AIDS: Court Case Exposes Scientific Contradictions

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AIDS: Court Case Exposes Scientific Contradictions

 

http://www.newmediaexplorer.org/sepp/2007/02/07/aids_court_case_exposes_scientif\

ic_contradictions.htm

 

 

A legal case in South Africa has brought the

scientific contradictions of the pharmaceutical

AIDS paradigm into the spotlight. The defendant,

Andre Chad Parenzee is appealing against his

conviction on three counts of endangering life.

The case challenges the validity of the theory

that a virus called HIV does indeed cause a large

number of disrelated illnesses that,

individually, have been known before AIDS became

a household word.

 

Helen Lobato, in an article that first appeared

on Melbourne Indymedia, put it this way:

" An appeal case has HIV-AIDS specialists on

tender hooks awaiting the outcome which they say

could set a dangerous precedent for public health

campaigns and the criminal law. He is in custody

awaiting sentencing and could face up to 15 years

in prison. He was found guilty in February 2006

of endangering the lives of three women because

he had unprotected sex with them without telling

them he had HIV. "

 

The court has heard witnesses for the defense for

a week and is now listening to what the

prosecution's experts have to say. What is being

discussed amid heated arguments against " aids

deniers " are the scientific merits of the

isolation of the virus that is said to cause

AIDS, and thus of the tests that determine

whether a person is infected or not. Next step:

Does the virus actually cause all the illnesses

that are ascribed to it or is it an innocent

passenger that all of us carry while other 'risk

factors', such as intravenous drug use, do the

major damage.

 

Beldeu Singh's recent article GALLO vs MONTAGNIER

gives us a good look into the inconsistencies

that exist, even in the words of the proponents

of the " HIV-causes-AIDS " theory. Those are

serious doubts which plague the debate the South

African judge will have to resolve. Typically, we

don't find the " HIV-Aids experts " willing to

discuss the merits of their views when

challenged, but in this case, they have no

choice. Here the article by Beldeu Singh:

 

GALLO vs MONTAGNIER

Beldeu Singh

 

The two persons who claimed to have discovered a

new virus, they called it the HIV, are Robert

Gallo and Luc Montagnier. Dr. Gallo claimed in

his seminal paper that HIV is the probable cause

of AIDS. It was supposed to be a virulent

pathogen that targeted the cells of the immune

system, including T4 cells that eventually

weakened or destroyed the immune system and

opened it for opportunistic infections.

 

A close study of AIDS shows that there are people

who test seropositive and show no symptoms of the

disease and there are those who have AIDS but do

not test positive. Of course, all the test kits

carry a disclaimer, saying that you must do

another test for an affirmative diagnosis. In

other words, these tests are not specific as one

would expect them to be in diagnosing viral

infections, but that is quite another matter.

 

The establishment does not like anyone

questioning the official " HIV causes AIDS " dogma.

One wonders why, for that is not scientific

temper. Montagnier has actually stated that " it

would be a tribute to their (dissidents like us)

courage and honour to abandon the HIV cause of

AIDS in the face of overwhelming evidence (Virus,

New York, WW Norton & Company Inc, 1999). Ok

fine. Lets abandon the questioning of that dogma

but lets look at their science. At least that

should be allowed. It is still science, isn't it?

 

In that book, Montagnier states that in AIDS

patients, oxidative stress is massive and it

occurs at an early stage. The cause of the

oxidative stress is HIV!. In an interview in

1995, Montagnier said that for the progression of

AIDS, oxidative stress is a key factor. The

contradiction here is that we should expect

massive oxidative stress only at a later stage,

after a large number of HIV replications have

occurred, certainly not in the initial stages.

 

Montagnier gave a talk to the European Union

Parliament on 8th December 2004. He told the EU

Parliament that " the clinical phase of

opportunistic infections and cancers which result

in death of the patient is principally due to a

decline in T4 cells and the decline in T4 cells

is due to their propensity to die from apoptosis. "

 

Apoptosis or programmed cell death (PCD) is a

highly regulated process. In contrast to

necrosis, which is a form of cell death that

results from acute cellular injury, apoptosis is

carried out in an orderly process that generally

confers advantages during an organism's life

cycle, as for example in the process of

differentiation in the embryo. The way the

apoptotic process is executed facilitates the

safe disposal of dead cells and fragments.

Apoptosis can occur if a cell is damaged beyond

repair or if it is infected with a virus. If a

cell's capability of apoptosis is impaired by

mutation or if the initiation of apoptosis is

blocked by a virus, that cell can continue

dividing without restrictions, developing into

cancer. So, HIV does not block apoptosis.

 

Chronic infections by viruses have been strongly

linked to the development of disease conditions

including carcinomas. Viruses are also known to

induce oxidative stress as seen from elevated

levels of reactive oxygen species (ROS). Usually

when a clear link with any particular viral gene

product is elusive, the elevated levels of ROS

are thought to cause death of cells that harbour

these viruses.

 

The genetic material of HIV is RNA. Hence, it is called a retrovirus.

 

" HIV depends on the cells it infects to make new

copies of itself. One of the main targets of HIV

is a white blood cell called a CD4. HIV infects a

CD4 cell by attaching itself to the surface of

the cell and injecting its genetic material (RNA)

into the host cell. Since RNA can not direct the

reproduction of new viruses, HIV must first turn

its RNA into DNA. HIV turns its viral RNA into a

viral DNA copy through the use of an enzyme

called reverse transcriptase. This process is

know as " reverse transcription " . Once the

conversion process is done, the viral DNA may

become part of the host cell's DNA. This process

is known as " integration " . At this point

infection is complete. Once the viral DNA copy is

successfully made part of the infected cell's

DNA, the infected cell goes on to produce new HIV

every day. As many as 10,000 new HIV copies can

be produced before the infected CD4 cell is

destroyed. The new copies of HIV then go on to

infect other cells. In this way, HIV spreads

throughout the body. This allows for billions of

new viruses to be produced every day. Forced to

manufacture new HIV (viral replicas), the host

cell neglects its own life processes. Gradually,

like a machine wearing out, the CD4 cell starts

to fail and dies " (Ch. 2: HIV: Biology and Safe

Sex; UCSD, Official Web Page of the Univ. of

California, San Diego, undated).

 

That mechanism was the classic explanation of how HIV causes AIDS.

In the Journal Of Infectious Diseases (1997;

176;655-64), Montagnier wrote that " in AIDS

pathogenesis oxidative stress is proposed as a

metabolic alteration that favours disease

progression by inducing both viral replication

and apoptotic (cell) death. " He claims to have

" evidence that oxidative stress indeed induces,

while antioxidants inhibit, HIV replication and

apoptosis " and suggests the use of these

molecules as an antiretroviral therapy to reduce

cell death in AIDs patients. Firstly, note that

here we have Montagnier suggesting the use of

antioxidants in AIDS patients to reduce apoptosis

of T4 cells and as an antiretroviral therapy.

Secondly, the " oxidative stress induces HIV

replication " assertion by Montagnier creates

another paradox.

 

Earlier on it was claimed that HIV attacked T4

cells. Then, it is claimed that HIV causes

oxidative stress and that oxidative stress

induces viral replication. In medical science

pathogens are killed by oxidative stress that

causes oxidative injury but it is just the other

way around with the HIV. The key factor that is

lethal at the molecular and submolecular level is

now claimed to induce HIV replication, making HIV

the first pathogen whose replication is free

radical induced! Oxidative stress in cells

impairs protein synthesis. That ought to slow

down and prevent the replication of the virus.

Oxidative stress also damages enzymes that are

required in converting HIV RNA into HIV DNA and

other enzymes involved in the processes that

result in viral replication.

 

Montagnier fails to explain why the " massive

oxidative stress that occurs at the early stage "

does not kill the T4 cell. Originally, it was

claimed that the HIV attacked T4 cells and how

such an oxidative stress could be managed by the

virus in such a way that it does not kill the

" infected " T4 cells but it remains under its

control for the purpose of inducing its

replication. Dr. William Haseltine at the Harvard

University School of Medicine, says T4 cells are

primed to die once they are infected. The reason,

he said, is that the virus ends up destroying the

cell membrane of any T4 cell that it infects.

 

Next, there is a need to explain how such a small

virus, with so little genetic information, can

utilize oxidatively damaged molecules in T4 cells

for its replication when the cells themselves do

not have such machinery. An elevated level of ROS

causes oxidative stress and it is toxic for

living cells by reason of oxidative injury to

membranes and oxidative damage of cellular

structures and free radical reactions with

proteins, lipids, nucleic acids etc. Reactive

oxygen and nitrogen intermediates are important

antimicrobial defense mechanisms of macrophages

and other phagocytic cells. What protects the HIV

protein and its genetic material from oxidative

injury? Yet, the free-radicals-generating drugs,

like AZT are supposed to be anti-HIV.

 

All the drugs used in AIDS patients are toxic and

induce free radical damage. AZT is probably the

most toxic of them. They generate oxidative

stress as well as create oxidative damage to a

large number of molecules and going by what

Montagnier wrote in the Journal Of Infectious

Diseases, that oxidative stress induces HIV

replication, it would mean that these drugs are

inducing HIV replication in " HIV-infected "

patients. That is simply astounding indeed, but

medical authorities take no cognizance of it for

therapy.

 

The observation that " virtually all T4 cells

eventually die in people with AIDS " may in fact

be due to the susceptibility of T4 cells to

oxidative stress rather than the fact that they

are infected with a virus.

 

Oxidative stress is the key to the progression of

AIDS, for there are people with AIDS who are not

HIV positive and many respond to antioxidant

interventions while AZT, a highly toxic chemical

that generates free radicals in the body that can

destroy cell membranes as well as a large number

of molecules, can produce the very symptoms of

AIDS. AZT, benzene and many compounds made from

benzene can deplete glutathione which is very

essential to the functioning and which determines

the life span of T4 cells.

In a 1998 study, researchers from Canada reported

that supplementation of vitamin E and C reduces

oxidative stress in " HIV " positives. " This study

is the first randomised controlled trial to

demonstrate that, in an HIV-positive population,

daily supplementation of 800 IU vitamin E and

1000 mg vitamin C significantly decreases

oxidative stress and produces a trend towards a

reduction in HIV viral load suggesting that there

may be some clinical benefit worthy of larger

clinical trialsŠconsideration of the potential

for this antioxidant therapy remains important

for the developing world " (Allard et al. 1998,

Effects of vitamin E and C supplementation

onoxidative stress and viral load in HIV infected

subjects. AIDS, 12:1653). Antioxidant activity

clearly reversed whatever was triggered by

oxidative stress.

 

On one hand we understand that viral toxins can

create oxidative stress and that appears to be

the cause of secondary diseases eg carcinomas or

leukemia, especially in cases of chronic

infections but the Montagnier-HIV (M-HIV) is

actually induced by oxidative stress and its

replication is inhibited by antioxidants. This

sounds more like a latency disease wherein a

certain length of a viral genome forms a

conjugate with a protein molecule produced by the

T4 cell or cells of the immune system and can

enter the cells without inhibition, only to be

reactivated later on by oxidative stress that

breaks the conjugate. Oxidative stress could

break the viral genome-protein conjugate,

releasing the viral genome to produce a latency

disease. Naturally, antioxidants would prevent

such breakage and help prevent the latency

disease (see: THE EPSTEIN-BARR VIRUS IN AIDS). If

so, HIV cannot be a new virus, but one that is

already a harmless passenger in many people. And

if oxidative stress is a factor in the

reactivation, it is nothing more than a problem

of nutrition and antioxidant intake as a measure

of curbing the AIDS problem and by direct

implication, toxic medication and drugs and

chemical stressors work towards initiating the

disease and aid its progression. It appears,

quite clearly that Gallo and Montagnier proved

this point.

 

Interestingly, in 1983 Luc Montagnier and in 1984

Robert Gallo stimulated cell cultures from

tissues of AIDS patients with numerous oxidising

agents (Sekkat et al, 1988, Oxidative phenomena

are implicated in human T-cell stimulation.

Immunology, 63:431-437) and mitogenic chemical

agents. These are toxic chemicals that generate

free radicals that cause oxidative stress. By

1986 Montagnier and Gallo acknowledged that the

" HIV " phenomenon cannot be detected unless the

cells are " stimulated " .

 

Montagnier and his associate David Klatzmann were

the first to draw attention to the fact that LAV

infection of T4 cells in vitro does not lead to

HIV expression unless the cells are stimulated.

" Infection of resting T4 cells does not lead to

viral replication or to expression of viral

antigens on the cell surface, while stimulation

by lectins or antigens of the same cells results

in production of viral particles, antigenic

expression and the cytopathic effect " (Klatzmann

and Montagnier, 1986, Approaches to AIDS therapy.

Nature 319:10-1).

 

Gallo also expressed the view that without

" activation " the T4 cells do not express the

virus (Zagury et al (1986) Long term cultures of

HTLV-III-infected T cells: A model of

cytopathology of T-cell depletion in AIDS.

Science, 231:850-853). In other words, here is a

virus that infects T4 cells but cannot replicate

in them. So, there is no HIV-AIDS without

oxidative stress and this turns out to be starkly

at variance from the classic explaination on how

HIV infection kills T4 cells. What eluded those

scientists was the fact that oxidative stress, in

some manner, had triggered the reactivation of a

viral genome that is already present in many of

us. It is certainly not a case of a " stimulated "

cell that causes the virus to replicate.

 

The point to note is that T4 cell exposure to

oxidizing agents produces both cytopathic effect

and results in the production of viral particles.

Glutathione deficiency in CD4 T cells from AIDS

patients is associated with markedly decreased

survival time and glutathione deficiency is a key

determinant of survival rates in AIDS patients.

Several studies, quite expectedly show positive

response by selenium supplementation.

T4 cell susceptibility to oxidative stress

arising from toxic chemicals that deplete

glutathione is the key in understanding AIDS and

the progression of the disease. Quite possibly,

mature T4 cells and mature macrophages may not be

able to produce glutathione or selenoproteins

from selenium. This may also explain why

narcotics and recreational drugs like ecstasy and

most of the drugs used as medications reduce T4

cell counts and white blood cell counts.

 

The role of oxidative stress as the causative

mechanism of AIDS was already proposed by the

Perth Group as long ago as 1983 and E

Papadopulos-E has argued in favour of oxidation

as being a critical factor in the pathogenesis of

AIDS (Res. Immunol. 1992, 143, 145-148 .

Oxidative Stress, HIV and AIDS. E.

Papadopulos-Eleopulos (1) V.F. Turner (2) and

J.M. Papadimitriou (3). (1) Department of Medical

Physics, (2) Emergency Department and (3)

Department of Pathology, (University of Western

Australia), Royal Perth Hospital, Wellington St.,

Perth 6001 (Western Australia)) and elaborated in

other articles by other researchers. Oxidative

stress is a factor in the progression of every

disease and disease condition. A wide range of

diseases are associated with altered levels of

antioxidant enzymes, especially glutathione and

coenzyme Q10. Selenium deficiency, low levels of

antioxidant enzymes in the blood and low

antioxidant activity in the body are independent

predictors of cell death, mortality and

progression of diseases.

 

Selenium deficiency was found to produce an

elevated risk in AIDS patients (High Risk of

HIV-Related Mortality Is Associated With Selenium

Deficiency. Marianna et al, 1997, JOURNAL OF

ACQUIRED IMMUNE DEFICIENCY SYNDROMES AND HUMAN

RETROVIROLOGY, 15:370-374). And there are many

studies that show that " nutritional problems have

been a part of the clinical aspects of AIDS from

its earliest recognition as a new disease. In

fact, in many AIDS patients, death seems to be

determined more by the individual's nutritional

status than by any particular opportunistic

infection. This is, when wasting of lean body

mass approaches 55% of what is normal for age,

sex, and height, death is imminent regardless of

the forces resulting is such profound

malnutrition. Furthermore, the severity of the

clinical manifestations of AIDS is proportional

to the degree of the nutritional deficiencies "

(cf: John Kirkham and James Whitehead, Some

immune stimulating treatments and the scientific

bases for them, undated). Natural vitamin C can

recharge glutathione directly or indirectly

through alpha-lipoic acid while natural vitamin E

can recharge it through alpha lipoic acid and

that explains why supplementation helps reduce

viral loads or produces clinical benefits.

Montagnier had been obviously ignored by the

pharmaceutical industry and the medical

authorities and the EU governments as far as his

assertion on the use of antioxidants to prevent

replication of HIV is concerned. What comes out

loud and clear is that while Gallo and Montagnier

propounded the HIV-causes-AIDS hypothesis, they

told people that after HIV infects cells in the

immune system, it replicates in them and it leads

to the ultimate devastation of the immune system,

while on the other the standard prescription is

primarily large doses of immunotoxic and

immunosuppressive " medicine " !

Montagnier stated that for the progression of

AIDS, oxidative stress is a key factor. Drugs

that create oxidative stress, in that light, are

in fact aiding the progression of AIDS in

patients. Why is that assertion by Montagnier

largely ignored by medical science? It would mean

throwing out all of the toxic medications.

 

But there is a bigger contradiction between these

discoverers of the HIV. In 1984 Dr. Gallo

registered a patent which shows that he has been

mass producing his HIV in an immortal line of T4

cells, which means the T4 cells are alive and

intact after 21 years. This Gallo-HIV (G-HIV)

does not produce oxidative stress in T4 cells and

does not cause apoptosis either. And that means

we have a very strange situation that can be

rolled up into a question as follows:

 

" If HIV is claimed to cause AIDS by killing

T-cells, how is the mass production of HIV in

immortal T-cell lines possible, as shown in the

1984 patent for a source of HIV proteins for

" AIDS tests " by Gallo/NIH, Weiss/Burroughs

Wellcome (UK), and Montagnier/Pasteur?. "

 

If HIV actually attacks these infected cell

lines, how come they are still producing HIV 21

years later! The proponents of the Gallo-HIV

(G-HIV) theory have exactly the opposite view -

the G-HIV does not kill cells, unlike the HIV

that was made out to be the cause of AIDS. So,

even in the T4 cells that are " stimulated " by

oxidizing agents in his culture in which they are

growing, the HIV does not attack and kill the T4

cells. Which ever way you look at it, this boils

down to the fact that their experiments prove

that the so called HIV does not kill T4 cells and

does not deplete them by replicating in them.

Something else is independently at work, either

as direct damage from oxidative stress or as an

oxidative stress that triggers a latency disease.

 

Gallo and Montagnier need to reconcile the

difference between G-HIV and M-HIV, but that does

not seem to bother those who hold the HIV dogma

sacred, nor has it stirred a controversy on how

AIDS patients ought to be treated - drug or

antioxidant therapy. The bottom line is not the

science and the ethics of science but the money

from drugs. Science is secondary to the treatment

already decided. There is more money in drugs

than antioxidants, but science will kill the AIDS

industry. Much of the controversy rests in the

fact that HIV has never been isolated and that

there is no antibody test that is specific to

such an isolated virus. There also is no

explaination as to why the antibodies that are

used to test for HIV are not able to stop the

infection just like in all other viral

infections. These antibodies must work if they

are specific to the viral coat proteins etc.

 

The dissidents have challenged the

HIV-causes-AIDS dogma. They have said their

piece. They are correct in pointing out all the

inconsistencies, but I am not saying, listen to

the dissidents. I am saying, listen to the the

two persons who claim to have discovered the

virus and it was Dr. Gallo who said HIV is the

" probable cause of AIDS. " Listen to their

inherent contradictions.

 

See also:

<http://www.newmediaexplorer.org/sepp/2006/05/30/why_retroviruses_appear_in_aids\

_cancer_and_autoimmune_diseases.htm>Why

Retroviruses Appear in AIDS, Cancer and

Autoimmune Diseases

 

 

--

 

 

The individual is supreme and finds its way through intuition.

 

Sepp Hasslberger

 

 

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