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Acid Horse and Bupleurum

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Just FYI at this point:

T & T is Transportation and Transformation-

 

 

Thanks - I just about remember all the functions, but I didn't recognise your

abbreviation!

 

> Ya, it is hard to interpret the behavior of a horse- unless maybe you're

> a " horse whisperer " ? ;-)

 

 

LOL, I do all that stuff - and it's still hard!!

 

>

> Another solution to excess acidity after eating is

> Transformation Enzymes' Gastro product. Not sure

> what the cost of dosing a horse would be though. How

> much more do they weigh than humans?

 

 

Just a little...670kg! Trouble is because they produce acid 24/7, you'd have

to treat every 2 hrs to really have an effect with some things. Horses don't

have a gallbladder you see, they just pump out acid all the time, so their

protection mechanisms are vital.

 

>

> As for chaihu and prostaglandins-

>

> The NSAIDS (ibuprofen, etc.) interrupt the COX-2 pathway.

 

 

Yes, the end result of which is the inhibition of prostaglandins apparently:

 

" Because of its involvement in inflammation, [prostaglandin E2] PGE2 is the

key indicator of non-steroidal anti-inflammatory drugs efficacy via

inhibition of COX-1 and COX-2 "

 

>

> The following MEDLINE references are about a different form of bupleurum but

> may shed some light-

 

 

That's one problem - I fell foul of this with the info that poria cocus has a

direct sedative effect - Fu Ling does not Michael said, it is the centre of

the fungus which has a different chinese name that does!

 

> There were also 4 references on Bupl. scorzonerifolium, the specific kind

> that is chai hu... only phytochemical info, nothing about its effects on

> humans.

 

 

But if you then look up the active constituent:

 

 

Antagonism of saikosaponin-induced prostaglandin E2 release by baicalein in

C6 rat glioma cells.

 

There are several Kampo medicines (Chinese herbal medicines) containing both

Bupleuri Radix and Scutellariae Radix, which are used for the treatment of

inflammation. Saikosaponins are derived from Bupleuri Radix, and baicalein is

from Scutellariae Radix. The present study was undertaken to investigate the

pharmacological interaction of saikosaponin b1 and baicalein in prostaglandin

E2 (PGE2) release from C6 rat glioma cells in vitro. Saikosaponin a, b1 and d

potently stimulated PGE2 release, while saikosaponin b2 and c moderately

stimulated PGE2 release.

 

 

>

> So I wonder where you heard that about chai hu, and what that person's

>

 

I just searched under bupleurum on Google, and if you search under

'bupleurum prostaglandins' many references come up. Maybe they are all wrong?

 

One paper says: " The medicinal species of Bupleurum in Shanxi, Gansu,

Ningxia, Qinghai provinces and Xingjiang Autonornous Region have been

investigated. It is shown that there are twenty one medicinal species in the

genus, of which nine are main stream species "

 

Another:

 

[Dynamic variation of saikosaponin contents]

 

Du X, Jiang C, Liu M.

 

Pharmacy Department, Heilongjiang College of Traditional ,

Harbin.

 

In this paper both the cultivated and wild Bupleurum chinense and B.

scorzonerifolium from northeast China of different growing periods and ages

were analyzed for the contents of saikosaponins a and d by HPLC and total

saikosaponins by spectrophotometry. The results indicated that the

saikosaponin content of B. chinense was higher than that of B.

scorzonerifolium, and that the saikosaponin contents of the cultivated

species were higher than or almost equal to those of the wild ones.

 

So it seems all the commonly used Bupleurums used as Chai Hu have

saikosaponins, which have a positive effect, as does a polysacharride in

bupleurum fulcatum at least - who knows if that is in the others. The fly in

the ointment is this extract in one paper on saikosaponins:

 

" The effects of these compounds on swelling

and other inflammatory parameters are described. In screening for in vitro

effects of saikosaponins on cellular systems generating cyclooxygenase (COX)

and lipoxygenase (LOX) metabolites, we observed that most saikosaponins

showed a significant effect. The action is more marked on LOX metabolite

LTC4. Our data support the inhibition of arachidonic acid metabolism as one

of the biochemical mechanisms that might be the rationale for the putative

antiphlogistic activity of these saikosaponins. "

 

I can't translate this and am not sure what the end result is - but where did

the prostglandin reference come from? It is possible the saponins have both

properties but who knows which is more potent? If there is another herb that

has the advantages without the disadvantages, I'd rather use it.

 

I don't know what to think really - I'll ask Michael if he knows for sure the

effect of the chai hu he buys on folk with gastric ulcers, or if he can

prescribe an alternative if there is any doubt. But horses may need the

protaglandin protection more than people because of their constant production

of acid. I'm not sure he welcomes such questioning - but I have to after 3

prescriptions (counting the first two which came from the TCM vet) all of

which made the horse infinately worse!

 

Thanks again,

 

Jackie

 

 

 

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Jackie wrote:

> Yes the last formula addressed Spleen Qi and food stagnation well, but

> without liver support - but what is T & T??

 

Just FYI at this point:

T & T is Transportation and Transformation-

The Stomach 'rots and ripens'/decomposes 'grain

and water'/food. The Spleen then extracts the

essence of food and dispatches it to the other organs...

this equals digestion, assimilation and distribution of

nutrients (this is from Wiseman/Ellis' Fundamentals of

.)

 

> So we tested and I'm pretty sure I've confirmed that - there was a marked

> improvement on the addition of a LOT of antacids.

 

Ya, it is hard to interpret the behavior of a horse- unless maybe you're

a " horse whisperer " ? ;-)

 

Another solution to excess acidity after eating is

Transformation Enzymes' Gastro product. Not sure

what the cost of dosing a horse would be though. How

much more do they weigh than humans?

 

As for chaihu and prostaglandins-

 

The NSAIDS (ibuprofen, etc.) interrupt the COX-2 pathway. That is not

the only way to reduce inflammation. E.g. steroids like prednisone work

through another pathway.

 

Traditional sources say it may cause nausea and vomiting, CI in yin xu cough

or Liv fire ascending to head.

 

> I'm a little worried about using bupleurum because I have read it inhibits

> protaglandin production, which inhibiiton causes gastric ulcers (that's

why

> NSAID's cause them). I don't know if anyone knows if that is so??

 

The following MEDLINE references are about a different form of bupleurum but

may

shed some light-

 

searched on " bupleurum prostaglandin " , " bupleurum cox " , " bupleurum stomach "

 

this one talks about anti-inflammatory actions,

it does not talk about the pathway it uses- does not mention COX-2

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_ui

ds=9763210 & dopt=Abstract

 

These four talk about Bupl. healing gastric ulcers... the opposite effect

you mentioned.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_ui

ds=1682444 & dopt=Abstract

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_ui

ds=11807976 & dopt=Abstract

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_ui

ds=8103101 & dopt=Abstract

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_ui

ds=5389580 & dopt=Abstract

 

There were also 4 references on Bupl. scorzonerifolium, the specific kind

that is chai hu... only phytochemical info, nothing about its effects on

humans.

 

So I wonder where you heard that about chai hu, and what that person's

references were?

 

B

 

 

 

Brian Benjamin Carter

Editor, The Pulse of Oriental Medicine

Columnist, Acupuncture Today

 

The PULSE of Oriental Medicine:

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