Stargardt's disease - fundus flavimaculatus

[Guest guest]

Hello Dear Marilette,

 

I have also requested this protocol from the website, but as I have not

had a reply I am trying through my e-mail address.

 

I hope you do not mind.

 

This lady has stargadts type three disease in her eyes. The central vision is

effected and therefore she finds she can not read or recognise people's faces

with her left eye.

Her right eye is also effected but is not as bad as the left one

 

After the healing,She was able to see a bit better with her left eye but this

was only temporary.

 

However, it is not blurred and she can see something but sometimes it is

difficult to read with her left eye.

 

 

She has had 2 to 3 healings of pranic healing But I would like a proper protocol

with a better understanding of the condition of her eyes.

 

Awaiting your reply,

 

thanking you in anticipation

 

With Kind Regards.

 

Love

Gita Shah

 

=================================

Dear Gita,

 

Namaste.

 

Thank you for your email.

 

Medical Background:

 

Stargardt's disease (also known as fundus flavimaculatus and Stargardt's macular

dystrophy) is the most common form of inherited juvenile macular degeneration.

Inherited as an autosomal recessive trait, it is a severe form of MD that begins

in late childhood, leading to legal blindness. Stargardt's disease is

symptomatically similar to age-related macular degeneration, and it affects

approximately one in 10,000 children.

 

Stargardt's disease is usually diagnosed in individuals under the age of twenty,

when decreased central vision is first noticed. It causes a progressive loss of

central vision and, in the early stages, patients may have good visual acuity,

but they may experience difficulty with reading and seeing in dim lighting.

Other common symptoms of Stargardt's disease include blurriness and distortion.

On examination, the ophthalmological findings vary significantly with the

progression of the disease. In fundus photos, patients with early Stargardt's

disease appear to have simple macular degeneration. Children with the disease

typically begin experiencing dark adaptation problems and central vision loss

between six and twelve years of age, but symptoms may also first appear in

adulthood.

 

As the disease progresses, lipid rich deposits accumulate in the retinal pigment

epithelium (RPE) layer beneath the macula. This " lipofuscin " appears as

yellowish-tinted flecks. The RPE is a layer of cells that lies between the

retina and the choroid, where it serves the purpose of nourishing the

photoreceptor cells. In advanced Stargardt's disease, the buildup of lipofuscin

causes atrophy of the macula and the underlying RPE. The progression of vision

loss is variable and can start with a visual acuity of 20/40 and decrease

rapidly (especially in children) to 20/200 (legal blindness). By age 50,

approximately 50% of all of those studied in clinical trials had visual acuities

of 20/200 to 20/400. In late stages of this disease, there may also be color

vision impairment.

 

Stargardt's disease is almost always inherited as an autosomal recessive

disorder, with only ten percent of cases resulting from a dominant mode of

inheritance. Autosomal recessive means that both parents are carriers, having

one gene for the disease paired with one normal gene. As a consequence, each of

their children has a 25 percent chance of inheriting the two copies of the

Stargardt gene (one from each parent) needed to cause the disease. Carriers are

unaffected because they have only one copy. At this time, it is impossible to

determine who is a carrier for Stargardt's disease until after an affected child

is diagnosed.

 

In 1997, researchers isolated the gene for Stargardt's disease. The ABCA4 gene

produces a protein involved in energy transport to and from photoreceptor cells

in the retina. Mutations in the ABCA4 gene, which cause Stargardt's disease,

produce a dysfunctional protein that cannot perform its transport function. As a

result, photoreceptor cells degenerate, and vision loss occurs. One of nineteen

mutations in the gene (causing deletions and substitutions of amino acids) has

been identified to cause Stargardt's disease. The non-functional ABCA4 protein

permits the accumulation of yellow fatty material to accumulate in the retina.

This material causes flecks and, ultimately, loss of vision. Further research is

needed to find out how the mutated ABCA4 genes affect the biochemistry of the

retina and lead to vision loss.

 

This discovery allows researchers to study the underlying biochemical

interactions that result from mutations in this gene. Understanding how genetic

mutations lead to retinal degeneration is critical for the development of

experimental therapies.

 

Current research also shows that patients with Stargardt's disease could slow

its progression by wearing UV-protective sunglasses and avoiding exposure to

bright light. Researchers have observed that mice which had a mutation of the

ABCA4 gene, and which were reared in dark environments had virtually no

lipofuscin deposits.

 

The disease is often misdiagnosed, or not diagnosed in the first few years of

onset, and this could be the result of little evidence being found during eye

examinations. The discovery of the Stargardt gene could help in a test for the

direct diagnosis of the disease. It is possible that the effect of this newly

discovered gene may not be limited only to juvenile MD, in that it could also

aid in the search for causes for age-related macular degeneration, the leading

cause of vision loss in people over age 65.

 

Currently, there is no effective treatment for Stargardt's disease, but having

the genetic " instruction manual " may assist in developing new strategies for

therapy. It is also important that the learning and working environment be

adapted for people with Stargardt's disease. Appropriate low vision aids and

lighting are two important considerations for helping both children and adults

to function as normally as possible.

 

Source: Macular Degeneration Support Library, www.mdsupport.org

 

Pranic Healing:

 

Pranic Treatment:

>

> 1. Invoke and scan before, during and after

> treatment.

>

> 2. Teach the patient proper pranic breathing.

> Instruct the patient to do 12 cycles before start of

> treatment.

> Continue pranic breathing during entire treatment.

>

> 3. General sweeping.

>

> 4. Localized thorough sweeping on the front and back

> solar plexus chakra and liver.

> Energize solar plexus with LWG, LWB then LWV.

>

> 5. Localized thorough sweeping on the front and back

> heart chakras. Energize through the back heart with

> LWG and then with more of ordinary LWV.

>

> 6. Localized thorough sweeping on both eyes

> alternately with LWG and ordinary LWV. Visualize the

> energy penetrating as you gently sweep the blood

> vessels behind the retina.

>

> 7. Project LWG on the ajna and back head chakras.

> Localized thorough sweeping on the ajna chakra and

> back head minor chakra. Energize the eyes through

> the

> ajna chakra and the back head minor chakra with LWGV

> (

> 70% white, 20% ordinary violet and 10% green)then

> with

> Gold.

>

> 8. Localized thorough sweeping on the entire head

> area, the forehead chakra, temple minor chakras and

> crown chakra. Energize the chakras with ordinary

> LWV.

>

> 9. Localized thorough sweeping on the lungs. Apply

> the blood cleansing technique.

>

> 10. Localized thorough sweeping on the arms and legs

> with emphasis on their minor chakras, the navel

> chakra, sex chakra and basic chakra. Energize the

> chakras with LWR.

>

> 11. Localized thorough cleansing on the front and

> back spleen chakra, the meng mein chakra and both

> kidneys.

>

> Energize the kidneys with white.

>

> 12. Stabilize and release projected pranic energy.

>

> Repeat treatment three times per week for as long as

> necessary.

>

> Recommmend:

>

> 1. Diet to include more fresh fruits and fresh dark

> leafy vegetables, with proper amounts of fresh water

> .

>

> 2. Regular physical exercise.

>

> 3. No smoking.

>

> 4. Consult a medical practioner for multi vitamins food

> suppliments with zinc.

>

> 5. Regular proper practice of the Meditation on Twin

> Hearts unless there are other medical or health

> conditions that do not permit the practice of the

> Meditation.

>

> 6. Avoid stress and over-working the eyes.

>

> 7. Regular practice of proper Pranic Breathing for 5

> minutes everyday, several times per day to improve

> the

> energy level and help deal with stress.

>

> Love,

>

> Marilette

>

 

Source: MASTER CHOA KOK SUI - Miracles Through Pranic Healing, Advanced Pranic

Healing, Pranic Psychotherapy, Pranic Crystal Healing.

 

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NOTICE:

1. Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or if the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer.

2. Pranic Healers who are are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments. ~ Master Choa

Kok Sui

 

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