YOU CAN MAKE SOMEONE SCHIZOPHRENIC - it's easy.

May 12, 2005

YOU CAN MAKE SOMEONE "SCHIZOPHRENIC" - it's easy. Apparently there has been the intentional administration of taraxein into humans! It appears it does NOT naturally occur- even in those diagnosed or labeled as 'schizophrenic'- it does make you wonder how many people are being GIVEN 'schizophrenia' through experiments?--"Cheyenne Cin"- P.S. Note that I have not looked at the linked info with the information below.

The GOOD NEWS is that if it's from severely reduced serotonin levels and we KNOW THAT, we can FIX IT! (Hopefully) -- I am SOOO suspicious, aren't I? Go fig!

=======================================

Administration of taraxein in humans.... Administration of taraxein in humans. HEATH RG, COHEN SB, SILVA F, LEACH BE,COHEN M. PMID: 13652807 [PubMed - OLDMEDLINE for Pre1966]www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=13652807 & dopt=Abstract -

Effect on behavior in humans with the administration of taraxein.... Effect on behavior in humans with the administration of taraxein. HEATH RG,MARTENS S, LEACH BE, COHEN M, ANGEL C. PMID: 13424746 [PubMed - OLDMEDLINE ...www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=13424746 & dopt=Abstract

=

http://www.theallengroup.com/members/Schizo_11.html

3) Biophysical View:

a) BIOCHEMICAL APPROACHES:

Are chemical substances responsible for the bizarre behavior of the schizophrenic? It has been known for a long time that administration of small amounts of certain substances such as mescaline and LSD can produce transient psychotic reactions. They are capable of producing visual and auditory hallucinations and feelings of unreality in normal people. Laboratory studies have provided data indicating that there may be molecular similarities between hallucinogenic agents such as LSD and naturally occurring bodily substances. Thus, perhaps there is an association between schizophrenia and the quantitative differences in the metabolism of certain normally occurring substances. Confirmation of this possibility is , however, lacking. One group of researchers has directed his attention to the chemical similarities between mescaline and adrenaline. Mescaline is a substance obtained from the Mexican peyote cactus; adrenaline occurs naturally in body. In 1954 Hoffer, Osmond, and Smythies observed that some asthmatic patients taking adrenaline experienced hallucinations and altered perception when they had an overdose. Earlier, during the Second World War, it had been noted that pink adrenaline - adrenaline that had deteriorated and turned pink- produced the same effects on behavior. Chemical analyses suggested that these effects are caused by one of the constituents of pink adrenaline - adrenochrome. While some researchers claim to have detected the presence of adrenochrome in human body, the variance of the findings is so great as to cause doubt that it occurs naturally in either schizophrenics or normals. The possibility remains that in some persons adrenaline may become metabolized into either mescaline or something like mescaline, or that some other type of metabolic error results in schizophrenia. Another substance implicated in biochemical hypothesis is serotonin. While found in many parts of the body, serotonin seems to be an especially important contributor to the functioning of limbic system. Experimental research suggests that the limbic system of the brain is related to emotional states and their regulation. Its possible link with schizophrenia was initially suggested when it was found that certain hallucinogens, notably LSD could block the effects of serotonin in smooth muscle. Wooley and Shaw, building on this and related findings, concluded that : The demonstrated ability of such agents to antagonize the action of serotonin in smooth muscle and findings of serotonin in the brain suggest that the mental changes caused by drugs are the result of a serotonin-deficiency which they induce in the brain. If this be true, then the naturally occurring mental disorders which are mimicked by these drugs, may pictured as being the result of a cerebral serotonin deficiency arising from a metabolic failure. Their interpretation leans heavily on the assumption of a similarity between the behavioral effects of substances like LSD and schizophrenia. Various kinds of evidence, including introspections of the normal people who have taken LSD, suggest that the behavior of the under the influence of the hallucinogenic drugs and the schizophrenic individual are not identical, Although they do have similarities. This doesn't mean that investigations of metabolism of serotonin in the brain will not lead to new insights into the nature of schizophrenia. Excesses and deficiencies of substances like serotonin might well cause a variety of behavioral aberrations. As understanding of the nature of both naturally occurring substances and those compounded in laboratory increases, it will become possible to evaluate more accurately speculations such as following : Schizophrenia is regarded as starting with a failure to form enough serotonin in the brain, and this is seen in the shyness and depression which are usually forerunners of the disease. With sharply increased emotional strain, the control mechanism which governs the level of serotonin in the brain begins to fail. The production of serotonin may increase sharply (or its rate of destruction may decrease), and this probably coincides with the agitated state. Subsequently, the decreased production may again take place (Wooley , 1962). Another example of a biochemical approach to schizophrenia is work that has been carried out on the substance taraxein, a protein involved in the creation of compounds that influence the central nervous system. Heath injected taraxein into monkeys and recorded behavioral and electroencephalographic changes. Several brain wave changes were observed. Heath also made EEG recordings in the septal region of the brains of schizophrenic patients during periods of psychosis. A number of EEG abnormalities were noted. He then found that intravenous injection of taraxein from schizophrenic patients produced similar EEGs in septal regions of rhesus monkeys. Taraxein also produced schizophrenic-like symptoms in human volunteer subjects. Injection of substances from the bodies of schizophrenic patients into the bodies of animals have produced a variety of findings, including atypical web construction in spiders and reduced learning in rats. Not uncommonly, enthusiasm and hope about biochemical causation has been dashed by the failure of different investigators to agree on the results of experimental procedures. Some investigators have got dramatic positive findings, whereas others studying similar problems have obtained negative or conflicting results. Some of these differences are attributable to uncontrolled variables. For example, differences in the composition of the blood of schizophrenics and non-schizophrenics may be attributable to differences in diets, particularly if the schizophrenics have been hospitalized for a long time. Body chemistry and behavior might be affected by the low iodine content in the table salt used in some hospitals or the lack of physical activity during hospitalization. Although their number is small, studies on periodic catatonics have uncovered significant relationships that support the use of homogenous groups. A group of patients studied by Gjessing have alternated over a period of many years between intervals of relative normality and periods of frozen immobility or, in some cases, violent excitement. Gjessing discovered that these bizarre swings corresponded with abnormal swings in the retention and over-excretion of nitrogen. His work suggests that the cause of the disorder may be an accumulation of a substance , such as one or a group of amines , in the hypothalamus or a flaw in the functioning of the pituitary gland. Examination of metabolic products in the urine and blood showed that periodic catatonic behavior coincided with periodic surges of autonomic nervous activity, "autonomic storms". Gjessing's extensive program of biochemical research led to the conclusion that a defect in the functioning of the thyroid gland (caused by either a defect in the hypothalamus or in the pituitary gland) was a major factor in periodic psychoses. When eh administered thyroid extract to one group of his periodic catatonic patients, psychotic symptoms completely disappeared and never returned. Those patients who did not take thyroid extract regularly showed relapses. Gjessing found that following administration of thyroid extract, not only did his patients' behavior improve and their basal metabolism rates return to normal, but the amount of excreted nitrogen compounds returned to normal.

===============================

Levi G. Ledgerwood, Paul W. Ewald, and Gregory M. Cochran - Genes ...... Effect on behavior in humans with the administration of taraxein. Am. J.Psychiatry 114:4-24. Ho-Yen, DO, and AWL Joss. 1992. Human toxoplasmosis. ...http://muse.jhu.edu/journals/perspectives_in_biology_and_medicine/v046/46.3ledgerwood.html

[PDF] lo ti onFile Format: PDF/Adobe Acrobat - View as HTML... in Humans with the Administration of Taraxein, 114 Am. J. Psychiatry. 94.Farmworth, Editorial, 185 J. Am. Med. Ass'n 878, 879 (1963). ...www.maps.org/psychedelicreview/v1n4/014394bar.pdf

[PDF] PSYCHIATRY 1File Format: PDF/Adobe Acrobat... ditism in 65 ambiguously sexed people, and demonstrated the unwisdom of .Experiments with human subjects included the administration of taraxein .http://dx.doi.org/10.1146/annurev.me.09.020158.001533

Full Article... Taraxein was identified as auto-Ab to some nuclear antigens in neurons of ...The administration of such partly purified Ab in the general blood flow of ...www.protein.bio.msu.ru/biokhimiya/contents/v67/full/67050721.html - 52k

http://au.FoodHerbHealth/

Made by www.yttepigen.dk

May 13, 2005

Kathy:

I have read this article only partially but I am very interested. My ex

husband appears to suffer from schizophrenia although he has never been

diagnosed. I will have to read up on my chemistry, he would sweat out a

smell that was like vinegar before he headed into an unstable state. I have

talked to another spouse of a schizophrenic and she also noted that vinegar

smell in his perspiration before he had periods of unstability. Is vinegar

similar to what they talk about in this article?

I have had the gut feeling that his disease was not from natural causes.

But it has just been a hunch.

Frog

-

" Kathy " <vanokat

Thursday, May 12, 2005 8:46 AM

YOU CAN MAKE SOMEONE " SCHIZOPHRENIC " - it's

easy.

YOU CAN MAKE SOMEONE " SCHIZOPHRENIC " - it's easy. Apparently there has been

the intentional administration of taraxein into humans! It appears it does

NOT naturally occur- even in those diagnosed or labeled as 'schizophrenic'-

it does make you wonder how many people are being GIVEN 'schizophrenia'

through experiments?-- " Cheyenne Cin " - P.S. Note that I have not looked at

the linked info with the information below.

The GOOD NEWS is that if it's from severely reduced serotonin levels and we

KNOW THAT, we can FIX IT! (Hopefully) -- I am SOOO suspicious, aren't I? Go

fig!

=======================================

Administration of taraxein in humans.

.... Administration of taraxein in humans. HEATH RG, COHEN SB, SILVA F, LEACH

BE,

COHEN M. PMID: 13652807 [PubMed - OLDMEDLINE for Pre1966]

www.ncbi.nlm.nih.gov/entrez/query

fcgi?cmd=Retrieve & db=PubMed & list_uids=13652807 & dopt=Abstract -

Effect on behavior in humans with the administration of taraxein.

.... Effect on behavior in humans with the administration of taraxein. HEATH

RG,

MARTENS S, LEACH BE, COHEN M, ANGEL C. PMID: 13424746 [PubMed - OLDMEDLINE .

..

www.ncbi.nlm.nih.gov/entrez/query

fcgi?cmd=Retrieve & db=PubMed & list_uids=13424746 & dopt=Abstract

=

http://www.theallengroup.com/members/Schizo_11.html

3) Biophysical View:

a) BIOCHEMICAL APPROACHES:

Are chemical substances responsible for the bizarre behavior of the

schizophrenic? It has been known for a long time that administration of

small amounts of certain substances such as mescaline and LSD can produce

transient psychotic reactions. They are capable of producing visual and

auditory hallucinations and feelings of unreality in normal people.

Laboratory studies have provided data indicating that there may be molecular

similarities between hallucinogenic agents such as LSD and naturally

occurring bodily substances. Thus, perhaps there is an association between

schizophrenia and the quantitative differences in the metabolism of certain

normally occurring substances. Confirmation of this possibility is , however

lacking. One group of researchers has directed his attention to the

chemical similarities between mescaline and adrenaline. Mescaline is a

substance obtained from the Mexican peyote cactus; adrenaline occurs

naturally in body. In 1954 Hoffer, Osmond, and Smythies observed that some

asthmatic patients taking adrenaline experienced hallucinations and altered

perception when they had an overdose. Earlier, during the Second World War,

it had been noted that pink adrenaline - adrenaline that had deteriorated

and turned pink- produced the same effects on behavior. Chemical analyses

suggested that these effects are caused by one of the constituents of pink

adrenaline - adrenochrome. While some researchers claim to have detected the

presence of adrenochrome in human body, the variance of the findings is so

great as to cause doubt that it occurs naturally in either schizophrenics or

normals. The possibility remains that in some persons adrenaline may become

metabolized into either mescaline or something like mescaline, or that some

other type of metabolic error results in schizophrenia. Another substance

implicated in biochemical hypothesis is serotonin. While found in many parts

of the body, serotonin seems to be an especially important contributor to

the functioning of limbic system. Experimental research suggests that the

limbic system of the brain is related to emotional states and their

regulation. Its possible link with schizophrenia was initially suggested

when it was found that certain hallucinogens, notably LSD could block the

effects of serotonin in smooth muscle. Wooley and Shaw, building on this and

related findings, concluded that :

The demonstrated ability of such agents to antagonize the action of

serotonin in smooth muscle and findings of serotonin in the brain suggest

that the mental changes caused by drugs are the result of a

serotonin-deficiency which they induce in the brain. If this be true, then

the naturally occurring mental disorders which are mimicked by these drugs,

may pictured as being the result of a cerebral serotonin deficiency arising

from a metabolic failure.

Their interpretation leans heavily on the assumption of a similarity between

the behavioral effects of substances like LSD and schizophrenia. Various

kinds of evidence, including introspections of the normal people who have

taken LSD, suggest that the behavior of the under the influence of the

hallucinogenic drugs and the schizophrenic individual are not identical,

Although they do have similarities. This doesn't mean that investigations of

metabolism of serotonin in the brain will not lead to new insights into the

nature of schizophrenia. Excesses and deficiencies of substances like

serotonin might well cause a variety of behavioral aberrations. As

understanding of the nature of both naturally occurring substances and those

compounded in laboratory increases, it will become possible to evaluate more

accurately speculations such as following :

Schizophrenia is regarded as starting with a failure to form enough

serotonin in the brain, and this is seen in the shyness and depression which

are usually forerunners of the disease. With sharply increased emotional

strain, the control mechanism which governs the level of serotonin in the

brain begins to fail. The production of serotonin may increase sharply (or

its rate of destruction may decrease), and this probably coincides with the

agitated state. Subsequently, the decreased production may again take place

(Wooley , 1962).

Another example of a biochemical approach to schizophrenia is work that has

been carried out on the substance taraxein, a protein involved in the

creation of compounds that influence the central nervous system. Heath

injected taraxein into monkeys and recorded behavioral and

electroencephalographic changes. Several brain wave changes were observed.

Heath also made EEG recordings in the septal region of the brains of

schizophrenic patients during periods of psychosis. A number of EEG

abnormalities were noted. He then found that intravenous injection of

taraxein from schizophrenic patients produced similar EEGs in septal regions

of rhesus monkeys. Taraxein also produced schizophrenic-like symptoms in

human volunteer subjects. Injection of substances from the bodies of

schizophrenic patients into the bodies of animals have produced a variety of

findings, including atypical web construction in spiders and reduced

learning in rats. Not uncommonly, enthusiasm and hope about biochemical

causation has been dashed by the failure of different investigators to agree

on the results of experimental procedures. Some investigators have got

dramatic positive findings, whereas others studying similar problems have

obtained negative or conflicting results. Some of these differences are

attributable to uncontrolled variables. For example, differences in the

composition of the blood of schizophrenics and non-schizophrenics may be

attributable to differences in diets, particularly if the schizophrenics

have been hospitalized for a long time. Body chemistry and behavior might be

affected by the low iodine content in the table salt used in some hospitals

or the lack of physical activity during hospitalization. Although their

number is small, studies on periodic catatonics have uncovered significant

relationships that support the use of homogenous groups. A group of patients

studied by Gjessing have alternated over a period of many years between

intervals of relative normality and periods of frozen immobility or, in some

cases, violent excitement. Gjessing discovered that these bizarre swings

corresponded with abnormal swings in the retention and over-excretion of

nitrogen. His work suggests that the cause of the disorder may be an

accumulation of a substance , such as one or a group of amines , in the

hypothalamus or a flaw in the functioning of the pituitary gland.

Examination of metabolic products in the urine and blood showed that

periodic catatonic behavior coincided with periodic surges of autonomic

nervous activity, " autonomic storms " . Gjessing's extensive program of

biochemical research led to the conclusion that a defect in the functioning

of the thyroid gland (caused by either a defect in the hypothalamus or in

the pituitary gland) was a major factor in periodic psychoses. When eh

administered thyroid extract to one group of his periodic catatonic patients

psychotic symptoms completely disappeared and never returned. Those

patients who did not take thyroid extract regularly showed relapses.

Gjessing found that following administration of thyroid extract, not only

did his patients' behavior improve and their basal metabolism rates return

to normal, but the amount of excreted nitrogen compounds returned to normal.

===============================

Levi G. Ledgerwood, Paul W. Ewald, and Gregory M. Cochran - Genes ...

.... Effect on behavior in humans with the administration of taraxein. Am. J.

Psychiatry 114:4-24. Ho-Yen, DO, and AWL Joss. 1992. Human toxoplasmosis. ..

http://muse.jhu.edu/journals/perspectives_in_biology_and_medicine/v046/46

3ledgerwood.html

[PDF] lo ti on

File Format: PDF/Adobe Acrobat - View as HTML

.... in Humans with the Administration of Taraxein, 114 Am. J. Psychiatry. 94

Farmworth, Editorial, 185 J. Am. Med. Ass'n 878, 879 (1963). ...

www.maps.org/psychedelicreview/v1n4/014394bar.pdf

[PDF] PSYCHIATRY 1

File Format: PDF/Adobe Acrobat

.... ditism in 65 ambiguously sexed people, and demonstrated the unwisdom of

Experiments with human subjects included the administration of taraxein .

http://dx.doi.org/10.1146/annurev.me.09.020158.001533

Full Article

.... Taraxein was identified as auto-Ab to some nuclear antigens in neurons

of ...

The administration of such partly purified Ab in the general blood flow of .

..

www.protein.bio.msu.ru/biokhimiya/contents/v67/full/67050721.html - 52k

http://au.FoodHerbHealth/

----------

----

Version: 7.0.308 / Virus Database: 266.11.9 - Release 5/12/05

May 13, 2005

Interesting points about the 'vinegary' smell .... I have found that if I go

into a state of protein depletion - hard physical work and living on fruit

vegies and little meat [steak, lamb, chicken, etc], my sweat would smell

vinegary or ammonia-like, and I would finf that I began to lose mental

sharpness, thought stability, get the 'shakes', and feel extremely jittery

inside.

Maybe all they need is a good steak and salad.......

Very telling if the blood group is " O " !!!!

Regards

Jorge

-

" Frog " <frogmushroom

Friday, May 13, 2005 5:12 PM

Re: YOU CAN MAKE SOMEONE " SCHIZOPHRENIC " -

it's easy.