AIDS - Retrovirus Expression Regulated by Methylation?

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18 May 2006 21:06:35 -0000

Health Supreme Update: AIDS - Retrovirus Expression Regulated

by Methylation?

sepp

 

 

http://www.newmediaexplorer.org/sepp/2006/05/18/aids_retrovirus_expression_regul\

ated_by_methylation.htm

 

 

 

 

Health Supreme Update: AIDS - Retrovirus Expression Regulated by

Methylation?

 

2006.05.18 23:06:34

 

 

May 18, 2006

 

AIDS - Retrovirus Expression Regulated by Methylation?

 

 

AIDS may not be what we are told it is - a retrovirus-mediated disease.

 

Illustration of what HIV is thought to look like. Image source

 

Rebecca Culshaw, a statistical mathematician and biologist came to the

conclusion that the official explanation of how AIDS is caused does

not make sense, when analyzing the statistical data with a view to

finding ways to overcome the syndrome. Culshaw quit her AIDS related

work and she explains why in her recent article.

 

Looking for the real cause of AIDS, geo-epidemiologist Dr Harold

Foster discovered that selenium, as well as three aminoacids are

generally found to be low in AIDS victims and are also low in the food

supply where AIDS is found in higher concentrations. In a recent

newsletter, the Orthomolecular Medicine News Service makes reference

to Harold Foster's discovery saying that high doses of the trace

element selenium, and of the amino acids cysteine, tryptophan, and

glutamine can together rapidly reverse the symptoms of AIDS.

 

Chinese researchers have found selenium to be an important mineral

nutrient to prevent both AIDS and Avian Flu. The article of Qu Yuan,

Qu Lai and Qu Shaozhong Avian influenza's " internal factor " of

selenium deficiency and an epidemic prevention strategy of " treating

the cause " makes interesting reading.

 

Beldeu Singh from Malaysia says that AIDS and CFS are Caused By

Oxidative Damage.

 

Now to tie this together even more, Cal Crilly, an artist from

Brisbane Australia and long time AIDS activist, adds an important

piece to the puzzle. He explains that retrovirus expression is

suppressed by proper methylation of our DNA, which in turn depends on

a selenium compund, S-adenosyl-methionine.

 

Here is Cal's contribution to the discussion. Perhaps one day -

hopefully soon - we will see this accumulating knowledge on the real

etiology of AIDS translated into actual solutions for those who are

suffering... CDC take note.

 

- - -

 

Cal Crilly found these connecting links and posted them as a comment to

Glutathione Peroxidase - Selenium, Aminoacids Overcome AIDS

 

I have to point out what everyone may be missing

 

Our genome is made up of about 40% transposable elements in the DNA.

 

Transposable elements means they can move around.

 

These are kept in check by a thing called DNA methylation, if your DNA

isn't properly methylated with the right nutrients then the

transposons can move around.

 

The transposons include 8% retroviruses and one of them is called

HERV-K and cross reacts with the HIV antibody test.

 

The main nutrient for methylation is a selenium compund called

S-adenosyl-methionine.

 

Folic acid and Choline as well.

 

When you look at studies on HIV and our own retroviruses (my opinion

is that HIV is normally in everyone's body) you find over and over

again that Hypomethylation (lack of methylation) makes the

retroviruses come out.

 

Or if you hypermethylate (over-methylate) they get suppressed.

 

" These results suggest that DNA hypermethylation of the HIV LTR may

change the binding characteristics between LTR sequences and cellular

proteins, thereby suppressing HIV LTR transcription and modulating

viral expression. "

Inactivation of the HIV LTR by DNA CpG methylation: evidence for a

role in latency

http://embojournal.npgjournals.com/cgi/content/abstract/9/4/1157

 

These studies show this phenomenon.

 

" Several cytidine analogs with known mutagenic capability were tested

for their effects on DNA methylation and on induction of endogenous

murine retrovirus. For each of the compounds tested it was found that

DNA methylation was inhibited at the same concentrations that were

required to induce virus expression. With each compound it was

observed that increased dose levels produced an increase in the

ability to inhibit methylation and an increase in the ability to

induce virus. "

Effects of cytidine analogs on methylation of DNA and retrovirus

induction.

CpG Methylation Directly Regulates Transcriptional Activity of the

Human Endogenous Retrovirus Family HERV-K(HML-2)

 

The problem is that all illness including cancer, AIDS and the

autoimmune diseases have an increase in retroviral expression so how

can we tell if HIV is a transmitted retrovirus when all our own

retroviruses come out when we are ill?

 

HERV-W, another of our retroviruses is also involved in pregnancy and

this is why pregnant women in Africa are at risk from the HIV test.

 

Here it turns up in cancer but only because our DNA is hypomethylating.

 

L1 and HERV-W retrotransposons are hypomethylated in human ovarian

carcinomas

 

This is one of the reasons why Glutathione a Selenium compound helps

with HIV and cancer.

 

It is also cell protective.

 

" Intracellular levels of glutathione are depleted in patients with

acquired immunodeficiency syndrome in whom the risk of tuberculosis,

particularly disseminated disease is many times that of healthy

individuals. In this study, we examined the role of glutathione in

immunity against tuberculosis infection in samples derived from

healthy and human immunodeficiency virus infected subjects. Our

studies confirm that glutathione levels are reduced in peripheral

blood mononuclear cells and in red blood cells isolated from human

immunodeficiency virus-infected subjects (CD4>400/cumm). Furthermore,

treatment of blood cultures from human immunodeficiency virus infected

subjects with N-acetyl cysteine, a glutathione precursor, caused

improved control of intracellular M. tuberculosis infection. "

Glutathione and growth inhibition of Mycobacterium tuberculosis in

healthy and HIV infected subjects.

 

" In AIDS patients, chronic inflammation and elevated levels of

cytokines seem to be associated with lower levels of GSH: GSH levels

decrease rapidly upon infection with HIV and continue to decline as

the disease progresses. Investigators have shown that agents that

increase intracellular levels of GSH inhibit HIV replication. "

Potential role of reduced glutathione as an antiviral agent

 

Now they knew as far back as 1991 that Glutathione stopped HIV but in

this study they also mentioned AZT causes Glutathione depletion, this

means AZT causes HIV expression and AIDS in the long run.

 

AZT only seems to work in the short term because it hypermethylates DNA.

 

" Azidothymidine causes functional and structural destruction of

mitochondria, glutathione deficiency "

Suppression of Human Immunodeficiency Virus Expression in Chronically

Infected Monocytic Cells by Glutathione, Glutathione Ester, and

N-Acetylcysteine 1991

 

Cal Crilly

- - -

 

 

 

posted by Sepp Hasslberger on Thursday May 18 2006

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