Sutherlandia OPC and Sodium Dichloroacetate (DCA) Flu Vac

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In a message dated 8/4/2009 7:09:58 P.M. Pacific Daylight Time, mikeleekc01 writes:

Thanks Marc for your detailed explanation.I may not understand fully the intricacies of celluar biology you have explained but I am taking her off DCA right away.oleander soup , "Marc Swanepoel - PhD" <marcswan wrote:>> Being on DCA (sodium dichloroacetate) treatment and using Sutherlandia OPC (with the associated protocol), could be counterproductive for the reasons below. > > > > All cells require energy in the form of Adenosine Triphosphate (ATP). This substance cannot be stored in the cells and at any stage we have enough to last for about 3 to 5 seconds. It is thus a continuous process. A few billion years ago, before there was oxygen in the atmosphere, primitive cells (eukaryotes) evolved the process of producing ATP energy without the use of oxygen by a process of glycolysis. Later on in the evolutionary process, when oxygen became part of the atmosphere, certain types of bacteria evolved a much more efficient way of producing ATP by utilizing oxygen through a process known as oxidative phosphorylation (OXPHOS). This new process was roughly 20 times more efficient than the glycolysis process. Roughly two billion years ago, these bacteria and the original eukaryotes fused into a symbiotically functioning and more complex cell system. Researchers now acknowledge that the bacterial component of cells is the cellular mitochondria. All cells, with the exclusion of red blood cells, can have thousands of the mitochondria. These new "symbionts" can switch between the two systems of ATP production, depending on the demands of the cells. During the fetal stage and repair of cell damage, it switches to the ATP production from glucose in the cytoplasm. During the cell differentiation stage, it switches to the OXPHOS system of oxidative ATP production in the cell mitochondria. The switching system itself is controlled by an intricate signaling system that depends on the permeability of the mitochondrial membrane and its influence on the Ca 2+ cycle, the electrical charge across the mitochondrial membrane and other factors. All these factors, in turn, are modulated by the so-called thiol pool of which glutathione is the most important component.> > When body cells are under oxidative or nitrosative stresses caused by environmental factors and/or life style, the thiol pool normally gets depleted and, with it, the ability of the mitochondria to produce ATP through the OXPHOS system. As a defensive measure, the mitochondrial switch puts the cells into the more primitive way of survival by reverting to the much less efficient enzymatic production of ATP in the cytoplasm. If the aforementioned stresses are chronic, the switch remains in this position and we call the condition "cancer" - identified by the undifferentiated division of cells.> > If the cut-off of ATP production by the OXPHOS system is sudden, the cells will die and the process is known as 'necrosis' or sudden cell death. If it is more gradual, the affected cells can 'commit suicide', a process know as 'apoptosis'. Treatments like the acetogenins in Paw-Paw extract, Graviola and similar plants, Cantron, Protocel, and DCA (sodium dichloroacetate) have the effect of reducing the ATP and/or glutathione levels in all cells and it is then hoped that the described 'apoptosis' of cancer cells will take place. Because of the fact that the ATP production of ALL cells is reduced, one can feel very tired when using these supplements.> > Another very important fact is that the apoptosis of cancer cells will only take place if the mitochondrial membrane is still permeable and the CA 2+ cycle is still working. In all advanced cancers, this permeability is blocked as part of the defense mechanism of cells and apoptosis CANNOT take place, however much Paw-Paw extract or any ATP/Glutathione reducing supplement one takes. One can thus see that the treatment will work in some cases where the cancer is at a stage where the mitochondrial switch is still fluctuating between the two methods of ATP production. This 'window of opportunity' is relatively short and when metastases have appeared, it is normally a sign that the mitochondrial membranes are blocked. The only alternative is then to reactivate the mitochondrial switch by getting rid of the oxidative and nitrosative stresses, and by topping up the depleted glutathione. Nothing else will work.> > > > The danger in using substances that will reduce ATP and/or glutathione is twofold. As the reduction of ATP and/or glutathione is not restricted to cancer cells it can (a) result in cells that are not yet cancerous becoming cancerous and (b) result in fungal and bacterial overgrowth that can be dangerous for weakened patients. If one looks up the known side-effects of some of the pharmaceutical agents used for this purposes, one will see that they are all associated with a reduction in energy and the inability of cells to defend themselves against internal pathogens (fatigue, rash, infection, mouth sores, etc).> > > > So, if one uses the S/OPC and the suggested NAC, then this will work against the intended action of the Sodium Dichloroacetate.> > > > Marc Swanepoel> > > - > Michael LEE > oleander soup > Tuesday, August 04, 2009 5:33 PM> Sutherlandia OPC and Sodium Dichloroacetate (DCA)> > > Hi everybody,> My dear wife is suffering from advanced pancreatic cancer.> I just received my order of Sutherlandia OPC today and I immediately put her on it > My wife is already on DCA (sodium dichloroacetate) and hydrogen peroxide therapy for her illness.> You can find info about DCA, from http://www.thedcasite.com/ > Does anyone know whether these 2 therapies are compatible with the Sutherlandia OPC treatment?> > Thanks> -- > > Best Regards,> Michael Lee>---