Foot Drop

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Dear Master Fe,

Greetings!

I require your help in treating my patient, an elderly genlteman, age

82.

He has the following problem.

Foot Drop: Demilination of sural nerve.

Poor hearing in the right ear - the eustachian tube closes and

hinders

hearing, if one blows into the ear, has better hearing.

Thank you for all your help.

Regards

Sumi----------

Dear Sumi,

Greetings.

MEDICAL

INFORMATION: FOOT

DROP

" Foot drop, a disorder of the distal aspect of a lower extremity,

like most foot ailments, has little to do with the intrinsic function of

the foot. This neurological disturbance, like most common to the foot, is

caused by nerves proximal to it's location. This impairment is a

malfunction of either a peripheral nerve or a part of the central nervous

system

The five muscles of importance are the tibialis anterior, peroneus longus

and brevis, posterior tibial, gluteal medius, and quadriceps. A weakness

of any of these muscles may indicate a peroneal nerve lesion. It is also

important to remember the unique role of the short head of the biceps

femoris, one of the three knee flexors. It is the only muscle above the

knee supplied by the peroneal nerve.

The peroneal nerve is named only below the knee, but functionally, it can

be considered a distinct structure up to the origin of the superior

gluteal nerve. After the nerve divides, the superficial branch innervates

the peroneal longus and peroneal brevis muscles. The deep branch supplies

the tibialis anterior, the extensor digitorum longus, the extensor

hallucis longus, and the extensor digitorum brevis. While the tibialis

anterior dorsiflexes the foot, the extensor digitorum longus and brevis,

along with the extensor hallucis longus, extend the toes. Therefore,

weakness of the dorsiflexors of the foot causes foot drop that is so

characteristic of this nerve lesion.

A keen knowledge of the anatomy of the musculature and its nervous

intervention will greatly help the clinician determine his site of

lesion. A lesion of the peroneal nerve at knee level will cause weakness

of the tibialis anterior muscle and the peroneal longus and brevis

musculature, while the strength in the muscles supplied by the peroneal

nerve above the knee remain normal. Muscle atrophy is an objective sign

that a nerve lesion in present. Observation of the muscle may help in

revealing a peroneal nerve lesion with a sharp anterior margin of the

tibia. This represents atrophy of the muscles innervated by the peroneal

nerve. Besides a peripheral nerve lesion, weakness of the tibialis

anterior can also be cause to a lesion involving the L4 or L5 nerve

roots. A differential diagnosis can be made by checking the integrity of

the muscles innervated by the L4-L5 roots by other nerves. These muscles

would include the quadriceps muscle, and the hip adductors supplied by

the femoral nerve and obturator nerve respectively. Muscles innervated by

the L5 root include the hip abductors, which include the gluteus medius

and minimus, which are supplied by the superior gluteal nerve. The

posterior tibialis muscle and the flexor digitorum longus are innervated

by the posterior tibial nerve. These muscles invert the foot and flex the

toes, respectively.

Due to the superficial position of the peroneal nerve, both trauma and

compression may be the causes of peroneal neuropathy. Lacerations over

the lateral surfaces of the knee or fractures in the vicinity of the knee

can cause lesions. Hip surgery and hip fractures are lessor but

noteworthy causes. Prolonged squatting and habitually sitting with knees

crossed may compress the peroneal nerve. Local masses and cysts may also

cause peroneal neuropathy.

The diagnosis of peroneal neuropathy is usually self-evident with

complaints by the patient of weakness and/or numbness of the foot and

foot drop or steppage gait. Examination will reveal weakness of foot

dorsiflexors and evertors as well as loss of sensation over the dorsum of

the foot. Therapy is required to maintain ankle motion and protect the

strength in the functioning musculature. If recovery is taking longer

than it should, then electrophysiological testing should be performed to

evaluate nerve function.

The use of electromyography may be used after a complete physical exam,

which includes orthopedic, neurological, and muscle testing exams. The

use of an EMG will give a more precise location of the lesion.

Electromyography involves inserting a needle electrode into a muscle and

displaying the potential detected by the electrode on an oscilloscope

screen after suitable amplification. "

PRANIC HEALING TREATMENT:

Invocation and thanksgiving before and after the treatment.

Scan and re-scan before, during, and after the treatment.

1. Apply general sweeping.

2. Apply localized sweeping on the spine with LWG.

3. Apply localized sweeping on the front and back solar plexus and

on the liver with LWG.

4. Energize the solar plexus with LWG, LWB, then LWV.

Apply ordinary sweeping.

5. Apply localized sweeping on the front and back heart

chakra. Energize the back heart chakra with LWG, then with more of

LWV.

6. Apply localized sweeping on the lower spine alternately and

thoroughly with LWG & LWO.

7. Energize the lower spine with LWG, LWB, then LWY.

8. Apply localized sweeping on the basic chakra with LWG &

LWO. Energize with LWR.

9. Apply localized sweeping on the perineum minor chakra.

9. Apply localized sweeping on the navel chakra. Energize

with WHITE.

10. Apply localized sweeping on the hip, the leg minor, and the

sole minor chakras of the affected foot alternately and thoroughly with

LWG & LWO. Sweep also the muscles and the nerves running from

the hip down to the foot with LWG & LWO. Energize them

with LWG, LWB, then with more of LWV.

11. Stabilize and cut the connecting cord.

12. Repeat treatment 2 to 3 times a week,

Love and light, masterfe