Kaishore vs.Yogaraj Guggulu in Arthritis treatment

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> Message: 1

> Tue, 05 Aug 2003 02:09:31 -0000

> "dwellsohm" <dwellsohm

> Kaishore vs.Yogaraj Guggulu in Arthritis treatment

>

> I have been treating a patient,a 42 year old female of strongly Pitta

> Prakruti with Pitta and Vata Vikruti. She has been variously

> diagnosed my various Western MDs as having; Fibromyalgia, Reitters

> Syndrome, Systemic Lupis and Arthritis( no overall agreement). She

> suffers severe joint pain which manifests in various locations from

> day to day. I began treating with Kaishore Guggulu 6 caps per day,

> she has still not felt any relief. After one month, would it be

> correct to consider this a case of "upashaya" and assume that her

> imbalance was not Pitta to be treated with Kaishore Guggulu but

> actually a Vata imbalance to be treated with Yogaraj Guggulu? Any

> thoughts of advice from Vaidyas would be most welcome. Thank You!

> David Wells

 

Hi David,

 

I think that more information regarding case history is important to

make an intelligent assessment, but the one clue you provide, that of

migrating body pain, is typically manifest in a clearly established

Vattic syndrome. Apart from determining the vikriti/prakriti, have you

come up with an Ayurvedic diagnosis? Sounds like amavata to me, but I

personally would need more details since the medical dx isn't clear.

 

That said, any condition like this is essentially an autoimmune

disorder, and you should treat her as such and make the necessary

changes in diet/nutrition that are key to stopping the symptoms and

reversing the condition. The modern concept of leaky gut or intestinal

permeability describes almost the same etiology as amavata. So, I

would recommend that you deepen your understanding of leaky gut, even

if to support the Ayurvedic diagnosis, and direct your attention to

what things in her diet cause gut inflammation and permeability.

Here's a published excerpt from a piece I wrote and presented on

amavata for a recent symposium (and its copyright folks so I don't want

to see it on somebody's website):

 

"The Madhava Nidana provides three basic causes of amavata:

 

•Weakness of digestion: The digestive fire is one of the prime

motivators of all human function, ensuring the proper absorption and

metabolism of nutrition. When digestion is impaired ama accumulates,

the doshas become vitiated, and the vitality (ojas) diminishes.

 

•Incompatible foods in the diet, including foods consumed out of season

or without respect to local bioclimactic factors; unfamiliar foods

(asatmya, i.e. opposite of the healthy norm [satmya], non-traditional

foods); spoilt and contaminated foods, food additives, refined flour

products, feed-lot meat, commercial dairy, etc.

 

•Lack of physical activity: Physical exercise (vyama) is considered an

important aspect to dinacharya, the daily regimen recommended in

Ayurveda. A lack of exercise predisposes one to amavata because the

circulation of blood to and the removal of wastes (ama) from the

periphery is impaired.

 

Pathogenesis of Amavata

 

The Madhava Nidanam states that when the digestive fire is weak and ama

is allowed to accumulate, it moves to the different locations of Kapha

in the body (Srikantha Murthy 1995, 95). Kapha is derived from the

Sanskrit root word 'shlesh,' which means 'to embrace.' Thus, Kapha

binds the joints together, nourishing and protecting the articular

surfaces (Srikantha Murthy 1994, 169). Like ama, Kapha is heavy (guru),

moist (snigdha) and cold (shita) in nature. For this reason, ama

typically associates with Kapha before the other two doshas. According

to the Madhava Nidanam, when ama is allowed to accumulate in the joints

they become congested with a "…hard, waxy material" (i.e. Kapha). Soon

the circulatory channels (dhamanis) that supply these regions become

congested as well. Eventually this blockage affects the heart (hrdaya),

which then becomes the "…seat of the disease" (Srikantha Murthy 1995,

95). Once Kapha has become vitiated the other doshas eventually become

involved. To restore homeostasis the body will initiate local

inflammatory processes (i.e. Pitta) in the joint in order to 'cook' the

accumulated ama. Despite the inflammatory component of this condition

however, the hallmark of amavata is the progressive pathological

influence of Vata in the synovial joints, and the resultant joint

degeneration.

 

The pathogenesis of amavata bears some similarity to the recently

described intestinal permeability syndrome (IPS). The impetus for IPS

is a process by which some agent or combination of agents initiates an

inflammatory response in the digestive tract. Persistent

gastrointestinal inflammation eventually disrupts the integrity of the

mucosal lining of the gut, and tiny perforations allow for molecules

larger than usual to pass across this barrier, including molecules from

dietary protein and fats, bacteria, parasites and fungi. In response to

this infiltration, an immune response is initiated and the body begins

to manufacture specific antibodies to these antigens. Unfortunately,

many tissues have antigenic sites almost identical to those substances

that pass across a permeable intestinal wall. Once activated, these

antibodies then circulate and 'look' for more antigens. When an antigen

is found, such as a tissue that has similar markers to an exogenous

antigen, the antibody initiates an immune response and the tissue

begins to be destroyed (Galland 1993). The differences between IPS and

amavata are obviously significant, with each using an entirely

different physiological model. Nonetheless if we can translate the

antigens described in IPS into the ama identified in Ayurvedic

medicine, the two models become strikingly congruent (Kumar 1997, 94).

 

Although amavata is primarily a disease of Vata, it is differentiated

into three basic subtypes, namely, Vata, Pitta, and Kapha. This

differentiation allows the practitioner to identify a greater range of

subtlety within the diagnosis and treatment of amavata. Where Pitta is

involved the joints appear red and feel hot, and the patient complains

of a burning, searing pain. With Vata the pain is severe, and migrates

from place to place. With Kapha the pain is less, but there is more

stiffness and immobility, often combined with sensations of itching.

There may also be a combination of any two or three of the doshas. If

one dosha is involved the condition is said to be easy to cure. With

two doshas the situation is more difficult, and with all three doshas

in a state of vitiation the condition is said to be incurable.

Similarly, when there is migrating pain and severe inflammation in the

joints of the hands, feet, head, heels, waist, knees and thighs,

amavata is said to be incurable (Srikantha Murthy 1995, 95-96)."

 

My approach is to remove all potential antigenic foods from the diet

including grains, legumes and dairy, along with all sweeteners,

tropical fruit, raw vegetables (unless juiced), and all food additives.

I also routinely supplement with EPA/DHA (1000 mg daily ea) and fish

liver oil (1000 IU vit D daily), and then add whatever else is

appropriate, e.g. Yogarajaguggulu, MSM/glucosamine, Ashvagandha with

dipanapachana dravyas, Haridra, Guduchi, acidophilus/bifidus etc..

Without making these changes and/or having the patient undergo pancha

karma first I doubt that treatment will be all that successful.

Caldecott

phyto

http://www.wrc.net/phyto

[Guest guest]

Because the pain is a migrating one, there are chances that she doesnt suffer

from arthritis at all (or only early onset of OsteoArthritis) which shouldent be

causing the severe pain). She is most likely suffering from Vitamin deficiency.

The most likely culprits are Vitamin B12 and D4 deficiency. Get a blood test

done on her for checking all neceesary vitamins, and mineral including Iron and

Calcium.

Regards

Mustafa

[Guest guest]

I think you mean D3? never heard of D4. D3 deficiency is all too

common, esp in NA and Europe, due to limited exposure of sunlight and

lack of D3 in the diet. D2 (derived from irradiated yeast) is only 25%

as active as D3.