Guest guest Posted January 18, 2002 Report Share Posted January 18, 2002 - <RowenaThursby Friday, January 18, 2002 6:20 AM Murdering the truth > MAD COW DISEASE > > Following is an interesting story, full of intrigue, about Mad Cow Disease, > from Dr. Mercola's site: > http://www.mercola.com/2001/feb/4/insecticides_mad_cow.htm > > Insecticides Cause Mad Cow Disease > > Pharmaceutical interests in the UK are ignoring new scientific research that > shows the insecticide used in the UK government's own warble-fly campaigns > triggered the UK surge of 'Mad Cow' disease. > > Latest experiments by Cambridge University prion specialist, David R. Brown, > have shown that manganese bonds with prions. Other researchers work shows > that prions in the bovine spine --along which insecticides are applied-- can > be damaged by ICI's Phosmet organophosphate(OP) insecticide -causing the > disease. > > British scientists have led the current theory that an infectious prion in > bonemeal fed to cattle causes bovine spongiform disease (BSE). Infectious > prions are also claimed to cause new variant Creutzfeld-Jakob Disease (CJD) > in humans -from ingesting beef. But the infectious prion theory serves to > obscure a tragic chemical poisoning scandal behind the majority of BSE cases. > > The new work proves that the prions can bond with manganese in animal feeds > or mineral licks. These manganese prions cause the neurological degeneration > seen in BSE. By a similar process, prions in human brains are damaged by lice > lotions containing organophosphate. This can result in neurological diseases > like CJD and Alzheimer's -later in life. > > Many might be surprised to hear that organophosphates were developed by Nazi > chemists during the course World War Two, as a chemical weapon nerve agent. > > The marginalized research has devastating financial implications for ICI. It > would provide a firm basis for litigants -who could include CJD sufferers, > farmers across the world and families of the many British farmers who > committed suicide during this BSE debacle. > > Scientist and organic farmer, Mark Purdey gave evidence to the UK BSE > inquiry, that warble fly insecticide was the cause of the disease. The > scientist wheeled out to rubbish Purdy's evidence -Dr. David Ray, later > turned out to have been receiving funding from the insecticide manufacturer > ICI. > > Purdey has been consistently denied even exploratory funding to extend his > privately supported research. Yet the Purdey/Brown chemical poisoning model > matches with the epidemiological spread of CJD clusters in humans. It also > predicts the incidence of BSE-type diseases in animals. The accepted > infectious model fits neither. > > The pharmaceutical industry is all the more determined to hide the chemical > source of BSE and CJD, because a spotlight on chemicals would expose the role > the insecticides in Alzheimer's --another neurodegenerative disease--that > might lead to claims which would dwarf those from BSE and CJD litigants. In > fact, two leading brain researchers into CJD and Alzheimer's have died in > suspicious circumstances in recent years. > > In the United States, the Environmental Protection Agency is already > reviewing Phosmet's safety. The Centers for Disease Control in the US has > recently conducted experiments on mice that confirm the organophosphate risk. > > Not only is the EC beef slaughter campaign futile -because BSE disease is > mostly noninfectious, but unless the underlying chemical cause is addressed, > BSE will simply reappear from chemical causes. A new warble fly campaign is > already underway in France using the organophosphate insecticide. > > Of greater concern is that some lotions for scabies and head lice are now > priming children and adults, for CJD and Alzheimer's in later life. > > Bonding the Prion > > Cambridge University prion biochemist, David R. Brown is dismissive of the > science behind the infectious model of BSE. He terms it " a very limited > amount of science by a few assumed- reputable scientists. " He insists there > is " no evidence an infectious agent is present in either meat or milk. " > > " Simple tests on udder walls of cows --which could easily detect an > infectious prion-- have not been done, why I don't understand. " > > A number of researchers have found that organophosphate(OP) in systemic > warble fly insecticide can deform the prion molecule, rendering it > ineffective at buffering free radical effects in the body. Worse still, the > prion is then partial to bond with manganese and become a 'rogue' prion. A > chain reaction whereby rogue prions turn others to rogues also, can explain > the bovine spongiform disease mechanism. > > Brown showed how prion protein bonds benignly with copper, but lethally with > manganese. Even natural variations in relative environmental availability of > manganese versus copper can trigger prion degradation. > > The CJD and BSE symptoms mirror 'manganese madness', an irreversible fatal > neuro-psychiatric degenerative syndrome that plagued manganese miners in the > first half of the last century > > Shining A Light On Spongiform > > Organic dairy farmer and peer-review-published independent scientist, Mark > Purdey, says the accepted theory of transmission from BSE-infected cattle to > human CJD -by bonemeal or meat, is dependent on a mutant prion that has never > been isolated under the scientific protocol called Koch's postulates. > > Purdey's insistence on sticking to the letter of this scientific law earned > him the condemnation of UK officialdom when he first mooted his theory. But > Purdey pointed to CJD clusters downwind of a British Phosmet production plant > to back his case. > > He gave evidence to the UK Government BSE inquiry and was supported by > Conservative MP, Thessa Gorman. His views were discounted, but his subsequent > research and the new Cambridge prion work have confirmed the alternative > theory. Despite this, and the backing of a British peer, he is denied even > exploratory funding. > > Why does CJD degeneration in humans begin in the retina, and why are CJD > disease clusters found in high altitude locations? > > The prion molecule has a known natural role as a shock absorber of damaging > energy from ultraviolet rays and other oxidizing agents. > > Once this prion defense system is rendered ineffective by organophosphates - > for example in human head lice lotions, these oxidizing effects have an > unmediated impact on tissues. Eventually, UV radiation damages the retina and > oxidative stress destroys the brain tissues of CJD patients. This theory > would expect to find higher CJD incidence in mountain regions -where UV > radiation levels are elevated. That prediction holds true. > > A similar but accelerated mechanism could be driving BSE. ICI's Phosmet > organophosphate warble fly insecticide -applied on the backs of animals along > the spinal column, similarly degrades prions. " Systemic versions of the > insecticide are designed to make the entire cow carcass toxic to warble fly, " > explains Purdey. " Unfortunately it's toxic to prions too -especially those > prions located just millimeters from the point of application. " > > The damaged prions are then ready to react with manganese in animal feed, or > manganese sprayed on land or in mineral licks -to become the driving force of > BSE neurodegeneration. Purdey says manganese-tipped prions set off lethal > chain reactions that neurologically burn through the animal. > > Chickens notoriously excrete most of the supplements fed to them -including > manganese. And their manganese-rich excreta have been blended into cattle > feed in the UK. Natural variations in the relative environmental availability > of copper and manganese can also spur prion degeneration says Purdey. > > From this research, any prudent person would conclude there is a significant > risk attaching to the use of organophosphate in humans. Preparations for head > lice and scabies are known to be overused in practice and might be priming > users for CJ disease. > > The Money Trail > > Critical scientists like Purdey are unlikely to prevail. The pharma industry > holds most research purse strings, and would hardly energetically explore an > avenue of research that could expose them to litigation for causing BSE. The > official theory is lavishly funded, alternative theories rarely, if at all. > > There are more explosive implications to his -and other's latest research. > Purdey says similar organophosphate-induced protein deformation could also > underlie Alzheimer's disease. If that were true, the litigation fallout would > destroy some pharmaceutical giants, and a lot of very influential noses would > be out of joint. > > Disturbingly, Purdey and other brain researchers seem to have had an undue > share of unfortunate accidents. Purdey's house was burned down and his lawyer > who was working with him on Mad Cow Disease was driven off the road by > another vehicle and subsequently died. The veterinarian on the case also died > in a car crash -locally reported as: 'Mystery Vet Death Riddle.' > > Dr. C. Bruton, a CJD specialist --who had just produced a paper on a new > strain of CJD-- was killed in a car crash before his work was announced to > the public. Purdey speculates that Bruton might have known more than what was > revealed in his last scientific paper. > > In 1996, leading Alzheimer's researcher Tsunao Saitoh, 46 and his 13 > -year-old daughter were killed in La Jolla, California, in what a Reuters > report described as a " very professionally done " shooting. > Quote Link to comment Share on other sites More sharing options...
Recommended Posts
Join the conversation
You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.