YOU CAN MAKE SOMEONE SCHIZOPHRENIC - it's easy.(Karl)

[Guest guest]

I am not sure Frog. Hopefully Karl (madcowcoverup) sees this and can answer better than I. He is one of the smartest guys I know - good on this kind of stuff.

Kathy

 

 

 

 

 

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Frog

05/13/05 02:56:39

 

Re: YOU CAN MAKE SOMEONE "SCHIZOPHRENIC" - it's easy.

Kathy:I have read this article only partially but I am very interested. My exhusband appears to suffer from schizophrenia although he has never beendiagnosed. I will have to read up on my chemistry, he would sweat out asmell that was like vinegar before he headed into an unstable state. I havetalked to another spouse of a schizophrenic and she also noted that vinegarsmell in his perspiration before he had periods of unstability. Is vinegarsimilar to what they talk about in this article?I have had the gut feeling that his disease was not from natural causes.But it has just been a hunch.Frog- "Kathy" <vanokatThursday, May 12, 2005 8:46 AM YOU CAN MAKE SOMEONE "SCHIZOPHRENIC" - it'seasy.YOU CAN MAKE SOMEONE "SCHIZOPHRENIC" - it's easy. Apparently there has beenthe intentional administration of taraxein into humans! It appears it doesNOT naturally occur- even in those diagnosed or labeled as 'schizophrenic'-it does make you wonder how many people are being GIVEN 'schizophrenia'through experiments?--"Cheyenne Cin"- P.S. Note that I have not looked atthe linked info with the information below.The GOOD NEWS is that if it's from severely reduced serotonin levels and weKNOW THAT, we can FIX IT! (Hopefully) -- I am SOOO suspicious, aren't I? Gofig!=======================================Administration of taraxein in humans.. Administration of taraxein in humans. HEATH RG, COHEN SB, SILVA F, LEACHBE,COHEN M. PMID: 13652807 [PubMed - OLDMEDLINE for Pre1966]www.ncbi.nlm.nih.gov/entrez/queryfcgi?cmd=Retrieve & db=PubMed & list_uids=13652807 & dopt=Abstract -Effect on behavior in humans with the administration of taraxein.. Effect on behavior in humans with the administration of taraxein. HEATHRG,MARTENS S, LEACH BE, COHEN M, ANGEL C. PMID: 13424746 [PubMed - OLDMEDLINE ..www.ncbi.nlm.nih.gov/entrez/queryfcgi?cmd=Retrieve & db=PubMed & list_uids=13424746 & dopt=Abstract=http://www.theallengroup.com/members/Schizo_11.html3) Biophysical View:a) BIOCHEMICAL APPROACHES:Are chemical substances responsible for the bizarre behavior of theschizophrenic? It has been known for a long time that administration ofsmall amounts of certain substances such as mescaline and LSD can producetransient psychotic reactions. They are capable of producing visual andauditory hallucinations and feelings of unreality in normal people.Laboratory studies have provided data indicating that there may be molecularsimilarities between hallucinogenic agents such as LSD and naturallyoccurring bodily substances. Thus, perhaps there is an association betweenschizophrenia and the quantitative differences in the metabolism of certainnormally occurring substances. Confirmation of this possibility is , howeverlacking. One group of researchers has directed his attention to thechemical similarities between mescaline and adrenaline. Mescaline is asubstance obtained from the Mexican peyote cactus; adrenaline occursnaturally in body. In 1954 Hoffer, Osmond, and Smythies observed that someasthmatic patients taking adrenaline experienced hallucinations and alteredperception when they had an overdose. Earlier, during the Second World War,it had been noted that pink adrenaline - adrenaline that had deterioratedand turned pink- produced the same effects on behavior. Chemical analysessuggested that these effects are caused by one of the constituents of pinkadrenaline - adrenochrome. While some researchers claim to have detected thepresence of adrenochrome in human body, the variance of the findings is sogreat as to cause doubt that it occurs naturally in either schizophrenics ornormals. The possibility remains that in some persons adrenaline may becomemetabolized into either mescaline or something like mescaline, or that someother type of metabolic error results in schizophrenia. Another substanceimplicated in biochemical hypothesis is serotonin. While found in many partsof the body, serotonin seems to be an especially important contributor tothe functioning of limbic system. Experimental research suggests that thelimbic system of the brain is related to emotional states and theirregulation. Its possible link with schizophrenia was initially suggestedwhen it was found that certain hallucinogens, notably LSD could block theeffects of serotonin in smooth muscle. Wooley and Shaw, building on this andrelated findings, concluded that :The demonstrated ability of such agents to antagonize the action ofserotonin in smooth muscle and findings of serotonin in the brain suggestthat the mental changes caused by drugs are the result of aserotonin-deficiency which they induce in the brain. If this be true, thenthe naturally occurring mental disorders which are mimicked by these drugs,may pictured as being the result of a cerebral serotonin deficiency arisingfrom a metabolic failure.Their interpretation leans heavily on the assumption of a similarity betweenthe behavioral effects of substances like LSD and schizophrenia. Variouskinds of evidence, including introspections of the normal people who havetaken LSD, suggest that the behavior of the under the influence of thehallucinogenic drugs and the schizophrenic individual are not identical,Although they do have similarities. This doesn't mean that investigations ofmetabolism of serotonin in the brain will not lead to new insights into thenature of schizophrenia. Excesses and deficiencies of substances likeserotonin might well cause a variety of behavioral aberrations. Asunderstanding of the nature of both naturally occurring substances and thosecompounded in laboratory increases, it will become possible to evaluate moreaccurately speculations such as following :Schizophrenia is regarded as starting with a failure to form enoughserotonin in the brain, and this is seen in the shyness and depression whichare usually forerunners of the disease. With sharply increased emotionalstrain, the control mechanism which governs the level of serotonin in thebrain begins to fail. The production of serotonin may increase sharply (orits rate of destruction may decrease), and this probably coincides with theagitated state. Subsequently, the decreased production may again take place(Wooley , 1962).Another example of a biochemical approach to schizophrenia is work that hasbeen carried out on the substance taraxein, a protein involved in thecreation of compounds that influence the central nervous system. Heathinjected taraxein into monkeys and recorded behavioral andelectroencephalographic changes. Several brain wave changes were observed.Heath also made EEG recordings in the septal region of the brains ofschizophrenic patients during periods of psychosis. A number of EEGabnormalities were noted. He then found that intravenous injection oftaraxein from schizophrenic patients produced similar EEGs in septal regionsof rhesus monkeys. Taraxein also produced schizophrenic-like symptoms inhuman volunteer subjects. Injection of substances from the bodies ofschizophrenic patients into the bodies of animals have produced a variety offindings, including atypical web construction in spiders and reducedlearning in rats. Not uncommonly, enthusiasm and hope about biochemicalcausation has been dashed by the failure of different investigators to agreeon the results of experimental procedures. Some investigators have gotdramatic positive findings, whereas others studying similar problems haveobtained negative or conflicting results. Some of these differences areattributable to uncontrolled variables. For example, differences in thecomposition of the blood of schizophrenics and non-schizophrenics may beattributable to differences in diets, particularly if the schizophrenicshave been hospitalized for a long time. Body chemistry and behavior might beaffected by the low iodine content in the table salt used in some hospitalsor the lack of physical activity during hospitalization. Although theirnumber is small, studies on periodic catatonics have uncovered significantrelationships that support the use of homogenous groups. A group of patientsstudied by Gjessing have alternated over a period of many years betweenintervals of relative normality and periods of frozen immobility or, in somecases, violent excitement. Gjessing discovered that these bizarre swingscorresponded with abnormal swings in the retention and over-excretion ofnitrogen. His work suggests that the cause of the disorder may be anaccumulation of a substance , such as one or a group of amines , in thehypothalamus or a flaw in the functioning of the pituitary gland.Examination of metabolic products in the urine and blood showed thatperiodic catatonic behavior coincided with periodic surges of autonomicnervous activity, "autonomic storms". Gjessing's extensive program ofbiochemical research led to the conclusion that a defect in the functioningof the thyroid gland (caused by either a defect in the hypothalamus or inthe pituitary gland) was a major factor in periodic psychoses. When ehadministered thyroid extract to one group of his periodic catatonic patientspsychotic symptoms completely disappeared and never returned. Thosepatients who did not take thyroid extract regularly showed relapses.Gjessing found that following administration of thyroid extract, not onlydid his patients' behavior improve and their basal metabolism rates returnto normal, but the amount of excreted nitrogen compounds returned to normal.===============================Levi G. Ledgerwood, Paul W. Ewald, and Gregory M. Cochran - Genes .... Effect on behavior in humans with the administration of taraxein. Am. J.Psychiatry 114:4-24. Ho-Yen, DO, and AWL Joss. 1992. Human toxoplasmosis. ..http://muse.jhu.edu/journals/perspectives_in_biology_and_medicine/v046/463ledgerwood.html[PDF] lo ti onFile Format: PDF/Adobe Acrobat - View as HTML. in Humans with the Administration of Taraxein, 114 Am. J. Psychiatry. 94Farmworth, Editorial, 185 J. Am. Med. Ass'n 878, 879 (1963). ...www.maps.org/psychedelicreview/v1n4/014394bar.pdf[PDF] PSYCHIATRY 1File Format: PDF/Adobe Acrobat. ditism in 65 ambiguously sexed people, and demonstrated the unwisdom ofExperiments with human subjects included the administration of taraxein .http://dx.doi.org/10.1146/annurev.me.09.020158.001533Full Article. Taraxein was identified as auto-Ab to some nuclear antigens in neuronsof ...The administration of such partly purified Ab in the general blood flow of ..www.protein.bio.msu.ru/biokhimiya/contents/v67/full/67050721.html - 52khttp://au.FoodHerbHealth/--Version: 7.0.308 / Virus Database: 266.11.9 - Release 5/12/05«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§«¤»¥«¤»§ - PULSE ON WORLD HEALTH CONSPIRACIES! §Subscribe:......... - To :.... - Any information here in is for educational purpose only, it may be news related, purely speculation or someone's opinion. Always consult with a qualified health practitioner before deciding on any course of treatment, especially for serious or life-threatening illnesses.**COPYRIGHT NOTICE**In accordance with Title 17 U.S.C. 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