Global Transient Amnesia (TGA), high white blood cell count

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Atma Namaste, I have a male patient, 53 years old who

has been diagnosed with Global Transient Amnesia, also

with a higher level of white blood cells, he is

otherwise healthy.

 

 

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Dear Magic,

 

Namaste.

 

Thank you for your email.

 

Medical Background:

 

The syndrome of TGA was described initially by Morris

Bender in the Journal of the Hillside Hospital in

1956. Fisher and Adams later wrote extensively about

TGA in Acta Neurologica Scandinavica in 1964. Since

that time, TGA has become a well-described syndrome,

but one whose exact etiology is not yet completely

understood.

 

TGA specifically affects memory function. Patients can

register information, but retentive memory ability is

affected dramatically. Patients are generally 50

years old and over.

 

Many mechanisms have been proposed, but no single

cause can explain fully all the features of TGA.

 

These include migraine variant, temporal lobe seizure,

and TIA. If a patient is young or has repeated

attacks, then the possibility of seizure or even

migraine is higher. Some authors have stated that

patients with TGA have age and risk factor profiles

similar to those of patients with stroke or TIA

(Shuping et al), but patients with TGA have a low

incidence of strokes on follow-up.

 

Precipitants of TGA frequently include

-physical exertion,

-overwhelming emotional stress,

-pain,

-cold-water exposure,

-sexual intercourse,

-and Valsalva maneuver.

 

These triggers may have a common physiologic feature:

increased venous return to the superior vena cava.

 

-The effects of drugs must be considered. For

instance, sedative-hypnotic medications, either

over-the-counter or prescribed for sleep (especially

if used in conjunction with a transoceanic flight), or

premedication with midazolam for medical procedures,

may cause similar symptoms. Excessive alcohol can

cause a blackout phenomenon. Hence, any history of

drug-related amnesia may help clarify mitigating

causes.

 

-Social history and family history is relevant.

Pantoni et al found that patients with TGA have a

higher incidence of personal or family background of

psychiatric conditions compared with patients who have

had a TIA. Prognostically, patients with TGA are less

likely to experience a cardiovascular or

cerebrovascular event compared with patients who have

had a TIA.

 

Neurologic examination of the patient typically fails

to demonstrate any abnormalities (other than memory

dysfunction).

 

The exact mechanism that produces TGA is unclear.

 

The most compelling evidence in favor of migraine is

that patients who suffer from a TGA event have a

slightly higher incidence of a previous migraine.

 

However, patients with TGA rarely report an associated

headache.

 

They also do not report nausea, photophobia, or

phonophobia.

 

Seizure (eg, temporal lobe) is unlikely.

 

TGA events are not associated with alteration of

consciousness or stereotypical movements.

 

EEG does not demonstrate epileptiform activity.

 

TIA as indicative of cerebrovascular disease is

unlikely.

 

Studies have demonstrated that patients with TGA have

fewer cerebrovascular risk factors than those with

known cerebrovascular or coronary artery disease.

 

The prognosis for TGA is often better than for TIAs.

 

One theory proposed by Lewis is that venous congestion

causes disrupted blood flow to the thalamic or mesial

temporal structures.

 

The frequently cited triggers for TGA can increase

either sympathetic activity and/or intrathoracic

pressure.

 

This, in turn, could cause back-pressure in the

jugular venous system, disrupting intracranial

arterial flow with secondary venous

congestion/ischemia to memory areas in the brain.

 

Conditions predisposing to this scenario might include

venous anatomy anomalies, integrity of jugular vein

valves, timing of the trigger, and severity of the

inciting event. In support of the above concept of

venous congestion is Schreiber et al's finding of a

higher prevalence of internal jugular vein valve

incompetence in patients with TGA versus normal

controls. However, the authors of this study could

find no particular internal jugular vein valve

incompetence associated venous circulatory patterns

that could indicate a direct cause/effect with TGA.

 

Reference: Roy Sucholeiki, MD

 

 

Pranic Healing:

 

1. Invoke and scan before, during and after

treatment.

 

2. Teach the patient proper deep abdominal pranic

bretahing 6-3-6-3. Ask the patient to do 12 cycles

before start of treatment and continue prani

cbreathing during treatment.

 

3. After patient has done 12 cycles of pranic

breathing, apply general sweeping.

 

4. Localized thorough sweeping on the front and back

solar plexus chakra and the liver. Energize the solar

plexus with LWG, LWB then ordinary LWV.

 

5. Localized thorough sweeping on the front, sides

and back of the lungs. Energize the lungs directly

through the back of the lungs with LWG then with more

of LWO. Point your fingers away from the patient's

head when energizing with O.

 

6. Localized thorough sweeping on the front and back

spleen chakra and the kidneys.

 

7. Localized thorough sweeping on the spine, the

upper back and both sides of the neck.

 

8. Localized thorough sweeping on the front, sides

and back of the lungs. Energize the lungs directly

through the back of the lungs with LWG then with more

of LWO. Point your fingers away from the patient's

head when energizing with O.

 

9. Localized thorough sweeping on the front and back

heart chakra. Energize through the back heart chakra

with LWG then with more of ordinary LWV simultaneously

visualize the heart chakra becoming bigger and

brighter.

 

10. Localized thorough sweeping on the entire head,

the different sections of the brain, the ajna chakra,

crown chakra, forehead chakra, back head chakra and

throat chakra. Energize the chakras with LWG then

with more of ordinary LWV.

 

11. Localized thorough sweeping on the sex chakra,

navel chakra and basic chakra. Energize them with

LWR.

 

If the patient has fever, do not energize the basic.

 

If the patient has venereal disease, do not energize

the sex chakra.

 

12. Stabilize and release projected pranic energy.

 

13. Repeat treatment several times a day during an

episode. If not having an episode, apply treatment 3

times per week as preventive measure for as long as

necessary.

 

14. For the patient:

 

-Do pranic breathing for 12 cycles per session,

several sessions everyday.

 

-Practice the Meditation on Twin Hearts regularly to

energize and activate the brain cells and to promote

general well being..

 

-Do low impact physical exercise regularly.

 

-Eat healthy balanced fresh food diet, drink proper

amounts of fresh water daily.

 

Love,

 

Marilette

 

 

 

 

 

 

 

 

 

 

 

1. Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer.

 

2. Pranic Healers who are are not medical doctors should not prescribe nor

interfere with prescribed medications and/or medical treatments. ~ Master Choa

Kok Sui

 

Miracles do not happen in contradiction to nature, but only to that which is

known to us in nature. ~ St. Augustine

 

Reference material for Pranic Healing protocols are the following books

written by Master Choa Kok Sui:

Science and Art of Modern Pranic Healing, Advanced Pranic Healing, Pranic

Psychotherapy, Pranic Crystal Healing.

 

Ask or read the up to date Pranic Healing protocols by joining the group through

http://health./

 

MCKS Pranic Healing gateway website: http://www.pranichealing.org.