Mineral Balance tied to HIV cure]

[Guest guest]

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab

finding points to potential new class of HIV treatments. Johns Hopkins

scientists have found that simply increasing manganese in cells can

halt HIV's unusual ability to process its genetic information

backwards, providing a new way to target the process's key driver, an

enzyme called reverse transcriptase.

 

Some of the recent data on HIV shows

that the trace metal balance in the blood and cells is directly linked

to the cure for HIV. Metals change the viral replication factors within

cells, See Ref below.

 

Fluorides in the body bind up these

trace metals and make them insoluble and not available for cell

functions. The manganese and reverse transcription factor is the main

principle for the HIV infection of T-cells.

 

It comes down the Govt. helping to

cover up the main cause of HIV due to the fluorides-metals effects and

this helping to make drug companies huge profits for making reverse

transcription drugs that can be patented. The trace mineral methods are

too simple for patents and the reporting of this effect leads to huge

liability problems for DOE and its reckless fluoride-uranium operations.

 

Metals like gold and silver colloid

compete for the fluorides in the blood and complex with them to allow

their rapid excretion as soluble salts, and this process cuts down on

the fluorides complexing with manganese and other essential metals for

the cellular functions.

 

The article below shows that the

simple trace metal manganese blocks the key infection method of reverse

transcription in T-cells.

 

Take note that metals like manganese

are directly linked to mad cow brain effects. Perhaps this is the cow

brain and immune response trying to protect itself from viral invasion

in a similar fashion?

 

Jim Phelps

 

http://www.eurekalert.org/pub_releases/2002-04/jhmi-mbh042502.php

 

Manganese blocks HIV replication; Lab

finding points to potential new class of HIV treatments

 

Johns Hopkins scientists have found

that simply increasing manganese in cells can halt HIV's unusual

ability to process its genetic information backwards, providing a new

way to target the process's key driver, an enzyme called reverse

transcriptase. By measuring DNA produced by a related reverse

transcriptase in yeast, the Hopkins team discovered that higher than

normal levels of manganese, caused by a defective gene, dramatically

lowered the enzyme's activity. The scientists then proved that HIV's

reverse transcriptase responds to manganese in the same way.

 

Hopkins graduate student Eric Bolton

determined that the defective gene is PMR1, whose protein carries both

manganese and calcium out of cells. Using special yeast developed by

others at Hopkins, he discovered that manganese stops reverse

transcriptase, the team reports in the April 26 issue of Molecular Cell.

 

"These results really point to a

never-before-proposed way to try to stop HIV in its tracks -- that

simply manipulating concentrations of a metal, manganese, can have a

profound effect on reverse transcriptase," says Jef Boeke, Ph. D.,

professor of molecular biology and genetics at the school's Institute

for Basic Biomedical Sciences. "We expect the human equivalent of PMR1

could be a good target for developing new drugs against HIV."