Guest guest Posted April 17, 2005 Report Share Posted April 17, 2005 My 6 year old daughter has molluscom. She contracted it last June. Since then we've been through numerous treatments of " beetle juice " which, ultimately, were ineffective and extremely painful. This fall, her pediatrician suggested a treatment using Tagamet. She takes it orally, once a day. She has been taking it since November and still, we have had no success. On top of everything, it has continued to spread. Can anyone out there reccommend or suggest anything? I have found several treatments on the web, but would love to hear from someone who has actually had success with something. Thanks in advance, A Mom on the Edge Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 18, 2005 Report Share Posted April 18, 2005 Hello " kaetidaniel " , I had a friend who's children had this. It was epidemic in their school system here in Katy, outside of Houston.... Most all the children at the elementary school have it... and pass it around. This is the information I collected for her while trying to find some help for this little known situation. She ended up using the homeopathic remedies listed below, and topically used Oregano essential oil in olive oil and a bandade.... Eventually, they disappeared. She has three children, and all three had this. All three are free of it. Most MD's had no experience with it, and actually recommended a antacid for a venereal " type " skin disease!!! Absurd! One doctor DID recognize it, and kicked this lady out of her office saying it was extremely contagious, handing her a prescription that did nothing.... So, here's what I collected. Hope it is helpful. Jennifer There's not much information on this... I think I counted like 10 hits! That's amazing! I've inquired on two professional lists, and it may take a couple days. Don't know if it's similar to Herpes or not? If you read down to the bottom by the Janine Boyd on Wednesday, February 12, 2003, she suggests Showering, not bathing, and she said in both her children, it took 6 months to go away. But, it also says in other posts, that it comes and goes, popping up later. So far, These are the responses that have been reported. It seems very common in Europe, probably because of their bath houses. It seems that swimming pools, bath tubs, etc... are really a problem, and may contaminate even between children if bath tub isn't sterilized with bleach between different children's baths. One site called it a water blister.... It seems to spread with water, possibly sweat, etc. Some diseases, a entire family, class, etc... has to be treated if the treatment is able to work to it's fullest potential. This " may " be one of them. ---- Original Message ----- " Patricia Hatherly " <triciah <minutus > Sunday, October 24, 2004 10:32 PM Re: [Minutus] Digest Number 1761: Molluscum contagiosum Like most of you I see the obvious sycosis connection with my little patients who present with mollusca. For what it's worth Sil comes up often which underlines the possible NWS vaccination aetiology. However, lately I've been expediting matters in those situations where mothers insist on putting something on them by suggesting their breast milk (if they are feeding) or some Lac-h 0/1 or Lac-h 7C if I've hand-succussed some of that mother's milk at some earlier time. (It's something I routinely do with all my lactating mothers; why?? looong story; you'll have to read my book for the answer to that and how I hand-succuss). That aside; this management strategy is based on a study that was published in The New England Journal of Medicine vol 350 (26) 2663-2672 June 24 2004 entitled Treatment of Skin Papillomas with Topical a-Lactalbumin-Oleic Acid by Gustafsson L et al. This group has isolated a tumour-necrosing factor in a-lactalbumin (the main protein in human milk) and has called it Hamlet [because it was discovered by that famous castle] and is an acronymn for human alpha-lactalbumin made lethal to tumour cells. What the researchers have found is that when applied to viral papilloma the human milk kills the cells. This was tried following the original use against cancer cells and bacteria. Apparently when applied to cancer cells the alpha-lactalbumin reconfigures itself by binding to oleic acid and creates the more powerful Hamlet compound. There is, of course, mention in the various news reports of marketing this new " discovery " for topical application but " expensive " is also mentioned. Human milk is cheap enough and (of course) does the trick all by itself and, to my clinical observation, Lac-h in physiological doses does the same thing. Those who use Lac-h often will observe that there's a strong tendency for that Rx to throw warts to the skin. Mueller has written much of this in his books. regards Patricia PS check out: http://content.nejm.org/cgi/content/short/350/26/2663 Therese said: > It does look sycotic and I have used Thuja, silica, sulphur and, yes, > medorrhinum 200 weekly - " Julian Winston " <jwinston <minutus > Sunday, October 24, 2004 9:43 AM Re: [Minutus] Molluscum contagiosum I do believe that in a recent issue of Homeopathy NewZ (the mag I edit in NZ) that Bruce Barwell from Auckland said he found the nodode of Molluscum contagiosum useful in the treatment. >Silica does seem to be commonly indicated but not all my patients have been >vaccinated. Vaccine damage is often an inherited thing - I see rabies miasm symptoms in dogs/people who have never had the vaccine, but whose parents or grandparents have. And Silicea is such a commonly needed remedy anyway.... vaccines need not be a factor. Mary Marlowe -- marlowe Yes I have also had success with the nosode but only in 40.00 %, Med. has been excellent Health, Hope, Joy & Healing : May you Prosper, even as your Soul Prospers 3John 2 Jennifer Ruby Email advice is not a substitute for medical treatment. http://www.rubysemporium.com SymphonicHealth ____________ «¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤ ¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯¯ - " kaetidaniel " <kaetidaniel Sunday, April 17, 2005 11:47 AM Molluscom > My 6 year old daughter has molluscom. She contracted it last June. > Since then we've been through numerous treatments of " beetle juice " > which, ultimately, were ineffective and extremely painful. This fall, > her pediatrician suggested a treatment using Tagamet. She takes it > orally, once a day. She has been taking it since November and still, > we have had no success. On top of everything, it has continued to > spread. Can anyone out there reccommend or suggest anything? I have > found several treatments on the web, but would love to hear from > someone who has actually had success with something. > Thanks in advance, > A Mom on the Edge Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 18, 2005 Report Share Posted April 18, 2005 I spent four years in a clinical study, in regard to effective treatment for the properly diagnosed Molluscum contagiosum virus. It is often misdiagnosed. The red pussles have a black volcano dip center. At its conclusion two herbs assisted in possible clearance. The herbs Yarrow and Yucca when taken orally as well as applied midly abbrasively to a 2' x 2 " patch on the skin initiated clearance within 3 days of 65% patients. These patients in a double blind study including adult and children with NO reported side effects. I am not a Medical physician. I am a Molecular Biologist, the principal researcher in this study monitored which was facilitated by group of 3 Dermatologists. In the worst case it will usually 99% suddenly clear on its own if left untreated within 8 weeks. All during this time it is highly contagious to susceptible individuals surviving even in bromalaided or chlorinated pools. Be Well, Dr. Gregory Keyock, Ph.D. Principal Research Director Antiviral Research Institute (AVRI) Xigent Technologies Consortium™®© www.X-igent.com kaetidaniel <kaetidaniel wrote: My 6 year old daughter has molluscom. She contracted it last June. Since then we've been through numerous treatments of " beetle juice " which, ultimately, were ineffective and extremely painful. This fall, her pediatrician suggested a treatment using Tagamet. She takes it orally, once a day. She has been taking it since November and still, we have had no success. On top of everything, it has continued to spread. Can anyone out there reccommend or suggest anything? I have found several treatments on the web, but would love to hear from someone who has actually had success with something. Thanks in advance, A Mom on the Edge Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 18, 2005 Report Share Posted April 18, 2005 dr.b_helthi wrote: Hello Kaetidaniel Mom, the results of any treatment of an otherwise, average-healthy, six-yr-old child can be evaluated at the end of 14 days. 1. If it is working, a discernable amelioration of the condition will occur. If nothing else, its spread will either slow down, or stop. 2. If it is not working, no change -or a worsening- is discernable. 3. If no change is discernable, the medication(s) aren't working, and 4. The meds should be stopped, immediately or sooner. ? Tagamet for mollucum in a 6-yr-old, female child ? For info about a preparation that works, goto: http://www.molluscum-contagiosum.net/ ============================ kaetidaniel wrote: My 6 year old daughter has molluscom. She contracted it last June. Since then we've been through numerous treatments of " beetle juice " which, ultimately, were ineffective and extremely painful. This fall, her pediatrician suggested a treatment using Tagamet. She takes it orally, once a day. She has been taking it since November and still, we have had no success. On top of everything, it has continued to spread. Can anyone out there reccommend or suggest anything? I have found several treatments on the web, but would love to hear from someone who has actually had success with something. Thanks in advance, A Mom on the Edge Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 20, 2005 Report Share Posted April 20, 2005 Molluscum Contagiosum Synopsis Gregory Keyock Graduate School Department of Molecular Biology Lehigh University FALL, 1980-1990 Bethlehem Pa. Molluscum contagiosum (MC) infection is found throughout the world. MC virus is specifically a human disease and any experiments to infect other animals have failed. MC infection is described as a harmless, though unsightly, skin infection. The presence may be an indication of other coincidental venerial infections, although MC is not necessarily linked to sexually transmitted diseases. There are only a few reports of fatigue associated with the MC skin infection and any associations have not been substantiated. MC is caused by a pox-virus and is not serologically related to other pox-viruses. MC infection is confined to the skin and propagates by localized topical spread. The virus is not implicated as causing infection in or being transmitted through the blood. Data on MC infection clearly indicates a dramatic increase in the U.S. and Britian since 1981. The increase in MC infection parallels the AIDS epidemic and AIDS or immunodeficient patients are at higher risk for acquiring the MC infection as well as spreading the virus. Molluscum contagiosum was first described by Thomas Bateman in 1817. Very little molecular characterization of the virus has been made on this virus since that time. Molluscum Contagiosum virus can be found in swimming pools, health spas, tanning beds or the floors and mats of aerobic excercise rooms. The virus found on floors, carpets, tables and objects is typically inactivated and destoyed by strong cleansing agents. Transmission of the virus is mainly mechanical requiring close contact of the skin. Lesions can be anywhere on the skin although they are found to be mostly clustered or localized. The MC virus can also be transmitted from an infected host to an unwary individual by way of an object. The MC virus can remain active in a protective coat for up to two days. Uninfected individuals can acquire the MC virus infection through subsequent contact with the affected site of an object. Molluscum Contagiosum sores are composed of skin (epidermal) tissue whose infected cells are either localized or found on the genitals. Infected cells are both increased in size and in number. The MCV tumor is benign and is a visible pearly, flesh colored nodule about 2 to 5 mm in diameter. The MCV tumors extend from the dermis basement and project out, above the immediate layers of the skin. Adjacent and parallel to the basement membrane is a varying thickness band of stromal proliferation composed of loose collagen bundles and fibrocytes along with almost a complete lack of elastic tissue fibers. Superficial layers of the tumor stained with alcian blue at ph 4.5 indicated no melanin pigment granules within the keratinocytes of the involved acanthotic epidermis. MCV sores are frequently misdiagnosed as acne. The sores of MCV infection can cover an area from about 2 inches up to large or entire regions of the body. MC infection on the palms or soles is rare; however, a few cases have been documented. MCV lesions have a characteristic dimple at its summit which appears as an opening through which a small white core can be seen. Simple, quick and inexpensive 'in office' procedures exists which allow proper diagnosis of molluscum contagiosum. One procedure involves the use of a topical skin stain and a magnifying glass to confirm the presence of the characteristic core found in the center of the sore. A second procedure involves a minimal incision of the papule and extraction of the infectious core. The pearly, flesh colored tissue is squashed between a slide and cover slip with a drop of Sedi-stain. The slide is visualized under a common light microscope. The field is described at 850x as a projection of dark particles from an amorphous compressed lesion. The dark particles are the molluscum virions. One of three major areas of research being initiated at Lehigh is the pursuit of a possible vaccine development as well as holistic topical creams that retard the spread of viral sores and infection. Current treatment however, can include phenol, electrocautery, cryopathy, trichloroacetic acid, podophylum, and inosoplex. Inosoplex is termed an immunomodulator which solicits host cell defense. Inosoplex is helpful in moderately immunosuppressed individuals and AIDS patients prior to final stages of the disease. It has been suggested that for the general cases involving children the infection should go untreated and allowed to heal on its own. MCV infected cells are larger than normal. The infected cell cytoplasm is swollen with a large hyaline acidophilic granular mass characteristic of the MC infection and termed the " Molluscum body " . The molluscum body consists of a spongy matrix divided into cavities containing clustered masses of viral particles. The core of the MCV sore is made of degenerating epidermal cells and inclusion bodies (molluscum bodies). Keratin is found in the core and is being produced by both infected and uninfected cells. The cytoplasmic inclusion bodies correspond with the B-type inclusions known as 'viral factories' charactersitic of pox-viruses. Infected cells containing inclusion bodies, in essence, become a sack of virus particles. The incubation period for molluscum contagiosum has been determined to be 14 to 50 days. The disease is chronic which means after the initial infection has cleared additional reinfections may occur. Recurrence of infectious manifestations is frequent and can be due to reinfection, poor hygeine, as well as typical chronic resurfacing. MCV lesions tend to heal spontaneously, a phenomenon known as spontaneous disappearance. Trauma or bacterial infection may initiate spontaneous disappearance giving rise to certain courses of treatment. In the course of spontaneous disappearance a usually intense inflammation develops around several lesions. Biopsy specimens have been studied at various stages in the course of the inflammation. A dense infiltrate of lymphocytes, large mononuclear cells, histiocytes and cells with basophilic and vacuolar cytoplasm were found surrounding and engulfing infected cells. These findings support that the regression of the lesions is most likely mediated by a cellular immune response. Histiological studies confirmed a cell mediated rejection reaction. There have been no reports of successful culturing of human skin tissue infected with the virus which has made laboratory research on the virus difficult. At Lehigh efforts to determine a successful culture system are being initiated. This will be another one of three major efforts in the study of this virus. Lack of molecular characterization of this virus is mostly due to the absence of large quantities of viral progeny. Without human skin tissue culture which will support the production of viral particles more intense investigations on the virus cannot readily be pursued. MC virions can be readily extracted from lesions. Most virions found in the molluscum bodies, however, are inactivated. The MC virus genome is single stranded DNA with a molecular weight of 118MDa. The viral genome is approximately 178 kilobase pairs. The virus also contains a virus-specified DNA-dependent RNA polymerase and at least 40 other associated polypeptides as determined by SDS polyacrylamide gel using a highly sensitive silver stain technique. Another major area of research to concomitantly begin at Lehigh is a more detailed analysis and characterization of proteins of the virus and its surrounding tumor structure. The molluscum contagiosum virus has the distinction of being the largest human virus and is visible under the light microscope allowing it to be uniquely visible to the clinician. The virus particle is brick shaped and measures about 0.3 x 0.2 x 0.1 um. Restriction endonuclease analysis has revealed the existence of two major viral subtypes. Restriction endonucleases analysis has shown two different cleavage patterns corresponding to the two MCV types termed MVC type 1 and MCV type 2. It has also been determined from these studies that MCV type 1 was derived from lesions taken from the body in localized clusters whereas MCV type 2 has been taken from vaginal lesions. Several additional variations within each of the two major subtypes were found. These may correspond to various infectious potentials of the virus. The relationship of the immune system and its state is strongly linked to the MC viral infection. It is suspected that the virus remains active on the skin in molluscum bodies and thereby goes undetected by the immune system. The MCV infection does induce synthesis of interferon. In most normal cases MCV solicits nominal immunity. Infections with lesions occur from two weeks to two years and reinfection is commom. As previously indicated, trauma or agitation to the skin lesions has initiated spontaneous disappearance of the infection. It is believed that the trauma allows antibody-immune response to detect the hidden infection. Along with herpes virus HSV-1 and HSV-2 and the Epstein Barr Virus (EBV), AIDS patients are extremely susceptible to the molluscum contagiosum virus. Not only has the frequency of MCV infections increased among AIDS patients and the general public, but it is suspected that the AIDS virus and its genetic interaction with other viruses like HSV-1, HSV-2 and EBV may also play some role in the possibility of more contagious strains of molluscum contagiosum. More severe cases of MC such as 'disseminated MC' are suspected as being linked to a syndrome termed 'selective immunoglobulin M deficiency'. Other immunotype deficiencies include a total absence of Langerhans and indeterminant cells during molluscum contagiosum infection. Langerhans and indeterminant cells of the skin are dendritic cells that are involved in the initiation of the cutaneous contact hypersensitivity response and in allograft rejection. The exact role of Langerhans and indeterminant cells with respect to viral infection is poorly illucidated. Some patients sera indicate a strong anticellular staining with the IgM class of antibody which was confined to prickle cell, granular and keratin layers. It was also observed in the epidermis of normal skin cells immediately adjoining the lesion. IgM antibodies are implicated as being directed toward protein fibrils. The presence of IgM antibody suggests that there must be some exposure of contractile cellular antigens during viral growth. A substantial amount of research is still needed to make any significant progress in the understanding of the molecular and cellular biology of Molluscum Contagiosum. Research is being initiated at Lehigh in an effort to provide medical treatment toward this and possible other viral diseases. Quote Link to comment Share on other sites More sharing options...
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