Ureaplasma Urealyticum (Uro-genital infection)

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SUSANA

January 21, 2010 3:15:22 AM EST (CA)

Ureaplasma urealyticum

 

ATMA NAMASTE

 

GRACIAS POR SU TRABAJO¡¡¡

 

UNA PACIENTE DE 40 AÑOS TIENE REITERADAMENTE UN

HONGO VAGINAL:

UREOPLASMA UREOLITICUM, DESEARIA QUE ME ENVIARA

PROTOCOLO PORQUE ES MUY

RESISTENTE.MUCHAS BENDICIONES

GRACIAS

 

SUSANA CERRUTI

 

=========================================

 

Dear Susana,

A Loving Atma Namaste.

 

Thank you for your e-mail.

 

Medical Background:

 

Synonyms and related keywords: Ureaplasma infection,

mycoplasma, Mycoplasma pneumoniae, M pneumoniae,

Mycoplasma hominis, M hominis, Ureaplasma, Ureaplasma

urealyticum, U urealyticum, Ureaplasma parvum, U

parvum, Mycoplasma genitalium, M genitalium,

Mycoplasma fermentans, M fermentans, Mycoplasma pirum,

M pirum, Mycoplasma penetrans, M penetrans, urogenital

disease, urethritis, urogenital infection, genital

mycoplasmal organisms, ureaplasmas, ureaplasmal

infection, cervicitis, pelvic inflammatory disease,

PID, pneumonia, bacterial pneumonia, infectious

arthritis, nongonococcal urethritis, nonchlamydial

nongonococcal urethritis, female urethral syndrome,

acute epididymoorchitis, acute pyelonephritis,

bacterial vaginosis, salpingitis, congenital

pneumonia, congenital bacteremia, congenital

meningitis, bronchopulmonary dysplasia, osteomyelitis,

meningitis, endometritis, chorioamnionitis, surgical

wound infections, neonatal pneumonia, neonatal

meningitis, septic arthritis, pneumonitis

 

Ureaplasma urealyticum is a species of gram-negative

bacteria found in the human genitourinary tract

(UROGENITAL SYSTEM), oropharynx, and anal canal.

 

In humans, both Mycoplasma and Ureaplasma species may

be transmitted by direct contact between hosts (ie,

venereally through genital-to-genital or

oral-to-genital contact), vertically from mother to

offspring (either at birth or in utero), or by

nosocomial acquisition through transplanted tissues.

 

Mycoplasma species are the smallest free-living

organisms and are unique among prokaryotes in that

they lack a cell wall. This feature is largely

responsible for their biologic properties, including

lack of a Gram stain reaction and nonsusceptibility to

many commonly prescribed antimicrobial agents,

including beta-lactams. Mycoplasma organisms are

usually associated with mucosae. They reside

extracellulary in the respiratory and urogenital

tracts and rarely penetrate the submucosa, except in

the case of immunosuppression or instrumentation, when

they may invade the bloodstream and disseminate to

numerous organs and tissues.

 

Among the 17 species isolated from humans, 4 types of

organisms are of major concern. Mycoplasma pneumoniae

is a well-established pathogen; it is rarely isolated

from healthy persons. See Mycoplasma Infections for a

discussion of M pneumoniae infection. Mycoplasma

hominis and Ureaplasma species, known collectively as

the genital mycoplasmal organisms, are generally

considered opportunists that cause invasive infections

in susceptible populations. The 2 Ureaplasma biovars,

Ureaplasma urealyticum and Ureaplasma parvum, have now

been designated as separate species. Separation of

these species is not possible except via molecular

techniques such as polymerase chain reaction (PCR).

Therefore, they are considered together as Ureaplasma

species (Waites, Clin Microbiol Rev, 2005).

 

Serologic studies and PCR have enhanced knowledge of

several other fastidious and slow-growing mycoplasmal

organisms, including Mycoplasma genitalium, Mycoplasma

fermentans, Mycoplasma pirum, and Mycoplasma

penetrans, and their possible roles in certain

pathologic conditions in humans. Because of their

extremely fastidious nature and the lack of reliable

means for cultivation on artificial media, detection

of these mycoplasmal organisms rests primarily with

molecular techniques. Relatively little is known about

their importance as human pathogens, with the notable

exception of M genitalium, an organism that has been

the focus of a considerable number of clinical

research studies in recent years. This research and

the subsequent data are made possible by the

availability of PCR assays, which can detect the

presence of these organisms.

 

Pathophysiology: Although M hominis and Ureaplasma

species are frequently detected in the lower

urogenital tracts of healthy adults, they can also

produce localized urogenital diseases. In some

settings, they can produce infection in extragenital

sites, as does M genitalium. Recent studies with PCR

assays expanded the understanding of sites of

mycoplasmal localization within the human body. The

presence of M fermentans was demonstrated in the

throats of children with pneumonia and in the synovial

fluid of persons with rheumatoid arthritis. M

genitalium is found in the lower urogenital tracts of

men with urethritis and women with cervicitis and

pelvic inflammatory disease. M penetrans is found in

the urine of children and homosexual males infected

with HIV, but the clinical significance of this is not

known.

 

The newest mycoplasmal species to be detected in

humans is Mycoplasma amphoriforme, an organism

detected in the lower respiratory tract of

immunosuppressed persons with chronic bronchitis

(Webster, 2003). Its true role as a human pathogen has

not yet been determined.

 

In humans, both Mycoplasma and Ureaplasma species may

be transmitted by direct contact between hosts (ie,

venereally through genital-to-genital or

oral-to-genital contact), vertically from mother to

offspring (either at birth or in utero), or by

nosocomial acquisition through transplanted tissues.

 

Ureaplasma species and M genitalium are causes of

nonchlamydial nongonococcal urethritis in men (Waites,

Clin Microbiol Rev, 2005; Jensen, 2004). No evidence

indicates that that M hominis causes female urethral

syndrome; however, Ureaplasma species may be involved.

Ureaplasma organisms have been recovered from an

epididymal aspirate from a patient with acute

epididymoorchitis, and these organisms may be an

infrequent cause of the disease. M hominis has been

isolated from the upper urinary tract of patients with

symptoms of acute pyelonephritis and may cause

approximately 5% of cases.

 

Mycoplasma species do not cause vaginitis, but they

may proliferate in patients with bacterial vaginosis

and may contribute to the condition. M hominis has

been isolated from the endometria and fallopian tubes

of approximately 10% of women with salpingitis; M

genitalium may also be involved in pelvic inflammatory

disease and cervicitis. Whether Ureaplasma infection

causes involuntary infertility remains speculative.

Ureaplasma species can cause placental inflammation

and may invade the amniotic sac early, causing

persistent infection and adverse pregnancy outcomes,

including premature birth. M hominis has been isolated

from the blood of approximately 10% of women with

postpartum or postabortal fever, but not from afebrile

women who had abortions or from healthy women who are

pregnant. Similar observations have been made for

Ureaplasma species.

 

Colonization of infants by genital mycoplasmal

organisms may occur by ascension of the microorganisms

from the lower genital tract of the mother at the time

of delivery or by direct invasion of the fetus in

utero. Congenital pneumonia, bacteremia, meningitis,

and death have occurred in infants with very low birth

weight due to Ureaplasma or Mycoplasma infection of

the lower respiratory tract. In several large studies,

chronic lung disease of prematurity or

bronchopulmonary dysplasia has also been associated

with the presence of Ureaplasma organisms in the lower

respiratory tract, presumably because of low-grade

inflammation in the airways that causes a prolonged

need for supplemental oxygen coupled with barotrauma

of mechanical ventilation and oxidant damage due to

oxygen administration (Waites, Clin Microbiol Rev,

2005).

 

Both M hominis and Ureaplasma species have been

isolated from maternal blood, umbilical cord blood,

and neonatal blood. Both organisms can invade the

cerebrospinal fluid (CSF) and induce pleocytosis.

While M fermentans has been detected in pure culture

from placentae and amniotic fluid in the presence of

inflammation, no studies confirm its occurrence and

significance in neonates.

 

Both Mycoplasma and Ureaplasma species can cause

invasive disease of the joints and respiratory tract

with bacteremic dissemination, particularly in persons

with antibody deficiencies (Furr, 1994). Ureaplasma

species are the most common nonbacterial etiologies of

infectious arthritis in persons who are

hypogammaglobulinemic. M hominis bacteremia has been

demonstrated following renal transplantation, trauma,

and genitourinary manipulations. This organism has

also been found in surgical wound infections, fluids

from pericardial effusions, prosthetic valves affected

by endocarditis, and subcutaneous abscesses. Both

organisms can cause osteomyelitis. M fermentans, M

hominis, and Ureaplasma species can be detected with

culture or PCR in the synovial fluid of persons with

rheumatoid arthritis. Their precise contribution to

this disease is uncertain (Waites, Mycoplasmas:

Pathogenesis, Molecular Biology, and Emerging

Strategies for Control, 2005).

 

The significance of M fermentans, M penetrans, M

pirum, and other mycoplasmal infections in persons

also infected with HIV has received a great deal of

attention and is a matter of debate. M fermentans has

also been detected in adults with an acute

influenzalike illness and in the bronchoalveolar

lavage fluids of patients with AIDS and pneumonia.

Apparently, respiratory tract infection with M

fermentans is not necessarily linked with

immunodeficiency, but it may behave as an

opportunistic respiratory pathogen.

 

Frequency: In the US - Ureaplasma species have been

isolated from cervicovaginal specimens in 40-80% of

women who are asymptomatic and sexually active. M

hominis has been isolated from cervicovaginal

specimens in 21-53% of women who are asymptomatic and

sexually active. These rates are somewhat lower in

males. Only a subgroup of adults who are colonized in

the lower urogenital tract develop symptomatic illness

from these organisms. Nongonococcal urethritis is the

most common sexually transmitted infection. Ureaplasma

species and M genitalium may account for a significant

portion of cases that are not due to chlamydiae. More

than 20% of liveborn infants may be colonized by

Ureaplasma, and infants born preterm most likely

harbor the organisms. Colonization declines after age

3 months. Less than 5% of children and 10% of adults

who are not sexually active are colonized with genital

mycoplasmal microorganisms.

 

Immunosuppression (eg, from antibody deficiency

or prematurity) increases the likelihood of developing

disseminated disease. Much less is known about the

epidemiology of species such as M genitalium and M

fermentans. Some organisms, such as M pirum and M

penetrans, have been primarily isolated from persons

with HIV infection.

 

* Internationally: Although few studies have

investigated the geographic distribution of genital

mycoplasmal infections, the facts that they (1) are

present on mucosal surfaces in so many healthy persons

and (2) can be transmitted venereally suggest that

variation in prevalence of these organisms in adults

is more likely related to behavioral variables such as

number of sexual partners and socioeconomic status

rather than to geographic or climatic differences.

 

Mortality/Morbidity:

 

* Assessing morbidity and mortality for diseases

specifically caused by genital mycoplasmal infections

is difficult because few studies systematically

evaluate them and some conditions with which they are

involved can be polymicrobial (eg, pelvic inflammatory

disease, urethritis). Difficulty in detecting the more

fastidious species, such as M genitalium and M

fermentans, further complicates such assessments.

 

* In adults with an intact and functional immune

system, infections associated with genital mycoplasmal

organisms are usually localized and do not result in

severe illness, attesting to their relatively low

virulence and perceived status as opportunists.

 

* Persons with antibody deficiencies reportedly

have developed severe pulmonary infections,

destructive arthritis and osteomyelitis associated

with subcutaneous abscesses, and other disseminated

infections of various organ systems.

 

* Deaths have occurred in neonates with

bloodstream invasion by Ureaplasma species and

meningitis caused by M hominis; however, in some

instances, the organisms spontaneously disappeared

from CSF without treatment (Waites, Clin Microbiol

Rev, 2005).

 

* Sporadic case reports document fatal infections

caused by Mycoplasma species of animal origin,

including Mycoplasma arginini in immunosuppressed

hosts, but these are extremely rare (Waites,

Mycoplasmas: Pathogenesis, Molecular Biology, and

Emerging Strategies for Control, 2005).

 

Race:

 

* Differences in carriage of genital mycoplasmal

organisms and subsequent disease are more likely

related to sexual behavior and socioeconomic status

than to race.

 

Sex:

 

* No obvious sex predilection is reported for

infections due to genital mycoplasmal species, except

for the differences in urogenital diseases such as

salpingitis and endometritis, which are sex specific.

The carriage rate of genital Mycoplasma species in the

lower urogenital tract is somewhat greater for females

than for males.

 

* Ureaplasma species have been isolated from

cervicovaginal specimens in 40-80% of women who are

asymptomatic and sexually active, and M hominis has

been isolated from cervicovaginal specimens in 21-53%

of women who are asymptomatic and sexually active.

This prevalence is somewhat lower in males.

 

Age:

 

* M hominis and Ureaplasma species are common

commensal inhabitants of the lower genitourinary tract

in adolescents and adult men and women who are

sexually active. The organisms can be transmitted

venereally and vertically from mother to offspring.

 

* Neonates who acquire the organisms are usually

colonized in the upper and sometimes lower respiratory

tracts with occasional dissemination to the

bloodstream and CSF. Clinically significant infections

may ensue in individuals who are sexually active and

in neonates but are rare to nonexistent in older

children and adolescents who are not sexually active,

with the exception of those with immunodeficiencies.

 

* M fermentans has been recovered from the throats

of children with pneumonia; however, the frequency of

its occurrence in healthy children is unknown.

 

Little is known about the occurrence of other

mycoplasmal species in different populations and

specific associations with disease.

 

Many clinicians are unfamiliar with Mycoplasma and

Ureaplasma species as etiologic agents. This

unfamiliarity is further complicated by a lack of

facilities to diagnose mycoplasmal infections in many

clinical settings. Subsequently, identification of

these organisms may be achieved only as a last resort,

particularly if initial treatment with drugs that are

ineffective against Mycoplasma or Ureaplasma species

is unsuccessful.

 

The following conditions may be caused by infection

with Mycoplasma and Ureaplasma species:

 

-Urethritis

 

-Pyelonephritis

 

-Pelvic inflammatory disease

 

-Endometritis or chorioamnionitis

 

-Infectious arthritis

 

-Surgical wound infections

 

-Neonatal pneumonia

 

-Neonatal meningitis

 

 

Mycoplasma and Ureaplasma organisms often play minor

roles as causes of the above-named conditions, which

may be caused by various microorganisms. When present

in patients with some of these conditions, such as

salpingitis, urethritis, and septic arthritis, one of

several etiologic organisms may be present

simultaneously.

 

Source - Ureaplasma Urealyticum by Ken B. Waites,

M.D., Director of Clinical Microbiology, Professor,

Department of Pathology, Division of Laboratory

Medicine, University of Alabama at Birmingham,

EMedicine from WebMD, January 5, 2007.

 

 

PRANIC HEALING:

 

1. Invoke and scan before, during and after treatment.

 

2. Apply advanced general sweeping with LWG 2 to 3 times.

 

3. Energize the sex chakra and the affected part(s)

with LB for localizing effects.

 

4. Do localized thorough sweeping on the sex chakra and

affected part(s) alternately with LWG and LWO.

Energize with LWG, LWB then ordinary LWV.

 

5. Do localized thorough sweeping on the basic chakra.

DO NOT energize the basic chakra.

 

6. Do localized thorough sweeping on the arms and legs.

 

Energize the hand and sole minor chakras with ordinary

LWV.

 

Do not apply this step more than once a day.

 

7. Do localized thorough sweeping on the front and back

of the lungs.

 

Rescan. Continue sweeping until the energy is clean

and stable.

 

Energize the lungs thoroughly through the back of the

lungs with LWG then LWO. This may take several

minutes.

 

* Rescan after every 15 minutes. Step 7 may be

repeated after every 15 minutes for the next several

hours until the energy remains clean and stable.

 

8. Do localized thorough sweeping on the front and back

solar plexus chakra and the liver. Energize the solar

plexus chakra with LWG, LWB then ordinary LWV.

 

9. Do localized thorough sweeping on the front and back

spleen chakra. Energize with LWG then with ordinary

LWV. This has to be done with caution.

 

10. Do localized thorough sweeping on the front and back

heart chakra. Energize through the back heart chakra

with LWG then with more of ordinary LWV.

 

11. Do localized thorough sweeping on the throat chakra,

crown chakra, forehead chakra, ajna chakra, and back

head minor chakra. Energize them with LWG then with more

of ordinary LWV.

 

12. Stabilize and release projected pranic energy.

 

13. Repeat entire treatment 3 times a week.

 

With Loving Blessings,

The PHQandA Team

 

Source: The books written by MASTER CHOA KOK SUI including:

 

Miracles Through Pranic Healing

Advanced Pranic Healing

Pranic Psychotherapy

Pranic Crystal Healing

 

NOTICE:

 

1. Pranic Healing is not intended to replace orthodox medicine, but rather

to complement it. If symptoms persist or if the ailment is severe, please

consult immediately a medical doctor and a Certified Pranic Healer.

 

2. Pranic Healers who are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments.

 

~ Master Choa Kok Sui

 

MCKS website: http://www.pranichealing.org