Article: World Aids Day - Time To Rethink Prevention, Treatment?

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revention_treatment.htm

 

 

 

 

 

November 30, 2006

 

 

 

World Aids Day - Time To Rethink Prevention, Treatment?

 

 

1 December is World Aids Day - time to think about Aids and the

virus that is thought by orthodox medicine to be selectively

devastating the immune system of ... mainly gays, intravenous drug

users and the poor both in the West and in developing countries. Aids

campaigners say we need to " raise awareness " about what they describe

as a sexually transmitted disease. They say we need to invest more

money to bring drugs to those in the developing countries who can't

afford them.

 

Others are convinced that we should be helping to improve living

conditions - sanitation, clean drinking water and basic nutrition.

They add that treating the single diseases such as malaria and

dysentery and a host of other infections prevalent in poor Africa and

rural India would go a long way in eliminating " Aids " .

 

 

Aids Rethinkers or Aids Denialists?

 

Those suggesting to abandon the fixation on a virus never properly

isolated and on highly toxic medications that cause many of the

symptoms of " Aids " call themselves Aids rethinkers. A less

complimentary term, used by the pro-Aids campaigners to position the

independent thinkers is " Aids-denialists " . Which of the two camps has

truth on their side is hard to say for a casual observer. Greg

Hambrick of the Charleston City Paper in a recent article titled

Rethinking AIDS - Doubters abandon traditional HIV/AIDS theories and

treatment strikes a balance between the two sides and gives an

understandable introduction to their respective arguments.

 

Liam Scheff, an independent journalist who exposed scandalous

experiments with toxic Aids drugs in a New York home for orphans and

wards of the State, has been called an Aids denialist. His articles

Inside Incarnation and The Truth about Nevirapine are indictments

against human indifference and pharmaceutical profiteering at the cost

of lives and untold suffering.

 

Scheff's investigative work has triggered the ire of professional

defenders of Aids orthodoxy, of those who sustain the viral paradigm

and the chemical solution to the problem. In Correcting The AIDS Lies

- Who Is Nick Bennett? Liam challenges one of these self-styled

experts who pounce on what they perceive as the enemies of the

infective-Aids paradigm.

 

Is Scheff an Aids denialist because he investigated and published his

findings? Andrew Maniotis, Ph.D. a program director at the University

of Illinois Department of Pathology, Anatomy and Cell Biology does not

think so. In his paper The ABC's of AIDS denialism (it's a rather

largish PDF file) Maniotis points up a myriad of inconsistencies in

the official dogmatic stance which, summed up, could be described as

HIV = AIDS = Retroviral Drugs = Delayed Death. Maniotis takes

Montagnier, Gallo and a great number of other supporters of Aids

orthodoxy to task for what he says are obvious contradictions and

flawed arguments in the science of Aids. It's them who are the

denialists, argues Maniotis in a very tongue-in-cheek paper.

 

In a more succinct but not less convincing way, Malaysian researcher

Beldeu Singh sums up the facts of Aids-as-an-infection vs. Aids seen

as a condition of cellular stress. His article, Don't Question the

Gallo-HIV AIDS Dogma, is in the second part of this post. Beldeu has

also written a celebratory piece - showing how Gallo himself has

demonstrated that HIV does not attack T-cells and therefore cannot be

the cause of Aids:

 

CELEBRATING WORLD AIDS DAY - DECEMBER 1 - MOURNING DEATH BY AZT AND

CELEBRATING GALLO's PROOF THAT HIV DOES NOT CAUSE AIDS

 

 

One trait the Aids rethinkers cum " denialists " have in common is a

plea for real prevention and for standard medical treatment of the

specific, known diseases that set in when the immune defenses have

been down for a considerable period of time.

 

 

Real prevention

 

Officially, Aids prevention involves sexual abstinence, condoms, and

even - who would have thought it - circumcision. The use of a cocktail

of toxic drugs is also advocated, but there is no word of improving

the economic outlook in developing nations, of bettering the living

conditions and providing basic nutrition to increase the population's

natural resistance. No word of nutrients like selenium and the three

amino acids that are found to be low in most Aids patients.

Geo-epidemiologist Harold Foster has identified these specific

deficiencies as the likely culprit for increased susceptibility to

contracting immune deficiency.

 

 

But those things are considered inappropriate to even seriously

research. No wonder - they are not expected to bring great profits. As

a matter of fact, they would tend to ruin a perfectly good business.

 

But the ranks of rethinkers and " denialists " are steadily swelling.

Quietly, they are making progress, even without funding from Bill and

Melinda. Small scale trials and not-so-small-scale pilot programs to

supply needed nutrients such as the efforts of the Dr Rath Foundation

in South Africa are showing positive results, derisive hoots and cries

by the real denialists notwithstanding.

 

 

Don't Question the Gallo-HIV AIDS Dogma, says Beldeu Singh from

Malaysia, as he proceeds to tell us how this dogma is full of holes ...

 

.... and at the end of this post, you will also find a World Aids Day

Message from Dr Leo Rebello.

 

With all these inconsistencies coming to the surface, this may indeed

be a time for re-thinking Aids and our relation to health and disease.

 

- - -

 

DON'T QUESTION THE GALLO-HIV AIDS DOGMA

Beldeu Singh

 

According to the HIV theory of AIDS, the virus is sexually

transmitted and that should produce an HIV-AIDS explosion in the

heterosexual population within 25 years. But it did not happen.

 

This point is well put forth by Dr. Robert Root-Bernstein. Female

prostitutes often have 200-300 sexual partners per year and are

therefore assumed to have much higher rates of exposure to HIV and

AIDS than the vast majority of heterosexuals. Many AIDS researchers

assumed that female prostitutes would be the vectors (or means of

transmission) of HIV and AIDS to the heterosexual community based on

the fact that a single HIV-infected intravenous drug user or bisexual

man could infect one female prostitute, who in turn could infect

dozens or perhaps even hundreds of non-drug using heterosexual men.

These men could, in turn, infect their other sexual partners, and an

explosion of HIV and AIDS could occur among people without any obvious

risk for AIDS. Paradoxically, no heterosexual epidemic has occurred

and no evidence of female prostitutes transmitting HIV or AIDS into

the heterosexual community exists for any Western nation. Transmission

almost always seems to be drug related. In fact, sexual acquisition of

HIV and AIDS among female prostitutes themselves is almost unknown in

the absence of concomitant intravenous drug use. Cell-free viral

particles have never been found directly in semen. In `American

Journal of Epidemiology' (Vol. 146, No.4), Nancy S. Padian et al reported:

 

" We estimate that HIV infectivity for male-to-female

transmission is low, approximately 0.0009 per contact, and that

infectivity for female-to-male transmission is even lower. "

 

In New York City, for example, 40 to 50 percent of streetwalkers

(a very low caste of prostitute) who have used IV drugs over the past

decade are HIV seropositive. Among call girls in New York City (a

higher caste of prostitute), no seropositivity was found among those

who were drug free. These figures were constant between 1984 and 1989.

These statistics have significant implications on the causative factor

or factors of AIDS.

 

The biggest problem in the gallo-HIV theory which says that a

pathogenic virus attacks the T4 cells of the immune system is this:

 

" If HIV is claimed to cause AIDS by killing T-cells, how is it

possible that there is mass production of HIV in immortal T-cell lines

as shown in the patent in 1984 as source of HIV proteins for " AIDS

tests " by Gallo/NIH, Weiss/Burroughs Wellcome (UK), and

Montagnier/Pasteur? "

 

If the gallo-HIV actually attacks these infected cell lines, how

come they are still producing HIV 21 years later! Here the proponents

of the gallo-HIV theory have been saying excactly the opposite - that

HIV does not kill cells, just like all other retroviruses. But that

does not seem to bother those who hold the gallo-HIV dogma sacred, nor

has it stirred a controversy or outcry in the mainstream media!

 

HIV is not behaving like a typical sexually transmitted disease

 

There is only one possible conclusion: as Japanese physician Y.

Shiokawa has suggested, it is probable that drug use, multiple

concurrent diseases, malnutrition, and other immunosuppressive factors

are required to increase susceptibility. In fact, it better fits a

model based on oxidative stress in cases of malnutrition and selenium

deficiency and on oxidative damage and oxidative injury to cells of

the immune system and cells in organs targeted by recreational drugs

or immunotoxic medication.

 

The most difficult aspect of the HIV postulate is to have first

decided that their HIV virus is an aggressive pathogen - which they

claim targets the immune system itself, as HIV was said to infect the

key CD4+ T cells that regulate the immune response, modifying or

destroying their ability to function - and to reconcile this 'science'

with scientific data and evidence that some people " appear better able

than others to resist progression of HIV infection or developing

AIDS, " resulting in " long-term survivors " who can be divided into

three groups;

 

1. Long-term nonprogressors who maintain healthy or steady

levels of CD4+ T cells despite many years of infection,

 

2. those tested-HIV-positive individuals who lose a

significant proportion of CD4+ T cells but remain healthy, and

 

3. the people who remain uninfected despite repeated " exposure

to HIV " .

 

So, to save the HIV postulate for AIDS they also claim that, once

the virus infects CD4+ T cells, the virus' genetic material is

permanently integrated into the cell's chromosomes, establishing

permanent latency within infected cells. After infection, the HIV

incorporates its genetic material into the host cell DNA. If a cell

reproduces itself, each new cell also contains the integrated HIV

genes. The virus can hide its genetic material for prolonged periods

until the cell is activated and makes new viruses. So, its not an

aggressive pathogen.

 

They also claim that other cells act as HIV reservoirs, harboring

intact viruses that may remain undetected by the immune system while

it " targets " the cells of the immune system.

 

There is no explaination on how an infected cell remains normal

and remains undetected as an abnormal cell by NK cells or activated

macrophages after the HIV incorporates its genetic material into the

chromosomes of the cell. Such a virus, with such a capability, having

a sophisticated enzyme system to incorporate its genetic material into

the cells' chromosomes and activate it later on into replicating

itself cannot be so small and elusive that it avoids isolation and

replication by other virologists. It must have a large amount of

genetic material to do all of those things but retroviruses, such as

the fictitious gallo-HIV, are " gifted " with too little genetic material.

 

The causal relationship between HIV and any disease is not settled

 

" HIV is an ordinary retrovirus. There is nothing about this virus

that is unique. Everything that is discovered about HIV has an

analogue in other retroviruses that don't cause AIDS. HIV only

contains a very small piece of genetic information. There is no way it

can do all these elaborate things they say it does, " according to Dr.

Harvey Bialy (Molecular Biologist and former editor of Bio/Technology

and Nature Biotechnology as reported in Spin June 1992). Dr. Gordon

Stewart (Emeritus Professor of Public Health, University of Glasgow)

agrees that " AIDS is a behavioral disease. It is multifactorial,

brought on by several simultaneous strains on the immune system -

drugs, pharmaceutical and recreational, sexually transmitted diseases,

multiple viral infections, " (Spin June 1992). Dr. Alfred Hässig,

(1921-1999) was professor emeritus in immunology at the University of

Bern, Director of the Swiss Red Cross Transfusion Service, and

President of the Board of Trustees of the International Society of

Blood Transfusion. His Swiss research group doesn't believe that HIV

causes AIDS either.

 

So, how did the world get duped into believing that HIV causes

AIDS? Very simply but on a very serious note, one of the reasons is

aptly stated by Dr. Richard Strohman (Emeritus Professor of Cell

Biology at the University of California at Berkeley) when he said, " In

the old days it was required that a scientist address the

possibilities of proving his hypothesis wrong as well as right. Now

there's none of that in the standard HIV-AIDS program with all its

billions of dollars, " (Penthouse April 1994).

 

Work by scientists, shoddy clinical trials and highly improper

statistics concerning HIV and AIDS have been passed off as science.

Unsuspectingly, non-governmental organizations, medical practitioners

and the top members of the scientific establishment have carelessly

supported or joined the media in spreading misinformation about the

nature of AIDS and HIV as the pathogenic factor in AIDS although there

are hundreds of thousands of healthy people who tested HIV-positive

and live as long as HIV-negative people and there are many people with

non-HIV AIDS who respond to proper nutrition.

 

And the `establishment' has created bigger problems. There is no

Universal Gold Standard `HIV' test to prove `HIV' positivity. The

`HIV' antibody test does not detect a `virus' but an assortment of

proteins that are non-specific to the hypothetical `HIV'. The proteins

that are used in the `HIV' test are merely the biological outcome of

stressed white blood cells used in the lab. There can be no Gold

Standard `HIV' test because there is no Gold Standard `HIV' isolate.

Methods of classical virology require that all evidence of `HIV'

positivity must be confirmed by pure culturing of a patient's

lymphocytes and detection of whole, cell-free viral particles; so far

this has never been achieved. The proteins that are used in the `HIV'

test are merely the biological outcome of stressed white blood cells

used in the lab. In `Bio/Technology', June 1993, `Aids' analyst, Dr

Eleni Papadopulos exposed the non-specificity and unreliability of the

`HIV' `antibody test'.

 

`HIV' is an artefact of cell-culture invented by Dr Robert Gallo.

The phenomena collectively known as `HIV' are non-specific: reverse

transcriptase is non-specific; PCR is non-specific; Viral Load is

non-specific. Each property relating to `HIV' can be shown to pertain

to the cells used in co-cultivation experiments. No particle of `HIV'

has ever been obtained pure, free of contaminants; nor has a complete

piece of `HIV' RNA (or the transcribed DNA) ever been proved to exist.

Moreover, Dr David Ho admits that 99.8 per cent of putative `HIV

particles' are non-infectious; the remaining 0.2 per cent of `viral

particles', being defective, are not capable of replication. As a

transmittable entity, `HIV' could not survive in nature. This

indicates that what we are calling `HIV' is a misinterpreted,

non-transmissible, endogenous epiphenomenon that should never have

been classed as a virus.

 

Dr John Papadimitriou states that the proper controls have never

been done: " They have not proven that they actually have detected a

unique, exogenous retrovirus. The critical data to support that idea

have not been presented. You have to be absolutely certain that what

you have detected is unique and exogenous, and a single molecular

species…('Aids: The failure of contemporary science', Neville

Hodgkinson, Fourth Estate, 1996, page 375). Since 1989, detection of a

24,000 molecular weight protein (p24) in cell cultures, (T cells from

persons presumed to be infected), or co- cultures, (of T cells from

persons presumed to be infected, with T cells from normal

individuals), has been used to quantify HIV in cells, " cellular

viremia " (Masquelier et al., 1992). Detection of p24 in cultures of T

cells from normal individuals with plasma from those presumed to be

infected has been used to quantify HIV in plasma, " plasma viremia "

(Coombs et al., 1989; Ho et al., 1989; Clark et al., 1991). There are

many reasons why p24 cannot be used to quantify or even detect the

presence of " HIV infectious particles " . There is ample evidence that

the p24 protein is not HIV specific (Papadopulos-Eleopulos et al., 1993a).

 

Look at some of the key points, as follows:

 

1. HIV is not behaving like a typical sexually transmitted

disease.

 

2. Other immunosuppressive factors are required to increase

susceptibility.

 

3. " AIDS is a behavioral disease. It is multifactorial,

brought on by several simultaneous strains on the immune system -

drugs, pharmaceutical and recreational, sexually transmitted diseases,

multiple viral infections. "

 

4. There is nothing about this virus that is unique.

Everything that is discovered about HIV has an analogue in other

retroviruses that don't cause AIDS. HIV only contains a very small

piece of genetic information.

 

5. Like in all other viral infections, eg smallpox, you have

to be absolutely certain that what you have detected is unique and

exogenous, and a single molecular species, not particles.

 

6. The `HIV' antibody test does not detect a `virus' but an

assortment of proteins that are non-specific to the hypothetical

`HIV'. The proteins that are used in the `HIV' test are merely the

biological outcome of stressed white blood cells used in the lab.

 

7. " 99.8 per cent of putative `HIV particles' are

non-infectious; the remaining 0.2 per cent of `viral particles', being

defective, are not capable of replication. As a transmittable entity,

`HIV' could not survive in nature " .

 

8. " It is multifactorial, brought on by several simultaneous

strains on the immune system - drugs, pharmaceutical and recreational,

sexually transmitted diseases, multiple viral infections. "

 

9. Gallo claimed that the interaction of gp41 with antibodies

found in AIDS patient sera is proof that gp41 is coded by the " HIV

genome " , and that both gp41 and the antibodies are specific to a

retrovirus.

 

10. The Epstein-Barr virus (EBV) resides as a persistent

infection in human leukocyte antigen (HLA) class II+ B lymphocytes and

is associated with a number of malignancies. The EBV lytic-phase

protein gp42 serves at least two functions: gp42 acts as the

coreceptor for viral entry into B cells and hampers T-cell recognition

via HLA class II molecules through steric hindrance of T-cell

receptor-class II-peptide interactions (Maaike E. Ressing et al,

Epstein-Barr Virus gp42 Is Posttranslationally Modified To Produce

Soluble gp42 That Mediates HLA Class II Immune Evasion, Journal of

Virology, January 2005, p. 841-852, Vol. 79, No. 2).

 

11. In 1980, two research groups, one from the Laboratory of

Cellular and Molecular Biology, National Cancer Institute and the

other from the Laboratory of Viral Oncology, Memorial Sloan-Kettering

Cancer Center, using the " viral glycoproteins " , found that the

antibodies present in human sera which reacted with these proteins

were " directed against carbohydrate structures " and concluded that

" The results are consistent with the idea that the antibodies in

question are elicited as a result of exposure to many natural

substances possessing widely cross-reacting antigens and are not a

result of widespread infection of man with replication competent

oncoviruses " .

 

12. Exposure of RAW 264.7 macrophages to JP, a cell permeant

analog of phalloidin that increases and stabilizes polymerized actin

in living cells, reduced the ability of RAW 264.7 macrophages to

phagocytose fluorescent Klebsiella by 50%. This indicates that

increased actin polymerization is a potential mechanism explaining

impairment of phagocytosis by oxidative stress and since AIDS is a

condition caused by excess free radicals (in malnourished people) (see

Philip J et al, Hyperoxia Impairs Antibacterial Function of

Macrophages Through Effects on Actin, American Journal of Respiratory

Cell and Molecular Biology. Vol. 28, pp. 443-450, 2003), it quite

clearly proves that oxidative stress on macrophages leads to increased

actin polymerisation and formation of prominent stress fibers and

actin aggregates which could occur in people recovering from malaria,

influenza or in people suffering from chronic fatigue due to

mitochodrial oxidative stress or ethanol toxicity or drug induced

oxidative stress.

 

13. The serological diagnosis of HIV infection is usually made

on the basis of the detection of circulating antibodies specific for

viral antigens gp41, gp120 and gp160 and possibly gp42. Studies show

them to be non-viral glycoproteins or polymer actins that have

suffered cleavage at different points. Quite possibly hydrogen

peroxide that accumulates in people who are low in glutathione or the

seleno-antioxidant enzyme that converts it into water and oxygen, can

cause cleavage in polymer actins produced by cells of the immune

system under oxidative stress. With relatively lower oxygen generated

by the enzymatic conversion of hydrogen peroxide, people with chronic

malnutrition and low selenium intake could create a cellular

environment of hyperoxia thereby initiating marked changes, including

an increase in the degree of actin polymerization, loss of cortical

actin, and the formation of prominent stress fibers and actin aggregates.

 

What appears to be consistent is the observation supported by

scientific tests that the viral antigens in the Gallo Isolate are

actin polymers produced by cells or white blood cells in oxidative

stress and consistent with the free radical theory of AIDS that

oxidative stress produces a broad range of symptoms and illnesses and

that includes suppression of the immune system or immunodeficiency.

One certainty that emerges is that this diametrically opposes the

claim of a specific disease caused by a specific virus that has

antigenic specificity like other viruses.

 

But could the body be producing antibodies to these polymer actins

produced under oxidative stress and hence being recognized as non-self

or could some of these actins especially gp41 and gp42 act as

conjugating glycoproteins that bind with EBV viral genomes that can

hide in cells of the immune system and that conjugate is later broken

by excess hydrogen peroxide during chronic oxidative stress or severe

oxidative stress caused by chemicals and drugs? Quite possibly both,

which means that AIDS is more likely to be an EBV latency disease

triggered by oxidative stress, whether by chronic malnutrition or

drugs. That does explain the different rates of " progression " of AIDS

and opportunistic infections in the very poor societies and in the

developed world (see: THE EPSTEIN-BARR VIRUS IN AIDS on

www.newmediaexplorer.org/sepp - scroll down three pages ).

 

On one hand their HIV-causes-AIDS hypothesis tells people that

after the HIV infects cells in the immune system, it replicates in

them and it leads to the ultimate devastation of the immune system

while on the other the prescription is primarily large doses of

immunotoxic and immunosuppresive " medicine " !

 

WHY is this enigma approved by the medical authorities? Other

drugs such as, sulfonamides and trimethoprim are also used in the

treatment of PCP, which cause severe hematological complications,

including agranulocytosis, hemolytic and megaloblastic anemia and

thrombocytopenia. The results of clinical trials on AZT and protease

inhibitors have revealed that these agents are poisons and not cures.

AZT is a very toxic poison that promotes cancer formation in the human

body. " Granulocytopenia " , is a deficiency of the most numerous cells

of our immune system, which in turn leads to opportunistic infections.

Prolonged use of AZT attacks the immune system through free radical

toxicity.

 

And now, the situation as described by Dr. Roger Cunningham

(Immunologist, Microbiologist and Director of the Centre for

Immunology at the State University of New York at Buffalo) has become

rather grave because " Unfortunately, an AIDS `establishment' seems to

have formed that intends to discourage challenges to the dogma on one

side and often insists on following discredited ideas on the other, "

(Sunday Times (London) 3 April 1994).

 

The greatest intrigue remains locked in the question - " How is it

possible for the mass production of HIV in immortal T-cell lines to

continue, if Gallo claims that they attack these cells as pathogens? "

Then comes another riddle - " How is a small retrovirus, with so little

genetic material, able to infect and incorporate itself into the cell

DNA and later become virulent, killing it? "

 

The gallo-HIV enters the Guiness Book Of Records as the tiniest

fictitious artefact that ever created a multi-billion dollar industry.

 

Ooops, I forgot, we are not supposed to question the gallo-HIV

dogma. Science is meant to be applied elsewhere, not within the haloed

confines of the gallo-HIV disease and the AIDS industry it spurned.

 

- - -

 

Some other articles of Beldeu Singh that can be found on this site:

 

AIDS, NON-HIV AIDS AND PRESCRIPTION AIDS

 

NATURAL BIOMOLECULES vs DRUGS

 

WHY OMEGA-3 FISH OIL PROTECTS YOUR HEART AND BRAIN

 

Flowers and Spices: Ayurveda or Toxic Chemotherapy?

 

 

- - -

 

WORLD AIDS DAY MESSAGE FROM DR. LEO REBELLO

 

1st December is a World AIDS Day -- AIDS Racket Day would be an

apt description. My 10-point advice to you is given below :

 

 

1.. HIV has NOT been identified. Therefore to say that it causes

AIDS is the biggest con of our time.

 

 

2.. AIDS is NOT a new disease. It is called Oja-kshaya in

Ayurveda, Vettai Noi in Siddha and Al-Zabool in Unani. It is due to

depleted immunity.

 

 

3.. AIDS is NOT (repeat not) caused by normal/natural sex. So

enjoy your natural sex without guilt or stress. Because by creating

scare they create psycho-neuro immunologic defects in your system.

 

4.. The only known cause of AIDS is antibiotics, anti-retroviral

drugs, vaccines and other deadly carcinogenic chemicals that are

indiscriminately being given to all and sundry. It is madness to take

medicines for anything and everything. Medicines do not heal, they kill.

 

5.. I am NOT against condoms per se. But please note that condoms

offer only 60% protection. But because of over exposure to condoms,

today, boys and girls as young as 12 are experimenting with sex,

leading to condom culture, condom civilisation, condom ethics, condom

morals and condemned society.

 

6.. Remember that all diseases start and end with Diet and Stress.

So take care what you eat, and what you think and how you work or react.

 

7.. Beware of Vaccines. They are the root cause of Tuberculosis,

Cancers, Autism AIDS and other debilitating diseases. If at all, you

may take Homeopathic Nosodes, Urine Vaccine, Veggie Vaccines or

Auto-Blood Vaccines.

 

8.. For Venereal Diseases use Herbal pastes, pessaries and douches

and maintain physical and mental hygiene.

 

9.. Eat well, sleep well, meditate, play music, dance, exercise,

laugh, take part in sports, go for movies to relax and re-create yourself.

 

10. Dr. Water, Dr. Diet, Dr. Sleep, Dr. Exercise, Dr. Sunshine,

Dr. Humour and Dr. Leo Rebello are the seven most wonderful doctors of

the world. If you follow their advice properly, you will live to be

healthy hundred without ills, pills and doctors bills.

 

LET US WORK FROM AIDS SCARE TO AIDS CARE.

 

For more details read my books, more particularly, AIDS AND

ALTERNATIVE

MEDICINE

www.healthwisdom.org

 

 

See also:

 

 

Happy Think, Live and Love World (Non-AIDS) Day

There's an illusion running through the world that many pitiable

and poor persons must now, more than ever, be protected from having

sex (and procreating), because they risk killing each other (after

many years), by giving each other this thing called AIDS (or is it

HIV?). But it's all a myth. AIDS has nothing to do with sex, or with

any particular, single cause. AIDS is, first and foremost, a clinical

diagnosis...

 

 

 

posted by Sepp Hasslberger on Thursday November 30 2006

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talk about HIV and Aids, the presence of antibodies means something

completely different? Are the standard rules of medical treatment not

valid for Aids? Something... [read more]

August 29, 2005 - Sepp Hasslberger