Guest guest Posted September 7, 2006 Report Share Posted September 7, 2006 Namaste Marilette, One of the healers in our group has been diagnosed with both Hepatic Hemoangioma and Hughes syndrome. We'd appreciate it a whole lot if you could help us with a good healing protocol for her... Thank you very much in advance, and may you always be blessed! Jaime Graterol PH Mexico ========================================== Dear Jaime, Namaste. Thank you for your email. Medical Background: The antiphospholipid antibody syndrome, also known as Hughes Syndrome, is a disorder characterized by multiple different antibodies that are associated with both arterial and venous thrombosis (clots). There are three primary classes of antibodies associated with the antiphospholipid antibody syndrome: 1) anticardiolipin antibodies, 2) the lupus anticoagulant and 3) antibodies directed against specific molecules including a molecule known as beta-2-glycoprotein 1. Historically, antiphospholipid antibodies were first noted in patients who had positive tests for syphilis without signs of infection. Subsequently, a clotting disorder was associated with two patients with systemic lupus erythematosus in 1952. In 1957 a link between recurrent pregnancy loss and what is now called the lupus anticoagulant was established. Ultimately, the lupus anticoagulant was further described in 1963 and in 1972 the term lupus anticoagulant was given. In 1983, Dr. Graham Hughes described the association between antiphospholipid antibodies and arterial as well as venous thrombosis. There are two main classifications of the antiphospholipid antibody syndrome. If the patient has an underlying autoimmune disorder, such as systemic lupus erythematosus, the patient is said to have secondary antiphospholipid antibody syndrome. If the patient has no known underlying autoimmune disorder, it is termed primary antiphospholipid antibody syndrome. Mechanism of the Antiphospholipid Antibody Syndrome: The antiphospholipid antibody syndrome is an autoimmune phenomenon. The immune system's function is to watch for and defend against foreign substances in the human body (for instance, bacteria or viruses). One component of this defence system is the antibody. An antibody is a protein that can recognize and bind to a foreign substance. Once it has bound to this substance, it can attract other molecules and cells to destroy the offending molecule. In some disease states, the immune system is not able to differentiate between foreign invading substances and normal components of the body; this is referred to as autoimmunity. There are a number of well known autoimmune disorders, including systemic lupus erythematosus, and studies on other diseases have suggested autoimmune components in a number of other illnesses. In the antiphospholipid antibody syndrome, the body produces antibodies that recognize various molecules in the body that, under normal circumstances, it would not. These molecules (phospholipids for example) play a role in the coagulation cascade along with other functions. The exact mechanism by which the antiphospholipid antibodies and anticardiolipin antibodies induce thrombophilic state is not known. A great deal of research is being done to explore the interactions these antibodies have with the components of the coagulation cascade and ultimately their role in the hypercoaguable state. At this time, there are numerous theories as to how these antibodies cause a hypercoaguable state; each of these theories has supporting evidence and evidence that calls it into question. For reference, a depiction of the clotting process is below. Epidemiology of the Antiphospholipid Antibody Syndrome: The prevalence in the general population is around 2-4%. Of patients with the antiphospholipid antibody syndrome, over half (50%) of them have the primary antiphospholipid antibody syndrome. In persons with systemic lupus erythematosus, around 30% will develop the antiphospholipid antibody syndrome. In general, anticardiolipin antibodies are more common that the lupus anticoagulant; anticardiolipin antibodies occurs approximately 5 times more often than the lupus anticoagulant in patients with the antiphospholipid antibody syndrome. In patients with an initial presentation of primary antiphospholipid antibody syndrome, around 10% will eventually go on to be diagnosed with an autoimmune disorder such as systemic lupus erythematosus or a mixed connective tissue disorder. Risks of the Antiphospholipid Antibody Syndrome: The role of the antiphospholipid antibody syndrome in both arterial and venous thrombotic disorders is an active area of clinical research. To date, studies examining the role of the antiphospholipid antibody syndrome in thrombosis are numerous. Clearly, the antiphospholipid antibody syndrome is associated with both arterial and venous thrombosis. However, a review of the literature clearly demonstrates continued controversy regarding the degree of risk these antibodies confer. Studies have not shown any clear differences between patients with the primary antiphospholipid antibody syndrome versus the secondary antiphospholipid antibody syndrome. A risk of recurrent thrombi, both arterial and venous, is associated with the antiphospholipid antibody syndrome as well. Most studies suggest that patients who have a recurrent episode will have it in a similar blood vessel type. In other words, patients who have a stroke initially will most often have a stroke if they have a recurrence. None-the-less, patients are reported that have multiple different types of thrombotic events. The antiphospholipid antibody syndrome is also associated with miscarriages as well as other complications of pregnancy including preterm labor and preeclampsia. An association with thrombocytopenia (low platelets) has also been established. This occurs in 20-40% of patients with the antiphospholipid antibody syndrome. Treatment of the Antiphospholipid Antibody Syndrome: Studies of the optimal treatment for the antiphospholipid antibody syndrome are currently under way. Treatment of the initial thrombosis in patients with the antiphospholipid antibody syndrome does not generally differ from treatment of patients with the same disorder who do not have the antiphospholipid antibody syndrome. Anticoagulation with heparin and then subsequently with oral anticoagulation is initiated. The duration of anticaogulation in patients without the antiphospholipid antibody syndrome is generally 3-6 months. In patients with the antiphospholipid antibody syndrome, the risk of recurrence is relatively high for both arterial and venous thrombotic events. As a result, patients are generally started on long-term (in some cases life-long) oral anticoagulation. The treatment of women who are pregnant and have the antiphospholipid antibody syndrome can result in a much higher success rate for the pregnancy. Several regimens have been studied including heparin. The role of medications generally used in autoimmune disorders to try and control the immune system is extremely limited. The primary role of these medications is in patients who have secondary antiphospholipid antibody syndrome, and they generally have no effect on the antiphospholipid antibody syndrome, but can help control the systemic lupus erythematosus, for example. Anti-platelet drugs, such as aspirin, are also used. At this time, a large study looking at the use of aspirin versus oral anticoagulation is underway in patients with the antiphospholipid antibody syndrome and stroke. Use of low-molecular-weight heparins instead of warfarin or in combination with other medications is also sometimes used. In the case of patients who are discovered to have the antiphospholipid antibodies without any known thrombotic problems, the question of preventative (prophylactic) treatment is unresolved. Currently, aspirin is the general recommendation. The use of long-term anticoagulation has risks associated with it (approximately a 3% chance per year of having a major hemorrhage, of which approximately 1/5 are fatal). Beginning long-term anticoagulation is influenced by the patient's overall risk of recurrent thrombosis balanced against the risks associated with long-term anticoagulation on an individual basis. Pregnancy and the Antiphospholipid Antibody Syndrome: As mentioned above, the antiphospholipid antibody syndrome is associated with complications in pregnancy. These complications can include miscarriages, preterm labor, low birth-weight and preeclampsia. A hemangioma is a benign tumor consisting of a mass of blood or lymphatic vessels; some appear as birthmarks. Source - University of Illinois - Urbana/Champaign Carle Cancer Center, Hematology Resource Page Pranic Healing: 1. Invoke and scan before, during and after treatment. 2. Instruct the patient to do 12 cycles of Pranic Breathing before start of treatment, then to continue Pranic Breathing during treatment. 3. After the patient has completed 12 cycles of Pranic Breathing, apply general sweeping several times using LWG. 4. Localized thorough sweeping and energizing on the ajna chakra with EV. 5. Localized thorough sweeping on the front, sides and back of the lungs. Energize through the back of the lungs with LWG, LWO then LWR. 6. Localized thorough sweeping on the basic chakra alternately with LWG and LWO. Energize the basic chakra with LWR. 7. Localized thorough sweeping on the arms and legs and their minor chakras alternately with LWG and LWO. Energize the minor chakras of the arms and legs with LWR. 8. Localized thorough sweeping on the affected parts alternately with LWG and LWO. Energize the affected part with LB for localizing effect. Energize the affected part with LWG then LWO. If the affected part is on the head, near the heart or spleen, just cleanse and energize the affected part with EV. 9. Localized thorough sweeping on the front and back heart chakra. Energize through the back heart chakra with LWG then more of ordinary LWV. 10. Localized thorough sweeping on the front and back solar plexus chakra. Energize with LWG then with more of ordinary LWV. For experienced advanced Pranic Healers, apply localized thorough sweeping on the front and back solar plexus chakra and on th eliver alternately with LWG and LWO. Energize the solar plexus chakra with LWB, LWG then LWO. 11. Localized thorough sweeping on the front and back spleen chakra with LWG. Energize the spleen chakra with LWG then with ordinary LWV. This has to be done with caution. 12. Localized thorough sweeping on the kidneys alternately with LWG and LWO. Energize the kidneys with LWR. 13. Localized thorough sweeping on the meng mein chakra. 14. Localized thorough sweeping on the throat chakra alternately with LWG and ordinary LWV. Energize with LWG then with more of ordinary LWV. 15. Stabilize and release projected pranic energy. 16. Play the Meditation on Twin Hearts CD. Instruct the patient to follow the guided meditation. 17. Repeat entire treatment 3 times per week. For the patient: 18. Practice forgiveness, loving kindness and mercy towards yourself and towards others. 19. Practice the Meditation on Twin Hearts properly everyday, including the physical exercises before and after the meditation. 20. Bathe in water with salt daily before meditation and before Pranic Healing treatment. Love, Marilette Source materials for all MCKS Pranic Healing protocols are exclusively from the following books by Master Choa Kok Sui: Miracles Through Pranic Healing, Advanced Pranic Healing, Pranic Psychtherapy and pranic Crystal Healing. NOTICE: 1. Pranic Healing is not intended to replace orthodox medicine, but rather to complement it. If symptoms persist or if the ailment is severe, please consult immediately a medical doctor and a Certified Pranic Healer. 2. Pranic Healers who are are not medical doctors should not prescribe nor interfere with prescribed medications and/or medical treatments. ~ Master Choa Kok Sui Miracles do not happen in contradiction to nature, but only to that which is known to us in nature. ~ St. Augustine Ask or read the up to date Pranic Healing protocols by joining the group through http://health./ MCKS Pranic Healing gateway website: http://www.pranichealing.org. Quote Link to comment Share on other sites More sharing options...
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