Myelodysplastic Syndrome -MDS, 80 yrs. old

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Dear Marilia,

 

Namaste.

 

Thank you for the additional information.

 

Scanning will indicate the energy condition and

requirement. The adjusted protocol below may be used

using white or color prana. If the the condition has

deteriorated to any form of leukemia, kindly apply the

protocol given for leukemia in the Advanced Pranic

Healing book by Master Choa Kok Sui.

 

Medical Background:

 

Myelodysplastic syndrome (MDS) refers to a

heterogeneous group of closely related clonal

hematopoietic disorders. All are characterized by a

cellular marrow with impaired morphology and

maturation (dysmyelopoiesis) and peripheral blood

cytopenias, resulting from ineffective blood cell

production.

 

The initial hematopoietic stem cell injury can be from

cytotoxic chemotherapy, radiation exposure, viral

infection, chemical exposure, or genetic

predisposition. A clonal mutation predominates over

bone marrow, suppressing healthy stem cells.

 

In early stages, the main cause of cytopenias is

increased apoptosis (programmed cell death). As the

disease progresses and converts into leukemia, a rare

gene mutation occurs and a proliferation of leukemic

cells overwhelms the healthy marrow.

 

MDS primarily affects elderly people, with the median

onset in the seventh decade of life. The median age of

these patients is 65 years, with ages ranging from the

early third decade of life to older than 80 years.

 

The syndrome may occur in persons of any age group,

including the pediatric population.

 

Causes:

 

-Primary, or idiopathic, MDSs are the most common.It

affects mainly over 60yo. However, a nonspecific

history of exposure to indeterminable chemicals or

radiation 10-15 years prior to onset of disease may be

present in approximately 50% of patients. This

relationship to pathogenesis remains unproved.

Other chemicals are leukemogenic.

Compounds such as benzene have been implicated.

Insecticides, weed killers, and fungicides are

possible causes of MDS and secondary leukemia.

Less evidence supports genetic predisposition, but

familial incidences have been described.

Viral infections have also been implicated. Affects

mainly over 60yo.

 

-Patients who survive malignancy treatment with

alkylating agents, with or without radiotherapy, have

a high risk of developing MDS or secondary acute

leukemia.

 

-Approximately 60-70% of patients do not have an

obvious exposure or cause for MDS and are classified

as primary MDS patients.

 

-Secondary MDS describes the development of MDS or

acute leukemia after known exposures to chemotherapy

drugs that can cause bone marrow damage. These drugs

are associated with a high prevalence of chromosomal

abnormalities (following exposure and at the time of

MDS or acute leukemia diagnosis).

 

Because the diagnostic criteria are new, an

international group of hematologists, the

French-American-British (FAB) Cooperative Group,

classified these disorders into 5 subgroups,

differentiating them from acute myeloid leukemia. An

underlying trilineage dysplastic change in the bone

marrow cells of the patients is found in all subtypes.

 

 

The 2 subgroups of refractory anemia (RA)

characterized by 5% or less myeloblasts in bone marrow

are (1) RA and (2) RA with ringed sideroblasts (RARS),

defined morphologically as having 15% erythroid cells

with abnormal ringed sideroblasts, reflecting an

abnormal iron accumulation in the mitochondria. Both

have a prolonged clinical course and a low prevalence

of progression to acute leukemia.

 

The 2 subgroups of RAs with greater than 5%

myeloblasts are (1) RA with excess blasts (RAEB),

defined as 6-20% myeloblasts, and (2) RAEB in

transformation (RAEB-T), with 21-30% myeloblasts. The

higher the percent of myeloblasts, the shorter the

clinical course and the closer the disease is to acute

myelogenous leukemia.

 

Patient transition from early to more advanced stages

indicates these subtypes are merely stages of disease

rather than distinct entities. Elderly patients with

MDS with trilineage dysplasia and greater than 30%

myeloblasts who progress to acute leukemia are often

considered to have poor prognoses because their

response to chemotherapy is worse than de novo acute

myeloid leukemia patients. The 1999 World Health

Organization (WHO) classification proposes to include

all cases of RAEB-T (patients with >20% myeloblasts)

in the category of acute leukemia because these

patients have similar prognostic outcomes. However,

their response to therapy is worse than patients with

the de novo or more typical acute myelogenous leukemia

or acute nonlymphocytic leukemia.

 

The fifth type of MDS, the most difficult to classify,

is called chronic myelomonocytic leukemia (CMML). This

subtype can have any percent of myeloblasts but

manifests as a monocytosis of 1000/mL or more. It may

be associated with splenomegaly. This subtype overlaps

with a myeloproliferative disorder (MPD) and may have

an intermediate clinical course. CMML must be

differentiated from the classic chronic myelocytic

leukemia, which is characterized by a negative Ph

chromosome. The 1999 WHO classification proposes that

juvenile and proliferative CMML be listed separately

from the FAB classification under MDS/MPD with

splenomegaly and greater than 13,000/mL total WBC

count. CMML in the FAB classification under MDS is

limited to monocytosis, has less than 13,000/mL total

leukocytes, and requires trilineage dysplasia.

 

A slight male predominance is noted in all age groups.

 

 

 

Median survival is from 9-29 months but some go 5 or

more years. Progression to AML is 10-40%. Outlook is

worse for therapy related MDS (4-8 month survival). Tx

is limited, mostly treat with antibiotics and blood

product transfusions.

 

Source: Dr. Emmanuel C Besa, MD, Coauthor(s): Ulrich

Woermann, MD,

 

 

 

Pranic Healing:

 

1. Invoke and scan before, during and after

treatment.

 

2. General sweeping several times.

 

3. Localized thorough sweeping on the front and

back heart chakra. Energize through the back heart

chakra with LWG then with more of ordinary LWV.

 

4. Localized thorough sweeping on the skull, the

spine and the ribs.

 

5. Localized thorough sweeping on the basic

chakra alternately with LWG and LWO.

Energize the basic chakra with LWR. Simultaneously

visuaiize the energy going into the spine, ribs and

skull bones.

 

6. Localized thorough sweeping on both arms and

both legs with emphasis on their minor chakras.

Energize the minor chakras of the arms and legs with

LWR while visualizing the energy going into the bones.

 

7. Localized thorough sweeping on the front and back

solar plexus chakra and the liver. Energize the

solar plexus chakra with LWG then with more of

ordinary

LWV.

 

8. Localized thorough cleansing on the front, sides

and back of the lungs. Energize the lungs directly

through the back of the lungs with LWG, LWO and LWR.

Point your fingers away from the patient's head when

energizing with O.

 

9. Localized thorough sweeping on the front and

back spleen chakra. Energize the spleen chakra with

LWG, then with ordinary LWV. This has to be done with

caution.

 

10. Localized thorough sweeping on the navel

chakra, the small and large intestines. Energize the

navel chakra with a little LWG, then with LWR.

 

11. Localized thorough sweeping on the meng mein

chakra and the kidneys.

Energize the kidneys with white.

 

12. Localized thorough sweeping on the sex

chakra.

Energize with white.

 

13. Localized thorough sweeping on the ajna, crown,

forehead and back head chakra.

Energize thee with a little of LWG then

with more of ordinary LWV.

 

14. Stabilize and release projected energy.

 

15. Repeat treatment twice per week or adjust

frequency based on the particular case's energy

requirement per your scanning.

 

Encourage the patient to practice the Meditation

on Twin Hearts regularly to raise his energy level and

facilitate healing. Considering patient's age,

Meditation may be practiced twice or once per week

with both eyes open and light physical exercise or

physiotherapy before and after meditation.

 

 

Source: Advanced Pranic Healing by Master Choa

Kok Sui.

 

Love,

 

Marilette

 

=======================================================

 

 

--- Marilia Tavares <mariliatavares

wrote:

 

> Dear Marilette,

> Namaste

>

> Thank you very much for the protocol you sent me.

> I have just found out the name of his desease,

> whish is mielodisplasy. I

> found mds name for this desease, which is

> myelodisplastic syndrome.

> I wonder if the protocol you sent me based on the

> symptoms is ok for this

> malady. Another question: since he is very old, is

> it ok to use bunch of

> colours or better to use only white, or maybe the

> lightest shade of color

> possible?

>

> The surgery he had some years ago was in his neck to

> correct a vein which

> was obstructing the flow of blood. Do not know

> itsname in english now...but

> can check if you need it.

> Thank you lots,

> Love,

> Marilia

>

>

 

 

=====

Pranic Healing is not intended to replace orthodox medicine, but rather to

complement it. If symptoms persist or the ailment is severe, please consult

immediately a medical doctor and a Certified Pranic Healer . ~ Master Choa Kok

Sui

 

Miracles do not happen in contradiction to nature, but only to that which is

known to us in nature. ~ St. Augustine

 

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