Pituitary Hormones (PRL, GH, *TSH*, FSH, LH, ACTH Oxytocin)in Dairy Bioactive

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Pituitary Hormones (PRL, GH, *TSH*, FSH, LH, ACTH Oxytocin)

in Dairy Bioactive

JoAnn Guest

Jan 05, 2005 19:54 PST

======================================================================

The milk controversy has heated up as a result of the recent Canadian

rejection of MONSANTO'S application for the genetically engineered

growth hormone, rBST.

 

That hormone was NOT approved in Canada because HEALTH CANADA, Canada's

equivalent regulatory agency to America's FDA,

determined that rbST might prove to be unsafe for dairy cows.

 

Previous to the official denial, Canadian scientists noted that

laboratory

animals suffered a vast array of biological effects from this hormone,

and questioned the lack of integrity by FDA and MONSANTO who ignored

this TRUTH.

 

These data from the KEY study were clearly not reviewed by

FDA. The results of that study (Richard, Odaglia and Deslex, 1989) had

to have been known to MONSANTO and American regulators.

 

FDA now has information indicating that data were manipulated and

withheld from peer review.

 

Most critically important is an indication that IGF in milk is absorbed

intact, exerting powerful growth effects on

the human body.

 

Should there be a cancer present in a human system,

that too would be greatly influenced by this growth hormone.

 

A LIST OF HORMONES IN MILK

 

MILK, long thought to be a wholesome food for children, actually

contains powerful growth hormones.

 

Endocrinologist Clark Grosvenor published a review of the known hormones

and growth factors in milk ( " Hormones and Growth Factors in Milk, "

Endocrine Reviews, volume 14, number 6, 1992).

 

Each sip of cow's milk includes pituitary, hypothalamic, pancreatic,

thyroid, parathyroid,

adrenal, gonadal, and gut hormones.

 

The list does not include other important milk factors such as

prostaglandins and neuropeptides.

 

The milk in your morning cereal still appears to be white and pure, but

what follows is a list of " bioactive " substances in milk taken from

Table One

of Grosvenor's paper:

 

PITUITARY HORMONES (PRL, GH, *TSH*, FSH, LH, ACTH Oxytocin)

 

STEROID HORMONES (Estradiol, Estriol, Progesterone, Testosterone, 17-

Ketosteroids, Corticosterone, Vitamin D)

 

HYPOTHALAMIC HORMONES (TRH, LHRH, Somatostatin, PRL- inhibiting factor,

PRL- releasing factor, GnRH, GRH)

 

GASTROINTESTINAL PEPTIDES (Vasoactive intestinal peptide, Bombesin,

Cholecystokinin,Gastrin, Gastrin inhibitory peptide, Pancreatic

peptide,Y peptide, Substance P, Neurotensin)

 

 

 

IGF AND CANCER - THE SCIENCE

 

Critics of the Dairy Education Board claim that there is NO REAL SCIENCE

 

behind the IGF-I CANCER CLAIM. The actual science exploding that

contention appears in highly regarded scientific journals including the

American, Japanese, British, European and International Journals of

Cancer.

 

In addition, there are important papers published in the

Journal of Clinical Endocrinology, Journal of Cellular Physiology and

American Journal of Clinical Pathology.

 

The " smoking gun " is an autopsy

study that appeared in the New York Times. That study revealed that

cancers, particularly BREAST CANCERS, are actually quite common and

appear at a SHOCKINGLY young age.

 

CANCER AND IGF-I

 

IGF-I has been identified as an autocrine and endocrine growth regulator

 

that ACCELERATES various types of cancer. IGF-I is considered to play a

key role in the proliferation of pancreatic cancer cells, according to

Gillespie.

 

--------------------

(J. Gillespie, et al. " Inhibition of pancreatic cancer cell growth in

vitro by the tyrphostin group of tyrosine kinase inhibitors. " Academic

Surgical Unit, St. Mary's Hospital Medical School, Imperial College of

Science, Technology and Medicine, London, UK. Br.-J-Cancer, December,

1993, 68(6), pp. 1122-1126.)

--------------------

 

Glick noted that IGFs play a considerable role in the regulation of

glucose metabolism in central nervous system tumors.

 

--------------------

(R.P. Glick, et al. " Identification of insulin-like growth factor (IGF)

and glucose transporter-I and -3 mRNA in CNS tumors. " Department of

Neurology, University of Illinois at Chicago, Cook County Hospital.

Regul-Pept., October, 20,1993, 48(1-2), pp. 251-256.)

--------------------

 

Atiq reported that IGF is associated with human colorectal tumors and

colon cancer growth.

 

--------------------

(Atiq, et al. " Alterations in serum levels of insulin-like growth

factors and insulin-like growth-factor-binding proteins in patients with

 

colorectal cancer. " Labor d'Immunologie, Faculte de Medecin, Marseille,

France. Int-J-Cancer, May 15, 1994, 57(4), pp. 491-497.)

--------------------

 

Yashiro found that IGF-I activity was significantly higher in cancer

extracts, suggesting that higher IGF-I activity in cancer tissue is

involved in regulating growth of thyroid cancer cells.

 

--------------------

(T. Yashiro, et al. " Increased activity of insulin-like growth factor-

binding protein in human thyroid papillary cancer tissue. " Department of

 

Sirgery, Tsukuba University, Ibaraki, Japan. Jpn-J-Cancer-Res., January,

 

1994, 85(1), pp. 46-52.)

--------------------

 

Robbins found that IGF-I increased lymphocyte numbers in every lymphoid

organ examined.

 

This increase had functional significance, and this scientist concluded

that IGF-I produced locally by bone marrow cells was

a key component of " lymphatic " cancer.

 

--------------------

(K. Robbins, et al. " Immunological effects of insulin-like growth

factor-enhancement of immunoglobulin synthesized. " Department of

Immunology, Genetech, Inc., South San Francisco, California. Clin-Exp-

Immunol., February, 1994, 95(2), pp. 337-342.)

--------------------

 

Yun demonstrated that IGF hormones were 32-64 times more abundant in

Wilms tumors than in the adjacent uninvolved kidneys.

 

--------------------

(K. Yun, et al. " Insulin-like growth factor II messenger ribonucleic

acid expression in Wilms tumor, nephrogenic rest, and kidney. "

Department of Pathology, University of Otago Medical School, Dunedin,

New Zealand. Lab-Invest., November, 1993, 69(5), pp. 603-615.)

--------------------

 

Minniti concluded that insulin-like growth factor (IGF-I) acts as an

autocrine growth and motility factor in human sarcoma cell lines.

 

Analyses of tumor biopsy specimens demonstrate high levels of IGF RNA

expression. All tumor specimens examined expressed the gene for IGF,

and this expression was localized to the tumor cells.

 

--------------------

(C.P. Minniti, et al. " Specific expression of insulin-like growth

factor-II in rhabdomyosarcoma tumor cells. " Institutes of Health,

Bethesda, Maryland. Am-J-Clin-Pathol., February, 1994, 101(2), pp. 198-

203.)

--------------------

 

Developing childhood bone cancers were researched by Kappel who wrote

that this type of cancer typically occurs during adolescent growth

spurts when growth hormone and insulin-like growth factor-I (IGF-I) may

be at their highest lifetime levels.

 

He noted that human bone cancer

cell lines are dependent on signaling through IGF-I receptors for

survival and proliferation.

 

--------------------

(C.C. Kappel, et al. " Human osteosarcoma cell lines are dependent on

insulin-like growth factor I for in vitro growth. " Molecular Oncology

Section, National Cancer Institute, Bethesda, Maryland. Cancer-Res., May

 

15, 1994, 54(10), pp. 2803-2807.)

--------------------

 

Lippman, as early as 1991, had implicated IGF-I as being critically

involved in the aberrant growth of human breast cancer cells.

 

--------------------

(M. Lippman. " Growth factors, receptors and breast cancers. " (IGF-I and

related growth factors are critically involved in aberrant growth of

human breast cancer cells.) J. Natl. Inst. Health Res., 1991, 3, pp. 59-

 

62.)

--------------------

 

Lee observed the processing of insulin-like growth factor by human

breast tissue and indicated that estrogen regulation of IGF-I in breast

cancer cells would support the hypothesis that IGF-I has a regulatory

function in breast cancer.

 

--------------------

(A.V. Lee, et al. " Processing of insulin-like growth factor-II (IGF-II)

by human breast cancer cells. " Imperial Cancer Research Fund, Breast

Biology Group, School of Biological Sciences, University of Surrey,

Guildford, UK. Mol-Cell- Endocrinol., March, 99(2), pp. 211-220.)

--------------------

 

Chen noted that IGFs are potent growth factors for cellular

proliferation in the human breast carcinoma cell line.

 

--------------------

(J.C. Chen, et al. " Insulin-like growth factor-binding protein

enhancement of insulin-like growth factor-I (IGF-I)-mediated DNA

synthesis and IGF-I binding in human breast carcinoma cell line. "

Department of Medicine, University of Maryland School of Medicine,

Baltimore, Maryland. J-Cell-Physiol., January, 1994, 158(1), pp. 69-78.)

 

 

--------------------

 

Figueroa confirmed that insulin-like growth factors (IGFs) are key

factors for breast cancer growth.

 

--------------------

(J.A. Figueroa, et al. " Recombinant insulin-like growth factor binding

protein-1 inhibits IGF-I serum, and estrogen-dependent growth of MCF-7

human breast cancer cells. " Department of Medicine, University of Texas

Health Science Center, San Antonio, Texas. J-Cell-Physiol., November,

1993, 157(2), pp. 229- 236.)

--------------------

 

Li treated breast cancer cells with IGF-I and observed a 10-fold

increase in RNA levels of cancer cells and concluded that IGF-I appears

to be an important step in cellular proliferation.

 

--------------------

(X.S. Li, et al. " Retinoic acid inhibition of insulin-like growth factor

 

I stimulation of c-fos mRNA levels in breast carcinoma cell lines. "

Department of Medicine, University of Maryland School of Medicine,

Baltimore, Maryland. Exp-Cell-Res., March, 1994, 211(1), pp. 68-73.)

--------------------

 

Krasnick furnishes another clue to this puzzle by revealing that IGF-I

plays a role in the regulation of human ovarian cancer.

His data

suggest that IGF-I and estrogen interact in a synergistic manner and

regulate the growth of cancer.

 

--------------------

(A. Krasnick, et al. " Insulin-like growth factor-I (IGF) and IGF-binding

 

protein-2 are increased in cyst fluids of epithelial ovarian cancer. "

Institute of Endocrinolgy, Chaim Sheba Medical Center, Tel-Hashomer,

Israel. J-Clin-Endocrinol-Metab, February, 1994, 78(2).)

--------------------

 

Musgrove states that growth factors play a major role in human breast

cancer cell growth.

 

--------------------

(E.A. Musgrove, et al. " Acute effects of growth factors on T-47D breast

cancer cell cycle progression. " Cancer Biology Division, Garvan

Institute for Medical Research, St. Vincent's Hospital, Darlinghurst,

NSW, Australia. Eur-J-Cancer, 29A (16), 1993, pp. 2273-2279.)

--------------------

 

THE MISSING LINK

 

On November 8, 1994, the New York Times published a story which revealed

two very critical facts.

 

(Gina Kolata, New York Times, Science Section, Nov. 8, 1994, p. C1.)

 

Although only 1 percent of women between the ages of 40

and 50 are diagnosed with breast cancer, autopsy studies reveal that 39

percent of women in that age group have breast cancer.

 

Although only 1 percent of men between the ages of 60 and 70 are

clinically diagnosed with prostate cancer, 46 percent actually have

prostate gland tumors. Gina Kolata. " New ability Find Earliest

Cancers: A Mixed Blessing? "

 

SUMMARY

 

IGF-I is the key factor in cancer's growth in the human body. IGF-I is

identical between humans and cows. Milk is a hormonal delivery system

and IGF-I is orally active, surviving digestive processes.

 

If you believe that breastfeeding mothers deliver substances to their

infants,

you then should understand that milk is a hormonal delivery system.

 

Lactoferins, immunoglobulins and protein hormones survive digestion and

exert powerful growth effects. Cancer is very common, but usually

controlled by the human body.

 

Cancerous tumors are the ones that have

somehow thrown off the usually tight genetic controls on unwanted

growth.

 

Robert Cohen 201-871-5871

_________________

 

JoAnn Guest

mrsjo-

DietaryTi-

www.geocities.com/mrsjoguest/Genes

 

 

 

 

 

AIM Barleygreen

" Wisdom of the Past, Food of the Future "

 

http://www.geocities.com/mrsjoguest/Diets.html

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

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