Pilonidal Disease (cyst and sinus)

[Guest guest]

Atma Namaste!

 

I wanted to know the protocol for Pinodial Sinus. It happens due a infection in

the cleft. A small hole is formed from where blood and puss is discharged. The

person is 23yrs old male.

 

Thank You very much

 

Regards

Deven

 

 

-

 

 

 

Dear Deven,

 

Atma namaste.

 

Thank you for your email.

 

Medical Background:

 

In 1833, Herbert Mayo described a cyst that contained hair just below the

coccyx. Hodge in 1880 coined the name " pilonidal " from the Latin words pilus,

which means hair, and nidus, which means nest. Pilonidal disease consists of a

spectrum of entities ranging from asymptomatic hair containing cysts and sinuses

to a large abscess in the sacrococcygeal area.

 

The controversies surrounding the origin of pilonidal disease first came to

light during World War II. From 1941-1944, 78,924 soldiers were treated for

pilonidal disease. The disease was the leading cause of nontraumatic sick days

taken by United States soldiers. Most soldiers had recovery times of

approximately 100 days.

Pathophysiology

 

The medical literature regarding the etiology of the pilonidal cyst has shifted.

Initially, these cysts were believed to be congenital in nature. One of the more

prevalent congenital theories believed that patients with pilonidal disease have

persistent remnants of the caudal segment of the neural canal. This caudal

segment formed multiple small cysts that remained in contact with the skin

surface. As the cyst increased in size, they eventually ruptured, resulting in

the formation of sinuses tracts. The congenital theory has been called into

question for multiple reasons. Most notable was multiple case reports describing

pilonidal cyst formation in jeep drivers in World War II. So many serviceman

were affected with pilonidal disease that it was renamed " jeep disease. " These

findings led to the belief that pilonidal cysts can be acquired by excessive

repetitive trauma to the sacrococcygeal region.

 

Although rare, pilonidal cysts have also been reported in other parts of the

body that are exposed to repetitive trauma such as barber's and sheepherder's

hands. The male predominance of pilonidal cyst, the presentation in the

adolescent period of life, and the recurrence after adequate surgical drainage

further encouraged the rejection of the congenital theory. These points lead the

way for the modern day acceptance of the acquired theory of pilonidal cysts.

 

The acquired theory postulates that pilonidal disease is a result of hair and

cellular debris finding a portal of entry into the skin and hair follicles. The

entering hair causes an inflammatory reaction and edema. The edema causes

occlusion of the skin opening increasing the hair follicle size. This results in

a build up in pressure in the hair follicle that eventually spreads its purulent

material into the subcutaneous tissue causing a foreign body reaction. This

reaction forms multiple microabscesses that eventually migrate further into the

subcutaneous tissue. A vacuum force caused by the tauting of skin when the

patient bends over is believed to aid in the hair migration. These

microabscesses eventually result in the creation of more sinus tracts and

abscesses. Karydakis later described the role of hair in the formation of

pilonidal disease and divided it into 3 phases: phase 1, the invader, a free

hair is available to invade into a portal of entry

into the skin; phase 2, the force that causes the insertion; and phase 3, the

vulnerability of the skin to the insertion of hair at the depth of the natal

cleft.

 

At surgery, only 50-75% of all pilonidal cysts actually contain hair.

Frequency

United States

 

Pilonidal disease affects approximately 26 per 100,000 people.

International

 

In England in 2000-2001, a total of 11,534 admissions were recorded for

pilonidal disease. The mean hospital stay was 4.3 days.

Sex

 

Pilonidal disease in the general population has a male preponderance. It occurs

in the ratio of 3 or 4:1. In children, however, the ratio is the opposite

occurring in 4 females for each male it afflicts.

Age

 

Pilonidal disease commonly affects adults in the second to third decade of life.

Pilonidal cysts are extremely uncommon after age 40 years, and the incidence

usually decreases by age 25 years. The average age of presentation is 21 years

for men and 19 years for women.

Clinical

History

 

Pilonidal disease has 3 major types of presentations.

 

* Completely asymptomatic sinus tracts that are noticed by the patient or

primary care physician

* Chronic disease: The average patient has 2 years of disease before seeking

medical treatment. More than 80% of presentations of pilonidal disease are

exacerbations of a chronic sinus tract.

 

Physical

 

The physical findings in pilonidal disease are dependent on the stage of disease

at presentation. In the early stages, the patient can notice a sinus tract or

pit in the sacrococcygeal region. This can progress to midline edema or abscess

formation.

 

As with any abscess, physical examination findings include tenderness to

palpation, fluctuance, warmth, purulent discharge, and induration or cellulitis.

Fever and other systemic signs of infection are uncommon.

 

Causes

 

* Pilonidal disease involves loose hair and skin and perineal flora.

o Risk factors for pilonidal disease include male gender, hirsute

individuals, Caucasians, sitting occupations, existence of a deep natal cleft,

and presence of hair within the natal cleft. Family history is seen in 38% of

patients with pilonidal disease. Obesity is a risk factor for recurrent disease.

o The most commonly reported bacteria cultured from pilonidal

abscesses differ by author. In one study, anaerobic cocci were present 77% of

the time; aerobic, 4%; and mixed aerobic and anaerobic, 17%. Other studies quote

Staphylococcus aureus, an aerobe, as being the most common bacterial pathogen.

 

Source: " Pilonidal Disease Cyst and Sinus " by Robert Ringelheim, MD, Attending

Physician, Memorial Regional Hospital for Medscape, WebMD.

 

Pranic Healing:

 

1. Invoke and scan before, during and after treatment.

 

2. General sweeping.

 

3. Localized thorough sweeping on the front and back solar plexus chakra.

Energize with LWg then LWB.

 

4. Localized thorough sweeping on the affected area alternately with LWg and

LWO.

 

Rescan. Continue sweeping until the energy is clean and stable.

 

5. Energize the affected part fully using your finger mini chakra. Project the

energy as a thin laser-like stream, as thin as the tip of a ball point pen.

Use LWG, LWB then gold.

 

Apply steps 1 to 5, three to four times daily for the next several days until

there is substantial improvement.

 

6. Apply the APH " Enhancing the Immunity and Defense System " .

 

7. Stabilize and release projected energy.

 

8. Repeat entire treatment 3 times per week for as long as necessary.

 

Love,

 

Marilette

 

 

 

 

 

 

 

 

 

 

Source: MASTER CHOA KOK SUI - Miracles Through Pranic Healing, Advanced Pranic

Healing, Pranic Psychotherapy, Pranic Crystal Healing.

 

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NOTICE:

1. Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or if the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer.

2. Pranic Healers who are are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments. ~ Master Choa

Kok Sui

 

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