow's milk alpha-casein, +/- alpha-lactalbumin, +/-

beta-lactalbumin, sensitivity/intolerance when antibodies to these

peptides of cow's milk are measured - as well over a thousand

patients since 1980. (cont.)

 

 

Under-investigation of symptoms in CFS and MCSS: (cont.)

 

When intolerant/hypersensitive to cow's milk albumin-globulin, then

these cross-react with gmo egg and beef albumin and globulin, and

also need to be avoided.

 

In addition, because of the suppressed/compromised immune

system and often low cortisol DHEA, MCSS patients (often being

coeliacs)

are at risk for opportunist pathogenes such as candida albicans,

mycoplasma, rickettsia, helicobacter pylori, chlamydia pneumoniae

and

campylobacter jejuni aggravating/complicating their treatment. These

pathogens need to be identified and killed off.

 

High levels of aluminium, copper and , less often, lead and mercury

are

seen in CFS/MCSS patients and show especially on hair analysis.

 

They

also have low serum cortisol +/- low serum DHEAS, i.e. adrenal

exhaustion, and many are hypothyroid. Most have a marked tendency to

hypoglycaemia and need to eat plant protein two-hourly in order not

to become hypyglocaemic. Severe amino acid deficiencies are common

in my

experience (also refer Newcastle University research) and may have

low Vit B3 in addition to nutrient deficiencies mentioned above.

 

High levels of pesticides/herbicides are common in CFS/MCSS patients

(work of Dr

Mark Donohoe and Tim Roberts et al of Newcastle University). They

also have abnormal metabolites in the urine (and Kryptopyrolles /

pyroluria ).

 

 

SPECIFIC INSTANCES OF UNDER-INVESTIGATION:

 

(a) I have seen moribund patients with severe pellagra who were

called " hysterical " and not allowed to have Vit. B3.

 

(Pellagra is a Vit. B3 deficiency). One patient at the Cummins Unit

of Royal North Shore

Hospital crawled out of hospital and caught a taxi to Manly Hospital

where she made a rapid recovery with B3 treatment, etc.

 

(b) Another with severe pallor/weakness at Camperdown Children's

Hospital with severe CFS picture was called hysterical until I

showed

the treating psychiatrist she had antibodies to alpha-casein,

alpha-lactalbumin, proving she had severe cow's milk

sensitivity/intolerance.

 

The ice-cream and milk products they were giving her out of kindness

were making her seriously ill and moribund.

Off all diary products she rapidly recovered.

 

© Another patient with tardive dyskinesia due to Serenace, and

wrongly diagnosed as schizophrenic instead of toxic

encephalopathy/chloropyrifos poisoning, etc., did well for many

months on extra nutrients and food allergy-free diet, etc., and no

neuroleptics at all, or minimal

Melleril, until she came into contact with more spraying with

pesticides/herbicides.

 

She was hospitalized and given high amounts of neuroleptics, making

her very ill. I saw her regularly while she was on

high dosage IMI neuroleptic weekly for her Acute Brain Syndrome

which

had severe side-effects, agitation, depression.

 

This was to be her

treatment indefinitely, all her complaints about side-effects being

ignored. Each day off the neuroleptics she rapidly improved.

 

 

Fortunately, she again did well on extra nutrients, special diet and

minimal Melleril. The evidence supporting increased investigation

into

causes of symptoms in CFS and MCSS is overwhelming.

 

JMS

 

(Originally published SOMA NEWSLETTER, October 1999, Vol.21, No.2.

Reprint by request.)

_________________

 

JoAnn Guest

mrsjo-

www.geocities.com/mrsjoguest/