tardive dyskinesia

June 26, 2003

Tardive duskinesia is a product of lipid peroxidation due to chronic

drug use. Thus it can be prevented with an elevated antioxidant

program. Here's something on glutathione for example.

and enter " tardive dyskinesia " glutathione

to see

" Biochemical analysis revealed that chronic haloperidol treatment

significantly induced lipid peroxidation, decreased glutathione

(GSH), superoxide dismutase (SOD), and catalase levels in the brains

of rats. "

You'll see the research also cites vitamin E, which has been used for

neurodegenerative disorders with small success, and selenium. They

are instrumental in creating glutathione(selenium), and keeping

glutathione in it's reduced form (antioxidants work together).

Because the damage is caused by oxidation, it can be prevented or

slowed as above, pursuant to the protocols for other

neurodegenerative disorders that also respond to a good antioxidant

program, such as ALS, MS, alzheimer's, Parkinson's disease etc... and

it can also be reversed by increasing HGH growth hormone levels

pursuant to protocols for these neurodegenerative disorders, some

data for which appears on

Although HGH injections were tested on the neurodegenerative

disorders in the peer-reviewed studies, the significance is that the

oral mix commonly used by doctors, for HGH increase, does also

include branched-chain amino acids.

Also a vitamin and mineral program especially one that includes b-

vitamins with 25 mg to 60 mg b-12 methylcobalamin (active form) daily

has been shown also to induce repair of neural sheaths. That

information appears somewhere on the Life extension Foundation site,

under ALS protocol. Note the ALS protocol is pretty thorough; it also

includes antioxidant therapy and growth hormone increasers.

Anyway, the data suggests that both avoidance and reversal of the

damage is possible, and a program is likely to work better than a

single supplement.

Duncan Crow

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