Fw: Multi-D News Fw: [Mr_Tracys_Corner] [Fwd: FW: question]

[Guest guest]

Lotus

TheMulti-DimensionalNewsPortal

Sunday, April 11, 2004 8:54 AM

Re: Multi-D News Fw: [Mr_Tracys_Corner] [Fwd: Re: FW: question]

 

 

Hi All.

 

First off, quite horrifying evidence that TSEs are transmissable,

whatever the infectious agent is;

 

Mad Cow Disease/Kuru/CJD

In The Fore Tribe

1-2-4

 

Before and during World War II, at the infamous Camp 731

in Manchuria, the Japanese military contaminated prisoners of

war with certain disease agents.

 

They also established a research camp in New Guinea in 1942.

There they experimented upon the Fore Indian tribe and

inoculated them with a minced-up version of the brains of

diseased sheep containing the visna virus which causes

" mad cow disease " or Creutzfeldt-Jakob disease.

 

About five or six years later, after the Japanese had been

driven out, the poor people of the Fore tribe developed

what they called kuru, which was their word for " wasting " , and

they began to shake, lose their appetites and die. The autopsies

revealed that their brains had literally turned to mush. They had

contracted " mad cow disease " from the Japanese experiments.

 

When World War II ended, Dr Ishii Shiro- the medical doctor

who was commissioned as a General in the Japanese Army so he

could take command of Japan's biological warfare development,

testing and deployment- was captured. He was given the choice

of a job with the United States Army or execution as a war criminal.

Not surprisingly, Dr Ishii Shiro chose to work with the US military

to demonstrate how the Japanese had created mad cow disease

in the Fore Indian tribe.

 

In 1957, when the disease was beginning to blossom in full among

the Fore people, Dr Carleton

< /config/login?/gajdusek.html > Gajdusek

of the US National Institutes of Health headed to New Guinea to

determine how the minced-up brains of the visna-infected sheep

affected them. He spent a couple of years there, studying the Fore

people, and wrote an extensive report. He won the Nobel Prize

for " discovering " kuru disease in the Fore tribe.

http://www.whale.to/m/scott7.html

[Mycoplasma- The Linking Pathogen in Neurosystemic Diseases]

 

'After Gibbs et al. were able to show conclusively in 1990 that the

Mission cattle Texas had indeed been infected with scrapie, the

USDA repeated the experiment at an ARS (Agricultural Research

Service) facility in Ames, Iowa under the direction of Randall Cutlip.

Dr. Cutlip's results mirrored those found in the earlier study: some

of the cows inoculated with scrapie did die of a TSE, but they did

not exhibit behaviors associated with " mad cow " disease in Britain

(i.e. British BSE). Rather, the behavior is more subtle and could be

mistaken as " downer cow. " As Cutlip et al. concluded, " Thus,

undiagnosed scrapie infection could contribute to the 'downer-cow'

syndrome [in the U.S.] " (Cutlip et al., 1994: 814).

...

As of January 23, 1997, some 5,342 cattle brains had been tested.

Yet only a couple hundred of the brains came from downer cows.

...

Given that the USDA tested only 1,000 cows for BSE in 1999 and

2,000 cows in 2000 -- this is a miniscule number. Also, the number

of downer cows examined out of the 2,000 test is even smaller.

Remember that it's commonly expected that 100,000 U.S. cows

are " downer cows " each year, meaning the U.S. is testing a fraction

of a percent of this population. '

http://www.vegsource.com/articles/bse_consumer.htm

 

The BSE epidemic in the UK is thought to have been

transmitted to cattle from scrapie infected sheep and spread via

the ever infamous rendering/recycling system.

 

['TSEs are a mysterious class of diseases that are called by different

names in different species. For instance some identified types of TSE

are Creutzfeldt-Jakob Disease (CJD), and its specific strain, new

variant Creutzfeldt-Jakob Disease (nvCJD) which is a human disease

apparently caused by the same agent which causes bovine spongiform

encephalopathy (BSE) or British " mad cow " disease, Fatal Familial

Insomnia (FFA), Gerstmann-Straussler-Scheinker Syndrome (GSS),

scrapie in sheep, transmissible mink encephalopathy (TME) in mink in

North America, and chronic wasting disease (CWD) in deer and elk

in North America. There may be different strains of TSE within species,

and new strains may be produced when TSEs move from one animal

species to another.

 

The common characteristics of TSE diseases are that they are

invariably fatal. '

http://www.icta.org/legal/madcow.htm

 

Yes, what enabled the spread was (is) the use of ag'chems/toxins

 

+ ingestion by livestock of excess manganese via supplementation

and feed..

 

'... Cambridge scientist David R. Brown is hot on the trail.

His recent research has shown that the prion proteins linked

to BSE can bond destructively with manganese found in

animal feeds or mineral licks. His latest, as yet unpublished

work has found a tenfold increase in the metal manganese in

brains of CJD victims.

 

All this is fully consistent with the Purdey hypothesis. These

manganese-tipped prions could be the principal cause of the

neurological degeneration seen in BSE. But manganese is

only the bullet -- organophosphate insecticide is the high-velocity

gun. It fires manganese into the brain by depleting copper which

the manganese then replaces. Purdey says the manganese-tipped

prions set off lethal chain reactions that neurologically burn

through the animal. ..

 

A number of researchers have found that organophosphate (OP)

in systemic warble fly insecticide can deform the prion molecule,

rendering it ineffective at buffering free radical effects in the body.

Worse still, the prion is then partial to bond with manganese and

become a 'rogue' prion. A chain reaction whereby rogue prions

turn others to rogues also, can explain the bovine spongiform

disease mechanism.

 

Brown showed how prion protein bonds benignly with copper,

but lethally with manganese. Even natural variations in relative

environmental availability of manganese versus copper can trigger

prion degradation.

 

Chickens notoriously excrete most of the supplements fed to them

-- including manganese. And their manganese-rich excreta have

been blended into cattle feed in the UK.

...

Purdey has built evidence from around the world that explains

and predicts the incidence in humans and animals: a cluster of

CJD in Slovakia, Eastern Europe -- around a manganese plant;

Rocky Mountain deer with Chronic Wasting Disease (CWD),

who were found to be eating pine needles rich in manganese;

the futile slaughter of sheep in Cyprus -- only for BSE to

reemerge within years.

....'

Organophosphates Implicated In Mad Cow Disease

http://www.cqs.com/opmadcow.htm

 

You'll find that similar ag' practices have been prevalent in the US.

 

So how did deer and other wild ruminants in the US contract it?

 

? US scrapie transmission or imported feed containing the UK

mutated-scrapie BSE -> rendered/recycled back through US

system = 'downers' -->

 

'..The most troubling explanation for CWD's appearance came from

neither state agency but from veteran agricultural and environmental

writer Mike Irwin, freelancing in Madison's Capital Times. Irwin's

groundbreaking reporting linked CWD to a group of landowners in

western Dane County who, in 1990, began a concentrated effort at

deer management in order to raise " super " bucks. The landowners,

who controlled 12 abutting square-mile sections in the northwestern

part of the town of Vermont, agreed to give young bucks six years to

grow so they'd develop the imposing antlers and muscular bodies that

would get them into the record books. Then they began long-term

feeding of nutritional supplements to wild deer. Their effort succeeded:

Between 1990 and 2000, Dane County recorded the third-highest

number of trophy bucks in North America.

 

Up until August 1997, when the FDA, reacting to Britain's mad cow

epidemic, banned all ruminant-to-ruminant feeding (sheep, cattle,

goats, elk, deer, antelope and buffalo) in the United States,

Midwestern rendering plants routinely processed Wisconsin deer

(and 'downer')

carcasses into meat and bone meal that went into feed mill products

fed back to ruminants, including deer. (Cows, sheep and deer can

still legally be processed into bone and blood meal feed for pigs,

pets and chickens; then they can be rendered and fed back to

cows, deer and other ruminants.)

 

The feeding practice may have amplified the disease in the same way

feeding spread TSEs among the Fore people, Britain's cows and the

Wisconsin mink, something further suggested by the fact that 11 of

the first 18 cases of CWD found in Wisconsin came from the " super

buck " area. The connection is especially vexing because " that kind of

feeding has been going on all over the state, " says author Stauber,

" more evidence that CWD is spread all over Wisconsin. " The DNR

did ban feeding of deer statewide once CWD was discovered, but

by then, much of the damage had been done. ..............'

http://www.milwaukeemagazine.com/122002/cwd.html

 

Whatever the cause and infectious agent, it's a killer, and

it is potentially present in ANY animal derived products.

 

Could Mad Cow Disease Already be Killing Thousands of

Americans Every Year?

by Michael Greger, M.D.

January 7, 2004

http://www.commondreams.org/views04/0107-07.htm !!!

 

* Go vegan.*

 

(Better late than never- for the animals and the planet too!!)

 

Best wishes. G-B.

 

L.

 

 

-

walt

TheMulti-DimensionalNewsPortal

Sunday, April 11, 2004 12:21 PM

Multi-D News Fw: [Mr_Tracys_Corner] [Fwd: Re: FW: question]

 

Hi, all,

I include all of you in the following response because so many people have asked

about it.

 

Hi, Kath,

My publisher forwarded your email to me. Thank you for your interest in my work.

 

There is no medical evidence that folded prions cause Mad Cow. The symptoms of

Mad Cow are similar to mercury contamination that

occurs from vaccines. There seems to be several reasons for creating a new

disease. Firstly, it is a smokescreen that prevents

pointing the finger at the pharmaceutical industry for using mercury in vaccines

that causes the nervous system to disintegrate.

 

Secondly, my research, and that of reporter Jon Rappaport, shows that the major

genetic food industry is behind the scare with

agents in the FDA, USDA, and CDC. The business plan seems to be to destroy all

faith in natural animals and replace faith with fear.

Presenting genetically altered " safe " animals that are patented would mean that

the manufacturers would receive royalties for every

animal if they wish, and completely control the food industry. It seems they are

doing the same with agricultured food. So, for

Monsanto and Dow and others, destruction of unpatentable animals is not only

good business sense but a must. If we discard the

bacteria theory, they would not get away with it. But, as you can see, everyone

follows the postulate that we should fear the

bacteria that humans and all animals have been living with for millions of years

and trust 600,000 new chemicals that create " clean "

living. Those chemicals are causing most of the diseases that exist today, not

germs.

 

If you think that that is a far-fetched conspiracy theory, you might miss the

boat to good health.

 

I ate Mad Cow meat for 3 1/2 years while I lived in Paris off and on from

1993-1997 and suffered no ill effects.

 

healthfully,

aajonus

 

Paul Kruhm wrote:

 

 

Katherine Correa [katherinecorrea]

Monday, February 23, 2004 8:28 AM

welive

question

 

Hi

 

I have a question about mad cow disease. It seems dangerous to eat

meats in

general now that this prion disease is likely to be rampant- or so I am

told. What information do you have or can I get to understand the

situation?

 

Thankyou

Kath Correa

 

The homepage and the place to sign up for Tracy's Corner is:

Mr_Tracys_Corner

 

 

 

 

[Guest guest]

- Lotus

TheMulti-DimensionalNewsPortal

Sunday, April 11, 2004 8:54 AM

Re: Multi-D News Fw: [Mr_Tracys_Corner] [Fwd: Re: FW: question]

Hi aajonus, All.First off, quite horrifying evidence that TSEs are transmissable,whatever the infectious agent is;Mad Cow Disease/Kuru/CJDIn The Fore Tribe1-2-4Before and during World War II, at the infamous Camp 731in Manchuria, the Japanese military contaminated prisoners ofwar with certain disease agents.They also established a research camp in New Guinea in 1942.There they experimented upon the Fore Indian tribe andinoculated them with a minced-up version of the brains ofdiseased sheep containing the visna virus which causes"mad cow disease" or Creutzfeldt-Jakob disease.About five or six years later, after the Japanese had beendriven out, the poor people of the Fore tribe developedwhat they called kuru, which was their word for "wasting", andthey began to shake, lose their appetites and die. The autopsiesrevealed that their brains had literally turned to mush. They hadcontracted "mad cow disease" from the Japanese experiments.When World War II ended, Dr Ishii Shiro- the medical doctorwho was commissioned as a General in the Japanese Army so hecould take command of Japan's biological warfare development,testing and deployment- was captured. He was given the choiceof a job with the United States Army or execution as a war criminal.Not surprisingly, Dr Ishii Shiro chose to work with the US militaryto demonstrate how the Japanese had created mad cow diseasein the Fore Indian tribe.In 1957, when the disease was beginning to blossom in full amongthe Fore people, Dr Carleton< /config/login?/gajdusek.html > Gajdusekof the US National Institutes of Health headed to New Guinea todetermine how the minced-up brains of the visna-infected sheepaffected them. He spent a couple of years there, studying the Forepeople, and wrote an extensive report. He won the Nobel Prizefor "discovering" kuru disease in the Fore tribe.http://www.whale.to/m/scott7.html[Mycoplasma- The Linking Pathogen in Neurosystemic Diseases]'After Gibbs et al. were able to show conclusively in 1990 that theMission cattle Texas had indeed been infected with scrapie, theUSDA repeated the experiment at an ARS (Agricultural ResearchService) facility in Ames, Iowa under the direction of Randall Cutlip.Dr. Cutlip's results mirrored those found in the earlier study: someof the cows inoculated with scrapie did die of a TSE, but they didnot exhibit behaviors associated with "mad cow" disease in Britain(i.e. British BSE). Rather, the behavior is more subtle and could bemistaken as "downer cow." As Cutlip et al. concluded, "Thus,undiagnosed scrapie infection could contribute to the 'downer-cow'syndrome [in the U.S.]" (Cutlip et al., 1994: 814)...As of January 23, 1997, some 5,342 cattle brains had been tested.Yet only a couple hundred of the brains came from downer cows...Given that the USDA tested only 1,000 cows for BSE in 1999 and2,000 cows in 2000 -- this is a miniscule number. Also, the numberof downer cows examined out of the 2,000 test is even smaller.Remember that it's commonly expected that 100,000 U.S. cowsare "downer cows" each year, meaning the U.S. is testing a fractionof a percent of this population. 'http://www.vegsource.com/articles/bse_consumer.htmThe BSE epidemic in the UK is thought to have beentransmitted to cattle from scrapie infected sheep and spread viathe ever infamous rendering/recycling system.['TSEs are a mysterious class of diseases that are called by differentnames in different species. For instance some identified types of TSEare Creutzfeldt-Jakob Disease (CJD), and its specific strain, newvariant Creutzfeldt-Jakob Disease (nvCJD) which is a human diseaseapparently caused by the same agent which causes bovine spongiformencephalopathy (BSE) or British "mad cow" disease, Fatal FamilialInsomnia (FFA), Gerstmann-Straussler-Scheinker Syndrome (GSS),scrapie in sheep, transmissible mink encephalopathy (TME) in mink inNorth America, and chronic wasting disease (CWD) in deer and elkin North America. There may be different strains of TSE within species,and new strains may be produced when TSEs move from one animalspecies to another.The common characteristics of TSE diseases are that they areinvariably fatal. 'http://www.icta.org/legal/madcow.htmYes, what enabled the spread was (is) the use of ag'chems/toxins+ ingestion by livestock of excess manganese via supplementationand feed..'... Cambridge scientist David R. Brown is hot on the trail.His recent research has shown that the prion proteins linkedto BSE can bond destructively with manganese found inanimal feeds or mineral licks. His latest, as yet unpublishedwork has found a tenfold increase in the metal manganese inbrains of CJD victims.All this is fully consistent with the Purdey hypothesis. Thesemanganese-tipped prions could be the principal cause of theneurological degeneration seen in BSE. But manganese isonly the bullet -- organophosphate insecticide is the high-velocitygun. It fires manganese into the brain by depleting copper whichthe manganese then replaces. Purdey says the manganese-tippedprions set off lethal chain reactions that neurologically burnthrough the animal. ..A number of researchers have found that organophosphate (OP)in systemic warble fly insecticide can deform the prion molecule,rendering it ineffective at buffering free radical effects in the body.Worse still, the prion is then partial to bond with manganese andbecome a 'rogue' prion. A chain reaction whereby rogue prionsturn others to rogues also, can explain the bovine spongiformdisease mechanism.Brown showed how prion protein bonds benignly with copper,but lethally with manganese. Even natural variations in relativeenvironmental availability of manganese versus copper can triggerprion degradation.Chickens notoriously excrete most of the supplements fed to them-- including manganese. And their manganese-rich excreta havebeen blended into cattle feed in the UK...Purdey has built evidence from around the world that explainsand predicts the incidence in humans and animals: a cluster ofCJD in Slovakia, Eastern Europe -- around a manganese plant;Rocky Mountain deer with Chronic Wasting Disease (CWD),who were found to be eating pine needles rich in manganese;the futile slaughter of sheep in Cyprus -- only for BSE toreemerge within years....'Organophosphates Implicated In Mad Cow Diseasehttp://www.cqs.com/opmadcow.htmYou'll find that similar ag' practices have been prevalent in the US.So how did deer and other wild ruminants in the US contract it?? US scrapie transmission or imported feed containing the UKmutated-scrapie BSE -> rendered/recycled back through USsystem = 'downers' -->'..The most troubling explanation for CWD's appearance came fromneither state agency but from veteran agricultural and environmentalwriter Mike Irwin, freelancing in Madison's Capital Times. Irwin'sgroundbreaking reporting linked CWD to a group of landowners inwestern Dane County who, in 1990, began a concentrated effort atdeer management in order to raise "super" bucks. The landowners,who controlled 12 abutting square-mile sections in the northwesternpart of the town of Vermont, agreed to give young bucks six years togrow so they'd develop the imposing antlers and muscular bodies thatwould get them into the record books. Then they began long-termfeeding of nutritional supplements to wild deer. Their effort succeeded:Between 1990 and 2000, Dane County recorded the third-highestnumber of trophy bucks in North America.Up until August 1997, when the FDA, reacting to Britain's mad cowepidemic, banned all ruminant-to-ruminant feeding (sheep, cattle,goats, elk, deer, antelope and buffalo) in the United States,Midwestern rendering plants routinely processed Wisconsin deer(and 'downer')carcasses into meat and bone meal that went into feed mill productsfed back to ruminants, including deer. (Cows, sheep and deer canstill legally be processed into bone and blood meal feed for pigs,pets and chickens; then they can be rendered and fed back tocows, deer and other ruminants.)The feeding practice may have amplified the disease in the same wayfeeding spread TSEs among the Fore people, Britain's cows and theWisconsin mink, something further suggested by the fact that 11 ofthe first 18 cases of CWD found in Wisconsin came from the "superbuck" area. The connection is especially vexing because "that kind offeeding has been going on all over the state," says author Stauber,"more evidence that CWD is spread all over Wisconsin." The DNRdid ban feeding of deer statewide once CWD was discovered, butby then, much of the damage had been done. ..............'http://www.milwaukeemagazine.com/122002/cwd.htmlWhatever the cause and infectious agent, it's a killer, andit is potentially present in ANY animal derived products.Could Mad Cow Disease Already be Killing Thousands ofAmericans Every Year?by Michael Greger, M.D.January 7, 2004http://www.commondreams.org/views04/0107-07.htm !!!* Go vegan.*(Better late than never- for the animals and the planet too!!)Best wishes. G-B.L.-waltTheMulti-DimensionalNewsPortal Sent: Sunday, April 11, 2004 12:21 PMMulti-D News Fw: [Mr_Tracys_Corner] [Fwd: Re: FW: question]Hi, all,I include all of you in the following response because so many people have asked about it.Hi, Kath,My publisher forwarded your email to me. Thank you for your interest in my work.There is no medical evidence that folded prions cause Mad Cow. The symptoms of Mad Cow are similar to mercury contamination thatoccurs from vaccines. There seems to be several reasons for creating a new disease. Firstly, it is a smokescreen that preventspointing the finger at the pharmaceutical industry for using mercury in vaccines that causes the nervous system to disintegrate.Secondly, my research, and that of reporter Jon Rappaport, shows that the major genetic food industry is behind the scare withagents in the FDA, USDA, and CDC. The business plan seems to be to destroy all faith in natural animals and replace faith with fear.Presenting genetically altered "safe" animals that are patented would mean that the manufacturers would receive royalties for everyanimal if they wish, and completely control the food industry. It seems they are doing the same with agricultured food. So, forMonsanto and Dow and others, destruction of unpatentable animals is not only good business sense but a must. If we discard thebacteria theory, they would not get away with it. But, as you can see, everyone follows the postulate that we should fear thebacteria that humans and all animals have been living with for millions of years and trust 600,000 new chemicals that create "clean"living. Those chemicals are causing most of the diseases that exist today, not germs.If you think that that is a far-fetched conspiracy theory, you might miss the boat to good health.I ate Mad Cow meat for 3 1/2 years while I lived in Paris off and on from 1993-1997 and suffered no ill effects.healthfully,aajonusPaul Kruhm wrote:Katherine Correa [katherinecorrea]Monday, February 23, 2004 8:28 AMweliveSubject: questionHiI have a question about mad cow disease. It seems dangerous to eatmeats ingeneral now that this prion disease is likely to be rampant- or so I amtold. What information do you have or can I get to understand thesituation?ThankyouKath CorreaThe homepage and the place to sign up for Tracy's Corner is: Mr_Tracys_CornerFor complaints or assistance contact xootsuit26