Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 My Wife has H.pylori, does anybody know a natural treatment for it? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 Since Helicobacter pylori is a bacteria, I would say colloidal silver. Also, raw garlic would probably be good for treating this. The others will be along soon to give wonderful advice, though. Shara bypass42000 <jalbers4 oleander soup Mon, Jan 25, 2010 8:18 am H. Pylori My Wife has H.pylori, does anybody know a natural treatment for it? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I was reading that Mutula tea and mastic gum are effective...When i had the bacteria 7 years ago, I was treated by my family doc, triple antibiotic.. for 2 weeks...Never re-occured after that. On Mon, Jan 25, 2010 at 11:13 AM, <showard208 wrote: Since Helicobacter pylori is a bacteria, I would say colloidal silver. Also, raw garlic would probably be good for treating this. The others will be along soon to give wonderful advice, though. Shara bypass42000 <jalbers4 oleander soup Mon, Jan 25, 2010 8:18 am H. Pylori My Wife has H.pylori, does anybody know a natural treatment for it? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 Second the Mastic Gum recommendation. - Steve oleander soup oleander soup On Behalf Of H & M Feld Monday, January 25, 2010 8:20 AM oleander soup Re: H. Pylori I would try lots of a good quality mastic gum first. It is a natural anti-biotic and has worked well for this. http://www.iherb.com/Source-Naturals-Mastic-Gum-Extract-500-mg-60-Capsules/1279?at=0 Another good product has been this stuff: http://www.ulcer-cure.com/H_Pylori/h-pylori-treatment.php?sl=2 Harry - " bypass42000 " <jalbers4 <oleander soup > Monday, January 25, 2010 9:18 AM H. Pylori > My Wife has H.pylori, does anybody know a natural treatment for it? > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I would say high quantities of colloidal silver, at the very least two ounces a day. Make sure she takes probotics to keep the beneficial bacteria in her intestines thriving. Also, since H Pylori is a communicable disease, I would suggest that you also take colloidal silver as a precaution, although not the entire two ounces, maybe one tablespoon daily. Other helpful aids are:4 to 5 teaspoons of raw apple cider vinegar (Bragg's) daily.2 oz aloe vera juice throughout the day.Fresh warm lemon juice in water (as a tea) in the morning.Cayenne extract will prevent any bleeding. We use Dr. Christopher's from iherb. If you are a first time buyer you receive $5.00 off by inserting code XOT949.As I mentioned before, this infection is communicable. It spreads through saliva. So, make sure you both wash your hands often and avoid sharing the same dining utensils, plates, drinking glasses, etc.My Best, Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 The communicability of H. Pylori should not be a concern. Infact, H. Pylori is generally a beneficial bacteria when kept under control. Anarticle in the January 2005 issue of Scientific American convincingly showsthat the decline of H. Pylori in developing countries over the past 100 yearshas paralleled an upsurge in potentially fatal diseases of the esophagus. See: http://www.nature.com/scientificamerican/journal/v292/n2/full/scientificamerican0205-38.htmlAn Endangered Species in the Stomach “Just as scientists were learning the importance of H. pylori,however, they discovered that the bacteria are losing their foothold in thehuman digestive tract. Whereas nearly all adults in the developing world stillcarry the organism, its prevalence is much lower in developed countries such asthe U.S. Epidemiologists believe that H. pylori has been disappearingfrom developed nations for the past 100 years thanks to improved hygiene, whichblocks the transmission of the bacteria, and to the widespread use ofantibiotics. As H. pylori has retreated, the rates of peptic ulcers andstomach cancer have dropped. But at the same time, diseases of the esophagus—includingacid reflux disease and a particularly deadly type of esophageal cancer—haveincreased dramatically, and a wide body of evidence indicates that the rise ofthese illnesses is also related to the disappearance of H. pylori.â€â€œWhat are the consequences of H.pylori's retreat? As noted, the incidences of both peptic ulcer disease(except those cases caused by aspirin and nonsteroidal anti-inflammatory agentssuch as ibuprofen) and stomach cancer are clearly declining in developedcountries. Because these illnesses, especially stomach cancer, develop overmany years, the drop in disease incidence has lagged several decades behind thedecline in H. pylori infection, but the falloff is startlingnonetheless. In 1900 stomach cancer was the leading cause of cancer death inthe U.S.; by 2000 the incidence and mortality rates had fallen by more than 80percent, putting them well below the rates for colon, prostate, breast and lungcancers. Substantial evidence indicates that the continuing extinction of H.pylori has played an important role in this phenomenal change. This is thegood news.At the same time, however,there has been an unexpected rise in the incidence of a new class of diseasesinvolving the esophagus. Since the early 1970s, epidemiologists in the U.S.,the U.K., Sweden and Australia have noted an alarming jump in esophagealadenocarcinoma, an aggressive cancer that develops in the inner lining of theesophagus just above the stomach. The incidence of this illness in the U.S. hasbeen climbing by 7 to 9 percent each year, making it the fastest-increasingmajor cancer in the country. Once diagnosed, the five-year survival rate foresophageal adenocarcinoma is less than 10 percent.Where are these terriblecancers coming from? We know that the primary risk factor is gastroesophagealreflux disease (GERD), a chronic inflammatory disorder involving theregurgitation of acidic stomach contents into the esophagus. More commonlyknown as acid reflux disease, GERD was not even described in the medicalliterature until the 1930s. Since then, however, its incidence has risendramatically, and now the disorder is quite common in the U.S. and otherwestern countries. GERD can lead to Barrett's esophagus, a premalignant lesionfirst described in 1950 by English surgeon Norman Barrett. The incidence ofBarrett's esophagus is rising in tandem with that of GERD, and patientssuffering from the condition have an increased risk of developing esophagealadenocarcinoma. It is becoming clear that GERD may initiate a 20- to 50-yearprocess: in some cases, the disorder slowly progresses to Barrett's esophagusand then to adenocarcinoma, paralleling the gradual changes that lead tocancers in other epithelial tissues [see illustration at right]. But whyare GERD and its follow-on disorders becoming more common?The rise of these diseases hasoccurred just as H. pylori has been disappearing, and it is tempting toassociate the two phenomena. When I began proposing this connection in 1996, Iwas greeted first by indifference and then by hostility. In recent years,though, a growing number of studies support the hypothesis that H. pyloricolonization of the stomach actually protects the esophagus against GERD andits consequences. What is more, the strains bearing the cagA gene—thatis, the bacteria that are most virulent in causing ulcers and stomachcancer—appear to be the most protective of the esophagus! In 1998, working withresearchers from the National Cancer Institute, we found that people carrying cagAstrainsof H. pylori had a significantly decreased risk of developingadenocarcinomas of the lower esophagus and the part of the stomach closest tothe esophagus. Then, in collaboration with investigators from the ClevelandClinic and the Erasmus Medical Center in the Netherlands, we showed a similarcorrelation for both GERD and Barrett's esophagus. Independent confirmationshave come from the U.K., Brazil and Sweden. Not all investigators have foundthis effect, perhaps because of differences in the methods of the studies.Nevertheless, the scientific evidence is now persuasive.â€â€œThe absence of H. pylori may have other physiologicaleffects as well. The stomach produces two hormones that affect eating behavior:leptin, which signals the brain to stop eating, and ghrelin, which stimulatesappetite. Eradication of H. pylori with antibiotics tends to lowerleptin and increase ghrelin; in one study, patients who had undergone treatmentto eliminate H. pylori gained more weight than the control subjects did.Could changes in human microecology be contributing to the current epidemic ofobesity and diabetes mellitus (an obesity-related condition) in developedcountries?†The article goes on to suggest that in some cases H. Pylorimight be beneficial as a probiotic. However, if you have an ulcer it is clearthat the H. Pylori is out of control. Just taking colloidal silver may not beeffective against H. Pylori for two reasons. First, H. Pylori creates a biofilmthat helps protect it against stomach acid. Colloidal silver can penetratebiofilms but it takes time. And that brings up the second problem, colloidalsilver does not remain in the stomach for very long. A better approach that Ihave seen is to first take an enzyme such as papain or serrapeptase, on anempty stomach, to help dissolve the biofilm. Wait 15 minutes and then take thecolloidal silver. What causes H. Pylori to become out of control? http://74.125.95.132/search?q=cache:LVoVkYHSS4IJ:www.antiagingdentist.com/Medicaltheories/cd/worddoc/biofilm%2520RNA%2520seed.docx+monolaurin+biofilm & cd=38 & hl=en & ct=clnk & gl=us “In some countries, as many as 90%of the population are ‘infected’ with H. pylori, yet the frequency ofgastritis and peptic ulcer disease in these countries is rather limited. The presence of H. pylori in the majority of peptic ulcer patients doesnot necessarily mean that these bacteria cause ulcers. These bacteria are invited by achanged local ecology, really caused by zinc, selenium or magnesium deficiencyor other factors uncoupling recycling of reduced glutathione (GSH). Lackof GSH encourages viral expression, replication and mutation, thus creatingaltered immune messaging, ulcers or fissures in the barrier tissues, as well aschanged gene expression in commensal microorganisms, converting friendlytolerance-inducing symbiotic biofilm to pathogenic inflammation-incitingparasitic biofilm.†Someone with stomach ulcers  should check to see if they aregetting adequate amounts of zinc, selenium and magnesium in their diet. - Steve oleander soup oleander soup On BehalfOf MayMonday, January 25, 2010 8:55 AMoleander soup Subject: Re: H. Pylori I would sayhigh quantities of colloidal silver, at the very least two ounces a day. Make sure she takes probotics to keep the beneficial bacteria in her intestinesthriving. Also, since H Pylori is a communicable disease, I would suggestthat you also take colloidal silver as a precaution, although not the entiretwo ounces, maybe one tablespoon daily. Other helpful aids are:4 to 5 teaspoons of raw apple cider vinegar (Bragg's) daily.2 oz aloe vera juice throughout the day.Fresh warm lemon juice in water (as a tea) in the morning.Cayenne extract will prevent any bleeding. We use Dr. Christopher's from iherb. If you are a first time buyer you receive $5.00 off by inserting code XOT949.As I mentioned before, this infection is communicable. It spreads throughsaliva. So, make sure you both wash your hands often and avoid sharingthe same dining utensils, plates, drinking glasses, etc.My Best, Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I sent this once but it does not appear to have posted. Here is another try.The communicability of H. Pylori should not be a concern. In fact, H. Pylori is generally a beneficial bacteria when kept under control. An article in the January 2005 issue of Scientific American convincingly shows that the decline of H. Pylori in developing countries over the past 100 years has paralleled an upsurge in potentially fatal diseases of the esophagus. See:http://www.nature.com/scientificamerican/journal/v292/n2/full/scientificamerican0205-38.htmlAn Endangered Species in the Stomach“Just as scientists were learning the importance of H. pylori, however, they discovered that the bacteria are losing their foothold in the human digestive tract. Whereas nearly all adults in the developing world still carry the organism, its prevalence is much lower in developed countries such as the U.S. Epidemiologists believe that H. pylori has been disappearing from developed nations for the past 100 years thanks to improved hygiene, which blocks the transmission of the bacteria, and to the widespread use of antibiotics. As H. pylori has retreated, the rates of peptic ulcers and stomach cancer have dropped. But at the same time, diseases of the esophagus—including acid reflux disease and a particularly deadly type of esophageal cancer—have increased dramatically, and a wide body of evidence indicates that the rise of these illnesses is also related to the disappearance of H. pylori.â€â€œWhat are the consequences of H. pylori's retreat? As noted, the incidences of both peptic ulcer disease (except those cases caused by aspirin and nonsteroidal anti-inflammatory agents such as ibuprofen) and stomach cancer are clearly declining in developed countries. Because these illnesses, especially stomach cancer, develop over many years, the drop in disease incidence has lagged several decades behind the decline in H. pylori infection, but the falloff is startling nonetheless. In 1900 stomach cancer was the leading cause of cancer death in the U.S.; by 2000 the incidence and mortality rates had fallen by more than 80 percent, putting them well below the rates for colon, prostate, breast and lung cancers. Substantial evidence indicates that the continuing extinction of H. pylori has played an important role in this phenomenal change. This is the good news.At the same time, however, there has been an unexpected rise in the incidence of a new class of diseases involving the esophagus. Since the early 1970s, epidemiologists in the U.S., the U.K., Sweden and Australia have noted an alarming jump in esophageal adenocarcinoma, an aggressive cancer that develops in the inner lining of the esophagus just above the stomach. The incidence of this illness in the U.S. has been climbing by 7 to 9 percent each year, making it the fastest-increasing major cancer in the country. Once diagnosed, the five-year survival rate for esophageal adenocarcinoma is less than 10 percent.Where are these terrible cancers coming from? We know that the primary risk factor is gastroesophageal reflux disease (GERD), a chronic inflammatory disorder involving the regurgitation of acidic stomach contents into the esophagus. More commonly known as acid reflux disease, GERD was not even described in the medical literature until the 1930s. Since then, however, its incidence has risen dramatically, and now the disorder is quite common in the U.S. and other western countries. GERD can lead to Barrett's esophagus, a premalignant lesion first described in 1950 by English surgeon Norman Barrett. The incidence of Barrett's esophagus is rising in tandem with that of GERD, and patients suffering from the condition have an increased risk of developing esophageal adenocarcinoma. It is becoming clear that GERD may initiate a 20- to 50-year process: in some cases, the disorder slowly progresses to Barrett's esophagus and then to adenocarcinoma, paralleling the gradual changes that lead to cancers in other epithelial tissues [see illustration at right]. But why are GERD and its follow-on disorders becoming more common?The rise of these diseases has occurred just as H. pylori has been disappearing, and it is tempting to associate the two phenomena. When I began proposing this connection in 1996, I was greeted first by indifference and then by hostility. In recent years, though, a growing number of studies support the hypothesis that H. pylori colonization of the stomach actually protects the esophagus against GERD and its consequences. What is more, the strains bearing the cagA gene—that is, the bacteria that are most virulent in causing ulcers and stomach cancer—appear to be the most protective of the esophagus! In 1998, working with researchers from the National Cancer Institute, we found that people carrying cagAstrains of H. pylori had a significantly decreased risk of developing adenocarcinomas of the lower esophagus and the part of the stomach closest to the esophagus. Then, in collaboration with investigators from the Cleveland Clinic and the Erasmus Medical Center in the Netherlands, we showed a similar correlation for both GERD and Barrett's esophagus. Independent confirmations have come from the U.K., Brazil and Sweden. Not all investigators have found this effect, perhaps because of differences in the methods of the studies. Nevertheless, the scientific evidence is now persuasive.â€â€œThe absence of H. pylori may have other physiological effects as well. The stomach produces two hormones that affect eating behavior: leptin, which signals the brain to stop eating, and ghrelin, which stimulates appetite. Eradication of H. pylori with antibiotics tends to lower leptin and increase ghrelin; in one study, patients who had undergone treatment to eliminate H. pylori gained more weight than the control subjects did. Could changes in human microecology be contributing to the current epidemic of obesity and diabetes mellitus (an obesity-related condition) in developed countries?â€The article goes on to suggest that in some cases H. Pylori might be beneficial as a probiotic. However, if you have an ulcer it is clear that the H. Pylori is out of control. Just taking colloidal silver may not be effective against H. Pylori for two reasons. First, H. Pylori creates a biofilm that helps protect it against stomach acid. Colloidal silver can penetrate biofilms but it takes time. And that brings up the second problem, colloidal silver does not remain in the stomach for very long. A better approach that I have seen is to first take an enzyme such as papain or serrapeptase, on an empty stomach, to help dissolve the biofilm. Wait 15 minutes and then take the colloidal silver.What causes H. Pylori to become out of control? http://74.125.95.132/search?q=cache:LVoVkYHSS4IJ:www.antiagingdentist.com/Medicaltheories/cd/worddoc/biofilm%2520RNA%2520seed.docx+monolaurin+biofilm & cd=38 & hl=en & ct=clnk & gl=us“In some countries, as many as 90% of the population are ‘infected’ with H. pylori, yet the frequency of gastritis and peptic ulcer disease in these countries is rather limited. The presence of H. pylori in the majority of peptic ulcer patients does not necessarily mean that these bacteria cause ulcers. These bacteria are invited by a changed local ecology, really caused by zinc, selenium or magnesium deficiency or other factors uncoupling recycling of reduced glutathione (GSH). Lack of GSH encourages viral expression, replication and mutation, thus creating altered immune messaging, ulcers or fissures in the barrier tissues, as well as changed gene expression in commensal microorganisms, converting friendly tolerance-inducing symbiotic biofilm to pathogenic inflammation-inciting parasitic biofilm.â€Someone with stomach ulcers  should check to see if they are getting adequate amounts of zinc, selenium and magnesium in their diet.- Steveoleander soup oleander soup On Behalf Of MayMonday, January 25, 2010 8:55 AMoleander soup Subject: Re: H. Pylori I would say high quantities of colloidal silver, at the very least two ounces a day. Make sure she takes probotics to keep the beneficial bacteria in her intestines thriving. Also, since H Pylori is a communicable disease, I would suggest that you also take colloidal silver as a precaution, although not the entire two ounces, maybe one tablespoon daily. Other helpful aids are:4 to 5 teaspoons of raw apple cider vinegar (Bragg's) daily.2 oz aloe vera juice throughout the day.Fresh warm lemon juice in water (as a tea) in the morning.Cayenne extract will prevent any bleeding. We use Dr. Christopher's from iherb. If you are a first time buyer you receive $5.00 off by inserting code XOT949.As I mentioned before, this infection is communicable. It spreads through saliva. So, make sure you both wash your hands often and avoid sharing the same dining utensils, plates, drinking glasses, etc.My Best, Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I sent this before but it does not appear to have posted. Here is another try but in a plain text format. The communicability of H. Pylori should not be a concern. In fact, H. Pylori is generally a beneficial bacteria when kept under control. An article in the January 2005 issue of Scientific American convincingly shows that the decline of H. Pylori in developing countries over the past 100 years has paralleled an upsurge in potentially fatal diseases of the esophagus. See: http://www.nature.com/scientificamerican/journal/v292/n2/full/scientificamerican\ 0205-38.html An Endangered Species in the Stomach “Just as scientists were learning the importance of H. pylori, however, they discovered that the bacteria are losing their foothold in the human digestive tract. Whereas nearly all adults in the developing world still carry the organism, its prevalence is much lower in developed countries such as the U.S. Epidemiologists believe that H. pylori has been disappearing from developed nations for the past 100 years thanks to improved hygiene, which blocks the transmission of the bacteria, and to the widespread use of antibiotics. As H. pylori has retreated, the rates of peptic ulcers and stomach cancer have dropped. But at the same time, diseases of the esophagus—including acid reflux disease and a particularly deadly type of esophageal cancer—have increased dramatically, and a wide body of evidence indicates that the rise of these illnesses is also related to the disappearance of H. pylori.†“What are the consequences of H. pylori's retreat? As noted, the incidences of both peptic ulcer disease (except those cases caused by aspirin and nonsteroidal anti-inflammatory agents such as ibuprofen) and stomach cancer are clearly declining in developed countries. Because these illnesses, especially stomach cancer, develop over many years, the drop in disease incidence has lagged several decades behind the decline in H. pylori infection, but the falloff is startling nonetheless. In 1900 stomach cancer was the leading cause of cancer death in the U.S.; by 2000 the incidence and mortality rates had fallen by more than 80 percent, putting them well below the rates for colon, prostate, breast and lung cancers. Substantial evidence indicates that the continuing extinction of H. pylori has played an important role in this phenomenal change. This is the good news. At the same time, however, there has been an unexpected rise in the incidence of a new class of diseases involving the esophagus. Since the early 1970s, epidemiologists in the U.S., the U.K., Sweden and Australia have noted an alarming jump in esophageal adenocarcinoma, an aggressive cancer that develops in the inner lining of the esophagus just above the stomach. The incidence of this illness in the U.S. has been climbing by 7 to 9 percent each year, making it the fastest-increasing major cancer in the country. Once diagnosed, the five-year survival rate for esophageal adenocarcinoma is less than 10 percent. Where are these terrible cancers coming from? We know that the primary risk factor is gastroesophageal reflux disease (GERD), a chronic inflammatory disorder involving the regurgitation of acidic stomach contents into the esophagus. More commonly known as acid reflux disease, GERD was not even described in the medical literature until the 1930s. Since then, however, its incidence has risen dramatically, and now the disorder is quite common in the U.S. and other western countries. GERD can lead to Barrett's esophagus, a premalignant lesion first described in 1950 by English surgeon Norman Barrett. The incidence of Barrett's esophagus is rising in tandem with that of GERD, and patients suffering from the condition have an increased risk of developing esophageal adenocarcinoma. It is becoming clear that GERD may initiate a 20- to 50-year process: in some cases, the disorder slowly progresses to Barrett's esophagus and then to adenocarcinoma, paralleling the gradual changes that lead to cancers in other epithelial tissues [see illustration at right]. But why are GERD and its follow-on disorders becoming more common? The rise of these diseases has occurred just as H. pylori has been disappearing, and it is tempting to associate the two phenomena. When I began proposing this connection in 1996, I was greeted first by indifference and then by hostility. In recent years, though, a growing number of studies support the hypothesis that H. pylori colonization of the stomach actually protects the esophagus against GERD and its consequences. What is more, the strains bearing the cagA gene—that is, the bacteria that are most virulent in causing ulcers and stomach cancer—appear to be the most protective of the esophagus! In 1998, working with researchers from the National Cancer Institute, we found that people carrying cagAstrains of H. pylori had a significantly decreased risk of developing adenocarcinomas of the lower esophagus and the part of the stomach closest to the esophagus. Then, in collaboration with investigators from the Cleveland Clinic and the Erasmus Medical Center in the Netherlands, we showed a similar correlation for both GERD and Barrett's esophagus. Independent confirmations have come from the U.K., Brazil and Sweden. Not all investigators have found this effect, perhaps because of differences in the methods of the studies. Nevertheless, the scientific evidence is now persuasive.†“The absence of H. pylori may have other physiological effects as well. The stomach produces two hormones that affect eating behavior: leptin, which signals the brain to stop eating, and ghrelin, which stimulates appetite. Eradication of H. pylori with antibiotics tends to lower leptin and increase ghrelin; in one study, patients who had undergone treatment to eliminate H. pylori gained more weight than the control subjects did. Could changes in human microecology be contributing to the current epidemic of obesity and diabetes mellitus (an obesity-related condition) in developed countries?†The article goes on to suggest that in some cases H. Pylori might be beneficial as a probiotic. However, if you have an ulcer it is clear that the H. Pylori is out of control. Just taking colloidal silver may not be effective against H. Pylori for two reasons. First, H. Pylori creates a biofilm that helps protect it against stomach acid. Colloidal silver can penetrate biofilms but it takes time. And that brings up the second problem, colloidal silver does not remain in the stomach for very long. A better approach that I have seen is to first take an enzyme such as papain or serrapeptase, on an empty stomach, to help dissolve the biofilm. Wait 15 minutes and then take the colloidal silver. What causes H. Pylori to become out of control? http://74.125.95.132/search?q=cache:LVoVkYHSS4IJ:www.antiagingdentist.com/Medica\ ltheories/cd/worddoc/biofilm%2520RNA%2520seed.docx+monolaurin+biofilm & cd=38 & hl=e\ n & ct=clnk & gl=us “In some countries, as many as 90% of the population are ‘infected’ with H. pylori, yet the frequency of gastritis and peptic ulcer disease in these countries is rather limited. The presence of H. pylori in the majority of peptic ulcer patients does not necessarily mean that these bacteria cause ulcers. These bacteria are invited by a changed local ecology, really caused by zinc, selenium or magnesium deficiency or other factors uncoupling recycling of reduced glutathione (GSH). Lack of GSH encourages viral expression, replication and mutation, thus creating altered immune messaging, ulcers or fissures in the barrier tissues, as well as changed gene expression in commensal microorganisms, converting friendly tolerance-inducing symbiotic biofilm to pathogenic inflammation-inciting parasitic biofilm.†Someone with stomach ulcers  should check to see if they are getting adequate amounts of zinc, selenium and magnesium in their diet. - Steve oleander soup oleander soup On Behalf Of May Monday, January 25, 2010 8:55 AM oleander soup Re: H. Pylori  I would say high quantities of colloidal silver, at the very least two ounces a day. Make sure she takes probotics to keep the beneficial bacteria in her intestines thriving. Also, since H Pylori is a communicable disease, I would suggest that you also take colloidal silver as a precaution, although not the entire two ounces, maybe one tablespoon daily. Other helpful aids are: 4 to 5 teaspoons of raw apple cider vinegar (Bragg's) daily. 2 oz aloe vera juice throughout the day. Fresh warm lemon juice in water (as a tea) in the morning. Cayenne extract will prevent any bleeding. We use Dr. Christopher's from iherb. If you are a first time buyer you receive $5.00 off by inserting code XOT949. As I mentioned before, this infection is communicable. It spreads through saliva. So, make sure you both wash your hands often and avoid sharing the same dining utensils, plates, drinking glasses, etc. My Best, Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I sent this before but it does not appear to have posted. Here is another try but in a plain text format. The communicability of H. Pylori should not be a concern. In fact, H. Pylori is generally a beneficial bacteria when kept under control. An article in the January 2005 issue of Scientific American convincingly shows that the decline of H. Pylori in developing countries over the past 100 years has paralleled an upsurge in potentially fatal diseases of the esophagus. See: http://www.nature.com/scientificamerican/journal/v292/n2/full/scientificamerican\ 0205-38.html An Endangered Species in the Stomach “Just as scientists were learning the importance of H. pylori, however, they discovered that the bacteria are losing their foothold in the human digestive tract. Whereas nearly all adults in the developing world still carry the organism, its prevalence is much lower in developed countries such as the U.S. Epidemiologists believe that H. pylori has been disappearing from developed nations for the past 100 years thanks to improved hygiene, which blocks the transmission of the bacteria, and to the widespread use of antibiotics. As H. pylori has retreated, the rates of peptic ulcers and stomach cancer have dropped. But at the same time, diseases of the esophagus—including acid reflux disease and a particularly deadly type of esophageal cancer—have increased dramatically, and a wide body of evidence indicates that the rise of these illnesses is also related to the disappearance of H. pylori.†“What are the consequences of H. pylori's retreat? As noted, the incidences of both peptic ulcer disease (except those cases caused by aspirin and nonsteroidal anti-inflammatory agents such as ibuprofen) and stomach cancer are clearly declining in developed countries. Because these illnesses, especially stomach cancer, develop over many years, the drop in disease incidence has lagged several decades behind the decline in H. pylori infection, but the falloff is startling nonetheless. In 1900 stomach cancer was the leading cause of cancer death in the U.S.; by 2000 the incidence and mortality rates had fallen by more than 80 percent, putting them well below the rates for colon, prostate, breast and lung cancers. Substantial evidence indicates that the continuing extinction of H. pylori has played an important role in this phenomenal change. This is the good news. At the same time, however, there has been an unexpected rise in the incidence of a new class of diseases involving the esophagus. Since the early 1970s, epidemiologists in the U.S., the U.K., Sweden and Australia have noted an alarming jump in esophageal adenocarcinoma, an aggressive cancer that develops in the inner lining of the esophagus just above the stomach. The incidence of this illness in the U.S. has been climbing by 7 to 9 percent each year, making it the fastest-increasing major cancer in the country. Once diagnosed, the five-year survival rate for esophageal adenocarcinoma is less than 10 percent. Where are these terrible cancers coming from? We know that the primary risk factor is gastroesophageal reflux disease (GERD), a chronic inflammatory disorder involving the regurgitation of acidic stomach contents into the esophagus. More commonly known as acid reflux disease, GERD was not even described in the medical literature until the 1930s. Since then, however, its incidence has risen dramatically, and now the disorder is quite common in the U.S. and other western countries. GERD can lead to Barrett's esophagus, a premalignant lesion first described in 1950 by English surgeon Norman Barrett. The incidence of Barrett's esophagus is rising in tandem with that of GERD, and patients suffering from the condition have an increased risk of developing esophageal adenocarcinoma. It is becoming clear that GERD may initiate a 20- to 50-year process: in some cases, the disorder slowly progresses to Barrett's esophagus and then to adenocarcinoma, paralleling the gradual changes that lead to cancers in other epithelial tissues [see illustration at right]. But why are GERD and its follow-on disorders becoming more common? The rise of these diseases has occurred just as H. pylori has been disappearing, and it is tempting to associate the two phenomena. When I began proposing this connection in 1996, I was greeted first by indifference and then by hostility. In recent years, though, a growing number of studies support the hypothesis that H. pylori colonization of the stomach actually protects the esophagus against GERD and its consequences. What is more, the strains bearing the cagA gene—that is, the bacteria that are most virulent in causing ulcers and stomach cancer—appear to be the most protective of the esophagus! In 1998, working with researchers from the National Cancer Institute, we found that people carrying cagAstrains of H. pylori had a significantly decreased risk of developing adenocarcinomas of the lower esophagus and the part of the stomach closest to the esophagus. Then, in collaboration with investigators from the Cleveland Clinic and the Erasmus Medical Center in the Netherlands, we showed a similar correlation for both GERD and Barrett's esophagus. Independent confirmations have come from the U.K., Brazil and Sweden. Not all investigators have found this effect, perhaps because of differences in the methods of the studies. Nevertheless, the scientific evidence is now persuasive.†“The absence of H. pylori may have other physiological effects as well. The stomach produces two hormones that affect eating behavior: leptin, which signals the brain to stop eating, and ghrelin, which stimulates appetite. Eradication of H. pylori with antibiotics tends to lower leptin and increase ghrelin; in one study, patients who had undergone treatment to eliminate H. pylori gained more weight than the control subjects did. Could changes in human microecology be contributing to the current epidemic of obesity and diabetes mellitus (an obesity-related condition) in developed countries?†The article goes on to suggest that in some cases H. Pylori might be beneficial as a probiotic. However, if you have an ulcer it is clear that the H. Pylori is out of control. Just taking colloidal silver may not be effective against H. Pylori for two reasons. First, H. Pylori creates a biofilm that helps protect it against stomach acid. Colloidal silver can penetrate biofilms but it takes time. And that brings up the second problem, colloidal silver does not remain in the stomach for very long. A better approach that I have seen is to first take an enzyme such as papain or serrapeptase, on an empty stomach, to help dissolve the biofilm. Wait 15 minutes and then take the colloidal silver. What causes H. Pylori to become out of control? See: http://74.125.95.132/search?q=cache:LVoVkYHSS4IJ:www.antiagingdentist.com/Medica\ ltheories/cd/worddoc/biofilm%2520RNA%2520seed.docx+monolaurin+biofilm & cd=38 & hl=e\ n & ct=clnk & gl=us “In some countries, as many as 90% of the population are ‘infected’ with H. pylori, yet the frequency of gastritis and peptic ulcer disease in these countries is rather limited. The presence of H. pylori in the majority of peptic ulcer patients does not necessarily mean that these bacteria cause ulcers. These bacteria are invited by a changed local ecology, really caused by zinc, selenium or magnesium deficiency or other factors uncoupling recycling of reduced glutathione (GSH). Lack of GSH encourages viral expression, replication and mutation, thus creating altered immune messaging, ulcers or fissures in the barrier tissues, as well as changed gene expression in commensal microorganisms, converting friendly tolerance-inducing symbiotic biofilm to pathogenic inflammation-inciting parasitic biofilm.†Someone with stomach ulcers  should check to see if they are getting adequate amounts of zinc, selenium and magnesium in their diet. - Steve Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 I sent this before but it does not appear to have posted. Here is another try but in a plain text format. Has anyone received the original message? - Steve Norton, Steve Monday, January 25, 2010 2:24 PM oleander soup RE: H. Pylori The communicability of H. Pylori should not be a concern. In fact, H. Pylori is generally a beneficial bacteria when kept under control. An article in the January 2005 issue of Scientific American convincingly shows that the decline of H. Pylori in developing countries over the past 100 years has paralleled an upsurge in potentially fatal diseases of the esophagus. See: http://www.nature.com/scientificamerican/journal/v292/n2/full/scientificamerican\ 0205-38.html An Endangered Species in the Stomach “Just as scientists were learning the importance of H. pylori, however, they discovered that the bacteria are losing their foothold in the human digestive tract. Whereas nearly all adults in the developing world still carry the organism, its prevalence is much lower in developed countries such as the U.S. Epidemiologists believe that H. pylori has been disappearing from developed nations for the past 100 years thanks to improved hygiene, which blocks the transmission of the bacteria, and to the widespread use of antibiotics. As H. pylori has retreated, the rates of peptic ulcers and stomach cancer have dropped. But at the same time, diseases of the esophagus—including acid reflux disease and a particularly deadly type of esophageal cancer—have increased dramatically, and a wide body of evidence indicates that the rise of these illnesses is also related to the disappearance of H. pylori.†“What are the consequences of H. pylori's retreat? As noted, the incidences of both peptic ulcer disease (except those cases caused by aspirin and nonsteroidal anti-inflammatory agents such as ibuprofen) and stomach cancer are clearly declining in developed countries. Because these illnesses, especially stomach cancer, develop over many years, the drop in disease incidence has lagged several decades behind the decline in H. pylori infection, but the falloff is startling nonetheless. In 1900 stomach cancer was the leading cause of cancer death in the U.S.; by 2000 the incidence and mortality rates had fallen by more than 80 percent, putting them well below the rates for colon, prostate, breast and lung cancers. Substantial evidence indicates that the continuing extinction of H. pylori has played an important role in this phenomenal change. This is the good news. At the same time, however, there has been an unexpected rise in the incidence of a new class of diseases involving the esophagus. Since the early 1970s, epidemiologists in the U.S., the U.K., Sweden and Australia have noted an alarming jump in esophageal adenocarcinoma, an aggressive cancer that develops in the inner lining of the esophagus just above the stomach. The incidence of this illness in the U.S. has been climbing by 7 to 9 percent each year, making it the fastest-increasing major cancer in the country. Once diagnosed, the five-year survival rate for esophageal adenocarcinoma is less than 10 percent. Where are these terrible cancers coming from? We know that the primary risk factor is gastroesophageal reflux disease (GERD), a chronic inflammatory disorder involving the regurgitation of acidic stomach contents into the esophagus. More commonly known as acid reflux disease, GERD was not even described in the medical literature until the 1930s. Since then, however, its incidence has risen dramatically, and now the disorder is quite common in the U.S. and other western countries. GERD can lead to Barrett's esophagus, a premalignant lesion first described in 1950 by English surgeon Norman Barrett. The incidence of Barrett's esophagus is rising in tandem with that of GERD, and patients suffering from the condition have an increased risk of developing esophageal adenocarcinoma. It is becoming clear that GERD may initiate a 20- to 50-year process: in some cases, the disorder slowly progresses to Barrett's esophagus and then to adenocarcinoma, paralleling the gradual changes that lead to cancers in other epithelial tissues [see illustration at right]. But why are GERD and its follow-on disorders becoming more common? The rise of these diseases has occurred just as H. pylori has been disappearing, and it is tempting to associate the two phenomena. When I began proposing this connection in 1996, I was greeted first by indifference and then by hostility. In recent years, though, a growing number of studies support the hypothesis that H. pylori colonization of the stomach actually protects the esophagus against GERD and its consequences. What is more, the strains bearing the cagA gene—that is, the bacteria that are most virulent in causing ulcers and stomach cancer—appear to be the most protective of the esophagus! In 1998, working with researchers from the National Cancer Institute, we found that people carrying cagAstrains of H. pylori had a significantly decreased risk of developing adenocarcinomas of the lower esophagus and the part of the stomach closest to the esophagus. Then, in collaboration with investigators from the Cleveland Clinic and the Erasmus Medical Center in the Netherlands, we showed a similar correlation for both GERD and Barrett's esophagus. Independent confirmations have come from the U.K., Brazil and Sweden. Not all investigators have found this effect, perhaps because of differences in the methods of the studies. Nevertheless, the scientific evidence is now persuasive.†“The absence of H. pylori may have other physiological effects as well. The stomach produces two hormones that affect eating behavior: leptin, which signals the brain to stop eating, and ghrelin, which stimulates appetite. Eradication of H. pylori with antibiotics tends to lower leptin and increase ghrelin; in one study, patients who had undergone treatment to eliminate H. pylori gained more weight than the control subjects did. Could changes in human microecology be contributing to the current epidemic of obesity and diabetes mellitus (an obesity-related condition) in developed countries?†The article goes on to suggest that in some cases H. Pylori might be beneficial as a probiotic. However, if you have an ulcer it is clear that the H. Pylori is out of control. Just taking colloidal silver may not be effective against H. Pylori for two reasons. First, H. Pylori creates a biofilm that helps protect it against stomach acid. Colloidal silver can penetrate biofilms but it takes time. And that brings up the second problem, colloidal silver does not remain in the stomach for very long. A better approach that I have seen is to first take an enzyme such as papain or serrapeptase, on an empty stomach, to help dissolve the biofilm. Wait 15 minutes and then take the colloidal silver. What causes H. Pylori to become out of control? See: http://74.125.95.132/search?q=cache:LVoVkYHSS4IJ:www.antiagingdentist.com/Medica\ ltheories/cd/worddoc/biofilm%2520RNA%2520seed.docx+monolaurin+biofilm & cd=38 & hl=e\ n & ct=clnk & gl=us “In some countries, as many as 90% of the population are ‘infected’ with H. pylori, yet the frequency of gastritis and peptic ulcer disease in these countries is rather limited. The presence of H. pylori in the majority of peptic ulcer patients does not necessarily mean that these bacteria cause ulcers. These bacteria are invited by a changed local ecology, really caused by zinc, selenium or magnesium deficiency or other factors uncoupling recycling of reduced glutathione (GSH). Lack of GSH encourages viral expression, replication and mutation, thus creating altered immune messaging, ulcers or fissures in the barrier tissues, as well as changed gene expression in commensal microorganisms, converting friendly tolerance-inducing symbiotic biofilm to pathogenic inflammation-inciting parasitic biofilm.†Someone with stomach ulcers  should check to see if they are getting adequate amounts of zinc, selenium and magnesium in their diet. - Steve Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 25, 2010 Report Share Posted January 25, 2010 Steve, I don't know about everyone else, but I have received it 5x’s. Not sure why it is not showing up in your email. Be Well Dr.L Loretta Lanphier, ND, CN, HHP, CH OASIS ADVANCED WELLNESS, LLC Natural Solutions for Optimum Health  Follow us on Twitter Follow us on Facebook oleander soup oleander soup On Behalf Of Norton, Steve Monday, January 25, 2010 5:38 PM oleander soup Re: H. Pylori I sent this before but it does not appear to have posted. Here is another try but in a plain text format. Has anyone received the original message?  - Steve Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 26, 2010 Report Share Posted January 26, 2010 Thanks. I was wondering what was happening. It has never shownup in my email.- Steve oleander soup oleander soup On BehalfOf Dr. Loretta LanphierMonday, January 25, 2010 3:59 PMoleander soup Subject: RE: H. Pylori Steve, I don't know about everyone else, but I have received it5x’s. Not sure why it is not showing up in your email. BeWellDr.L LorettaLanphier, ND, CN, HHP, CHOASIS ADVANCED WELLNESS, LLCNatural Solutions forOptimum Health Follow us on Twitter Follow us on Facebook Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 26, 2010 Report Share Posted January 26, 2010 Everybody has it. As long as the bug does not take over a part of the stomach causing ulceration, it may be actually symbiotic. Normal orthodox treatment for stomach ulcers for the last 20 years has been antibiotics, which work quite well. You do not want to know what it was before the time an Australian doctor stood up to whole medical establishment proving that the ulcers were due to this bug. I have been through it [stomach ulcer] and there are probably several ways to do without antibiotics. Aside from being on Pepside and Prepulside for a few months some 15 years ago, both prescription drugs then and not exactly cheap, the second being much worse than the first, I got to do some thinking and I did my ulcer in with whisky. No kidding! One big shot of whisky on an empty stomach every morning and nothing to eat or drink for some 10 more minutes. The symptoms were gone in 2 weeks and I continued for another four, just to get an insurance. I have not had one since. Lots of people get the ulcers out by a long water fast, 2 weeks or longer. It appears that lowering the stomach acidity by coaxing it not to produce stomach juices for a long enough period does the job too. With kind regards, Slavek. bypass42000 wrote: > > > > My Wife has H.pylori, does anybody know a natural treatment for it? > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 5, 2010 Report Share Posted April 5, 2010 Zuo Jin Wan & #24038; & #37329; & #20024; - wu zhu yu is said to knock out h.pylori. It is one part wu zhu yu and 6 parts huang lian. Also for patients who don't know if they have h. pylori I've used the patent Xiang Lian Wan - simply Mu Xiang and Huang Lian - xiang lian 3 grams and Huang lian 9 if written out. Both work very well very quickly for what the patient describe as reflux or heartburn. Doug , Lola Fox <lolafox wrote: > > Are there any known herbs or formulas known to treat h. Pylori regardless of pattern presentation? I'd be willing to experiment on myself. Treating the pattern did not eliminate it. > > Sent from my iPhone > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 5, 2010 Report Share Posted April 5, 2010 Thanks Doug, how soon do you recommend retesting to see if h pylori is gone? Sent from my iPhone On Apr 5, 2010, at 2:33 PM, " " wrote: Zuo Jin Wan & #24038; & #37329; & #20024; - wu zhu yu is said to knock out h.pylori. It is one part wu zhu yu and 6 parts huang lian. Also for patients who don't know if they have h. pylori I've used the patent Xiang Lian Wan - simply Mu Xiang and Huang Lian - xiang lian 3 grams and Huang lian 9 if written out. Both work very well very quickly for what the patient describe as reflux or heartburn. Doug , Lola Fox <lolafox wrote: > > Are there any known herbs or formulas known to treat h. Pylori regardless of pattern presentation? I'd be willing to experiment on myself. Treating the pattern did not eliminate it. > > Sent from my iPhone > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 5, 2010 Report Share Posted April 5, 2010 masuka honey that you can buy in the US appears to lower h.pylori. --- On Mon, 4/5/10, Lola Fox <lolafox wrote: Lola Fox <lolafox H. Pylori " " Monday, April 5, 2010, 6:48 AM  Are there any known herbs or formulas known to treat h. Pylori regardless of pattern presentation? I'd be willing to experiment on myself. Treating the pattern did not eliminate it. Sent from my iPhone Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 5, 2010 Report Share Posted April 5, 2010 Do you mean Manuka honey? On Apr 5, 2010, at 6:09 PM, connie reynolds wrote: > masuka honey that you can buy in the US appears to lower h.pylori. > > --- On Mon, 4/5/10, Lola Fox <lolafox wrote: > > Lola Fox <lolafox > H. Pylori > " " < > > > Monday, April 5, 2010, 6:48 AM > > > > Are there any known herbs or formulas known to treat h. Pylori > regardless of pattern presentation? I'd be willing to experiment on > myself. Treating the pattern did not eliminate it. > > Sent from my iPhone > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 5, 2010 Report Share Posted April 5, 2010 I generally don't... just following the symptoms... doug , Lola Fox <lolafox wrote: > > Thanks Doug, how soon do you recommend retesting to see if h pylori is gone? > > Sent from my iPhone > > On Apr 5, 2010, at 2:33 PM, " " wrote: > > Zuo Jin Wan & #24038; & #37329; & #20024; - wu zhu yu is said to knock out h.pylori. It is one part wu zhu yu and 6 parts huang lian. Also for patients who don't know if they have h. pylori I've used the patent Xiang Lian Wan - simply Mu Xiang and Huang Lian - xiang lian 3 grams and Huang lian 9 if written out. Both work very well very quickly for what the patient describe as reflux or heartburn. > Doug > > , Lola Fox <lolafox@> wrote: > > > > Are there any known herbs or formulas known to treat h. Pylori regardless of pattern presentation? I'd be willing to experiment on myself. Treating the pattern did not eliminate it. > > > > Sent from my iPhone > > > > > > > > Quote Link to comment Share on other sites More sharing options...
Recommended Posts
Join the conversation
You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.