HIV cure cover up, for drug profits!

[Guest guest]

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab finding points to potential new

class of HIV treatments. Johns Hopkins scientists have found that simply

increasing manganese in cells can halt HIV's unusual ability to process

its genetic information backwards, providing a new way to target the

process's key driver, an enzyme called reverse transcriptase.

 

Some of the recent data on HIV shows that the trace metal balance in the

blood and cells is directly linked to the cure for HIV. Metals change

the viral replication factors within cells, See Ref below.

 

Fluorides in the body bind up these trace metals and make them insoluble

and not available for cell functions. The manganese and reverse

transcription factor is the main principle for the HIV infection of T-cells.

 

It comes down the Govt. helping to cover up the main cause of HIV due to

the fluorides-metals effects and this helping to make drug companies

huge profits for making reverse transcription drugs that can be

patented. The trace mineral methods are too simple for patents and the

reporting of this effect leads to huge liability problems for DOE and

its reckless fluoride-uranium operations.

 

Metals like gold and silver colloid compete for the fluorides in the

blood and complex with them to allow their rapid excretion as soluble

salts, and this process cuts down on the fluorides complexing with

manganese and other essential metals for the cellular functions.

 

The article below shows that the simple trace metal manganese blocks the

key infection method of reverse transcription in T-cells.

 

Take note that metals like manganese are directly linked to mad cow

brain effects. Perhaps this is the cow brain and immune response trying

to protect itself from viral invasion in a similar fashion?

 

Jim Phelps

 

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab finding points to potential new

class of HIV treatments

 

Johns Hopkins scientists have found that simply increasing manganese in

cells can halt HIV's unusual ability to process its genetic information

backwards, providing a new way to target the process's key driver, an

enzyme called reverse transcriptase. By measuring DNA produced by a

related reverse transcriptase in yeast, the Hopkins team discovered that

higher than normal levels of manganese, caused by a defective gene,

dramatically lowered the enzyme's activity. The scientists then proved

that HIV's reverse transcriptase responds to manganese in the same way.

 

Hopkins graduate student Eric Bolton determined that the defective gene

is PMR1, whose protein carries both manganese and calcium out of cells.

Using special yeast developed by others at Hopkins, he discovered that

manganese stops reverse transcriptase, the team reports in the April 26

issue of Molecular Cell.

 

" These results really point to a never-before-proposed way to try to

stop HIV in its tracks -- that simply manipulating concentrations of a

metal, manganese, can have a profound effect on reverse transcriptase, "

says Jef Boeke, Ph. D., professor of molecular biology and genetics at

the school's Institute for Basic Biomedical Sciences. " We expect the

human equivalent of PMR1 could be a good target for developing new drugs

against HIV. "