EFA deficiency may be the result of the use of canola and soy oils

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Just before I got the messages on how bad canola oil is for you, I had ordered a

case of six bottles. I found a good use for it. I gave it to my husband to use

to oil his clippers.

 

Tamika Polk

personal email and MSN

TAMIKAPOLK

 

Life is not a matter of milestones, but of moments. (Rose Kennedy)

 

 

 

 

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EFA deficiency may be the result of the use of canola and soy oils

 

Entrez-PubMed -http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=11593118 & dopt=Abstract -

 

Medical Department, Ross Products Division, Abbott Laboratories, Columbus,Ohio 43215-1724, USA. phyllis.acosta

 

BACKGROUND: Investigators in Italy and Spain have suggested that therapyforpatients with phenylketonuria (PKU) may result in essential fatty acid(EFA)deficiency. Objectives of this study were to determine if the diets ofpatients with PKU in the United States provided adequate EFA intakes andwhether patients could form long-chain polyunsaturated fatty acids.METHODS:Patients (1-13 years of age) with classic PKU undergoing therapy and theirnon-PKU sibling closest in age were compared. Nutrient intakes werecalculated from 3-day diet diaries. Fatty acids in plasma and erythrocyteswere identified and quantified. Paired t tests compared results for thepatients and their non-PKU siblings. RESULTS: Twenty-eight patients and 26siblings were studied. Mean fat intake was greatest by siblings (34.8 +/-1.3% of energy) and lowest by Phenyl-Free-fed patients (19.5 +/- 1.2% ofenergy; P < 0.05). Fat intake (30.4 +/- 1.8% of energy) by Phenex-fedpatients did not differ from that of siblings. Percentage of energyingestedas C18:2n-6 and C18:3n-3 did not differ significantly between patients andsiblings. No clinically significant, consistent differences were found infatty acid levels (wt%) in plasma or erythrocytes between patients withPKUand siblings. CONCLUSIONS: No patient in this study exhibited a Holmanindexof EFA deficiency. Siblings ingested animal protein containing C20:5n-3andC22:6n-3 fatty acids, and this may account for their greater wt% of theseplasma and erythrocyte fatty acids. Because patients with PKU do notingestfatty acids C18 but C20:4n-6, C20:5n-3, and C22:6n-3 were found in theirplasma and erythrocytes, in vivo synthesis from C18:2n-6 and C18:3n-3appears to occur. Lack of EFA deficiency in patients in this study may bethe result of the use of canola and soy oils containing C18:2n-6 andC18:3n-3 rather than olive oil in the diets.

 

NCBI NLM NIHDepartment of Health & Human ServicesFreedom of Information Act Disclaimersparc-sun-solaris2.8 Nov 15 2001 10:41:10